Ex psych readiness assessment 2 Flashcards

You may prefer our related Brainscape-certified flashcards:
1
Q

What is panic disorder? What reactions does it cause?

A

Intense anxiety that strikes without warning or an obvious source
Causes chest pains, short breaths, choking or dizziness

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2
Q

What is OCD? What is this due to?

A

Repetitive actions or rituals done to relieve anxiety

Due to intrusive thoughts

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3
Q

What is PTSD?

A

Anxiety that occurs in a delayed reaction in response to trauma or a stressful situation

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4
Q

What is GAD? Does it cause comorbidity?

A

Uncontrollable worry about multiple concerns that last at least 6 months
It has the highest rate of comorbidity

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5
Q

What are the 3 components of anxiety?

A

Cognitions = thoughts
Emotional responses
Physiological changes = muscle tension, autonomic NS hyperactivity)

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6
Q

Which of these components distinguishes anxiety from arousal? Why?

A
Cognitions and emotional responses
Because there can be non-specific physiological responses:
- increased muscle tension
- increased HR
- increased alertness
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7
Q

What is state anxiety? What is usually prescribed?

A

An immediate response to anxiety that is transient/temporary

Usually prescribed single bouts of ex

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8
Q

What is trait anxiety? What is usually prescribed?

A

A predisposition to perceive a situation as threatening and is an expression of personality
Usually prescribed multiple bouts of ex

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9
Q

What have anxiety+ex studies mostly examined?

A

Individuals without anxiety disorders

Individuals with medical conditions other than anxiety (e.g. stroke rehabilitation)

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10
Q

Why have they only examined this?

A

During the 1960-70s, PA was thought to induce panic attacks

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11
Q

What experiment did Pitts and McClure do in 1967? Was it correct or incorrect?

A

Assessed anxious people after intravenous infusion of sodium DL-Lactate
It was an incorrect conclusion

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12
Q

What were the results?

A

It caused increased anxiety and induced panic attacks in 13 of 14 patients

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13
Q

Why does this observation matter in terms of ex?

A

High intensity/anaerobic ex causes lactate production

So it was believed that ex would cause anxiety attacks

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14
Q

Why was this an incorrect conclusion?

A

There is a difference in the breakdown of sodium DL-Lactate infusion and lactate from ex
DL-Lactate caused metabolic alkalosis which led to hyperventilation
Ex induced lactate causes metabolic acidosis

Literature concludes the correct association between ex and panic attacks
444 ex bouts done by 420 panic disorder patients
Only 5 had panic attacks
Supramaximal treadmill tests also showed more lactate levels that infused DL-Lactate but had minimal panic attacks (1 of 35)

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15
Q

What conclusions were made about preventing anxiety in cross sectional studies?

A

Regular PA reduces the odds of anxiety symptoms and anxiety disorder diagnoses in the past year
A dose response reduction is seen

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16
Q

What conclusions were made about preventing anxiety in prospective cohort studies?

A

PA is associated with reduced risks of anxiety disorders (25-50%)

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17
Q

What does the meta-analysis say about state anxiety and ex?

A

There is an acute reduction in state anxiety post ex
The largest effects are seen 5-30 mins after ex
20-30 min bouts of ex produce the greatest change in state anxiety

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18
Q

What are the conclusions on the effectiveness of ex on state anxiety?

A

Ex is as effective as other treatments like biofeedback, meditation and medication
Ex is no more effective than quiet rest or distraction
(even though the effects of ex induced reductions in anxiety lasts longer)

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19
Q

What were some of the research limitations prior to 1993?

A

Most research could not quantify relative ex intensity (they were not based on one’s own fitness level)
Most research could not examine varying intensities with the same participants
Most research only utilised sub-max tests to determine aerobic capacity (which can be up to 20% off their true aerobic capacities)

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20
Q

What have been some of the research limitations/improvements since 1993?

A

Quantifying ex has become more standard using VO2max or peak (although inactive experience can cause higher metabolic strain)
The bulk of evidence still relies on self report which causes bias because it is harder to conceal the contents of an assessment (active people tend to have greater expectations with being active in reducing anxiety)
Majority of research only examines aerobic ex

21
Q

What does the meta-analysis say about trait anxiety?

A

Chronic ex lowers trait anxiety in people without anxiety disorders, people with chronic medical conditions and people with high trait anxiety
People with high trait anxiety experience the greatest change in anxiety

22
Q

What are the conclusions in the effectiveness of ex on trait anxiety?

A

Ex is as good as active treatment and better than controls
However the magnitude of change tends to be similar despite ex type, duration or pattern
And an optimal program length and ex time is still TBD

23
Q

What were 3 experimental studies to research patients with anxiety disorders?

A

Anaerobic ex vs SSRI
Walking vs Group Cognitive Behavioural therapy
Ex vs GAD

24
Q

What were the results of the anaerobic ex vs SSRI study?

A

10 week program
27% placebo and aerobic ex patients dropped out
0% SSRI patients dropped out

25
Q

What were the results of the ex vs GAD study?

A

Resistance training saw 60% remission rate
Aerobic training saw 40% remission rate
Control group saw 30% remission rate (could be placebo effect)

26
Q

What are some of the issues in studies of individuals without anxiety disorders?

A

Daily stressors can have a negative impact

Studies mostly examined low-moderately anxious people

27
Q

What are some of the limitations with studying individuals without anxiety disorders?

A

Generalisability
Can cause the floor effect (due to little room for improvement)
No strong evidence to say ex specific scales should be used for anxiety
Physiological changes (muscle tension and reflexes) can happen regardless of anxiety, so self report corroboration is needed

28
Q

Do anxiolytic effects result from the environment or ex?

A

We don’t know

29
Q

Is there a change in simple phobias after ex?

A

No

30
Q

Which anxiety disorder has a higher likelihood for change?

A

GAD

Only 25% are receiving treatment

31
Q

Do we know why/how ex reduces anxiety? Why/why not?

A

No
We need clarification on the optimal dose (intensity/mode/duration)
Anxiety and ex are multifaceted

32
Q

What is the meaning of cognitive appraisal?

A

When anxious individuals overestimate the intensity of a feared event and underestimate their ability to cope
Physiological symptoms associated with anxiety can be misinterpreted

33
Q

What is the relationship between ex and arousal?

A

Ex can redefine the subjective meaning of arousal

34
Q

What is the time-out hypothesis? Who invented it?

A

Breus and OConnor in 1998
Ex used as a distractor to test 18 high trait anxious females
Either cycling, cycling while studying, or quiet rest

35
Q

Which category of ex caused the highest change in state anxiety and which didn’t? What conclusion was made?

A

Cycling had the largest change
Cycling while studying and quiet rest saw no change
Studying blocks anxiolytic effects of ex

36
Q

What was shown about anxiety through animal models?

A

Anxiety can be inferred through

  • reduced locomotion
  • reduced approach to the centre of an open field
  • freezing
  • defecation
  • urination
  • shivering
37
Q

What is the function of the amygdala?

A

The emotion centre of the brain and a critical neural structure for anxiety

38
Q

Where does the amygdala receive info from?

A
The thalamus (the sensory gateway that mediates pain)
The locus coeruleus (in the Pons for norepinephrine causing changes in sleep, attention and emotional responses)
39
Q

What biological centres does this info get extended to?

A
Cognitive
Affective
Neuroendocrine 
Cardiorespiratory
Musculoskeletal
40
Q

What types of drugs impact serotonin release?

A

Antidepressants

Anxiolytic drugs

41
Q

What have animal studies shown about serotonin and ex?

A

Ex training increases the activation of the raphe nuclei which increases the synthesis of serotonin

42
Q

What have human studies shown by inference about ex and serotonin?

A

Serotonergic systems are impacted by ex

  • increased lipolysis
  • increased free tryptophan to the brain
  • increased serotonin production

Also increases free fatty acids (this is the best support for ex induced increase in serotonin

43
Q

What is norepinephrine?

A

The nonadrenergic system involved in anxiety

44
Q

Where is it released from?

A

The locus coeruleus releases this into the body to prepare it for action

45
Q

Does this increase or decrease anxiety behaviour?

A

Causes anxiety like behaviour in animals

which is why many anti-anxiety meds act on LC neurons

46
Q

Can ex impact the locus coeruleus?

A

Research is limited

47
Q

What is GABA?

A

A major neural inhibitory transmitter

Involved in the expression of anxiety

48
Q

What medications are there to target GABA?

A

Benzodiazepines (e.g. valium)

Increase the effect of GABA as it binds to GABA receptors and inhibits neutron activity

49
Q

What have animal studies on GABA and ex shown?

A

Chronic wheel running and treadmill running increases GABA and reduces GABAa receptors (inhibitors)
Increased open field locomotion