Evaluating Patients with Dyspnea Flashcards

1
Q

how is tachypnea defined?

A

RR > 20

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2
Q

in what kind of acid-base disturbance can a patient be observed to be breathing rapidly but may NOT feel short of breath?

A

metabolic acidosis - increased ventilation as a compensatory mechanism

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3
Q

what are the ABC’s of evaluating a patient with dyspnea?

A

Airway - obstruction?

Breathing - can patient speak full sentences?

Circulation

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4
Q

name a pulmonary cause of dyspnea for each category:
a. airway
b. parenchyma
c. vascular
d. pleura

A

a. airway: asthma, bronchiectasis, COPD, foreign body, anaphylaxis
b. parenchyma: interstitial lung disease (fibrosis), acute lung injury (ARDS), pneumonia
c. vascular: pulmonary embolism, pulmonary artery HTN
d. pleura: pleural effusion, pneumothorax

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5
Q

how can anemia be a cause of dyspnea?

A

not enough hemoglobin to transport sufficient oxygen

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6
Q

where does pleuritic chest pain come from?

A

lung parenchyma and visceral pleura do not have pain fibers

pleuritis is chest pain caused by inflammation of the parietal pleura - receives systemic circulation and has somatic nerve fibers, pain afferents go to intercostal and phrenic nerves

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7
Q

what are the clinical features (symptoms) of pleural inflammation that can differentiate it from cardiac chest pain?

A

sharp, severe pain that is well localized and can be lateral (one lung vs other)

worse with breathing, coughing, laughing, any chest wall movement

improves with shallow breathing and limiting movement

*recall pleuritic chest pain signals originate from parietal pleura, as there are no pain fibers in lung parenchyma or visceral pleura

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8
Q

at what point does a chronic cough typically necessitate further investigation? (when cough is only presenting symptom)

A

chronic cough of 8 weeks or more

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9
Q

what are the 3 phases of the cough reflex?

A
  1. inspiratory phase
  2. closure of glottis and diaphragmatic relaxation
  3. rapid contraction of expiratory muscles, causing a rise in intra-abdominal and intra-pleural pressures, followed by opening of the glottis
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10
Q

what is the most common etiology of acute (<3 weeks) vs subacute (3-8 weeks) vs chronic (>8 weeks) cough?

A

acute: most often viral URI

subacute: often post-infectious origin, secondary to asthma or bacterial sinusitis

chronic: post nasal drip (upper airway cough syndrome), asthma, GERD

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11
Q

hemoptysis

A

coughing or spitting up blood derived from lungs or bronchial tubes, secondary to pulmonary or bronchial hemorrhage

bronchial artery hemorrhage (90%) is worse because it is at systemic pressure, while pulmonary arteries are at much lower pressure

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12
Q

what are the 3 B’s of hemoptysis that is tracheobronchial in origin?

A
  1. Bronchitis
  2. Bronchogenic carcinoma
  3. Bronchiectasis (inflammation of airways, e.g. cystic fibrosis)

[hemoptysis = coughing up blood]

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13
Q

what does strider indicate?

A

upper airway obstruction

high-pitched musical sound, intense and heard without stethoscope

more clearly heard during inspiration, most prominent over neck

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14
Q

hyperpnea

A

increased tidal volume, often associated with increased respiratory rate

seen with acidosis

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15
Q

Kussmaul’s vs Cheyne-Stokes respiration?

A

Kussmaul: increased rate and depth of inspiration

Cheyne-Stokes: constant rate with variable depth and apneic periods (neurological disorders, congestive heart failure, high altitude, normal aging)

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16
Q

what pattern of abnormal breathing is associated with congestive heart failure?

A

Cheyne-Stokes: constant rate with variable depth and apneic periods

17
Q

what extra accessory muscle for breathing is activated when patients stand/sit in a tripod position (hands on knees)?

A

pectoralis major - helps lift chest

18
Q

what does prolonged exhalation indicate?

A

usually inspiration is longer/louder than expiration

prolonged expiration indicates obstruction - emphysema, asthma

19
Q

what does purse-lip exhalation indicate? what is the effect of breathing like this?

A

purse-lip exhalation maintains positive airway pressure during expiration - helps to improve gas transfer (increase arterial O2, decreases CO2)

present with obstructive lung disease - emphysema, asthma

20
Q

what does paradoxical breathing indicate?

A

normally, diaphragm descends with inspiration

paradoxical breathing: diaphragm RISES with inspiration, due to respiratory fatigue or diaphragmatic weakness

21
Q

match:
barrel chest or kyphosis
with
restrictive or obstructive lung disease

A

barrel chest - obstructive (hyperinflation)

kyphosis (sharp curve of spine) - restrictive

22
Q

what does finger clubbing represent?

A

capillaries in nail bed expand due to angiogenesis, responding to low oxygen tensions in the blood

23
Q

why is cyanosis difficult to detect in patients with severe anemia?

A

there is not enough hemoglobin

need 5gm/dL of reduced hemoglobin for cyanosis to be detected - need a fixed amount of hemoglobin

24
Q

which will be a stronger indication of hypoxemia, central or peripheral cyanosis?

A

finding of central cyanosis increases probability of hypoxemia (face, tongue, mucous membranes)

peripheral cyanosis can be due to peripheral vasoconstriction or stasis of blood in extremities

25
Q

what would increased tactile fremitus indicate?

A

consolidation of lung parenchyma, as with pneumonia (increased tissue density)

*only asymmetric tactile fremitus is a pathological finding

26
Q

how will obesity and bronchial obstruction affect tactile fremitus?

A

anything that separates the lung from the chest wall will decrease tactile fremitus

effusion, obesity, etc

27
Q

which of these would cause hyper-resonance upon posterior chest percussion?
a. consolidation
b. air-trapping
c. atelectasis
d. pleural effusion

A

air-trapping

others cause dullness

28
Q

how does the rhythm of breathing vary between tracheal, bronchial, bronchovesicular, and vesicular breath sounds?

A

tracheal (outside of chest): inspiration = expiration

bronchial (manubrium): inspiration &laquo_space;expiration

bronchovesicular (within lung): inspiration = expiration

vesicular (lung bases, peripheral fields): inspiration&raquo_space;> expiration

*these are all normal lung sounds as long as they are heard in the correct locations!

29
Q

vesicular vs bronchial breath sounds

A

vesicular: inspiration longer/stronger than expiration (2:1), normal finding in lung periphery

bronchial: expiration longer/stronger than inspiration (1:2) with pause between, harsh like Darth Vader, normal finding in trachea

30
Q

you are auscultating your patient’s lungs when you notice bronchial breath sounds over the lung periphery - what does this indicate?

A

bronchial breath sounds are harsh and have longer expiration and are normally heard around trachea; vesicular breath sounds have longer inspiration and are heard in lung periphery

bronchial breath sounds over lung periphery is associated with consolidation —> indicates pneumonia

31
Q

what is wheezing associated with? when is it heard and how does it sound?

A

obstructive disease, heard all over chest or localized

always associated with airflow limitation (but may be absent with severe obstruction)

long, continuous, musical sound, most often expiratory (airways get smaller)

32
Q

what are rhonchi associated with?

A

rhonchi: low-pitched version of wheeze that sounds like snoring, can clear with coughing

associated with rupture of fluid films/ abnormal airway collapse

33
Q

what are fine vs coarse crackles caused by?

A

crackles = rales: discontinuous sounds caused by opening of collapsed distal airways/alveoli (sound like velcro)

fine: interstitial lung disease, pneumonia, CHF

coarse: intermittent airway opening, may be related to secretions, associated with fluid in larger bronchi

34
Q

differentiate the clinical characteristics of fine vs coarse crackles on lung auscultation (how do they differ in sound and timing)?

A

fine: sounds like velcro, mid-to-late inspiration, unaffected by cough
(interstitial lung disease)

coarse: short/explosive, early inspiration/throughout expiration, affected by cough
(intermittent airway opening, fluid)

35
Q

what egophony change is noted with lung consolidation?

A

“eee” to “aaa” change

whispered sounds are also heard clearly and distinctly (“99”)

36
Q

how do the following affect breath sounds, tactile fremitus, and percussion?
a. consolidation
b. pleural effusion
c. pneumothorax
d. atelectasis

A

a. consolidation: bronchial breath sounds, increased fremitus, dullness on percussion

b. pleural effusion: decreased breath sounds, decreased fremitus, dullness

c. pneumothorax: decreased breath sounds, decreased fremitus, hyper-resonance on percussion

d. atelectasis: decreased breath sounds, decreased fremitus, dullness

37
Q

on physical exam, you note bronchial breath sounds and increased tactile fremitus. which of the following is most likely?
a. pleural effusion
b. atelectasis
c. consolidation
d. normal lung
e. pneumothorax

A

c. consolidation: bronchial breath sounds (in periphery, rather than vesicular), increased tactile fremitus, dullness on percussion (rather than resonance)

38
Q

on physical exam, you note hyper-resonance on percussion. which of the following is most likely?
a. pleural effusion
b. atelectasis
c. consolidation
d. normal lung
e. pneumothorax

A

e. pneumothorax: deceased breath sounds, decreased tactile fremitus, hyper-resonance on percussion