Etiology of malocclusions Flashcards

1
Q

Predisposing Factors to Malocclusion

4

A
  • Disturbances in Embryologic Development
  • Heredity (genetics)
  • Functional matrix
  • Traumas
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2
Q

• Functional matrix (2)

A
  • Muscular or functional disturbances

* Habits

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3
Q

• Traumas (1)

A

• Especially mandibular fractures.

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4
Q

Primary Etiologic Sites

• Neuromuscular system…

A

Cerebral Palsy

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5
Q

Primary Etiologic Sites

• Bone:

A

OI

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6
Q

Primary Etiologic Sites

• Teeth:

A

Ectodermic Dysplasia

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7
Q

Primary Etiologic Sites

• Soft tissues (excluding muscles):

A

Sclerodermia

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8
Q

Disturbances in Embryologic Development

• — may interfere with cellular differentiation during pregnancy

A

Teratogens

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9
Q

• Teratogens may interfere with cellular differentiation during pregnancy (4)

A
  • Alcool
  • Drugs
  • Pollution

• Exemple: Cleft and Lip Palate

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10
Q

Craniosynostosis syndromes Crouzon Syndrome

A

• Crouzon’s syndrome is the most frequently occurring member of this group. It is characterized by underdevelopment of the
midface and eyes that seem to bulge from their sockets .Crouzon’s syndrome arises because of prenatal fusion of the
superior and posterior sutures of the maxilla, along the wall of the orbit. The premature fusion frequently extends
posteriorly into the cranium, producing distortions of the cranial vault as well. If fusion in the orbital area prevents the
maxilla from translating downward and forward, the result must be severe underdevelopment of the middle third of the
face. The characteristic protrusion of the eyes is largely an illusion-the eyes appear to bulge outward because the area
beneath them is underdeveloped. There may be a component of true extrusion of the eyes, however, because when cranial
sutures become synostosed, intracranial pressure increases.

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11
Q

Treacher Collins (6)

A

•down slanting eyes with notched lower lids
•underdeveloped cheek and jawbones,
•prominent nose, broad mouth and characteristically small chin with
steep lower jaw angle.
• Ears are malformed or prominent and hair may extend towards the
face;
•hearing loss is also possible.
•may have: cleft lip and/or palate, heart defects, and strabismus

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12
Q

Cleidocranial dysplasia (7)

A

•Delayed closure (ossification) of the space between the bones of the
skull (fontanels)
•Premature closing of the coronal suture
•Protruding jaw (mandible) and protruding brow bone (frontal bossing)
•Wide nasal bridge due to increased space between the eyes
(hypertelorism)
•High arched palate or possible cleft palate
• Short stature
• Scoliosis of the spine

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13
Q

Cleidocranial dysplasia

• Dental abnormalities - (5)

A

failure to lose the baby teeth (deciduous) at the expected time; slow
eruption of secondary teeth; extra teeth; delayed or absent formation of teeth
• Ability to touch the shoulders together in front of the body

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14
Q

Dentinogenesis Imperfecta (DI) (3)

A
• DI is one of the most
significant aspect of OI
• Not all teeth are affected in
the same manner
• If one OI member of the
family has DI, all the other
OI member will have DI as
well
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15
Q

Down Syndrome (4)

A
  • Underdevelopment or hypoplasia of the midfacial region.
  • The bridge of the nose, bones of the midface and maxilla are relatively smaller in size.
  • Prognathic Class III occlusal relationship which contributes to an open bite (Vittek et al, 1994).
  • Molars, impacted maxillary canines, and missing premolars
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16
Q

Cleido Cranial Dysplasia: Some facial features (6)

A
  • Wide –flat forehead
  • Wide set eyes
  • Small sinuses
  • Prolongued retention of primary teeth
  • Supernumary teeth
  • Severe malocclusion
17
Q

Normal skeletal relationship (3)

A
• 1. Cranial base
• Position and projection of
the upper jaw
• Position and projection of
the lower jaw
18
Q
  • Malocclusion occurs in — dimensions

* Angle classification is based on — relationships

A

3

AP

19
Q

• Cluster 1=

A

mild maxillary
retrognathism and mandibular
prognathism, flat mandibular
plane

20
Q

Cluster 2=

A

mild maxillary
retrognathism and mandibular
prognathism, normal
mandibular plane

21
Q

Cluster 3=

A

Large mandible

expressed vertically

22
Q

Cluster 4= s

A

everely

mandibular prognathic

23
Q

Cluster 5=

A

severely maxillary
retrognathic
▫ (most common in this sample)

24
Q
Prevalence
• Depends on the population
• Southeast Asian ~---
• Middle eastern ~---
• Indian ~---
• European ~---
A

15%
10%
1%
1-4%

25
Q

Genetics of Class III Xue 2010 (3)

A
  • Focused on mandibular prognathism
  • Family and twin studies
  • Linkage Analysis studies
26
Q

Family and twin studies

• Suggest a — model as primary cause of Class III malocclusion

A

polygenic

27
Q

Linkage Analysis studies (2)

A
• Used to determine the
chromosome loci associated
with the condition
• Suggested loci: 1p22, 1p36,
3q26.2, 6q25, 11q22, 12q13,
12q23, 19p32.2
28
Q

Deep Bite and Openbite: Multifactorial Etiologies (4)

A
  1. Transitory
  2. Skeletal (genetic)
  3. Neuromuscular
    imbalance (CP)
  4. Combination
29
Q

Neuromuscular

imbalance (CP) (2)

A
  1. Oral habits
  2. Anterior tongue
    posture
30
Q

. Combination (1)

A

Sleep Apnea