Epi Mt. 2 Flashcards
Several virulence variants of African Swine Fever Virus exist
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African swine fever is shed in the saliva of the infected animals
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Stamping out of the infected herds is applied in the control of African Swine Fever
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In African swine fever cases of abortion are never observed
F
The leading clinical sign in African swine fever is excess salivation
F
In acute African swine fever the leading pathological findings are the haemorrhages
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The African swine fever infects the swine per os
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The African swine fever virus genome codes only 2 proteins
F
The African swine fever virus infects the swine by air
F
The primary replication site of African swine fever virus is the oronasal mucosa.
F
The tick species vectoring African swine fever are present in Portugal and Spain
T
The ASF virus is serologically uniform but several genotypes exist
T
In acute ASF we can observe high mortality in sows
T
Vaccines are available in EU to be used in the control of African swine fever
F
The African swine fever virus infects the swine by air
F
The African swine fever virus is highly resistant against the environmental conditions
T
The African swine fever causes hemadsorption in cell cultures
T
In the control of African swine fever thinning of the wild boar populations is helpful
T
ASF is always a peracute disease
F
ASF virus retains its infectivity for years in frozen meat
T
Picornaviridae are enveloped viruses
F
The resistance of Picornaviridae is low, they cannot survive in the environment
F
Asymptomatic animals can carry Teschovirus
T
Teschovirus is resistant, it remains infective in the faeces for a few weeks
T
Teschen and Talfan disease are caused by different virulence variants of the same virus
T
Dogs and cats can be infected with Teschen disease virus
F
Teschen disease virus can be present in pork meat
T
The virus of Teschen disease is shed in the faeces for several weeks after infection
T
The main clinical sign of Teschen disease in sheep is pneumonia
F
Lameness is a typical clinical sign of Teschen disease
T
Teschen disease occurs in cattle, sheep and pigs
F
Enteritis and pneumonia are frequent postmortem lesions of Teschen disease
F
Enlargement of the spleen and haemorrhages are typical postmortem lesions of Teschen disease
F
Teschen disease is frequent all over the world
F
The Teschen disease virus infects the animals per os
T
Inactivated vaccines are widely used in Europe to prevent Talfan disease
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Talfan disease can be seen in animals below 4 months of age
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Talfan disease is a disease of pigs
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The clinical signs of Talfan disease are more severe than that of Teschen disease
F
Talfan disease occurs in any age groups
F
Inactivated vaccines are widely used in Europe to prevent Talfan disease
F
Haemorrhages are typical postmortem lesions of Talfan disease
F
Flaccid paralysis is a typical clinical sign of Talfan disease
T
Vesicles can be seen in the liver in the case of swine vesicular disease
F
Swine vesicular disease virus cannot cause viraemia
F
Swine vesicular disease is a zoonosis
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Swine vesicular disease virus can cause vesicles on the snout of pigs
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The resistance of swine vesicular disease virus is very low; it cannot survive in the environment
F
Haemorrhages in the serous membranes can be frequently seen postmotem in the case of swine
vesicular disease
F
The mortality of swine vesicular disease is 40-50%
F
Swine vesicular disease is frequently seen in Europe
F
Swine vesicular disease virus replicates in the epithelium of the throat and tonsils
T
Only pigs are susceptible to swine vesicular disease virus
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The VES infection results in clinical signs similar to that of FMD
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The VES virus can survive in water for 2 weeks
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The primary source of VES infection is sea origin feed
T
VES causes the highest mortality in pregnant sows
F
VES causes mortality in piglets
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VES is more contagious than FMD
F
Haemorrhages in the parenchymal organs are frequent postmortem lesions of avian
encephalomyelitis
F
There is germinative spread in the case of avian encephalomyelitis
T
Tremor is a typical clinical sign of avian encephalomyelitis
T
Germinative infection can happen in the case of duck hepatitis
F
Spasmodical paddling is a frequent clinical sign of duck hepatitis
T
The resistance of duck hepatitis A virus is high
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Ducks are infected with the duck hepatitis A virus generally per os
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Duck hepatitis cannot be prevented with vaccination
F
Duck hepatitis A virus is shed in the faeces
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Encephalomyocarditis virus is shed in the faeces by infected animals
T
Encephalomyocarditis virus infects animals mainly per os
T
FMD is a chronic disease
F
FMD spreads slowly within the herd
F
FMD virus is shed by semen
T
Foot and mouth disease is serologically uniform
F
Because of the high variability of FMD virus cross protection cannot be observed even within the
serotype
T
Within the FMD serotypes, subtypes can be differentiated
T
The sample from the FMD suspected animal should be submitted to lab cooled but not frozen
T
FMD ulcers are usually sharp edged and regular round shaped
F
The foot and mouth disease is classified into 7 serotypes
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FMD can be transmitted by frozen meat
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The peracut form of FMD causes severe haemorrhages and death by shock
F
The chronic form of FMD means arthritis
F
FMD is transmitted by insect vectors
F
Vesicles of FMD can fully recover after immune response
T
FMD causes the loss of hoof in swine
T
The SAT-1,2 and 3 serotypes of FMD virus are present in Africa and Arabia
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In humid cold environment, the FMD virus can retain its infectivity for weeks
T
Even the vaccinated animals can carry and shed the FMD virus
T
The main host (reservoir) of foot and mouth disease virus is swine
F
FMD induces lameness in sheep and oral vesicles in goats
T
FMD virus is shed in milk
T
For laboratory tests foot and mouth disease samples should be submitted in buffered transport
medium
T
Within the FMD serotypes, subtypes can be differentiated
T
The foot and mouth disease virus can retain its infectivity for 40 days in manure
T
In Europe supportive therapy is applied in the treatment of FMD
F
Frothy and sticky nasal discharge is the characteristic sign of FMD
F
Even the vaccinated animals can carry and shed the FMD virus
T
The hedgehog is susceptible to FMD
T
Because of high variability of FMD virus cross protection cannot be observed even within the
serotype
T
All serotypes of foot and mouth disease can be detected worldwide
F
For laboratory tests Foot and mouth disease samples should be submitted in buffered transport
medium
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Foot and mouth disease is transmitted by insect vectors
F
Foot and mouth disease can cause myocarditis in young animals
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Ruminants can carry foot and mouth disease virus for up to 3 years
T
The Asia serotype of FMD virus is present in Turkey and in the Middle East
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The FMD virus is enveloped
T
FMD virus cannot be propagated in cell culture
F
24 hours after the inoculation of FMD virus into guinea pig paw pad vesicle formation can be
observed
T
The FMD mortality is nearly 100%
F
The FMD suspected sample should be sent to lab by post (in mail)
F
FMD virus can be transmitted by feed
T
The primary replication sites of foot and mouth disease are the lymph nodes
F
The primary replication sites of foot and mouth disease is the bone marrow
F
Horses are susceptible to foot and mouth disease
F
Cattle shed foot and mouth disease virus for a shorter time comparing to swine
F
The foot and mouth disease virus is very sensitive to the environmental conditions
F
Foot and mouth disease is transmitted by insect vectors
F
We can observe the most severe foot and mouth disease signs on sheep
F
In the control of foot and mouth disease vaccines are used in Europe
F
9 hours after infection foot and mouth disease virus can be shed by the infected animals
T
Swine shed foot and mouth disease virus for a shorter time than cattle
T
The foot and mouth disease virus is very sensitive to acidic pH
T
Calicivirus can cause persisting infections in the tonsils
T
Feline calicivirus is carried and shed by vaccinated animals too
T
The feline calicivirus is shed in excretes
T
The feline calicivirus infection can be recognised by the ulcers seen on the tongue
T
The feline calicivirus infection can cause the limping of kittens
T
RHD induces clinical signs mostly in animals younger than 2 months F
F
RHD virus is used for biological control of rural rabbits
T
RHD virus propagates in the liver of the infected animals
T
RHD virus can be propagated in cell culture
F
Vaccines against RHDV-1 induce protection against RHDV-2 too
F
The orthohepevirus A causes clinical symptoms only in human
T
The Orthohepevirus A can be zoonotic
T
The avian hepatitis E is a zoonotic disease
F
The avian hepatitis E causes drop in egg production
T
Avian nephritis is caused by astroviruses
T
The mortality of avian nephritis is 50-60%
T
Avian nephritis replicates in the gut
T
Broilers are regularly vaccinated against avian nephritis virus
F
Urate deposition is a postmortem lesion of avian nephritis
T
Clinical signs of avian nephritis can be seen in the first four weeks of life
T
Avian nephritis is more frequent in waterfowl than in chicken
F
Togaviruses are strongly resistant to the environmental conditions
F
Togaviruses are transmitted by mosquitoes
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The clinical appearance of togavirus infection is dose dependent
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Togaviruses cause airborne infection
F
Abortion is the main clinical sign of togavirus infection results in abortion of horses
F
The leading clinical sign of togavirus infection is the frothy nasal discharge
F
Togavirus infection in horses results in hepatitis
F
In the geographic distribution of equine togavirus migratory birds have an important role
T
Rodents serve as reservoirs for the Western equine encephalomyelitis virus
T
Equine encephalomyelitis viruses are zoonotic
T
Equine encephalomyelitis viruses can cause asymptomatic infections
T