Everything else Flashcards
Signs and symptoms if the left ventricle is affected
Shortness of breath, tachypnea and respiratory crackles
Signs and symptoms if the right ventricle is affected
distended neck veins, liver enlargement , anorexia and nausea
At risk for heart failure sections and classes
At Risk for Heart Failure
* Stage A. At high risk for heart failure, but without structural disease or symptoms of heart failure
* Stage B. Structural heart disease, but without signs or symptoms of heart failure
Cardiac Failure
* Stage C. Structural heart disease with prior or current symptoms of heart failure
* Stage D. Treatment-resistant heart failure requiring specialized intervention
Nursing care for CCF
Maintain the patient in high fowler’s position
Elevate extremities
Frequently monitor vital signs
Change position frequently
Monitor intake and output and daily weight
Restrict fluids as ordered
Signs and symptoms left-sided HF
ONE: Nearly all of these symptoms can be seen in heart failure, particularly left-sided failure (PND, orthopnea, crackles, S3 gallop).
TWO: Pulmonary Edema: Crackles, dyspnea, orthopnea
THREE: Shock/Hypoxia: Cold, clammy skin, cyanosis, fatigue,
Signs and symptoms right-sided HF
Peripheral oedema
jugular venous distension
Hepatomegaly
Abdominal pain
Nausea
Anorexia
Bloating
Diagnostic studies for CCF
- Electrocardiography (ejection fraction)
- MRI
- Computed tomography imaging
- Cardiac catheterization with angiography
- A biopsy of heart muscle may be needed
Complication/largest risk for CCF
Pulmonary edema
Acute pulmonary edema is defined as the sudden increase in PCWP - Pulmonary capillary wedge pressure (usually more than 25 mm Hg) as a result of acute and fulminant left ventricular failure. It is a medical emergency and has a very dramatic clinical presentation. The patient appears extremely ill, poorly perfused, restless, sweaty, tachypneic.
AKI Pathophysiology/phases/S+S
Acute Kidney Injury (AKI) is a sudden and almost complete loss of kidney function over a period of hours to days.
Glomerular injury, vasoconstriction of capillaries or tubular injury due to sepsis, trauma, nephrotoxins etc. can lead to:
Oliguria (less than 400 mL/day of urine) or anuria (less than 50 mL/day of urine.
High serum creatinine and blood urea nitrogen levels (azotemia) and retention of other metabolic waste products normally excreted by the kidneys.
AKI diagnostic studies
Kidney Function Tests:
Blood Urea Nitrogen (BUN) & Creatinine: Elevated levels indicate impaired kidney function.
Complete/Full Blood Count (CBC):
Used to detect underlying conditions that could contribute to or result from AKI.
Serologic Tests:
Assess for autoimmune or infectious causes.
Electrolytes:
Look for imbalances, particularly potassium, sodium, and bicarbonate, which are often disrupted in AKI.
Bladder Pressure Measurement:
AKI Nursing management
Auscultation (Lung and Heart Sounds):
Monitor for: Fluid overload, which may lead to pulmonary oedema and heart failure.
Signs: Adventitious breath sounds (crackles), extra heart sounds (e.g., S3, S4).
Assess Level of Consciousness:
Monitor for: Changes in mentation or restlessness.
Indicates: Potential fluid shifts, toxin accumulation, acidosis, electrolyte imbalances, or hypoxia.
Interventions:
Fluid Replacement:
Plan with patient: Set fluid intake within prescribed restrictions.
Suggestions: Offer a variety of drinks (hot, cold, frozen) spaced throughout the day.
Goal: Reduce feelings of deprivation and thirst.
Correct Reversible Causes of Acute Renal Failure (ARF):
Examples:
Replace lost blood.
Maximize cardiac output.
Stop nephrotoxic drugs.
Relieve obstructions (surgery).
Outcome: Restoring normal kidney function and preventing lasting damage.
Skin Care:
AKI clinical assessment/findings
Skin:
Digital ischemia, butterfly rash, palpable purpura: Suggests systemic vasculitis.
Maculopapular rash: Indicates allergic interstitial nephritis.
Track marks: Points to intravenous drug abuse.
Eyes:
Dry conjunctivae: Seen in autoimmune vasculitis.
Jaundice: Indicates liver diseases.
Band keratopathy: Associated with hypercalcemia.
Retinopathy: A sign of systemic diseases affecting the kidneys.
Pulmonary System:
Rales: Could indicate fluid overload or pulmonary edema.
Hemoptysis: May suggest an underlying pulmonary-renal syndrome or severe infections.
Complications AKI
Fluid Volume overload: Acute kidney failure may lead to an excess of fluid in the lungs, which can cause shortness of breath (Pulmonary oedema).
Neurological disorders: Siezures may occur with the increase in nitrogenous waste accumulating.
Hyperkalaemia: Elevated levels of potassium in blood are particularly dangerous. At risk of developing a life threatening arrhythmia.
Aetiology Resp Failure
Smoking
Obesity
^Age
Trauma
Near drowning
Sepsis
Lung diseases (COPD)
Asbestos/chemical inhalation
ARDS diagnostic criteria
1Acute onset
2Bilateral pulmonary infiltrates on CXR
3No evidence of left ventricular heart failure
resp failure pathophys types one and 2
T1 (HYPOXEMIC too little O2)
PO2 less than 60mmHg or 50PCO2 unaffected
T2 (HYPERCAPNIA too much co2)
Too much PCO2, greater than 50mmHg
Main S+S ARDS
^HR, ^RR, RESP alkalosis (Blood PH rising because not enough O2), resp crackles.
Treatment Resp failure
Antibiotics + Corticosteroids
chest exercise + suctioning
Fluid resuscitation
O2 therapy, nebs, humidify.
ARDS pathophys primary insult to resp failure chart
triggers inflammatory response - becomes too much for blood vessels in alveoli space - decreased vascular permeability - fluid shift alveoli and into lungs - decreased surfacant production and decreased lung compliance - poor gas exchange -
Aetiology of cardiac disturbances
Genetics, older age, CCF, AKI, pulmonary diseases
Cardiac disturbances pathophys
Electrical misfiring in the heart, further in the SA node, purjunki fibres and bundle of his. The electrical misbalance effects the contractibility and it cannot fill to pump correctly. Not enough blood gets through systemic circulation. Decreased tissue perfusion = cell ischemia.
Complications of a cardiac arrythmia
Stroke
Death
Signs and symptoms cardiac arrythmia
decreased GCS, Increased RR and decreased O2.
Treatment for cardiac disturbances
Cardioversion, echocardiogram, beta blockers, angiogram (stents).