Exam 1 Flashcards

1
Q

Lecture 1

A
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2
Q

Tooth Anatomy

What type of teeth anatomy do dogs, cats, primates, humans have?

A

Brachydont: tooth with shorter anatomical crowns than root(s) at maturity

-Having short crowns
-Well developed roots
-Only narrow canals in the roots

**Hypsodont: tooth with long anatomical crown at tooth maturity, continuously erupting as occlusal wear takes place (e.g., horses and ruminants).

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3
Q

Name all the parts of the tooth

A
  1. Enamel
  2. Dentin
  3. Pulp
  4. Cementoenamel Junction (neck
  5. Gingiva
  6. Periodontal Ligament
  7. Cementum
  8. Alveolar Bone
  9. Apex
  10. Cusp
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4
Q

What is the Cusp?

A

A pronounced point on the occlusal or most coronal portion of a tooth.

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5
Q

Describe the enamel and Dentin

What cells produce enamel and dentin?

What is the thickness of enamel in cats and dogs?

A

Enamel

-highly mineralized inorganic hard tissue that covers the dentin of the crown,
-produced by AMEOBLASTS.
-Amelogenesis: stops prior to tooth eruption. It can’t be rapider once it stops growing. Heat/cold sensitivity when damaged.
Dog: <0.1 to 0.3mm
Cat: <0.1mm

Dentin

-Hard tissue covered by enamel (crown) and cementum (root)
-Produced by ODONTOBLASTS throughout the life of the tooth.
-Dentin is porous
-Primary dentin: produced during development of the tooth
-Secondary dentin: produced throughout life of a vital tooth, causing the pulp cavity to become progressively narrower.
-Tertiary dentin: reparative dentin produced in response to injury and irritation

DENTINAL TUBULES: contain cytoplasmic extensions of odontoblasts. Each tubules contain fluid, and come contain nerve extending from the pulp.
-Tooth pain results from fluid shifts and nerve stimulation due to changes in temperature, desiccation, or osmotic changes (e.g., foods with high sugar content) in the area of exposed tubules.

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6
Q

Periodontium Components

  1. Gingiva
  2. Periodontal Ligament
  3. Cementum
  4. Alveolar bone
    These structures are affected by, and ultimately lost from periodontal disease

Describe Cementum and Periodontal ligament

A

Cementum

-Hard tissue that covers the root and is produced by CEMENTOBLASTS at apex of root
-Periodontal ligament and gingiva attach to cementum
-Width increases with age
-Hepercementosis: occurs from chronic irritation and “lock” the tooth into the alveolar socket.
-Loss of cementum prevents reattachment of periodontal ligament.

SHARPEY’S FIBERS (cementum)
-Embedded in cementum and transverse the periodontal space to anchor the tooth in alveolar bone.

Periodontal Ligament

-Shock absorption, transmits occlusal forces, attaches to bone, supplies nutrients, provides tactile and proprioceptive information
-SHARPEY’s FIBERS: travel transversely in the coronal portion of the tooth and more oblique toward the apex.
-Radiographically the PDL appears as a dark line surrounding the root.

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7
Q

Describe the Pulp

A

-Soft tissue, connective tissue containing nerves, blood, and odontoblasts, which produce dentin.
-Pulp proper: layer containing major vessels, nerves, and connective tissue. Sharp localized pain, myelinated A-delta fibers, Dull pain unmyelinated C fibers.

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8
Q

What is the gingival sulcus?
What is the normal sulcus depth?

A

The gingiva is keratinized and consists of four layers: Stratum corner, stratum granulosum, stratum spinous (prickle cell layer) and stratum basale.

Gingival sulcus: space between the tooth surface and gingiva.
Normal sulcus depth = DOGS: <3mm CATS: <1mm

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9
Q

Positional Terminology

A

-Mesial: inter proximal surface of the tooth that faces rostrally or towards the midline of the dental arch.
-Distal: inter proximal surface that faces caudally or away from midline of the dental arch.
-Vestibular: surface of the tooth facing the lips “buccal” and “labial” are acceptable alternatives
-Lingual: refers to the surfaces of the MANDIBULAR teeth that face the tongue:
-Palatal: refers to the surface of the MAXILLARY teeth that face the palate

-Rostral: referring to a location toward the tip of the nose
-Caudal: referring to a location towards the tail
-Ventral: referring to a location towards the lower jaw
-Dorsal: referring towards the top pf the head or muzzle

-Occlusal: refers to the surface of the tooth that faces the tooth of the opposing arcade
-Proximal: referring to the medial and distal surfaces of a tooth that come in close contact to an adjacent tooth
-Interproximal: referring to the space between adjacent teeth.
-Coronal: referring to a location or direction toward the crown of the tooth.

-Subgingival: referring to a structure or area that is apical to the gingival margin
-Supragingival: referring to a structure or area that is coronal to the gingival margin

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10
Q

Modified Triadan Tooth Numbering System CANINE

A

Permanent

2[I 3/3, C 1/1, PM 4/4, M 2/3] = 42 teeth

Right upper quadrant: 100 (500 when referring to deciduous teeth)

Left upper quadrant: 200 or 600 deciduous

Lower right quadrant: 400 or 800 deciduous

Lower left quadrant: 300 or 700 deciduous

Mesial to distal: 01 to 10 on maxilla
Mesial to distal: 01 to 11 on mandibule

Canine: 04
PM1: 05
M1: 09
M3: 11 mandible only

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11
Q

Which teeth are 1 rooted teeth?

A

Incisors and canine teeth mandibular and maxillary
-01 to 04
-Plus PM1 (05)

Mandibular
-M3 (11)

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12
Q

Which teeth are two rooted teeth?

A

Maxillary
-PM2 (06)
-PM3 (07)

Mandibular
-PM2 (06), PM3 (07), PM4(08), M1 (09), M2 (10).

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13
Q

Which teeth are three-rooted?

A

Maxillary
-PM4 (08)
-M1 (09)
-M2 (10)

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14
Q

Canine Tooth Eruption Schedule
(don’t have to memorize)

A

Deciduous teeth

2[ I 3/3, C 1/1, PM 3/3] = 28 teeth

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15
Q

Normal Occlusion - Dog

What teeth superimpose normally?

A

Mandibular M1 with Maxillary PM4

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16
Q

Feline Teeth

Dental formula - Modified Triadan System

A

Permanent teeth

2[ I 3/3, C 1/1, PM 3/2, M 1/1] = 30

I: 101-103 (Right upper) 201-203 (Left upper) 301-303 (Left lower) 401-403 (Right lower)

C: 104, 204, 304, 404

PM: NO 05 Maxillary
PM: 06-08
M: 09

PM: NO 05, 06 Mandibular
PM: 107-108
M: 09

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17
Q

Which teeth are one, two, and three-rooted? Feline

A

One rooted

Maxillary
I: 01-03
C: 04
PM: 06 (no 05 remember?)

Mandibular
I: 01-03
C: 04

Two Rooted

Maxillary
PM: 07
M: 09

Mandibular
PM: 07-08

Three rooted

Maxillary
PM: 08

Mandibular
No three-rooted

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18
Q

Which are the strategic canine teeth?
Which are more commonly taken out?

A

Canines

-Large, well developed roots
-Important to maintain the integrity of the mouth and to chew hard food

Carnassial teeth

-PM4 maxillary (08)
-M1 mandibular (09)

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19
Q

Lecture 2

A
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20
Q

Name the anatomical structures

A
  1. Parotid salivary gland
  2. Parotid duct: drains at level of the maxillary 4th premolar = build up
  3. Mandibular salivary gland
  4. Mandibular lymph nodes

Clinical importance

Avoid transecting ducts during oral surgery/surgical extraction of maxillary PM4 (08) and M1 (09)

Zygomatic salivary gland ducts open near maxillary M1(09) and M2 (10)

Mandibular salivary duct opens on the sublingual caruncle

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21
Q

Lingual Molar Salivary Gland in Cats

What can it be confused with?
Avoid when?

A

Lingual molar salivary gland near mandibular M1 (09)

Confused for an abnormal structure
Avoid during surgical extraction of mandibular M1

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22
Q

What are significant skeletal landmarks and used for what procedures?

A

Caudal Maxillary Nerve Block

Ventrodorsal view of caudal palate
-M2 (10)
-Maxillary tuberosity
Caudodorsal view of orbit
-Zygomatic process of frontal bone
-Maxillary foramen (leads to infraorbital canal)
-Maxillary tuberosity
-M2 (10)

Sites of nerve blocks

Inferior Alveolar Nerve Block

-Infraorbital foramen
-Middle mental foramen
-Mandibular Canal: location of major blood/nerve supply during surgical extraction of mandibular teeth
Be able to recognize normal radiolucency on radiographs
-Mandibular foramen site of the inferior alveolar nerve block

Palatine fissure
-Recognize maxilla on radiographs

Mandibular symphysis
-Fibrocartilagenous synchondrosis
-Normal radiolucent line

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23
Q

Sensory Nerves Innervation

A

Trigeminal nerve

  1. Maxillary nerve
    a. Infraorbital nerve
    -Alveolar branches
    b. Pterygopalatine nerve
    -Palatine nerves
  2. Mandibular nerve
    a. Inferior alveolar nerve
    -alveolar sensory branches
    b. Mental nerve
    -Mental nerve

Clinical importance

  1. Maxillary nerve
  2. Infraorbital nerve
  3. Inferior alveolar nerve
    19.” Mental nerve
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24
Q

Blood supply

A

Branches of Maxillary Artery

  1. Inferior Alveolar Artery: courses through mandibular foramen and canal to supply mandibular teeth and bone
    -Three mental branches: course through caudal, middle and rostral mental foramena. May encounter when performing surgical extraction of mandibular teeth.
  2. Major and Minor Palatine Arteries: supply hard and soft palate, periosteum, alveolar bone of maxillary teeth
  3. Infraorbital Artery: continuation of maxillary artery, emerges from infraorbital canal. Several branching
    -Alveolar arteries brach and supply the maxillary teeth.
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25
Q

Muscles of Mastication

A

-Masseter m.
-Temporal m.
-Pterygoid m.
-Digastricus m. (caudal belly, facial nerve)

Trigeminal nerve innervation (Facial branch)

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26
Q

Lecture 3

A
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27
Q

Periodontal Disease facts

A

Present in 70-90 % of patients to some degree

Most common cause of oral infection and tooth loss

Preventable disease, but once stablished only managed, chronically progressive

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28
Q

Gingiva

A

Normal: sharp, thin, non-inflamed margins. Firm, Coral-pink, stippled
-Aerobic gram positive bacteria

Abnormal: marginal gingivitis, rounded edges, erythematous, may bleed upon probing
-Accumulation of calculus/plaque in sub gingival location creates anaerobic environment
-Increased anaerobic, gram negative and spirochete bacteria.

Plaque Induced Gingivitis

-Bacterial plaque colonizes within 24-36 hours after cleaning
-Plaque is inorganic, transparent, adhesive biofilm, salivary glycoproteins, oral bacteria and extracellular polysaccharides.
-If not removed, within a few days, inflammatory response at gingival margin

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29
Q

Gingivitis

What type of bacteria predominates?
What does the inflammatory response consist of?

A

Normal: sharp, thin, non-inflamed margins. Firm, Coral-pink, stippled
-Aerobic gram positive bacteria

Abnormal: marginal gingivitis, rounded edges, erythematous, may bleed upon probing
-Accumulation of calculus/plaque in sub gingival location creates anaerobic environment
-Increased anaerobic, gram negative and spirochete bacteria.

Inflammation
-Vasculitis
-Edema
-Collagen loss

Diagnosed
-When inflammation is limited to the gingiva
-Advanced gingivitis can develop in periodontitis

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30
Q

Risk factor for periodontal disease
Tissue Destruction

A

-Black pigmented anaerobic bacteria is commonly associated with periodontitis

Inflammatory factors host’s immune response and anaerobic bacteria cause the destruction of the tissue surrounding the tooth (periodontium)

Risks

-Age: more common in older animals
-Breed: brachycephalic, toy breeds, dolicocephalic breeds
-Persistent deciduous teeth
-Malocclusions
-Genetic predisposition
-Host’s immune response
-Non-abrasive diet
-Chewing habits
-Periodontal trauma
-Oral foreign bodies

Periodontitis Tissue destruction

-Epithelium attachment loses integrity
-Gingival sulcus increases in depth
-Pockets are formed as periodontal ligament and alveolar bone are destroyed
-Gingival recession
-Tooth loss if not treated

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31
Q

Clinical features of Periodontal Disease

A

-Gingivitis
-Halitosis (bad breath)
-Gingival enlargement
-Gingival recession below neck of tooth
-Periodontal Pockets
-Abscessation (apical tooth abscess)
-Facial swelling
-Draining fistula
-Ophthalmic sequela
-Oronasal fistula - nasal discharge
-Osteomylitis
-Osteolysis
-Mandibular fractures (when bone loss is severe)
-Systemic organ lesions from bacteremia (kidney, liver), chronic pulmonary changes
-Oral pain
-Tooth mobility

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32
Q

How is Periodontal disease assessed and Diagnosed?
What are the stages of PD? How are they determined?

A

Periodontal probing

-Locating periodontal pockets
-Checking for gingival sulcuses depth

Diagnosis

-Definitive Dx requires general anesthesia
-Dental radiographs
-Periodontal probing
-PD (periodontal disease) refers to degree of severity on a single individual tooth.
Stages 0-4

Attachment Loss Concept

-Refers to the destruction of periodontium (gingiva, cementum, alveolar bone, PDL) due to periodontitis
-Determined by probing, measurement of gingival recession (if present) and radiographs
-Probing depth: mm from gingival margin to the most apical point that the probe reaches when gently inserted into the gingival sulcus or pocket.

Radiograph

-Measure distance from the alveolar margin (AM) to the cementoenamel junction (CEJ) relative to the length of the tooth. Ex: 11/15 = 0.73 = 73% attachment loss

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33
Q

Tooth Root Furcation

Furcation Index Stages 0-4

A

-Normal space in multicoated teeth
-When disease is present bacteria inhabits the space

Stage 1
-Involvement exits when the probe extends LESS than halfway under the crown in any direction of multicoated teeth with attachment loss

Stage 2
-Involvement exits when the probe extends MORE than halfway under the crown in any direction of multicoated teeth with attachment loss, but not through and through

Stage 3
-Involvement exits when the probe extends under the crown in any direction of multicoated teeth, through and through from one side of the furcation out the other.
-Tooth needs to be extracted bc it’s hard to keep it clean

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34
Q

Periodontal Disease Stages

A

Normal (PD 0)
-Clinically normal
-Gingival inflammation or periodontitis is not clinically evident

Stage 1 PD
-Gingivitis only without attachment loss
-Height and architecture of the alveolar margin is normal
-Mild gingivitis, mild calculus, no recession

Stage 2 PD
-Early periodontitis or at most there is stage 1 furcation
-Periodontal attachment loss <25%
-Or radiographic determination of the distance of the alveolar margin from the cemento-enamel junction relative to the length of the tooth.

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35
Q
A
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36
Q
A
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37
Q

Periodontal Disease

Stage 3
-Moderate periodontitis
-25-50% attachment loss
-Measured by probing of the clinical attachment level, radiographic determination of the distance of the alveolar margin from the CEJ relative to the length of the tooth, or
-There is stage 2 furcation involvement in multi-rooted teeth.

A

Stage 4
-Advanced periodontal disease
->50% attachment loss or
-Stage 3 furcation

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38
Q

Lecture 4

A
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39
Q

Indications for Dental Radiography

A

-Most dental pathology is below the level of the gingiva and cannot be detected through visual examination

-Diagnosis of periodontal disease
-Diagnosis of endodontic disease
-“missing” teeth
-Extractions, prior, maybe during, and after
-Resorptive tooth lesions
-Screening radiographs during routine dental cleaning
-Periorbital swelling, gingival swelling and oral neoplasia
-Developmental abnormalities
-Trauma
-Malocclussions
-Rule out dental cause of “nasal” disease

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40
Q

Dental radiographs

A

Conventional radiograph unit

-Useful in imaging mandibular/maxillary fractures, trauma, masses, TMJ
-Some practices without dental unit still may have regular unit
-Not great for diagnosis
-Difficult to isolate individual teeth
-Can’t take intra-operative films without moving patient from dental treatment table
-Inconvenient, usually not in location where dental procedures are performed

Dental Radiographs Unit

-kVp and mA are often fixed and only time is selected
-Most units have anatomic interface where operator sets the size and tooth the exposure is adjusted automatically

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41
Q

Radiographic Safety

A

-Less radiation than conventional units
-Required to be registered and inspected by the State Department of health
-In Virginia, stand >9 feet from the tube when making an exposure
-Stand at angle of 90-135 degrees from primary beam
-Use Disometry badges
-Use positioning aids (e.g., gauze sponge or play dough) to help stabilize the sensor/film to prevent exposure of staff by holding it

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42
Q

Obtaining Image

A
  1. Conventional Intraoral film
    -Least expensive
    -Requires no automation
    -Several sizes available
    -Takes more time
    -Requires developing chemicals
    -Must store and catalog films as part of the patient record.
    -Processing errors can be a problem
  2. Sensor based radiograph (DR) “digital radiograph”
    -Charged-coupled device (CCD)
    -Complementary metal oxide semiconductor (CMOS)
    -Need #2 sensor, laptop or desktop, software
    -Immediate images ~5 sec
    -Digital images, easy to store
    -More consistent quality
    -Less radiation required
    -Sensors can be expensive ~$2000
    -Only #1 and #2 size available
  3. Photostimulable phosphor plates (CR) “computed radiograph”
    -CR reader
    -Computer, software
    -Phosphos plare
    -Available in size 0, 1, 2, 3, 4. May be used in large animals
    -Digital images
    -#1 plate better for cats
    -Quality images
    -Slower ~30 sec for images
    -Hard-wired sensor
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43
Q

Radiographic Positioning

A
  1. Parallel Technique
    -Film or sensor is parallel to the long axis of the subject
    Distal mandibular (307/407)
    -Film sensor can be placed parallel to the tooth and perpendicular to the primary beam
  2. Bisecting angle technique
    -Due to anatomical interference with all maxillary teeth
    -Sensor can’t be aligned parallel to the long axis of the tooth
    -Mandibular rostral teeth also
    -Angle is between the plate and perpendicular to sensor angle, and plate and sensor perpendicular to long axis of tooth angle.
    Orient primary beam half-way between these those extremes, the image of the tooth will be same as the actual tooth
    -Some dental x-ray units come with angle indicators
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44
Q

Dental Radiograph Chart

A
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45
Q
A
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46
Q
A
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47
Q

Corrective techniques for superimposed PM4 maxillary

A
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48
Q

Foreshortening and Elongation Errors
Other errors

A

Foreshortening
-X-ray beam is too perpendicular to the sensor beam
-Correction: drop the angle to a more lateral position

Elongation
-The beam is too perpendicular to the tooth
-Correction: increase the angle to a more vertical position

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49
Q

Lecture 5

A
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50
Q

What are the components of the periodontium?
What is its function?

A

The function is to provide both structural and functional anchorage for the teeth within the mandible and maxilla

  1. Gingiva
  2. Periodontal ligament
  3. Cementum
  4. Alveolar bone
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51
Q

Pathogenesis of periodontitis

A

-Pellicle: protein film consisting of salivary glycoproteins which form within seconds of the teeth being cleaned
-Plaque biofilm: bacterial pellicle forms within 24 hours
-Calculus: forms via mineralization of the plaque biofilm by ions in the saliva. It is detectable within 48-72 hours after cleaning.
-Mechanical removal of the biofilm is required to prevent and treat periodontal disease
-supragingival bacteria influence the growth and pathogenicity of subgingival bacterial population

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52
Q

What are some locoregional and systemic periodontal disease?

A

Locoregional

-Periapical abscesses
-Oronasal fistula: +/- chronic rhinitis
-Retrobolbulbar cellulitis/ophthalmic inflammation
-Epiphora (insufficient tear drainage/blockage): due to compression/inflammation of nasolacrimal duct
-Pathologic fracture of the mandible in small/toy breed predisposition.

Systemic

-Bacteremia
-Kidney damage
-Liver damage
-Cardiovascular damage (myocardium)
-Left atrioventricular valve

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53
Q

Symptoms & PE findings & Diagnosis

A

Symptoms

-Reluctance to chew
-Head shyness
-Pawing at the face
-Dropping food when eating
-Sneezing/nasal discharge
-Exaggerated jaw motion when chewing
-Aversion to food or water

Most patients still eat with severe periodontal disease

PE findings

Intraoral
-Halitosis
-gingivitis
-Calculus
-Gingival recession/root exposure
-Missing teeth
-Mobile teeth

Extraoral
-Submandibular swelling
-Periorbital swelling
-Draining tracts

Diagnosis (can’t Dx on awake oral exam alone)

Minimum database
-CBS
-Chemistry profile with electrolytes
-UA with USPG if renal values borderline
-Species specific with ELISA testing: HWT, FIV/FeLV

Comprehensive Oral Health Assessment and Treatment (COHAT)

-Requires anesthesia
-Requires imaging
Dental radiographs is the standard of care Cone beam Computed Tomography for advanced diagnosis
-Probe Exam
-Dental chart for documentation in addition to written SOAP notes

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54
Q

Diagnostic Terminology

A

Stage: Periodontal disease

-Assessment of the extent of pathological lesions in the course of the disease. Ex: stages of periodontal disease, stages of oral tumor.

Grade: Furcartion Exposure

-The quantitative assessment of the degree of severity of a disease or abnormal condition at the time of diagnosis, irrespective of wether the disease is progressive (e.g., a grade 2 mast cell tumor based on mitotic figures)

Index: Gingivitis

-A quantitative expression of predefined diagnostic criteria whereby the presence and/or severity of pathological conditions are recorded by assessing a numerical value (e.g., gingival index, plaque index)

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55
Q

Diagnosis: Furcation Index

A

Stage 1 (F1)

-Furcation 1 involvement, periodontal probe extends less than halfway in any direction of multirooted tooth

Stage 2 (F2)

-Periodontal probe extends >halfway under the crown of a multirooted tooth WITH attachment loss, but not through and through

Stage 3 (F3)

-Periodontal probe extends under the crown of any multirooted tooth, through and through from one side to the other.

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56
Q

Diagnosis: Mobility Index

A

Stage 0 (M0)

-Physiological mobility up to 0.2 mm

Stage 1 (M1)

-Increased mobility in any direction other than axial >0.2mm up to 0.5mm

Stage 2 (M2)

-Increased mobility in any direction other than axial >0.5mm up to 1mm

Stage 3 (M3)

-Increased mobility in any direction other than axial >1mm

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57
Q

Diagnosis: Gingivitis Index (up for debate)

A

Gingival Index 1

-Inflammation and swelling, NO bleeding during periodontal probing

Gingival Index 2

-Inflammation and swelling, With bleeding during periodontal probing

Gingival Index 3

-Inflammation and swelling, spontaneous bleeding during periodontal probing

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58
Q

Diagnosis: Periodontal Disease Stages

A

Normal (PD0)

-0% attachment loss, 0 gingivitis

Stage 1 (PD1)

-0% attachment loss, gingivitis present

Stage 2 (PD2)

  • <25% attachment loss
    -Stage 1 furcation involvement

Stage 3 (PD3)

  • > 25% up to 50% attachment loss
  • Moderate periodontitis
    -Stage 2 furcation involvement

Stage 4 (PD4)

  • > 50% attachment loss
    -Advanced periodontitis
    -Stage 3 furcation involvement
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59
Q

Diagnosis: Probe Exam

-Performed after dental radiographs during COHAT
-Requires periodontal probe
-Requires dental chart to record findings

A

A. Marquis color-coded probe
-Calibrations are in 3mm sections
-Dog only

B. University of North Carolina-15 Probe
-15mm long probe
-With millimeter markings at each millimeter
-Color coding at 5th, 10th, and 15th millimeter
-Cat or dog

C. University of Michigan “O” probe with Williams marking
-Markings at 1,2,3,5,7,8,9, and 10mm

Requires dental chart to record findings

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60
Q

Measuring a periodontal pocket with probe

What is the normal pocket depth in a dog and cat?

A

Normal

-Dog: 1-3mm
-Cat: <1mm

1.Gently place the probe between the gingiva and tooth
2. Stop when you feel resistance
3. Record the pocket depth
Do not use too much force, can cause damage and create an accidental pocket

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61
Q

Treatment Options

A

Surgical

-Requires referral (except certain extractions)
-Defined as any procedure requiring a mucogingival flap
-Examples: open root canal, bone grafting, etc.

Non-Surgical

-Can be performed at a general practice
-Ultrasound scaling and polishing
-Closed root planning (with or without perioceutic application)
-Barrier/sealant application
-Homecare

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62
Q

Perioceutics

A

Parioceutics

-Antimicrobial products available for dental application (FDA approved on non lactating dogs)

8.5% doxycycline

-Powder contained within a slowly dissolving polymer gel

2% Clindamycin

-Hydrated salt in a matrix that gels at body temperature

Act as a physical barrier to pocket contamination, doxycycline in addition has anticollagenase activity
-Local Infusion of antibiotics

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63
Q

Treatment Planning

A

Early Stage PD

-PD0, PD1: managed with annual COHATs and homecare
-PD2/F1: closed root planning +/- perioceutic application

Advanced stage periodontal disease

-PD3: open root planning +/- perioceutic application, GTR, extraction

-PD4: extraction

Combined periodontal and endodontic lesions

-Extraction

Periapical Pathology

-Extraction

64
Q

PD0, PD1 Treatment

A

COHAT

-Ultrasonic scaling and polishing
<10 secs per tooth
-Can cause thermal damage to pulp
-Perio tip: appropriate for subgingival scaling
-Universal tip: thick, for modertae to heavy supragingival calculus removal, NOT appropriate for subgingival scaling

Homecare
-Daily tooth brushing #1 most effective way to prevent periodontal disease
-Daily dental chew, water additives, prescription diets
-Anti-plaque barrier gels (OraVet)

65
Q

PD2, PD3 Treatment

A

Root planning
-Closed
-Open
-With or without perioceutic application
-Tools: Gracey, Universal curettes. 5-0 Monocryl to close ALL periodontal surgeries

**Run the curette along the surface of the tooth into the periodontal pocket, “hook” the granulation tissue and calculus, scrape upward along the tooth to remove debris.

Bone grafting
-Performed to maintain alveolar bone height

Guided Tissue Regeneration (GRT)

Mucogingival flap osseous countouring/recontouring

66
Q

Advanced Procedures:
Bone Grafting
GTR

A

Bone Grafting

-Osteoconductive: materials that occupy space and acts as scaffold on which new bone can grow

-Osteoinductive: contains growth factors or hormones that signal host to produce new bone

-Bioceramic: Osteoconductive, Flap dehiscence. Ex: CONSIL

-Autologous: Osteoinductive and osteoconductive, preferred grafting material. Ex: SYNERGY, PERIOMIX

-Autogenous: Grafter from patient themselves, best integration, lowest risk of rejection. Osteogenic, osteoconductive, osteoinductive.

Guided Tissue Regeneration

-Allows for regeneration of the periodontal tissue, rather than repair.
-Requires recheck under anesthesia with dental radiographs in 6 months to monitor osseous integration

67
Q

Role of Antimicrobial Therapy

A

Antibiotic Prophylaxis

-COHATs cause transient bacteremia
-AVDC statement: systemically administered antibiotics are needed only for animals that are immune compromised, have underlying systemic disease (cardiac, hepatic, renal) and/or when severe oral infections are present

-Per human literature: NOT NEEDED in periodontal surgery

Clavamox (Broad-spectrum, aerobic and anaerobic bacteria)

Clindamycin (limited spectrum)

Subantimicrobial Dose Doxycycline (SDD)

-Clinical PD significantly improved
-2mg/kg SID for 8 weeks

68
Q

Lecture 6

A

Endodontics

-The study (practice) of the basic and clinical sciences of normal DENTAL PULP
-Etiology, diagnosis, prevention, and treatment of diseases and injuries of the dental pulp along with associated PERIARDICULAR conditions

69
Q

What are the 4 major tissue components of the tooth?

A
  1. Enamel
  2. Dentin
  3. Pulp
  4. Cementum

Apical delta
Cemetoenamel junction
Alveolar Bone

70
Q

Enamel and Dentin

A

Enamel

-96% hydroxyapatite
-4% water and fibrous organic material
-Avascular: no regenerative capability
-Thickest at the crown
-Thinnest at the cementoenamel junction
0.1-0.3mm CATS
0.1-0.6mm DOGS

Dentin

-Hard yellow substance covered by the enamel
-29,000-52,000 dentinal tubules per mm2 of cut dentin in the maxillary canine tooth
-Made up of tubules
-70% inorganic hydroxyapatite
-Number increases with patient size, and closer to the pulp
-Primary: forms prior to tooth eruption
-Secondary: forms after eruption
-Tertiary: forms in response to injury, brown discoloration
-Sclerotic dentin occurs when tubules are mineralized (age, trauma)

71
Q

Pulp, Dentin-Pulp Complex

A

Pulp

-Tissue contained within the root canal consisting of blood vessels, nerves, fibroblasts, collagen fibers, undifferentiated mesenchymal cells, odontoblasts, and other connective tissue cells.
-Produces dentin throughout the life of the tooth.
-Wide in young dogs and narrow with age
Zones
1. Odontoblastic
2. Cell-free (zone of wei)
3. Cell-rich
4. Pulp core

Dentin-Pulp complex

-Odontoblasts: move between the pulp and the dentin
-Critical for the formation of new dentin
-Odontoblasts themselves do not undergo cell division
-Potential for differentiation into new odontoblasts decreases with age: impacts therapeutic decision for treatment of teeth with vital pulp therapy or pulp capping procedures.

72
Q

Cementum

A

-Cementum formation begins at CEJ
-Exposure of dentin May cause tooth sensitivity
-Cervical half to 2/3 of the root is covered by acellular cementum
-Apical 1/3 of the root is covered by cellular cementum (cementocytes)

-Hypercementosis: cellular cementum of the root apex increases in thickness, common in cats due to occlusal stress of the tooth.
-Cementum has the ability to repair itself when injured

73
Q

Types of Endodontic Diseases

A

Traumatic Dentoalveolar Injuries

-Alveolar fracture
-Enamel fracture
-Concussion
-Tooth fracture: uncomplicated, complicated, root fracture
-Tooth avulsion
-Tooth subluxation
-Tooth luxation: lateral, extrusive, intrusive

Developmental Diseases

-Enamel defects: enamel hypoplasia, enamel hypomineralization, dentinogenesis imperfecta, amelogenesis imperfecta.

Intrinsically stained teeth

-Idiopathic pulpitis

74
Q

Symptoms Tooth Fractures

A

TDI
(66% Considered severe injuries with significant consequences if not treated promptly)

-Halitosis
-Dropping food
-Reluctance to play with toys
-Lethargy
-Inappetence
-Facial swelling
-Hypersalivation
-Maxillofacial trauma: 75% cases at least 1 TDI
-Most TDIs are incidental findings
Second most common oral disease to periodontal disease
-Canine>premolar>incisor>molar
-Most common PM4>canine>incisor>molar

75
Q

Tooth Fractures

A

Enamel Infarction

-Incomplete fractures of the enamel
-No treatment needed
-Monitor q6-12 mts
-Predisposed to pathology longterm

Enamel Fracture

-Confined to the enamel
-No exposed dentin
-Rare
-Treatment: Odontoplasty with or without bonded restoration
-Evaluate pathology, Radiographs
-Trauma usually involved, can lead to necrosis of pulp
-Monitor q12mts

Uncomplicated Crown Fracture

-Does not expose the pulp
-AKA enamel-dentin fracture
-Tooth surface is lost
-Evaluate, radiographs to ensure pulp is not involved
-Premolar teeth most commonly affected
-Treatment: acute - odontoplasty with bonding, full metal crown depending on location/tooth/amount lost. Chronic - removal of tertiary dentin, odontoplasty with bonding/restoration, root canal if pulp involvement.

Complicated Crown Fracture

-Pulp Exposed
-AKA Enamel-Dentin-Pulp fracture
-TDI most common
-Affects strategic teeth most commonly
-Painful, necrosis of pulp and periapical periodontitis if unattended
-Acute: hemorrhage of pulp
-Chronic: pulp necrosis (blackened or missing)
-Evaluate, radiographs
-Treatment: root canal or extraction

Uncomplicated root-crown fracture

-No pulp exposed
-Mostly PM4 maxillary
-AKA “slab fracture”
-Requires surgical exploration
-Treatment: root canal or extraction
Root canal therapy is made more challenging by root fracture, and often requires periodontal surgery

Complicated Crown-Root Fractures

-Pulp involvement/Exposed
-Canine teeth most often
-Frequently concurrent with alveolar bone fracture
-Treatment: root canal or extraction. Requires periodontal surgery

Root Fracture

-Involves ONLY the ROOT
-Cementum, pulp, and dentin involved
-Often incidental findings
-Prognosis depends on location, degree of mobility, timing of treatment
-Heal by 1. Hard tissue union between fracture segments IDEALLY 2. Connective tissue attachment only 3. Localized resorption of alveolar bone and tooth.
-Treatment: best <48hrs. Splinting, endodontic therapy, pulpitis.
-Guarded pronosis

76
Q

Consequences of Untreated TDI

A

-Abscess formation
-Endo-perio lesions
-Pain
-Infection
-Tooth death
-Behavioral problems: head shyness, poor performance of working dogs, aggression due to pain, food/toy aversion

Endo-Perio Lesions

-Class 1: Fractured crown with pulp exposure. Periodontal tissue secondarily affected by migration of bacteria through the apical foramen or lateral canal.
-Class2: Wide deep periodontal pocket extending to the apex of the tooth. Bacteria can gain access to the pulp through the apical foramen or a lateral canal.
-Class 3: Combined endodontic and periodontic lesion.

77
Q

Developmental disease: Enamel defects

A

Enamel hypoplasia
-Thin enamel layer
-Yellowish-brown discoloration due to visible dentin

Enamel hypomineralization
-Spots on the enamel, brown-yellow.
-Crowns are soft and wear fast
-Same density as dentin seen in radiograph

Dentinogenesis imperfecta
-Enamel separates from dentin
-Translucent or opalascent hue
-Border Collies

Amelogenesis imperfecta
-General term for enamel formation abnormalities
-Often hereditary

Intrinsic staining
-Results from a change of structure or composition within the hard surfaces of the tooth.
-Chronic pulpitis can develop. Narrower root canal prematurely.
-Radiographic evidence of pulp death: arrested tooth maturation (wider root canal), root resorption, apex resorption, changes in the trabecular bone pattern around the root apex.

Diagnosis of Instrinsically Stained Teeth

-Radiographs
-Transillumination: lacks accuracy

Treatment
-Monitor radiographically
-Root canal therapy (preferred)
-Extraction

78
Q

Non-vital Teeth

A

Treatment Options

79
Q

Fractures Treatment Option
Important Questions

A

What type of fracture?
When did the trauma occur?
Is the tooth vital or non-vital?
Is the apex open or closed?

Endodontic Treatment

-Standard root canal therapy
-Surgical root canal therapy
-Vital Pulp therapy: Complicated crown fractures <48 hours old.
-Odontoplasty and restoration
-Indirect pulp capping

TDIs

-Acute avulsion where owner recovers tooth: place tooth in milk and replace in alveolus within 24 hours
-Avulsions >24hrs: unlikely to save tooth
-Subluxation/Luxation: combination of intraoral splint and root canal therapy

80
Q

OdontoPlasty and Bonding

A

-Indicated for treatment of enamel fractures and uncomplicated crown fractures.
-Additional training in restorative dentistry
-Not recommended for cage chewers, high anxiety dogs, working dogs, stron chewers.
-Annual monitoring and dedicated homecare

81
Q

Standard Root Canal Therapy (RCT)

A

-Involves removal of the pulp to repair a decaying or infected tooth
-Preserves function of a critical chewing tooth
-Performed in referral practice: 1-Acces 2-Preparation 3-shaping and disinfecting 4. Obturation

82
Q

Vital Pulp Therapy

A

-Removal of infected pulp while allowing continued growth and tooth maturation
-Keep the tooth alive to allow root closure and dentinal wall thickening
-Performed in referral practice
-Highly successful if treated within <48 hours

83
Q

Lecture 7

A
84
Q

Anatomy Review

A

Gingivitis: inflammation of the gingiva

Mucositis: inflammation of the alveolar and buccal mucosa

Alveolar mucositis: inflammation of the gingiva and mucosa overlying the cheek teeth.

Caudal stomatitis: inflammation of the mucosa lateral to the palatoglossal arches
-Pathognomonic for FCGS

Faucitis: incorrectly refers to inflammation in the area lateral to the palatoglossal folds.

Glossitis: inflammation of the tongue

Palatitis: inflamation of the palate

85
Q

Differentials Diagnosis for Ulceroproliferative Diseases in Dogs

A

-Generalized Gingivostomatitis/CCUS
-Autoimmune skin diseases: Erythema multiforme, Lupus erythematosus, Pemphigus vulgaris, Mucous membrane pemphigoid.
-Uremia
-Eosinophilic granuloma complex
-Chemical or thermal injury
-Infection: Acute viral, Candidiasis, other systemic infections
-Neoplasia: Epitheliotropic lymphoma, amelanotic melanoma, Others.

86
Q

Local Inflammatory Diseases
Most Common

A

-Feline Chronic Gingivostomatitis (FCGS)
-Juvenile Periodontitis: particularly cats
-Canine Chronic Ulcerative Stomatitis (CCUS): “cups”

87
Q

Feline Chronic Gingivostomatits (FCGS)

A

-Stomatatis: widespread oral inflammation that is beyond gingivitis and periodontal disease and may extend into submucosal tissues.
-Multifactorial involving viruses, bacteria, individual immune response, and living conditions.
-No age predilection except more common in cats <7-8 yrs old
-No sex or breed predilection
-Ulceroproliferative caudal mucositis can be present

Symptoms, PE findings

-Dysphagia and pain
-Extensive inflammation and ulceration of oral soft tissues
-sublingual mucositis and contact mucositis can be present
-PALATOGLOSSAL ARCHES is the most consistently affected region
-Hard palate, labial mucosa, sublingual mucosa are usually spared
-Hypersalivation
-Halitosis

Diagnosis

-Oral exam findings
-IgG very high, hyperglobulinemia
-Dysphagia (swallowing difficulty), anorexia, pain.
-Vocalization at opening the mouth

Medical Management

-Unrewarding, only temporary improvement
-Surgical treatment including full or partial mouth dental extraction is the STANDARD of CARE for FCGS
-Preoperative evaluation should include workup for any concurrent comorbidities such as FIV/FeLV, heart disease, renal disease.

General Practitioner

-Small punch biopsy
-Correct oral diagnosis: bilateral caudal stomatitis, alveolar/labial/buccal/sublingual mucositis.
-CBC, chemistry panel, viral testing
-COHAT
-Score lesions (location, nature, extent, depth)
-Refer to specialist

-May take up to 6mts to resolve
-Revaluation at 3-4 weeks
-Consider immunomodulating treatment of a poor responder (refractory cases)

Refractory Cases

-Immunomodulation therapy
-Glucocorticoids: PREDNISOLONE preferred. 2mg/kg BID for 30 days. Re-check response, taper once inflammation has resolved, may require small dose for the rest of their lives.
-NSAIDs
-Cyclosporine: alternative to glucocorticoids. Whole blood levels >300ug/ml needed, dose varies from cat to cat. Careful, monitor for liver toxicity. Vomiting common sideeffect
-Adjunct pain meds: Gabapentin, Buprenorphine, Fentanyl patch.
-Antimicrobial therapy: ONLY IF secondary infection, immunocompromised or worsening lesions. Most are gram-negatives and anaerobes. AMOXICILLIN-CLAVULANATE, CLINDAMYCIN, METRONIDAZOLE.
-Recombinant Feline Interferon-Omega

88
Q

Juvenile Periodontitis

A

-<9mts of age
-Siamese, Maine Coon, DSH predisposed

PE Findings

-Rapid accumulation of plaque and calculus, with subsequent gingivitis
-NO CAUDAL mucositis
-Non-painful
-Significant early bone loss, periodontal pocket formation, gum recession and furcation exposure

Treatment: dental homecare: 1-TDC capsules applied to gums daily
-Effective treatment and management is difficult
-COHAT with extractions as indicated by radiographs

89
Q

Canine Chronic Ulcerative Stomatitis (CCUS) CUPS

A

-Severe inflammatory condition affecting the gingiva and mucosa of the oral cavity
-Alveolar, labial, buccal mucosa, palatal mucosa along the dental arches, and the lateral margins, and the lateral margins of the tongue may be affected.
**Characterized by contact mucositis, contact with the surface of the teeth “kissing lesions” **

Symptoms

-Depression
-Anorexia
-Inability to fully open mouth
-Restricted yawning
-Pain
-Drooling
-Severe halitosis
-Head shy

PE Findings

-Severe streaking erythema on attached and unattached mucosal surfaces
-Ulcerations on the labial and buccal mucosa
Maxillary soft tissue next to canines and carnassial teeth are common sites for these ulcers
-“kissing lesions” (localized vestibular contact ulcers)
-Intertrigo and dermatitis of the lower lip due to drooling
-Scarring of mucosal tissues at the lip comissures in chronic cases.
-Prevents complete opening of the mouth

Diagnosis

-Based on clinical history and excluding other causes
-Histopathology of lesions can support diagnosis

DDX
-Uremic ulcers
-Ulcers secondary to leptospirosis
-Mucosistis secondary to contact with irritants
-Autoimmune disease, etc.

Treatment Planning

-Consider patient and lifestyle
-Accurately identify and characterize distribution of lesions
-Minimum database CBC/Chem/UA required
-Workup for any concurrent comorbidities prior to anesthesia

Medical Management

-Unrewarding, surgical preferred
-Plaque control is critical
-If conservative treatment desired, start with COHAT
-Medications alone are not effective at controlling disease
-Multimodal pain management critical: NSAIDs + Gabapentin +/- Opioid
-Strategic extraction of teeth causing contact mucositis
-Repeat cleanings q6mts
-Diligent homecare, daily brushing

Surgical treatment

-Extraction of all teeth
-Required multimodal pain management
-Recommended for cases with severe inflammation or owners who can’t commit to COHATs q6mts

90
Q

Autoimmune Disease Conditions

A

-Erythema multiforme (EM)
-Lupus erythematosus (LE)
-Discoid Lupus erythematosus
-Mucocutaneous lupus erythematous
-Pemphigus vulgaris (PV)
-Eosinophilic Granuloma Complex in Cats
-Eosinophilic Stomatitis in the dog
-Wegner’s Granulomatosis

91
Q

Erythema Multiforme

A

-Uncommon
-Vesiculobullous and/or ulcerative skin disease
-Non-specific gingivitis and stomatitis can mimic this disease
-Affected areas include intraoral and extraoral sites: ventrum of trunk, footpads, ponnae, glossitis, etc.

92
Q

Lupus Erythematosus

A

-Discoid lupus erythematosus, mucocutaneous lupus
-DLE: linchenified, crusted, depigmented, and ulcerated lesions nasal planum, perioral, periocular, and pinnal leisons
-SLE: acute lesions on mucosa, skin and ORGANS

93
Q

Pemphigus vulgaris (PV)

A

-RARE
-Middle age dogs, SEVERE
-Antibodies against Desmoglein-3 at the base of the epidermis and oral mucosa
-Hard palate, gingiva, dorsal tongue affected
-Mucocutaneous junction affected
-Oral lesions before skin lesions occur in about 50% of the cases
-Characterized by fragile vesicles, patchy areas of ulceration, crusting and inflammation quickly follow when vesicles rupture and slough

-Subtypes: Paraneoplastic pemphigus has been described in rare cases in dogs

94
Q

Eosinophilic Granuloma Complex in Cats

A

-Oral mucosa, lip, tongue, and palate
-Histopath characteristic presence of Eosinophilic infiltrates
-Lesions are typically linear, well-circumscribed, raised, yellow-pink color
-May be seen in the skin, paws, cheek, lip commissure, chin, pinna of the ear and oral cavity.
-“POUTING” look in the cat from lesions
-C/S: Dysphagia, or ptyalism, interference with swallowing
More common in 2-6 yrs old
Higher incidence in females
Associated with
-Insect bites allergies
-Food allergy
-Atopy
-Immunosuppression
-Bacterial and viral (Calicivirus) infections
Eosinophilic Ulcer, usually concave when indolent
-Granular orange-yellow color upper lip near the philtrum (rodent ulcer)

95
Q

Eosinophilic Stomatitis in DOG

A

-Palatal lesions mostly
-Less involvement of the tongue
Cavalier King Charles Spaniels = dysphagia, paroxysmal coughing or anorexia. Mucosal ulceration, plaque formation and granulomatous lesions. Familial predisposition
Siberian Huskies
-Palatal lesions = no clinical signs
-Young 1-7 yrs old
-Treatment same as cats: Corticosteroids or surgical excision
-May be recurrent

Oral Eosinophilic Granuloma

Siberian Huskies
-Believed to be hereditary or familial
-Usually on the tongue, palatal mucosal
-Halitosis and oral discomfort

96
Q

Wegner’s Granulomatosis

A

-Uncommon
-Autoimmune inflammatory disorder
-C5a complementary system inhibition role in pathogenesis
-Multifocal, bruised, erythematous, expansible, friable gingival lesions
Grossly looks like Neoplasm
-Severe alveolar bone loss on radiographs
-Presence of GRANULOMATOUS inflammation, NOT secondary to fungal or infectious
-DDX: traumatic granuloma, necrotizing ulcerative stomatitis, pyogenic granuloma, neoplasia, immune-mediated conditions.

97
Q

Hyperparathyroidism Dogs

A

AKA: Rubber jaw, renal osteodystrophy, Fibrous osteodystrophy, Osteodystrophia Fibrosa

-Primary hyperthyroidism functional adenoma
-Secondary hyperthyroidism as a result of diet with low calcium-to-phosphorous ratio such as all meat diets.
Radiographic findings are pathognomonic of disease
-Pathophysiology: low phosphorous or low calcium:phosphorous ratio. In renal insufficiency the reabsorption of phosphate is impaired, which results in hyperphosphotemia.
-Hyperphosphotemia a decreased blood calcium stimulates parathyroid gland activity
-Concurrently the synthesis of 1,25-dihydroxyvitamin D in the kidney is also decreased, an intestinal calcium absorption and impaired mineralization of osteoid.

98
Q

Congenital Hypothryroidism Cats

A

-RARE
-Darwfism, soft coat, wide eyes, flat face, delayed skeletal growth, dull mentation (depressed, less alert)
-Historically megacolon and obstipation as primary presenting complain
-Treatment: Levothyroxine and Gingivectomy to allow normal dental eruption

Mentation Reminder
1) Bright, Alert and Responsive (BAR)–self explanatory. This is normal mentation.
2) Depressed or obtunded–the depressed pet is dull or less responsive to normal stimuli.
3) Stuporous–this means the pet is unresponsive until a noxious or painful stimulus is applied.
4) Comatose–this means the pet remains unresponsive with even a noxious or painful stimulus.

99
Q

Uremic Ulcers

A

-Cats more common than dogs
-Cause: chronic renal desease
-Oral bacteria is capable of producing URASE generate cytotoxic levels of ammonia from urea in saliva
-Multifocal oral ulceration
-Fibrinoid necrosis or arterioles within the oral mucosa, necrosis and ulceration by infarction
-Lesions along the ventral aspect of tongue and buccal mucosa
-Foul-smelling film coating the ulcerated surface
-Uremic odor may be detected

100
Q

Lecture 8

A

Interpretation of Dental Radiographs

101
Q

Orientation

A

Orientation Mandible

102
Q

Orientation Mandible

A
103
Q

Orientation Maxilla

A
104
Q

Root anatomy

A

Periodontal disease Percent attachment bone loss

105
Q

Periodontal disease

A

Periodontal disease

106
Q

Periodontal disease

A

Periodontal disease

107
Q

Periodontal disease

A

Endodontic disease

108
Q

Endodontic disease

A

Normal Lamina Dura

109
Q

Normal Lamina dura

A

Evaluation of Lamina Dura

110
Q

Periapical Lucency

A

Tooth Resorption

111
Q

Tooth Resorption

A

Tooth Resorption types based on radiographs

112
Q

Tooth Resorption Based on Radiographs

A

Tooth Resorption Based on Radiographs

113
Q

Tooth Resorption in Dogs

A

-Most often in mandibular premolars
-Often begins in the roots
-If it doesn’t extend beyond the level of gingiva, can monitor
-If extends into crown or is communication with oral cavity = extract, painful
-Roots can be ankylosed (fused, immobile)

114
Q

Radiographs and Extractions

A

Pre-extraction

115
Q

Pre-extraction radiographs

A

Pre-extraction radiographs

116
Q

Lecture 9 Oral Pathology 2

A
117
Q

Review Key-terms

A

-Sarcoma: characterizes tumors that arise from non-epithelial tissue

-En bloc: a procedure in surgical oncology aiming to remove a tumoral mass in its entirety, completely surrounded by a continuous layer of healthy tissue

-Palliative: palliative treatments aim to shrink cancer, slow down its growth or control symptoms, NOT cure
-Definitive: therapy aimed to minimize the long-term adverse effects of therapy and to enhance the long-term control of the cancer. Definitive intent radiation therapy generally means that small dosages of radiation are administered daily over 2-4 weeks.

118
Q

Neoplasia of the Oral Cavity

A
119
Q

Bening Odontogenic Tumors

A

General symptoms and PE Findings

-Generally Asymptomatic in early stages
-Advance stage interference with prehension and mastication

Symptoms include
-Dropping food
-Halitosis
-Reluctance to eat or drink
-Reluctance to play aggressively with toys
-Hypersalivation: blood-tinged saliva

PE findings
-Raised smooth non-pigmented gingival swelling associated with affected tooth
-Pseudopocket may be present
-May bleed upon palpation

120
Q

Overview of Diagnosis and Treatment

A

Minimum database
-CBC
-Chemistry profile with electrolytes
-UA with USPG if renal values are borderline
-Species specific ELISA test: HWT, FIV/FeLV
-Comprehensive Oral Health Assessment and Treatment (COHAT) requires anesthesia, imaging.
-Cone beam radiograph for advanced diagnostics
-Perform punch biopsy for for definitive diagnosis
-Request pathologist familiar with oral path

Treatment
-Local control of disease via surgery
-Aggressiveness of surgery is determined by tumor type

121
Q

Peripheral Odontogenic Fibroma

A

Second most common odontogenic tumor AKA “Epulis”

-Slow growing, bening, firm, sessile (immobile, attached, no stalk) to pedunculated (stalk present), non-ulcerated lesion
-Usually associated with free gingiva of tooth
-NOT invasive
-No predilection sex, age, breed or jaw location
-Can lead to secondary periodontitis
-Traumatized during mastication possible
1. Fibromatous: only soft tissue
2. Ossifying: osseous metaplasia
Create pseudopocket

Diagnostics
-Radiographs: soft tissue inflammation minimal osseous tissue involment
-Histopath: Base of proliferative neoplasia, FIBROUS tissue anchored to the periosteal surface of the alveolar crestal bone near the CEJ with NO PDL involvement

Treatment
-En bloc resection of tumor and associated tooth/teeth
-More aggressive Tx for recurrent cases

122
Q

Canine Acanthomatous Ameoblastoma

A

Most common OT in dogs

-Not seen in cats
-Locally infiltrative, NOT metastatic
-Commonly appear as extraosseous inflammation with or without tooth displacement and bone invasion
-Arises from odontogenic epithelium in gingiva of tooth-bearing areas of JAWS
-Location: rostral mandible»mandible>maxilla
Infiltration into surrounding bone, which makes it different from Central Ameloblastoma
Golden Retriever, Akita, Crocker, Sheltie

Radiographs
-Bone infiltration
-Alveolar bone resorption
-Tooth displacement

CT
-Interosseous cystic and appeared subjectively more aggressive than extraosseous

Histology
-Islands and sheets of mature cells that are clearly squamous epithelium.
-PALISADING cells bonded to island sheets…

Treatment of choice
-Current recommendations is wide excision (en bloc) with 1cm margins

Excellent prognosis with wide local exc

123
Q

Odontoma

A

-Bening, inductive tumor
-6-18 mts of age
-Cats and dogs
-Well differentiated cells
-Non-painful, expansile, MN or MX
-Associated with UNERUPTED tooth
-Alveolar inflammation

  1. Compound odontoma
    -Tooth-like structures
    -Orderly pattern
    -DENTICLES on radiographs
  2. Complex odontoma
    -Didorderly dental tissue

Radiographs
-Well defined tumor mass calcified material

Treatment
-Marginal excision or even intracapsular may be curative

124
Q

Amyloid-Producing Odontogenic Tumor

A

APOT

-RARE
-Suspected derived from ameloblasts
-Gingival enlargement
-Slow growing expansile mass

-Middle age to older dogs

Radiographs
-Unilocular to multilocular radiolucency
-Cystic appearance

Treatment
-Surgical resection with 1 cm margin appears to be curative in most cases
-Enucleation alone = high recurrence

125
Q

Feline Inductive Odontogenic Tumor

A

FIOT

-8-18 mts of age
-Inductive bc epithelium induces mesenchymal cells to form aggregated foci of dental pulp-like cells
-Raised, submucosal, rapidly expansive, locally destructive, soft tissue masses
-Usually unilateral on rostral maxilla
-Does not metastasize

Treatment
-Wide excision is curative

126
Q

Feline “Epulides”

A

-Term applies to a variety of gingival masses in the cat
-Combination of fibromatous, ossifying, and acanthomatous (small red raised growth) features

Treatment
-Surgical management recommended, but challenging
-High recurrence rate
-Some cases may require multiple extractions and gingivectomy

127
Q

Malignant (non-odontogenic) Tumors

A

General symptoms

-Generally asymptomatic at early stage of disease
-Late stage interference with prehension and mastication
-Halitosis
-Dropping of food
-Reluctance to eat or drink
-Hypersalivation
-Bleeding from the mouth
-Exophthalmia

Exam findings
-Destruction of local anatomy
-Wide-based, +/- ulcerative mass spanning multiple teeth
-Sometimes involves the tongue, lips, and tonsils
-Extraoral distortion and inflammation
-Regional lymphadenopathy because they are
-Metastatic

Overview Diagnosis and Treatment

-Minimum database: CBC, chemistry with electrolytes, UA with USPG, Species specific ELISA test.
-Consider stagging for metastasis if malignancy is suspected = Chest x-rays, biopsy.
-COHAT: requires anesthesia, requires imaging, dental radiographs standard of care. Cone beam computed tomography (CBCT) for advanced diagnostics
-Perform punch biopsy or incisional biopsy during COHAT for definitive diagnosis

Treatment
-Local control of disease
-Curative intent surgery vs. palliative intent
-Consider follow-up radiation and chemotherapy pending margins

128
Q

Squamous Cell Carcinoma (SCC)

A

Second most common oral tumor in dogs
First most common tumor in cats

-Aggressive local infiltration, associated with papillomavirus infections, flea collars, diet, prior radiation, chronic mucosal or periodontal inflammation, tobacco smoke.
-Masses are typically friable, red, and vascular
-On gingiva, tonsils, oral mucosa, lip, palate, tongue, pharynx.
-May present as ulceration with no evidence of a mass, loose teeth, history of extraction with subsequent tumor growth at the extraction site.

Radiographs
-May resemble osteomyelitis
-Primary osteolytic process, MOTH-EATEN appearance, radiolucent and ill-defined borders

Treatment Dog
-Surgery, aggressive resection with 1 cm margins
-Mandibulectomy/Maxillectomy

Treatment Cat
-Surgery often not possible because of the size of the tumor
-No tolerance for mandibulectomy/maxillectomy

Radiation (non-tonsilar)
-Limiting regional spread of disease

Chemotherapy
-NOT recommended

Combination of radiation and surgery are associated with a better prognosis

COX-2 inhibitors: NSAIDs
PEROXICAM

129
Q

Papillary SCC

A

-Maybe Papillomavirus association
- <2-5mts of age in dogs
- Adults up to 9 yrs also can be affected
-Locally aggressive, with underlying bone lysis
-Responds well to surgery and orthovoltage radiotherapy combined
-NO reports of METASTASIS

  1. Intraosseous: within the jaw bone (cavitating pattern = deep erosion, valley-like)
  2. Exophytic: mass grossly similar to benign papilloma (non-cavitating pattern)

Treatment
-Surgical excision with 1cm margin may be curative

130
Q

Oral Malignant Melanoma

A

-One of the most common oral malignancies
#1 Most common in Dogs

-Gingiva, labial mucosa, palate, buccal mucosa, tongue.
-Typically firm, grayish or brownish black, can also lack pigment
-Ulcerative, friable, readily bleeds
-Fast growing
-NECROSIS common feature
-Gingival tumors have the worst prognosis

Treatment
-Surgical resection for locoregional control
-1 cm margins may be curative
-May follow up with radiation
-METASTATIC high rate to lymphnodes and lungs
-Vaccine questionable efficacy

Prognosis
-Highly dependent on size at time of surgery
-Variable

131
Q

Fibrosarcoma

A

3rd most common malignant tumor in the DOG

2nd most common malignant tumor in the CAT

-More common in large breed dogs
-Average age 8 yrs old
-Lesions in gingiva, hard palate, labial mucosa, soft palate, tongue.
-Tends to present in broad-based, firm swelling.

Radiographs
-Infiltrative: challenging to discern full extent of tumor even with advanced imaging

Treatment
-Aggressive surgery 2cm margins
-Post surgical radiation combined, best outcome
-High level of local recurrence with surgery alone

132
Q

High-Low Fibrosarcoma

A

-Appears benign histiologically but behaves like an aggressive malignant neoplasma

Golden retrievers predisposed
-3-13 yrs old

Treatment
-Low-grade histo but high grade biologically must be aggressive, 1-2 cm surgical margins
-72% cases with bone lysis
-12% Lymph node metastasis

133
Q

Osteosarcoma

A

-Often present as soft tissue mass
-Red, friable, surrounding bone lysis rather than hard tissue proliferation
-OSA - more common in appendicular than axial skeleton
-Maxilla and mandible commonly affected sites

Treatment
-Surgery: median survival 9mts
-28% local recurrence in dogs
-Chemo NOT useful
-Radiation therapy may be helpful

Cats
-Poor prognosis as surgical options are limited

134
Q

Benign non-odontogenic Tumors

Papilloma

A

-Young dogs
-Multiple verrucous lesions on the tongue, lip, and cheeks.
-“SEA ANEMONE” appearance

Treatment
-Most regress on their own
-Cleated by immune system
-Refractory cases: concurrently immunocompromised
-Vaccine may be helpful

135
Q

Osteoma

A

-Can occur in dogs or cats
-Well circumscribed, firm raised boney lesions
-Located in hard palate, zygomatic arch, caudal mandible

Histiopath
-May not show neoplasia characteristics
-May show normal woven bone

Treatment
-Surgery
-Maxillectomy, mandibulectomy, debulking all resulted in > 1 yr survival

136
Q

Plasmacytoma

A

-RARE
-Well circumscribed red mass often <1 cm
-Biologically more aggressive than cutaneous counterparts
-May undergo local invasion to underlying bone

Treatment
-Surgical margins 1 cm have been curative

137
Q

Lecture 10 Extractions

A
138
Q

Review Anatomy

Which teeth are two rooted, three rooted, one rooted?

A

Review Anatomy

One root

-Incisors (01-03)
-Canine (04) 104, 204
-PM1 (05)
-M3 (11) mandible

Two Roots

-PM2 (06), PM3 (07), PM 4 (08), M1 (09), M2 (10) Mandible
-PM2 (06), PM3 (07) Maxilla

Three Roots

-PM4 (08), M1 (09), M2 (10) Maxilla

Cats

-PM2 (06), PM3 (07), M1 (09) Maxilla - Two Roots
-PM4 (08) Maxilla - Three Roots

139
Q

Review Terminology

A

-Simple extraction: not requiring a gingival incision or sectioning the tooth. AKA Closed, uncomplicated, or nonsurgical extraction
-Surgical extraction: requiring a gingival incision, bone removal, and/or sectioning of the tooth. AKA open or complicated extraction
-Elevation: The process by which the periodontal ligament is PDL FATIGUED or TORN and alveolar bone is expanded to facilitate removal of the tooth from alveolus. Using ELEVATOR as a lever, the tooth is lifted (elevated) from its alveolus.
-Luxation: the process by which the PDL is CUT or severed to loosen teeth from the surrounding alveolar bone

140
Q

Indications and Contraindications for Extractions

A

Indications

-Severe periodontitis
-Pulp necrosis
-Tooth resorption
-Stomatitis
-Fractures teeth: adult or deciduous
-Traumatic malocclusion
-Crowding
-Unerupted teeth
-Unhealthy teeth in jaw fracture line
-Failed endodontic treatment

Contraindications

-Ethics: disarming procedures
-Severe systemic disease: congestive heart failure, uremia, uncontrolled endocrine disease, severe coagulopathies, untreated leukemia/lymphoma
-Previous radiation therapy: leads to osteonecrosis, poor wound healing.

141
Q

Surgical Principles for the Oral Cavity

A

Fundamental steps for successful extraction

  1. Adequate visualization: of the tooth to be extracted
  2. Unimpeded pathway for the removal of the tooth
  3. Use of controlled force to luxate or elevate and remove the tooth
142
Q

Equipment and Intruments

A

A. Minnesota lip retractor
B. Feline lip retractor
C. Miller bone curette
D. Periosteal elevator
E. Straight and winged elevators
F. Small breed extraction forceps
G. Needle holders
H. Iris scissors
I. Thumb forceps
J. Scalpel blade handle

-High speed hand piece: used to modified pen grasps, resting figure on patient
-Burs: Round burs, taper fissure crosscut burs, Diamond burs.
-#15 or 15C scalpel blade
-Root tip pick
-Root tip extraction forceps (small tips)
-Miller bone curette for debriding granulation tissue, bone fragments, debris in alveolus
-Suture: 4-0 or 5-0 Monocryl, reverse cutting or tapered needle

143
Q

Biomechanical Principles for Extractions

A

Elevators = Levers

-Transmit a modest force into a small movement against great resistance. Mechanical advantage
-Rotation of elevator in PDL space utilizes the surrounding alveolar bone as a fulcrum
Elevator is never placed or used adjacent to teeth that will not be extracted

Luxator = Wedges

-Wedged into PDL space and pressure is directed apically
-Made of softer metal and can’t be used as a lever

144
Q
  1. Elevator
  2. Luxator
  3. Extraction forceps: Leverage
A

Twist once and pull down and out in a continuous movement

145
Q

Dental Machine

A

High-speed hand piece

146
Q

Air supply

A

Different Burs

147
Q

Surgical Principles and Techniques

Flap Design

A

-Good exposure: sufficient size
-Preserve blood supply: wide base compares to free margin
-Prevent dehiscence/delayed healing: edges of flap must lie over intact bone
-Non-tension closure: make vertical releasing incisions at line angles of adjacent teeth vs. parallel to allow for large non-tension flap

148
Q

Principles of Suturing Oral Mucosa

A

A. Insert at 90 degree angle
B. Rotated to easily pass through tissue

149
Q

Patient Preparation

A

-Intraoral radiographs PRIOR to any tooth extraction REQUIRED
-Post-extraction intraoral dental radiographs REQUIRED

150
Q

Retained Tooth Root Fragments (RTRF)

A

-Attempts should be made to remove retained tooth root fragments
-Leaving fragments <4mm may be considered if patient is at high risk, unstable under anesthesia, or damaging surrounding structures.
- >4mm poor practice
-Exception end-stage resorption
-If root tips are left for intentional resorption oral exam and x-ray follow up REQUIRED to track changes

Post-operative care

-Multimodal pain control: locoregional nerve blocks (perioperatively), NSAIDs, Gabapentin, Opioid +/-
-Homecare: soft food 5-7 days, avoid chewing on toys and treats for 7-10 days. Wait 3-4 days to resume brushing and avoid extraction area
-Ice packs can help with subcutaneous emphysema from high speed hand piece
-Oral rinses chlorhexidine q12hrs after meals
-Monitor for signs of halitosis, inappetence, lethargy
-Post-op check 2 weeks later
-Antibiotics typically NOT needed

151
Q

Simple Extraction Technique

A

Preparation
-Remove calculus and rinse mouth with 0.05%-0.012% chorhexidine gluconate
-Elevation technique
1. First class lever: between tooth and alveolar bone, which acts as a fulcrum (pivot point) to release the tooth
2. Wedge lever: elevator placed perpendicular to the long axis of the root and rotated so the force is directed outward from the alveolus
3. Luxator technique
-Not intended for rotational forces
-Always place it parallel to the root surface
-Placed into the PDL space and advanced apically
-Best place is along the mesial and distal surfaces of the tooth (not labial or lingual)

Forceps: once loosened, place on the root as apically as possible and with the beaks parallel to the long axis, place rotational force.

152
Q

Simple Extractions Tips/Tricks

A
153
Q

Surgical Extractions

A

-Requires gingival incision, bone removal, and or sectioning of the tooth
-Technically can apply to any tooth
-Commonly PM4 maxilla
-M1 mandibular
-Canine teeth
-Others not affected by advanced stage periodontal disease

PM4 Maxillary

Indications: fracture, periodontal disease, non-vital tooth

-15 blade, create incision for mucoperiosteal flap
-Using periosteal elevator, elevate gingiva and mucosa off tooth and alveolar bone
-Using either a diamond bur or 2-, or 4-, round bur, remove alveolar bone to expose the mesial and distal roots. Typically recommended to remove coronal 2/3 of alveolar bone
-Using cutting but (701, 701L) section the tooth into 3 positions along root segments.
-Using elevation technique of choice, remove each root individually. Remove distal, then mesial-buccal, then messial-palatal
-Using a diamond bur (smoothing bur) remove sharp edges of alveolar bone (Alveoloplasty)
-Using iris scissors perform a periosteal releasing incision on mucogingival flap to ensure tension-free closure
-Suture the flap closed in simple interrupted suture pattern using 4-0 MONOCRYL in dogs and 5-0 MONOCRYL in cats.

154
Q

Surgical Extractions 2-Rooted tooth

A

Indications: fracture, periodontal disease, non-vital tooth

Watch out for mandibular canal, periodontal risk for jaw fracture M1

-15 blade, create incision for mucoperiosteal flap
-Using periosteal elevator, elevate gingiva and mucosa off tooth and alveolar bone
-Elevate lingual mucosa as well as either before or after extraction for tension-free closure
-Using either a diamond bur or 2-, or 4-, round bur, remove alveolar bone to expose the mesial and distal roots. Typically recommended to remove coronal 1/2 of alveolar bone
-Using cutting but (701, 701L) section the tooth into 2 positions along root segments.
-Using elevation technique of choice, remove each root individually. Remove distal, then mesial-buccal, then messial-palatal
-Using a diamond bur (smoothing bur) remove sharp edges of alveolar bone (Alveoloplasty)
-Using iris scissors perform a periosteal releasing incision on mucogingival flap to ensure tension-free closure
-Suture the flap closed in simple interrupted suture pattern using 4-0 MONOCRYL in dogs and 5-0 MONOCRYL in cats.

155
Q

Surgical Extraction Maxillary Canine

A

Indications: Fracture, periodontal disease, non-vital tooth

Watch out for penetration into the nasal cavity

-15 blade, create incision for mucoperiosteal flap
-Using periosteal elevator, elevate gingiva and mucosa off tooth and alveolar bone
-Using either a diamond bur or 2-, or 4-, round bur, remove alveolar bone to expose the mesial and distal roots. Typically recommended to remove coronal 1/2 of alveolar bone
-Careful accidental iatrogenic ONF formation
-Using elevation technique of choice, remove the tooth.
-Can use butting bur (701, 701L) to “notch” either side of the alveolus (socket) at the level of the CEJ to create space to place your elevator or luxator
-Using a diamond bur (smoothing bur) remove sharp edges of alveolar bone (Alveoloplasty)
-Using iris scissors perform a periosteal releasing incision on mucogingival flap to ensure tension-free closure
-Suture the flap closed in simple interrupted suture pattern using 4-0 MONOCRYL in dogs and 5-0 MONOCRYL in cats.

156
Q

Surgical Extraction of Mandibular Canine

A

Indications: Fracture, periodontal disease, non-vital tooth

Watch out for mandibular canal, potential risk for mandibular fracture
Never remove bone on the lingual aspect of the tooth = JAW FRACTURE

-15 blade, create incision for mucoperiosteal flap
-Using periosteal elevator, elevate gingiva and mucosa off tooth and alveolar bone
-Using either a diamond bur or 2-, or 4-, round bur, remove alveolar bone to expose the mesial and distal roots. Typically recommended to remove coronal 1/2 of alveolar bone
-Using elevation technique of choice, remove the tooth.
-Can use butting bur (701, 701L) to “notch” either side of the alveolus (socket) at the level of the CEJ to create space to place your elevator or luxator
-Using a diamond bur (smoothing bur) remove sharp edges of alveolar bone (Alveoloplasty)
-Using iris scissors perform a periosteal releasing incision on mucogingival flap to ensure tension-free closure
-Suture the flap closed in simple interrupted suture pattern using 4-0 MONOCRYL in dogs and 5-0 MONOCRYL in cats.

157
Q
A