Evaluating Patients with Dyspnea Flashcards
how is tachypnea defined?
RR > 20
in what kind of acid-base disturbance can a patient be observed to be breathing rapidly but may NOT feel short of breath?
metabolic acidosis - increased ventilation as a compensatory mechanism
what are the ABC’s of evaluating a patient with dyspnea?
Airway - obstruction?
Breathing - can patient speak full sentences?
Circulation
name a pulmonary cause of dyspnea for each category:
a. airway
b. parenchyma
c. vascular
d. pleura
a. airway: asthma, bronchiectasis, COPD, foreign body, anaphylaxis
b. parenchyma: interstitial lung disease (fibrosis), acute lung injury (ARDS), pneumonia
c. vascular: pulmonary embolism, pulmonary artery HTN
d. pleura: pleural effusion, pneumothorax
how can anemia be a cause of dyspnea?
not enough hemoglobin to transport sufficient oxygen
where does pleuritic chest pain come from?
lung parenchyma and visceral pleura do not have pain fibers
pleuritis is chest pain caused by inflammation of the parietal pleura - receives systemic circulation and has somatic nerve fibers, pain afferents go to intercostal and phrenic nerves
what are the clinical features (symptoms) of pleural inflammation that can differentiate it from cardiac chest pain?
sharp, severe pain that is well localized and can be lateral (one lung vs other)
worse with breathing, coughing, laughing, any chest wall movement
improves with shallow breathing and limiting movement
*recall pleuritic chest pain signals originate from parietal pleura, as there are no pain fibers in lung parenchyma or visceral pleura
at what point does a chronic cough typically necessitate further investigation? (when cough is only presenting symptom)
chronic cough of 8 weeks or more
what are the 3 phases of the cough reflex?
- inspiratory phase
- closure of glottis and diaphragmatic relaxation
- rapid contraction of expiratory muscles, causing a rise in intra-abdominal and intra-pleural pressures, followed by opening of the glottis
what is the most common etiology of acute (<3 weeks) vs subacute (3-8 weeks) vs chronic (>8 weeks) cough?
acute: most often viral URI
subacute: often post-infectious origin, secondary to asthma or bacterial sinusitis
chronic: post nasal drip (upper airway cough syndrome), asthma, GERD
hemoptysis
coughing or spitting up blood derived from lungs or bronchial tubes, secondary to pulmonary or bronchial hemorrhage
bronchial artery hemorrhage (90%) is worse because it is at systemic pressure, while pulmonary arteries are at much lower pressure
what are the 3 B’s of hemoptysis that is tracheobronchial in origin?
- Bronchitis
- Bronchogenic carcinoma
- Bronchiectasis (inflammation of airways, e.g. cystic fibrosis)
[hemoptysis = coughing up blood]
what does strider indicate?
upper airway obstruction
high-pitched musical sound, intense and heard without stethoscope
more clearly heard during inspiration, most prominent over neck
hyperpnea
increased tidal volume, often associated with increased respiratory rate
seen with acidosis
Kussmaul’s vs Cheyne-Stokes respiration?
Kussmaul: increased rate and depth of inspiration
Cheyne-Stokes: constant rate with variable depth and apneic periods (neurological disorders, congestive heart failure, high altitude, normal aging)