ENT Flashcards

1
Q

Give 3 differentials for a sore throat.

A

Tonsillitis - viral and bacterial
Quinsy
Infectious mononucleosis (OM)

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2
Q

What are the common causative organisms of tonsillitis?

A

Viruses most common
Bacteria - Grp A Strep (GAS), esp strep pyogenes
Strep pneumoniae

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3
Q

How is tonsillitis managed? When would you give antibiotics?

A
Centor criteria:
Fever >38
Tonsillar exudates
Absence of cough
Lymphadenopathy
score 3 or more of these = 40-60% probability of bacterial tonsillitis, can give abx. Penicillin V 10 days first line.
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4
Q

What is quinsy? How does it present?

A

Peritonsillar abscess - bacterial infection, trapped pus.
Usually a complication of untreated tonsillitis. Usually bacterial (Grp A Strep, staph aureus, H. influenzae)

Presentation: similar to tonsillitis, sore throat, painful swallow, fever, neck pain, referred otalgia, lymphadenopathy.
Trismus
Hot potato voice

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5
Q

How is quinsy managed?

A

Refer to ENT for incision+drainage (I+D) under GA

Abx appropriate eg co-amoxiclav

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6
Q

Give 3 differentials for otalgia.

A
Otological:
-Middle ear - otitis media. Usually viral
-External ear - otitis externa
Referred (50% of otalgia):
dental
TMJ dysfunction, malocclusion
C spine osteoarthritis
Malignancy
Acute pharyngitis
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7
Q

How does otitis media present?

A

Otalgia, hearing loss in affected ear, generally unwell, upper URTI symptoms (cough, coryza, sore throat), discharge if perforated eardrum, bulging red tympanic membrane (TM).
May get vertigo/balance issues.

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8
Q

How would you manage otitis media? When would you give abx?

A

Usually resolves within 3-7 days without abx, give pcm/ibuprofen for pain/fever
Abx eg amoxicillin 5-7 days, clarithromycin if penicillin-allergic, erythromycin in pregnant penicillin-allergic.
Admit if systemically unwell <3 months old
Safety net for complications - mastoiditis, meningitis, abscess

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9
Q

How would you manage an adult patient with painful, discharging ear, with debris in the ear on examination?

A

Bacterial otitis externa (swimmers ear) - Usually pseudomonas. Topical abx may be needed. Use Otomize: neomycin (pseudomonas is resistant to many abx) + dexamethasone + acetic acid. EXCLUDE TM PERFORATION before giving neomycin.

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10
Q

Give 3 differentials for facial pain

A
Sinus: Sinusitis
Neuro: Trigeminal neuralgia, herpes zoster, 
Temporal arteritis
Eye: glaucoma
Cluster headaches, migraine, MOH
Dental abscess
Ear: OM, OE
Nose: URTI, nasal injury
Functional: Atypical Facial Pain (ATFP)
TMJ dysfunction
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11
Q

How does acute sinusitis present?

A

Dull constant aching pain over maxillary sinus, tenderness, postnasal drip, ethmoid/sphenoid pain (midline root on nose), worse on bending, lasts 1-2 weeks (but can be up to 12 weeks), asso coryza.
[Almost a doctor]

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12
Q

How is sinusitis managed?

A

Usually self-resolves in 2-3 weeks.
Sx <10 days: no treatment
>10 days: 2 weeks high-dose steroid nasal spray
No improvement: consider abx - Penicillin V 5 day course.

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13
Q

What are the most common causative organisms for sinusitis?

A
Usually viral.
Bacterial causes:
Group A Strep 
Strep Pneumoniae
H. influenzae
Morazella Catarrhalis
Staph aureus
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14
Q

What are the complications of sinusitis and what investigations would you do?

A
CT if suspecting complications eg spread of infection to orbit/intracranial region.
Chandler classification:
1 Preseptal cellulitis
2 Orbital cellulitis
3 Subperiosteal cellulitis
4 Orbital Abscess
5 Cavernous sinus thrombosis
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15
Q

What is chronic sinusitis?

A

Chronic = >12 weeks, >2 symptoms (nasal blockage, obstruction, congestion, discharge, nasal drip), change in sense of smell.

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16
Q

Give 5 differential diagnoses for a neck lump.

A

Developmental abnormalities: Thyroglossal cysts, branchial cysts
Cervical lymphadenopathy (most common in GP) eg infective (IMN, CMV), malignant (lymphoma, mets)
Thyroid masses eg goitre, nodule
Salivary gland swellings
Haematoma
Sardoidosis
Cancer eg oropharyngeal - ask about RFs eg tobacco, alcohol, odynophagia, otalgia, dyspnoea, wt loss)

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17
Q

What is the likely cause of a midline neck lump which is smooth, painless, and moves on movements of the tongue?

A

Thyroglossal duct cysts.= benign cyst that forms in midline of neck, base of tongue to sternal notch, embryological remnant.

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18
Q

How would you investigate a neck lump?

A

Hx - ca risk factors, odynophagia, otalgia, wt loss, exposure to HIV/travel
Examination: lump size, shape, appearance, relation to other tissues eg does it move with swallowing/ tongue protrusion
Flexible nasopharyngology

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19
Q

What causes a neck lump that moves on swallowing but not tongue protrusion?

A

A thyroid lump (nodule).

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20
Q

How can you differentiate a malignant neck lump from a benign one?

A

(Cancer til proven otherwise)
Benign: younger age, mobile, present from birth (congenital)/ 2-6 week hx (inflammatory)
Malignant red flags: older age, gradually increasing in size, hard, non-mobile, changed over 3-6 weeks, hoarse voice >3 weeks, new onset dysphagia, unexplained persistent salivary gland swellings, otalgia >4 weeks with normal otoscopy, sore throat, non-healing ulcers, white/red oral lesion.
If any of these, refer 2 week wait.

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21
Q

Give 5 causes of sensorineural hearing loss.

A
Congenital
Presbycusis (age-related)
Noise exposure (occupational)
Meniere's disease (unilat)
Drug toxicity (bilat - furosemide, gentamicin, cisplatin)
Acoustic neuroma (acute)
Infections eg Meningitis
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22
Q

How would you investigate hearing loss?

A

Hx - onset, rate, otalgia, otorrhea, tinnitus (pulsatile?), vertigo, nasal, drug hx (furosemide ototoxic), FHx, exposure to noise
Ex: pinna, mastoid, both ears,
External auditory canal: discharge, bony swellings, wax oedema
TM:
Pars tensa: perforations, retraction pockets, ossicles gromets
Pars flaccida: attic retraction pockets, cholesteatoma
Hearing assessment
CN examination (House-Brackmann scale)
Nasendoscopy
Pure tone audiogram (PTA)
Tympanometry

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23
Q

Describe Rinne’s and Weber’s tests and what they show.

A

Hearing assessment:
512 Hz tuning fork
Weber’s: Middle of forehead, louder on the opposite side to a SNHL or towards the side of a CHL
Rinne’s: Put tuning fork on mastoid, ask patient to tell you when it stops humming, then hover 1cm over ear and ask when it stops. Positive = normal = patient can still hear it after being taken off, so AC > BC. Negative (abnormal) = They cant hear it after taken off on the side with conductive hearing loss so BC > AC.

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24
Q

What type of hearing loss is indicated by the air and bone conduction lines being far apart on PTA?

A

Conductive hearing loss.

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25
Q

Describe the House-Brackmann scale.

A

Classification of facial nerve palsy.
I = normal
II = slight weakness
III = complete eye closure, obvious weakness
IV = incomplete eye closure, weakness, disfiguring asymmetry
V = flicker of motion
VI = no movement

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26
Q

Management of chronic deafness in adults?

A

OME - examine post-nasal space to exclude neoplasia
Unilat sx asso tinnitus + vertigo –> MRI to exclude acoustic neuroma
If chest infection/sinusitis/kidney problem this may suggest autoimmune/granulomatous disease –> ab screen, refer
Presbyacusis –> hearing aid

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27
Q

Management of acute deafness in adults?

A

If asso neurology –> CVA/MS - MRI and refer
Review medications - chemo, aspirin, ototoxic abx, loop diuretics
Ask abt recent head/barotrauma
Menieres episode - supportive
5-10% have identifiable cause, 50% have complete spontaneous recovery.
[lecture]

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28
Q

What causes vertigo with sudden onset, N+V, often following a sore throat, and can cause hearing loss?

A

Viral labyrinthitis. L for Loss of hearing

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29
Q

How does vestibular neuronitis present?

A

Viral infection, acute onset vertigo, gradually improves over weeks, N for No hearing loss

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30
Q

What is BPPV and what causes it?

A

Benign Paroxysmal Positional Vertigo. Otoconia (CaCO3 crystals) in the semicircular canals disrupt flow of endolymph. Attacks last minutes and are triggered by movement.

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31
Q

What factors in the history + examination point to a central cause of vertigo?

A

HINTS:
Head impulse: Jerk patients head laterally, patient should continue to focus on you. Saccades in vestibular dysfunction. Normal in central or no symptoms.
Nystagmus - unilateral/horizontal in peripheral, bilateral/vertical in central.
Test of skew/alternate cover test - cover each eye, should keep fixed on you, vertical drift/correction in central.
Gradual onset (except stroke)
Persistent
No hearing loss
Ataxia and abnormal neurology
No/mild nausea

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32
Q

What causes Ménière’s disease?

How is it treated?

A

Excessive endolymph in the semicircular canals, causing a higher pressure than normal, disrupting the sensory signals. Relapsing remitting attacks of rotatory vertigo, hearing loss and tinnitus lasting hours, with nystagmus.
Worse with stress. Treat symptomatic with betahistine.

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33
Q

Give 5 causes of conductive hearing loss.

A
Ear wax
Infection eg OM/OE
Middle ear effusion
Eustachean tube dysfunction
Fixation of the ossicular chain (otosclerosis)
Perforated TM
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34
Q

What would you see on audiometry in sensorineural hearing loss?

A

Both air and bone conduction readings more than 20db (need to be louder to hear so line is lower down on graph).

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35
Q

What is presbycusis and how does it present?

A

Age-related hearing loss, gradual loss of high-pitched sounds, +/- tinnitus. Increased risk of dementia in untreated hearing loss.

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36
Q

Give 4 risk factors for presbycusis.

A

Older age, male, FHx, loud noise exposure, diabetes, htn, ototoxic medications (furosemide, gentamicin, cisplatin), smoking

37
Q

What would audiometry show in presbycusus?

A

Sensorineural hearing loss pattern with hearing loss at higher frequencies (lines for both air and bone conduction are lower at high frequencies).

38
Q

What is the management for presbycusis?

A

Cannot be reversed

Optimise environment eg reducing ambient noise, hearing aids, cochlear implant last line

39
Q

What is sudden sensorineural hearing loss?

A

Unexplained hearing loss over less than 72 hours, usually unilateral, can be permanent, loss of 30 decibels in 3 consecutive frequencies on audiogram.

40
Q

What causes SSNHL?

A

90% idiopathic but can be caused by infection, MS, stroke, ototoxic meds, Cogan’s (eyes), acoustic neuroma etc.

41
Q

What is the management of SSNHL?

A

Otological emergency - refer urgently to ENT.
Exclude differentials - CHL, CT head for stroke and acoustic neuroma
Idiopathic (90%) –> steroids

42
Q

What is the eustachian tube and what is its function?

A

Tube between middle ear and throat. Equalises air pressure in the middle ear and drains fluid from middle ear. When is is dysfunctional/blocked, the pressure between the middle ear and the environment can become unequal and the middle ear can fill with fluid.

43
Q

What causes eustachian tube dysfunction?

A

Viral URTI, allergies eg hay fever, smoking

44
Q

How does eustachian tube dysfunction present?

A

Reduced/altered hearing; popping noises/sensations in the ear; sensation of fullness in the ear, pain/discomfort, tinnitus, worse in pressure change eg flying, climbing mountain, scuba diving.

45
Q

How is eustachian tube dysfunction diagnosed?

A

Clinical dx
Otoscopy - exclude other causes
If persistent/severe/problematic symptoms, investigate with tympanometry (measuring sound absorbed at different air pressures - peak admittance with negative ear canal pressures), audiometry, nasopharyngoscopy, CT scan

46
Q

What is the management for eustachian tube dysfunction?

A

Resolves spontaneously eg post URTI
Valsalva manoeuvre, Otovent (blowing balloon device)
Decongestant nasal spray (short term)
Antihistamines for allergies
Surgery in severe or persistent cases - grommets, balloon dilatation eustachian tuboplasty, adenoidectomy

47
Q

What is otosclerosis?

A

Remodelling of the small bones in the middle ear. mainly the base of the stapes, leading to conductive hearing loss. Autosomal dominant inheritance, more common in women, before 40 years

48
Q

What would you see on audiology in otosclerosis?

A

Conductive hearing loss, so reduced air conduction (>20dB, normal bone conduction (<20dB). Can be unilateral or bilateral. Loss of hearing in lower pitched sounds, so line looks lower down at lower frequencies.

49
Q

How does otosclerosis present?

A

Women under 40, family history, unilateral or bilateral hearing loss and tinnitus. Loss of low pitch sounds. Their own voice may sound louder so they might talk quietly.

50
Q

What would tympanometry show in otosclerosis?

A

Generally reduced admittance (absorption of sound through the TM).

51
Q

How is otosclerosis managed?

A

Conservative - hearing aids
Surgical - stapedectomy (remove stapes and put in a prosthetic stapes), stapedotomy (leave base of stapes attached to oval window, add prosthesis to transmit sounds from incus to cochlea via base of stapes).

52
Q

What is otitis media?

A

Infection in middle ear - between tympanic membrane and inner ear. Contains cochlea, vestibular apparatus and nerves. Bacteria enter from back of throat through eustachian tube. Viral URTI often precedes bacterial otitis media.

53
Q

What causes otitis media? Give 3 organisms

A
Usually viral but bacterial causes are:
Strep pneumoniae (most common)
H influenzae
Moraxella catarrhalis
Staph aureus
54
Q

What treatment can prevent otitis externa?

A

Acetic acid 2% (EarCalm). Antifungal and antibacterial, can be used before and after swimming in patients that are prone to otitis externa.

55
Q

What would you give in otitis externa if the TM is perforated?

A

Aminoglycosides eg neomycin/gentamicin are ototoxic if they get into the inner ear, so exclude perforated TM first. Give ciprofloxacin instead.

56
Q

What is cerumen? How does it cause hearing loss?

A

Ear wax. Usually healthy and prevents infections in ear canal. If impacted and stuck to TM, results in hearing loss, discomfort, feeling of fullness, pain, tinnitus.

57
Q

How is cerumen impaction managed?

A

Usually self-resolves. DO NOT use cotton buds. Methods for removing wax:

  1. Ear drops - olive oil/sodium bicarb 5%
  2. Ear irrigation - water (GP). If CI due to TM perforation or infection then–>
  3. Microsuction - specialist ENT.
58
Q

What is tinnitus?

A

Additional sound in the ear, eg ringing, buzzing, hissing. Sensory signal produced by the cochlea not effectively filtered out by the central auditory system. Can be primary (SNHL, idiopathic); secondary to wax, MS, Menieres, noise exposure etc, or part of systemic conditions eg diabetes.

59
Q

Give 4 causes of secondary tinnitus.

A
Impacted ear wax
Ear infection
Menieres 
Noise exposure
Medications eg furosemide, gentamicin
Acoustic neuroma
MS
Trauma
Depression

Systemic conditions: anaemia, diabetes, hypothyroidism/hyperthyroidism, hyperlipidemia.

60
Q

What is objective tinnitus? Give 2 causes.

A

Patient can hear an extra sound within their head, observable on examination by auscultating around the ear. Causes:
Carotid artery stenosis (pulsatile carotid bruit)
Aortic stenosis (radiating pulsatile murmur sounds)
AV malformations (pulsatile)
Eustachian tube dysfunction (popping or clicking noises.

61
Q

Give 5 red flags to ask about in a tinnitus history.

A

Unilateral
Pulsatile
Hyperacusis (hypersensitivity, pain or distress with environmental sounds)
Associated hearing loss - unilateral, sudden onset
Associated vertigo, dizziness
Headaches, visual symptoms, neuro sx
Suicidal ideation related to the tinnitus

62
Q

Give 4 blood tests you would do in tinnitus and why.

A

FBC - anaemia
Glucose - diabetes
TSH - thyroid disorder
Lipids - hyperlipidaemia

63
Q

What is the management of tinnitus?

A

Self resolving
Treat underling cause
Hearing aids, sound therapy (adding background noise to mask the tinnitus), CBT

64
Q

What are the DVLA rules in vertigo?

A

Sudden and unprovoked or unprecipitated episodes of disabling dizziness –> can’t drive and must inform DVLA.

65
Q

What is an acoustic neuroma?

A

Benign tumour of schwann cells surrounding auditory (vestibulocochlear) nerve which innervates inner ear. Aka vestibular schwannomas, or cerebellopontine angle tumours.

66
Q

What condition is associated with bilateral acoustic neuroma?

A

Neurofibromatosis type II.

67
Q

How does acoustic neuroma present?

A

40-60 years, gradual onset
Unilateral sensorineural hearing loss (often the first symptom)
Unilateral tinnitus
Dizziness or imbalance
A sensation of fullness in the ear
Facial nerve palsy if the tumour grows large enough to compress the facial nerve. Forehead NOT spared as it is an LMN lesion.

68
Q

What would you see on audiometry with acoustic neuroma?

A

Sensorineural hearing loss

69
Q

How is acoustic neuroma managed?

A

CT/MRI to assess
Conservative - eg monitoring if no symptoms
Surgery - partial or total removal
Radiotherapy to reduce growth
Risk of vestibulochoclear nerve or facial nerve injury –> hearing loss, dizziness, facial weakness.

70
Q

What is cholesteatoma?

A

Benign tumour of squamous epithelial cells in the middle ear. Can invade local tissues and nerves and erode the bones of the middle ear, and predispose to significant infections.
Negative pressure in the middle ear causes a pocket of the TM to retract into the middle ear, so it gets sucked inwards. The squamous epithelial cells in the picket proliferate into the surrounding space.

71
Q

How does cholesteatoma present?

A

Foul discharge, unilateral conductive hearing loss
Compression –> Infection, pain, vertigo, facial nerve palsy
Otoscopy may show buildup of whitish debris/crust in upper TM.

72
Q

What is the management for cholesteatoma?

A

CT hear to assess, +/- MRI

Surgical removal

73
Q

Describe the pathway, functions the 5 branches of the facial nerve.

A

Exits brainstem at cerebellopontine angle, passes through temporal bone and parotid gland, then branches into:
Temporal
Zygomatic
Buccal
Marginal mandibular
Cervical
MOTOR to facial expression, stapedius, and neck muscles
SENSORY - taste anterior 2/3 tongue
PARASYMPATHETIC to submandibular and sublingual salivary and lacrimal glands.

74
Q

How does facial nerve palsy present? Give 5 causes.

A

Facial weakness, altered taste or dry mouth or eyes.
LMN = forehead involvement.
Idiopathic: Bell’s palsy
Infection: Ramsay Hunt syndrome, OM, malignant OE, HIV, Lyme’s disease
Systemic: diabetes, sarcoidosis, leukaemia, MS, GBS
Tumour: acoustic neuroma, parotid tumour, cholesteatoma
Trauma: direct to nerve, during surgery, base of skull fracture

75
Q

What is Bell’s palsy? What is the prognosis?

A

Common idiopathic unilateral LMN facial nerve palsy.

Most patients recover in weeks, may take 1 year, 1/3 left with residual weakness.

76
Q

What is the management of Bell’s palsy?

A

If presenting within 72 hours, give prednisolone 50mg for 10 days (or 60mg for 5 days then reduce by 10mg a day)
Lubricating eye drops
Tape eye closed at night
Risk of exposure keratopathy

77
Q

What is Ramsay-Hunt syndrome? How is it treated?

A

HSV unilateral LWN facial nerve palsy. Tender vesicular rash around the ear. Treatment with prednisolone, aciclovir and lubricating eye drops.

78
Q

Where does epistaxis usually originate?

A

Kiesselbach’s plexus in Little’s area - area of nasal mucosa at front of nasal cavity. Think little fingers picking noses.

79
Q

Give 5 causes of epistaxis.

A
Nose picking
Colds
Sinusitis
Vigorous nose-blowing
Trauma 
Changes in the weather
Coagulation disorders (e.g., thrombocytopenia or Von Willebrand disease)
Anticoagulant medication (e.g., aspirin, DOACs or warfarin)
Snorting cocaine
Tumours (e.g., squamous cell carcinoma)
80
Q

Give 3 complications of epistaxis.

A

Blood loss

Aspiration - especially with posterior bleeding, which is suggested by bleeding from BOTH nostrils

81
Q

What is the management of epistaxis?

A
  1. Advice: sit up, tilt head forward, squeeze soft part of nostrils for 10-15 mins, spit out any blood in mouth rather than swallow. If still continues after 15 mins, from both nostrils, or haem unstable, admit
  2. Nasal packing using tampons/inflatable packs
  3. Nasal cautery using silver nitrate ticks
    Prescribe naseptin (chlorhexidine and neomycin) QDS for 10 days to reduce any crusting, inflammation and infection. Contains soy+peanuts tho
82
Q

Give the 3 most common causes of tonsillitis.

A

Usually viral
Bacterial is usually Group A strep (most to least common: strep pyogenes, strep pneumoniae, H influenzae, M camarillas, staph aureus)

83
Q

What is Waldeyer’s tonsillar ring? Which area is most commonly affected by tonsillitis?

A

Ring of lymphoid tissue in pharynx (back of throat). 6 areas of lymphoid tissue:
Adenoids
Lingual tonsil
Tubal tonsils x2
Palatine tonsils x2 - usually affected by tonsillitis.

84
Q

How does tonsillitis present?

A

Typical presentation sore throat, fever above 38 and pain on swallowing, red, inflamed and enlarged tonsils. Usually viral
Bacterial = plus exudate = white patches of pus, tender anterior cervical lymphadenopathy

85
Q

Describe the Centor criteria and how they are interpreted.

A

A score of 3 or more gives a 40 – 60 % probability of bacterial tonsillitis, and it is appropriate to offer antibiotics. A point is given if each of the following features are present:

Fever over 38ºC
Tonsillar exudates
Absence of cough
Tender anterior cervical lymph nodes (lymphadenopathy)

86
Q

Describe the FeverPAIN score and how it is interpreted.

A

A score of 2 – 3 gives a 34 – 40% probability, and 4 – 5 gives a 62 – 65% probability of bacterial tonsillitis:

Fever during previous 24 hours
P – Purulence (pus on tonsils)
A – Attended within 3 days of the onset of symptoms
I – Inflamed tonsils (severely inflamed)
N – No cough or coryza
87
Q

What is the management of tonsillitis?

A

Admit: immunocompromised, systemically unwell, dehydrated, stridor, respiratory distress or evidence of a peritonsillar abscess or cellulitis.

Educate patients with likely viral tonsillitis and give safety net advice about when to seek medical advice. Advise simple analgesia with paracetamol and ibuprofen to control pain and fever. Return if the pain has not settled after 3 days or the fever rises above 38.3ºC.

Abx if the Centor score is ≥ 3, or the FeverPAIN score is ≥ 4, if they are at risk of more severe infections, such as young infants, immunocompromised patients or those with significant co-morbidity, or a history of rheumatic fever.
This is with Pen V 10 days, or clarithromycin in pen allergy.

Delayed prescriptions - if the symptoms worsen or do not improve in the next 2 – 3 days.

88
Q

Give 5 complications of tonsillitis.

A

Peritonsillar abscess, also known as quinsy
Otitis media, if the infection spreads to the inner ear
Scarlet fever
Rheumatic fever
Post-streptococcal glomerulonephritis
Post-streptococcal reactive arthritis