Epi Mt. 2 Flashcards
1
Q
Several virulence variants of African Swine Fever Virus exist
A
T
2
Q
African swine fever is shed in the saliva of the infected animals
A
T
3
Q
Stamping out of the infected herds is applied in the control of African Swine Fever
A
T
4
Q
In African swine fever cases of abortion are never observed
A
F
5
Q
The leading clinical sign in African swine fever is excess salivation
A
F
6
Q
In acute African swine fever the leading pathological findings are the haemorrhages
A
T
7
Q
The African swine fever infects the swine per os
A
T
8
Q
The African swine fever virus genome codes only 2 proteins
A
F
9
Q
The African swine fever virus infects the swine by air
A
F
10
Q
The primary replication site of African swine fever virus is the oronasal mucosa.
A
F
11
Q
The tick species vectoring African swine fever are present in Portugal and Spain
A
T
12
Q
The ASF virus is serologically uniform but several genotypes exist
A
T
13
Q
In acute ASF we can observe high mortality in sows
A
T
14
Q
Vaccines are available in EU to be used in the control of African swine fever
A
F
15
Q
The African swine fever virus infects the swine by air
A
F
16
Q
The African swine fever virus is highly resistant against the environmental conditions
A
T
17
Q
The African swine fever causes hemadsorption in cell cultures
A
T
18
Q
In the control of African swine fever thinning of the wild boar populations is helpful
A
T
19
Q
ASF is always a peracute disease
A
F
20
Q
ASF virus retains its infectivity for years in frozen meat
A
T
21
Q
Picornaviridae are enveloped viruses
A
F
22
Q
The resistance of Picornaviridae is low, they cannot survive in the environment
A
F
23
Q
Asymptomatic animals can carry Teschovirus
A
T
24
Q
Teschovirus is resistant, it remains infective in the faeces for a few weeks
A
T
25
Teschen and Talfan disease are caused by different virulence variants of the same virus
T
26
Dogs and cats can be infected with Teschen disease virus
F
27
Teschen disease virus can be present in pork meat
T
28
The virus of Teschen disease is shed in the faeces for several weeks after infection
T
29
The main clinical sign of Teschen disease in sheep is pneumonia
F
30
Lameness is a typical clinical sign of Teschen disease
T
31
Teschen disease occurs in cattle, sheep and pigs
F
32
Enteritis and pneumonia are frequent postmortem lesions of Teschen disease
F
33
Enlargement of the spleen and haemorrhages are typical postmortem lesions of Teschen disease
F
34
Teschen disease is frequent all over the world
F
35
The Teschen disease virus infects the animals per os
T
36
Inactivated vaccines are widely used in Europe to prevent Talfan disease
F
37
Talfan disease can be seen in animals below 4 months of age
T
38
Talfan disease is a disease of pigs
T
39
The clinical signs of Talfan disease are more severe than that of Teschen disease
F
40
Talfan disease occurs in any age groups
F
41
Inactivated vaccines are widely used in Europe to prevent Talfan disease
F
42
Haemorrhages are typical postmortem lesions of Talfan disease
F
43
Flaccid paralysis is a typical clinical sign of Talfan disease
T
44
Vesicles can be seen in the liver in the case of swine vesicular disease
F
45
Swine vesicular disease virus cannot cause viraemia
F
46
Swine vesicular disease is a zoonosis
T
47
Swine vesicular disease virus can cause vesicles on the snout of pigs
T
48
The resistance of swine vesicular disease virus is very low; it cannot survive in the environment
F
49
Haemorrhages in the serous membranes can be frequently seen postmotem in the case of swine
vesicular disease
F
50
The mortality of swine vesicular disease is 40-50%
F
51
Swine vesicular disease is frequently seen in Europe
F
52
Swine vesicular disease virus replicates in the epithelium of the throat and tonsils
T
53
Only pigs are susceptible to swine vesicular disease virus
T
54
The VES infection results in clinical signs similar to that of FMD
T
55
The VES virus can survive in water for 2 weeks
T
56
The primary source of VES infection is sea origin feed
T
57
VES causes the highest mortality in pregnant sows
F
58
VES causes mortality in piglets
T
59
VES is more contagious than FMD
F
60
Haemorrhages in the parenchymal organs are frequent postmortem lesions of avian
encephalomyelitis
F
61
There is germinative spread in the case of avian encephalomyelitis
T
62
Tremor is a typical clinical sign of avian encephalomyelitis
T
63
Germinative infection can happen in the case of duck hepatitis
F
64
Spasmodical paddling is a frequent clinical sign of duck hepatitis
T
65
The resistance of duck hepatitis A virus is high
T
66
Ducks are infected with the duck hepatitis A virus generally per os
T
67
Duck hepatitis cannot be prevented with vaccination
F
68
Duck hepatitis A virus is shed in the faeces
T
69
Encephalomyocarditis virus is shed in the faeces by infected animals
T
70
Encephalomyocarditis virus infects animals mainly per os
T
71
FMD is a chronic disease
F
72
FMD spreads slowly within the herd
F
73
FMD virus is shed by semen
T
74
Foot and mouth disease is serologically uniform
F
75
Because of the high variability of FMD virus cross protection cannot be observed even within the
serotype
T
76
Within the FMD serotypes, subtypes can be differentiated
T
77
The sample from the FMD suspected animal should be submitted to lab cooled but not frozen
T
78
FMD ulcers are usually sharp edged and regular round shaped
F
79
The foot and mouth disease is classified into 7 serotypes
T
80
FMD can be transmitted by frozen meat
T
81
The peracut form of FMD causes severe haemorrhages and death by shock
F
82
The chronic form of FMD means arthritis
F
83
FMD is transmitted by insect vectors
F
84
Vesicles of FMD can fully recover after immune response
T
85
FMD causes the loss of hoof in swine
T
86
The SAT-1,2 and 3 serotypes of FMD virus are present in Africa and Arabia
T
87
In humid cold environment, the FMD virus can retain its infectivity for weeks
T
88
Even the vaccinated animals can carry and shed the FMD virus
T
89
The main host (reservoir) of foot and mouth disease virus is swine
F
90
FMD induces lameness in sheep and oral vesicles in goats
T
91
FMD virus is shed in milk
T
92
For laboratory tests foot and mouth disease samples should be submitted in buffered transport
medium
T
93
Within the FMD serotypes, subtypes can be differentiated
T
94
The foot and mouth disease virus can retain its infectivity for 40 days in manure
T
95
In Europe supportive therapy is applied in the treatment of FMD
F
96
Frothy and sticky nasal discharge is the characteristic sign of FMD
F
97
Even the vaccinated animals can carry and shed the FMD virus
T
98
The hedgehog is susceptible to FMD
T
99
Because of high variability of FMD virus cross protection cannot be observed even within the
serotype
T
100
All serotypes of foot and mouth disease can be detected worldwide
F
101
For laboratory tests Foot and mouth disease samples should be submitted in buffered transport
medium
T
102
Foot and mouth disease is transmitted by insect vectors
F
103
Foot and mouth disease can cause myocarditis in young animals
T
104
Ruminants can carry foot and mouth disease virus for up to 3 years
T
105
The Asia serotype of FMD virus is present in Turkey and in the Middle East
T
106
The FMD virus is enveloped
T
107
FMD virus cannot be propagated in cell culture
F
108
24 hours after the inoculation of FMD virus into guinea pig paw pad vesicle formation can be
observed
T
109
The FMD mortality is nearly 100%
F
110
The FMD suspected sample should be sent to lab by post (in mail)
F
111
FMD virus can be transmitted by feed
T
112
The primary replication sites of foot and mouth disease are the lymph nodes
F
113
The primary replication sites of foot and mouth disease is the bone marrow
F
114
Horses are susceptible to foot and mouth disease
F
115
Cattle shed foot and mouth disease virus for a shorter time comparing to swine
F
116
The foot and mouth disease virus is very sensitive to the environmental conditions
F
117
Foot and mouth disease is transmitted by insect vectors
F
118
We can observe the most severe foot and mouth disease signs on sheep
F
119
In the control of foot and mouth disease vaccines are used in Europe
F
120
9 hours after infection foot and mouth disease virus can be shed by the infected animals
T
121
Swine shed foot and mouth disease virus for a shorter time than cattle
T
122
The foot and mouth disease virus is very sensitive to acidic pH
T
123
Calicivirus can cause persisting infections in the tonsils
T
124
Feline calicivirus is carried and shed by vaccinated animals too
T
125
The feline calicivirus is shed in excretes
T
126
The feline calicivirus infection can be recognised by the ulcers seen on the tongue
T
127
The feline calicivirus infection can cause the limping of kittens
T
128
RHD induces clinical signs mostly in animals younger than 2 months F
F
129
RHD virus is used for biological control of rural rabbits
T
130
RHD virus propagates in the liver of the infected animals
T
131
RHD virus can be propagated in cell culture
F
132
Vaccines against RHDV-1 induce protection against RHDV-2 too
F
133
The orthohepevirus A causes clinical symptoms only in human
T
134
The Orthohepevirus A can be zoonotic
T
135
The avian hepatitis E is a zoonotic disease
F
136
The avian hepatitis E causes drop in egg production
T
137
Avian nephritis is caused by astroviruses
T
138
The mortality of avian nephritis is 50-60%
T
139
Avian nephritis replicates in the gut
T
140
Broilers are regularly vaccinated against avian nephritis virus
F
141
Urate deposition is a postmortem lesion of avian nephritis
T
142
Clinical signs of avian nephritis can be seen in the first four weeks of life
T
143
Avian nephritis is more frequent in waterfowl than in chicken
F
144
Togaviruses are strongly resistant to the environmental conditions
F
145
Togaviruses are transmitted by mosquitoes
T
146
The clinical appearance of togavirus infection is dose dependent
T
147
Togaviruses cause airborne infection
F
148
Abortion is the main clinical sign of togavirus infection results in abortion of horses
F
149
The leading clinical sign of togavirus infection is the frothy nasal discharge
F
150
Togavirus infection in horses results in hepatitis
F
151
In the geographic distribution of equine togavirus migratory birds have an important role
T
152
Rodents serve as reservoirs for the Western equine encephalomyelitis virus
T
153
Equine encephalomyelitis viruses are zoonotic
T
154
Equine encephalomyelitis viruses can cause asymptomatic infections
T
155
Horses encephalomyelitis vaccines defend against African horse sickness (cross protection)
F
156
Horse encephalomyelitis viruses are transmitted by ticks
F
157
Birds serve as reservoirs for the Eastern equine encephalomyelitis virus
T
158
Equine encephalomyelitis viruses can cause lameness in horses
T
159
Birds serve as reservoirs for Venezuelan horse encephalomyelitis virus
F
160
Western equine encephalitis causes the highest mortality
F
161
Equine encephalitis can be diagnosed easily by gross pathology
F
162
In Venezuelan horse encephalitis enteric symptoms can be observed too
T
163
Recovery from equine horse encephalitis induces immunity that lasts only 1 year
F
164
Equine encephalitis vaccines are available only for humans
F
165
Chikungunya virus causes joint pains, fever, rash and haemorrhages in humans
T
166
Chikungunya virus is transmitted by the Asian tiger mosquito
T
167
Serological cross reactions frequently occur between related flaviviruses
T
168
Pestiviruses are zoonotic
F
169
Pestiviruses are arboviruses
F
170
Louping ill is seen in cattle in North America
F
171
The louping ill was introduced to Austrailia to control rabbit population
F
172
The louping ill is seen in cattle in Great Britain and Scandinavia
F
173
The tick borne encephalitis is endemic in central Europe
T
174
The main vectors of tick borne encephalitis virus are sylvatic mammalian species
F
175
The tick borne encephalitis virus can be transmitted via consumption of raw milk
T
176
Migratory birds play a role in the transmission of West Nile virus
T
177
Mammals are mostly accidental hosts of West Nile Fever virus
T
178
West Nile Fever is a notifiable disease
T
179
In West Nile epidemics wild bird mortality is usually observed before horse and human cases can
be detected
T
180
To prevent West Nile Fever, vaccine is available for horses
T
181
The West Nile Fever can be diagnosed unambiguously by the clinical signs
F
182
The West Nile Fever virus infects mostly swine and cattle F
F
183
The West Nile virus may cause encephalitis in humans and in horses T
T
184
Horses and humans do not play a significant role in the transmission of West Nile virus
T
185
The West Nile fever epidemics occur usually at the end of summer and during Autumn
T
186
West Nile fever is a notifiable disease
T
187
The Japanese encephalitis virus causes abortion in swine
T
188
The Wesselsbron disease causes meningoencephalitis in turkey
F
189
The yellow fever virus causes liver damages
T
190
The zika virus causes fever, rash and conjunctivitis in humans
T
191
BVDV can be frequently detected in the background of respiratory diseases
T
192
Cytopathic and non-cytopathic biotypes of the Bovine Viral Diarrhoea (BVD) virus are known
T
193
In utero infection with non-cytopathic BVDV can cause seropositivity of the newborn calf
T
194
Non cytopathic strains of Bovine viral diarrhoea virus (BVDV) can cause hydrocephalus and
cerebellar hypoplasia
F
195
In utero infection with non cytopathic BVDV can cause immunotolerance
T
196
Pigs infected with BVDV turn seropositive to classical swine fever
T
197
Persistently infected cattle can maintain BVDV in a farm
T
198
Cytopathic strains of Bovine Viral diarrhoea (BVDV) alone can cause mucosal disease
F
199
The BVD virus can infect pigs also
T
200
Cytopathic strains of the Bovine Viral Diarrhoea virus (BVDV) can cause abortion
T
201
Non cytopathic strains of Bovine Viral Diarrhoea virus (BVDV) do not cause transplacental
infection
T
202
Cattle which are immune tolerant to BVD virus may develop mucosal disease
T
203
In sheep BVDV causes symptoms similar to border disease
T
204
Mucosal disease can develop in cattle persistently infected with BVDV
T
205
Non cytopathic strains of Bovine viral diarrhoea virus (BVDV) can cause immunosuppression
T
206
BVDV can be transmitted by semen
T
207
In mucosal disease sharp edged, usually round or oval ulcers can be observed on the oral mucosa
T
208
In mucosal disease haemorrhages can be observed under the serosal surfaces and in the kidney
cortex
T
209
Bloody diarrhoea is a clinical sign of the mucosal disease
T
210
The Border disease virus causes pneumonia in sheep
F
211
The border disease is present only in the United Kingdom
F
212
The border disease virus frequently causes encephalitis in ewes
F
213
Haemorrhagic enteritis is a characteristic pathological lesion of Classical swine fever
T
214
Classical swine fever virus can be transmitted by raw pork products
T
215
Classical swine fever virus can retain its infectivity for 6 months in frozen meat
T
216
During necropsy enlarged bloody lymph nodes can be seen in Classical swine fever cases
T
217
Because of endothelial damage Classical swine fever results in haemorrhages
T
218
In acute Classical swine fever, at the beginning obstipation, later diarrhoea can be observed
T
219
Classical swine fever targets the bone marrow stem cells
T
220
Weakness of the hind legs and ataxia are clinical signs of Classical Swine Fever
T
221
Ischaemic infarcts at the edges of the pleen are postmortem lesions of subacute Classical Swine
Fever
T
222
The boutons in the intestines are characteristic lesions in subacute Classical Swine Fever
T
223
Kidney haemorrhage is a characteristic lesion of Classical Swine Fever
T
224
During necropsy of acute Classical swine fever cases, haemorrhages can be seen in the gastric
mucosa
T
225
Among Classical Swine Fever clinical signs CNS and eye problems can be observed
T
226
Classical swine fever infection during the first half of pregnancy results in the birth of
immunotolerant piglets
T
227
Classical swine fever causes oronasal infection
T
228
Classical swine fever can be transmitted by mating
T
229
Classical swine fever targets lymphatic tissues
T
230
Clinical signs of Classical Swine fever are mainly seen in 6-12 week old piglets in vaccinated herds
T
231
Classical swine fever infection during the first half of pregnancy can lead to abortion
T
232
In Europe it is obligatory to vaccinate against Classical swine fever
F
233
In acute Classical swine fever skin haemorrhages and bloody nasal discharge can be observed
T
234
Normal sized spleen can be observed in uncomplicated cases of subacute Classical swine fever
T
235
Classical swine fever infection results in immunosuppression
T
236
Orbiviruses spread by droplet (aerosol) infection
F
237
Orbiviruses can be cultivated in vivo by inoculation of embryonated eggs
T
238
Genetic reassortments may cause significant antigenic changes in reovirus strains
T
239
Orbiviruses are serologically uniform
F
240
Orthoreoviruses frequently cause tenosynovitis of horses
F
241
Orthoreoviruses can cause pneumoenteritis in calves
T
242
Avian orthoreoviruses cause mainly nephritis and encephalitis
F
243
Infection of adult birds with orthoreoviruses usually remains subclinical
T
244
Avian orthoreoviruses usually transmitted by germinative route
T
245
Avian orthoreoviruses can cause damages in the bursa Fabricii
T
246
Mammalian orthoreoviruses can cause pneumoenteritis only in suckling animals
F
247
Rotaviruses are sensitive to environmental conditions
F
248
Rotavirus infection is sporadic within the herd
F
249
Rotavirus only infects mammals
F
250
Clinical signs of rotavirus infection are usually seen in animals older than 2 weeks of age
F
251
Swine is not susceptible to rotavirus infection
F
252
Rotaviruses predispose to E.Coli infection in suckling piglets
T
253
Rotaviruses are shed in the faeces in high titres
T
254
Rotavirus infection results in high mortality
F
255
Avian rotaviruses are transmitted by germinative infection
F
256
Losses due to rotavirus infection of young animals can be prevented by colostrum feeding
T
257
Rotaviruses usually cause enteritis in young (1-2 weeks old) animals
T
258
Clinical signs of rotavirus infection are usually seen in animals older than 2 weeks of age
F
259
Rotaviruses damage the mucosa of the large intestines
F
260
Rotaviruses are serologically uniform
F
261
Rotaviruses are typically transmitted via the faecal oral route
T
262
Goats are not susceptible to bluetongue virus
F
263
Ibaraki disease is clinically very similar to bluetongue
T
264
Lameness is a frequent symptom of bluetongue in sheep
T
265
Bluetongue virus can cause bloody diarrhoea in lambs
T
266
Bluetongue is named after cyanosis of the tongue
T
267
The bluetongue virus is vectored by midges/ gnats
T
268
Cattle can carry the bluetongue virus for years without symptoms
T
269
Wild birds play the most important role in the distribution of bluetongue
F
270
In Europe vaccination of small ruminants against bluetongue is mandatory
F
271
Rodents serve as reservoirs of bluetongue
F
272
Bluetongue virus is also foetopathic
T
273
Lameness and abortion are signs of bluetongue
T
274
Bluetongue occurs only in Africa and Australia
F
275
Bluetongue is transmitted by ticks
F
276
Bluetongue can be transmitted by semen
T
277
Goats are more resistant to the bluetongue than sheep T
T
278
Vaccination against Bluetongue results serotype specific immunity T
T
279
Bluetongue disease occurs only in Africa
F
280
The epizootic haemorrhagic fever is observed in the USA in wild deer
T
281
The Infectious equine arthritis and the African horse sickness ay have similar clinical signs
T
282
Oedemas and haemorrhages are the most frequent lesions in African horse sickness
T
283
African horse sickness is spread by ticks
F
284
African horse sickness is zoonotic
F
285
Frothy nasal discharge is a characteristic sign of African horse sickness
T
286
The subacute form of African horses sickness is causing oedema formation and heart failure
T
287
Encephalitis is the most frequent sign of African horse sickness
F
288
Acute form of African horse sickness occurs mainly in zebras and horses
F
289
Carnivores are susceptible to African horse sickness
T
290
The natural reservoirs of the African horse sickness virus are mainly zebras
T
291
Subcutaneous oedema is a frequent symptom of subacute African horse sickness
T
292
African horse sickness is mostly a peracute disease
F
293
Horse encephalosis is endemic in Africa
T
294
Equine encephalosis can result in abortion
T
295
Equine encephalosis is transmitted by mosquitoes
T
296
Equine encephalosis causes the most severe clinical signs in Zebras
F
297
Horse encephalosis occurs only in America
F
298
Equine encephalosis causes high mortality
F
299
Bursitis virus targets the premature B lymphocytes
T
300
The bursitis virus causes per os infection
T
301
Meat type hybrids are more sensitive to Gumboro disease than layers
F
302
Clinical signs of Gumboro disease can be seen in chickens older than 8 weeks
F
303
Some Gumboro disease vaccines can be applied only besides yolk immunity
T
304
Watery diarrhoea is typical clinical sign of Gumboro disease
T
305
There are several virulence variants of bursitis virus
T
306
Clinical signs of Gumboro disease can be seen in chickens younger than 8 weeks
F
307
Bursitis virus is highly contagious
T
308
Immunocomplex vaccines can be applied to prevent Gumboro disease
T
309
Clinical signs of Gumboro disease can be seen in chickens younger than 8 days
F
310
Before vaccination against Gumboro disease the level of maternal immunity in the flock should
be determined
T
311
Anaemia and cyanosis can be clinical signs of Gumboro disease
T
312
Bursitis virus causes the highest mortality in day old chicks
F
313
Viruses are available to control Gumboro disease
T
314
The bursitis virus is very sensitive to the environmental conditions
F
315
Bursitis virus causes disease only in chicken
F
316
The bursitis virus can retain its infectivity for 1-4 months in the litter
T
317
Both serotypes of bursitis virus are pathogenic
F
318
Bursitis virus infection can reduce the efficacy of vaccinations
T
319
Bursitis virus infection results in immunosuppression
T
320
Bursitis virus is shed in the faeces
T
321
Influenzaviruses have segmented genome
T
322
HPAI and LPAI strains differ in the number of basal amino acids in the HA protein
T
323
HA protein of influenza viruses is responsible for the attachment to the cell
T
324
Antigenic drift means serials of point mutations in the HA and NA genes
T
325
The NA protein in the influenza virus is responsible for the release from the infected cells
T
326
Enzymatic cleavage of the HA protein is needed for the influenza virus penetration into the cell
T
327
Genetic reassortment of human and animal influenza viruses result in the occurrence of new
human influenza strains
T
328
Bats have an important role in the epidemiology of influenza
F
329
Antigenic shift is behind the influenza pandemics
T
330
Influenza viruses are classified into genera by their HA and NA proteins
F
331
Influenza causes persistent infection in donkey
F
332
The serotype of influenza viruses is determined by their HA and NA proteins
T
333
The influenza viruses cause respiratory signs in sea mammals
T
334
The host range of influenza virus is determined by the NA protein
F
335
The bird to human host switch of influenza virus is mediated by swine
T
336
The main host of influenza is the swine
F
337
In humans inactivated vaccines are used for the immunisation against influenza
T
338
Antigenic drift is behind the seasonal influenza epidemics
T
339
Antigenic shift means the reassortment of the influenzavirus genome segments
T?
340
Influenzaviruses are sensitive to drying out
T
341
For the in vitro propagation of influenza viruses embryonated eggs is used
T
342
The high virulence of some influenza A viruses is the result of mutations in the HA gene
T
343
Influenza viruses are classified into genera by their HA and NA proteins
F
344
25) The HPAI strains can cause CNS Signs T
T
345
26) HPAI causes blood vessel damages and generalised infections T
T
346
27) The HPAI strains are usually developed from the H5 and H7 influenza viruses T
T
347
28) The LPAI causes immunosuppression T
T
348
29) Cat is susceptible to human seasonal influenza
F
349
Swine recovered from influenza should not be kept for breeding
T
350
Influenza causes high morbidity but low mortality in swine
T
351
The HPAI causes skin haemorrhages in swine
F
352
Swine influenza outbreaks occur usually during summer
F
353
In a horse influenza outbreak all infected horses must be killed
F
354
The generalized symptoms of horse influenza are result of the interferon response
T
355
Racehorses should be vaccinated before the influenza season
F
356
Breeding mares should be vaccinated before the influenza season
F
357
The eggs produced in LPAI infected flock can be hatched
T
358
The HPAI infection can be unambiguously differentiated from Newcastle disease by the clinical
signs
F
359
The HPAI strains can cause clinical signs in waterfowl
T
360
In poultry farms LPAI may be endemic without clinical signs
T
361
Waterfowl can carry the influenza viruses for long time
T
362
Influenza viruses are shed by birds exclusively in the nasal discharge
F
363
Avian influenza is zoonotic
T
364
The virulence of avian influenza is determined by the i.v. pathogeny index and mortality
T
365
Avian influenza can switch directly to human and cause epidemics with high case numbers as
result of human to human transmission
F
366
All Bunyaviruses are transmitted by mosquitoes
F
367
Bunyaviruses cause oral infection
F
368
The virus causing severe fever and thrombocytopenia syndrome is transmitted by tick
T
369
The Aino virus causes foetal development problems and abortion in sheep
T
370
Akabane virus is zoonotic
F
371
Akabane virus causes foetal damages in dogs
F
372
Schmallenberg virus causes disease only in Suffolk sheep breed
F
373
Schallenberg virus causes foetal damages in humans
F
374
Schmallenberg virus causes foetal development problems more frequently in cattle than in sheep
T
375
Schmallenberg virus is present worldwide
F
376
Human is a competent host of Rift Valley fever virus i.e. the viraemic titre is high enough to infect
the vector
T
377
The Rift valley fever virus damages the blood vessels thus causes haemorrhages
T
378
Wild rabbits are reservoirs of Rift valley fever virus
F
379
Rift valley fever is a human only disease
F
380
Nairobi sheep disease causes generalized infection in the animals
T
381
Vaccines are available in endemic regions to prevent Nairobi sheep disease
T
382
Nairobi sheep disease leads to abortion in the infected pregnant animals
T
383
Nairobi sheep disease causes haemorrhagic enteritis in the infected animals
T
384
Small rodents serve as reservoir hosts of Hantaviruses
T
385
Hantaviruses can cause renal failure in human
T
386
The Crimean-congo haemorrhagic fever is a human disease with headache, muscle pain and skin
haemorrhages
T
