ENT - Balance Flashcards

1
Q

What are some causes of secondary tinnitus?

A
  • impacted ear wax
  • ear infection
  • ménière’s disease
  • noise exposure
  • medications
  • acoustic neuroma
  • multiple sclerosis
  • trauma
  • depression
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2
Q

Which medications are associated with tinnitus?

A
  • loop diuretics
  • gentamicin
  • chemotherapy drugs (e.g. cisplatin)
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3
Q

Systemic conditions associated with tinnitus.

A
  • anaemia
  • diabetes
  • hyperthyroidism
  • hypothyroidism
  • hyperlipidaemia
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4
Q

What is objective tinnitus?

A

When a patient can objectively hear an extra sound within their head. This sound can also be observable on examination by auscultating with a stethoscope around the ear.

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5
Q

Causes of objective tinnitus.

A
  • carotid artery stenosis (pulsatile carotid bruit)
  • aortic stenosis (radiating pulsatile murmur)
  • arteriovenous malformation
  • Eustachian tube dysfunction
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6
Q

Investigations for tinnitus.

A

Laboratory investigations to exclude underlying causes:
- full blood count (?anaemia)
- HbA1c (?diabetes)
- TSH (?thyroid disorder)
- lipids (?hyperlipidaemia)

Specialist investigations may be audiometry and imaging (CT / MRI).

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7
Q

Tinnitus red flags.

A
  • unilateral
  • pulsatile
  • hyperacusis
  • unilateral hearing loss
  • sudden onset hearing loss
  • vertigo / dizziness
  • headaches / visual symptoms
  • neurological symptoms
  • suicidal ideation

May indicate a serious underlying pathology and the need for specialist assessment.

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8
Q

Management of tinnitus.

A

Tinnitus tends to improve or resolve over time without interventions.

Any identifiable causes should be managed.

Measures to help improve and manage tinnitus:
- hearing aids
- sound therapy
- CBT

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9
Q

What is vertigo?

A

A descriptive term for a sensation that there is movement between the patient and their environment.

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10
Q

Sensory inputs responsible for maintaining balance and posture.

A
  • vision
  • proprioception (cerebellum)
  • signals from the vestibular system
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11
Q

Vertigo can be caused by either a:

1)

2)

A

1) peripheral problem (affecting vestibular system)

2) central problem (affecting the brainstem or cerebellum)

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12
Q

Give some peripheral causes of vertigo.

A
  • benign paroxysmal positional vertigo (BPPV)
  • Ménière’s disease
  • vestibular neuronitis
  • labyrinthitis
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13
Q

Give some central causes of vertigo.

A
  • posterior circulation stroke
  • tumour
  • multiple sclerosis
  • vestibular migraine
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14
Q

Peripheral vertigo history

a) onset

b) duration

c) hearing loss or tinnitus

d) coordination

e) nausea

A

a) acute

b) short (seconds or minutes)

c) often present

d) intact

e) severe

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15
Q

Central vertigo history

a) onset

b) duration

c) hearing loss or tinnitus

d) coordination

e) nausea

A

a) gradual (except POCS)

b) persistant

c) rarely

d) impaired

e) mild

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16
Q

What should be examined when assessing a patient presenting with vertigo?

A
  1. Otoscopy - look for signs of infection or other pathology.
  2. Neurological examination - assess for central causes of vertigo.
  3. Cardiovascular examination - assess for arrhythmias or valve disease)
  4. Special tests
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17
Q

Which clinical signs indicate central vertigo.

A

DANISH - cerebellar components.

Dysdiadokinesia
Ataxic gait (heel-to-toe)
Nystagmus
Intention tremor
Speech (slurred)
Heel-Shin test

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18
Q

Which examination can be performed to distinguish between a central and peripheral vertigo?

A

HINTS examination

Head Impulse
Nystagmus
Test of Skew

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19
Q

What is the Head Impulse test?

A

The head impulse test involves the patient sitting upright and fixing their gaze on the examiner’s nose. The examiner holds the patient’s head and rapidly jerks it 10-20 degrees in one direction while the patient continues looking at the examiner’s nose. The head is moved slowly back to the centre before repeating in the opposite direction. Ensure they have no neck pain or pathology before performing the test.

A patient with a normally functioning vestibular system will keep their eyes fixed on the examiner’s nose.

In a patient with an abnormally functioning vestibular system (e.g., vestibular neuronitis or labyrinthitis), the eyes will saccade (rapidly move back and forth) as they eventually fix back on the examiner.

The head impulse test helps diagnose a peripheral cause of vertigo but will be normal if the patient has no current symptoms or a central cause of vertigo.

20
Q

What are the causes of nystagmus?

a) unilateral horizontal nystagmus

b) bilateral vertical nystagmus

A

a) peripheral vertigo

b) central vertigo

21
Q

What is the Test of Skew?

A

Involves the patient sitting upright and fixing their gaze on the examiner’s nose. The examiner covers one eye at a time, alternating between covering either eye. The eyes should remain fixed on the examiner’s nose with no deviation.

If there is a vertical correction when an eye is uncovered (the eye has drifted up or down and needs to move vertically to fix on the nose when uncovered), this indicates a central cause of vertigo.

22
Q

Pathophysiology of benign paroxysmal positional vertigo (BPPV).

A

Otoconia (calcium crystals) become displaced into the semicircular canals.

This disrupts the normal flow of endolymph through the canals, confusing the vestibular system.

Head movement creates the flow of endolymph in the canals, triggers episodes of vertigo.

23
Q

Which structure in the semicircular canal is most commonly affected in BPPV?

A

Posterior semicircular canal.

24
Q

Causes of otoconia displacement (BPPV).

A
  • viral infection
  • head trauma
  • ageing
  • idiopathic
25
Q

Presentation of BPPV.

A
  • head movements trigger vertigo
  • vertigo settles within 60s
  • asymptomatic between attacks

BPPV does not cause hearing loss or tinnitus.

26
Q

What is the diagnostic manoeuvre for BPPV?

A

Dix-Hallpipe manoeuvre

Triggers rotational nystagmus and symptoms of vertigo, towards the affected ear.

27
Q

What manoeuvre can be used to treat BPPV?

A

Epley manoevre

28
Q

What exercises can be prescribed to manage BPPV?

A

Brandt-Daroff exercises

Exercises are repeated several times a day until symptoms improve.

29
Q

Pathophysiology of vestibular neuronitis.

A

Inflammation of the vestibular nerve causes distortion of signals travelling from the vestibular system to the brain.

This results in episodes of vertigo, where the brain thinks the head is moving when it’s not.

30
Q

Triggers of vestibular neuronitis.

A

Viral upper respiratory tract infection.

31
Q

Presentation of vestibular neuronitis.

A
  • vertigo
  • nausea and vomiting
  • balance problems

NB: Tinnitus and hearing loss are not features, as the cochlear nerve is not affected.

32
Q

Management of vestibular neuronitis.

A
  • prochlorperazine
  • antihistamines

NICE recommend treatment can be used for up to three days - extended use may slow recovery.

NB: If there is no symptomatic improvement, patient can be offered vestibular rehabilitation therapy.

33
Q

Prognosis of vestibular neuronitis.

A

Symptoms are most severe for the first few days - gradually resolve over the following 2-6 weeks.

BPPV may develop after vestibular neuronitis.

34
Q

What is labyrinthitis?

A

Inflammation of the bony labyrinth of the ear:
- semicircular canals
- vestibule
- cochlea

35
Q

Trigger of labyrinthitis.

A

Viral upper respiratory tract infection.

NB: Rarely caused by secondary bacterial infection (e.g. otitis media, meningitis).

36
Q

Presentation of labyrinthitis.

A
  • acute vertigo
  • hearing loss
  • tinnitus

NB: patients may have symptoms associated with the causative virus.

37
Q

Diagnosis of labyrinthitis.

A

Clinical diagnosis based on history and examination.

Head impulse test used to diagnose peripheral causes of vertigo.

38
Q

Management of labyrinthitis.

A

Short-term use of medication:
- prochlorperazine
- antihistamines

Antibiotics are used to treat bacterial labyrinthitis.

39
Q

Pathophysiology of Ménière’s disease.

A

Excessive accumulation of endolymph in the labyrinth results in a high pressure, disrupting sensory signals.

40
Q

Symptoms of Ménière’s disease.

A
  • vertigo
  • hearing loss (sensorineural)
  • tinnitus

Other symptoms include:
- fullness in ear
- unexplained falls
- imbalance

41
Q

Management of Ménière’s disease.

A

Managing symptoms during an acute attack:
- prochlorperazine
- antihistamines

Prophylaxis:
- betahistine

42
Q

How to differentiate labyrinthitis and Ménière’s disease?

A

Both have symptoms of:
- vertigo
- tinnitus
- hearing loss

Only Ménière’s experiences fullness of the ear.

43
Q

What is a vestibular migraine?

A

Headache associated with a visual aura and dizziness.

Triggered by:
- stress
- bright lights
- strong smells
- dehydration
- menstruation
- abnormal sleep patterns

44
Q

How is vestibular migraine managed?

A

Avoid triggers and lifestyle changes (e.g. getting enough sleep, remain hydrated).

Medical management is similar to migraines:
- triptans for acute symptoms
- amitriptyline to prevent attacks

45
Q

DVLA advice on vertigo.

A

Patients must not drive and inform the DVLA if they are liable to sudden and unprovoked, or unprecipitated, episodes of disabling dizziness.