Essay Body Flashcards

1
Q

Descrobe the metacognition body?

A

Suggests that gad is developed through chronic worry.

Split into 2 types of worry (what if and worry about worry)

  • type 2.mainrains GAD through negative appraisals of worry
  • try and get rid if worry through suppression of worries and avoidance
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2
Q

What is type 1 worry?

A

Negative what if? Worry

Positively appraising worry
If I worry about all of thr outcomes, I will be prepared.

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3
Q

What is type 2 worry?

A

Worrying about worrying

Negative appraisals of worry occur triggering worry about worry and trigger8ng maladaptive behaviours

-maintains GAD

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4
Q

What is the vicious cycle of the metacog model?

A

The maladaptive eebhaviours (avoidance and suppression) prohibited indicidual to learn productive strategies for managing worry and stress

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5
Q

Who provides support evidence for the metacog model (Wells, 1997)?

A

Mahoney et al (2018)

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6
Q

What’s the aim of Mahoney et al (2018) Study?

A

Examined mediating effects of maladaptive behaviours on gad symptoms and key cognitive factors
And vice versa

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7
Q

What are the cognitive factors for mahoney et al (2018)?

A

Worry
Gad symptoms
Intolerance of uncertainty
Metacognition
Cognitive avoidance

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8
Q

What is the sample for mahoney et al (2018)?

A

2 samples
125 and 292 for validating maladaptive behaviours psychometrics

Mediation was explored

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9
Q

What did mahoney et al (2018) find?

A

Maladaptive behaviours mediated relationship between gad and intolerance of uncertainty, metacog and avoidance

Cog mediates maladaptive behaviours also

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10
Q

What are the takeaways for mahoney et al (2018l?

A

Supports model as metacog, maladaptive behaviours and worry associates with gad

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11
Q

What are the limitations of mahoney et al (2018l?

A

Cross sectional design prohibit# exploration of causality

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12
Q

What are the focused claims of Wells (1997 midel)?

A

Worry coe to gad
Worry 2 is the maintenance of gad
Worry causes damage to cognitive and emotion regulation
Those with gad having maladaptive thought patterns of tho8ght control

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13
Q

What is thr psychological intervention for GAD?

A

CBT

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14
Q

What paper for CBT treatment to GAD?

A

Stefan et al (2019)

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15
Q

What 3 types of CBT are investigated in Stefan et al (2019)?

A

Cognitive therapy/ borkovec’s treatment package
-Acceptance and Commitment therapy
- rational emotive behaviour therapy

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16
Q

What is Stefan et al (2019) study aims?

A

RCT to see wh8ch CBT is mist effective at treating GAD

17
Q

What is Stefan et al (2019l sample?

A

71 ppts diagnosed with GAD not under medicinal intervention

18
Q

What are fhe key results in the Stefan et al (2019 study?

A

No sign difference between each cbt therapy znd severity of symptoms
All improved severity of symptoms

19
Q

Results of sensitivity analysis?

A

Detect intersction effects as small as .25

20
Q

What was stefan et al (2019) power?

A

.30 to.39

21
Q

What is the limitations of Stefan et al (2019) study?

A

Small sample

Possible reason why no observable differences between intervention groups

22
Q

What are the bio intervention for GAD?

A

SSRIs and SNRIs

23
Q

What study is used fir thr bio intervention of GAD?

A

Perna et al (2016)

24
Q

What was Perna et al (2016) study?

A

Systematic review
7 RCTs for GAD
8 CTs for GAD

25
Q

Accord8ng to Stefan et al (2019) why are ssris and snris good for GAD?

A

Good long term treatment

Sognricsntt reduces relapse rates

26
Q

What SSRI and SNRI are good for GAD?

A

Escitalopram
Venlafaxine FR

27
Q

What we’re the relapse rates reduction between meds snd placebos?

A

10-19% vs 39-50%

28
Q

What is the limitations to the bio treatment of GAD?

A

These SSRIs and SNRIs are most specid8x to older adults

Relapsemore likely when undergoing cbt without medicinal treatment