ENT Flashcards
he health status of the oral cavity is linked to
cardiovascular disease
diabete
other systemic illnesses.
Assume any head and neck infection or swelling to be _______ in origin until proven otherwise.
odontogenic
Caries is what type of infection
bacterial
- causes demineralization and destruction of the hard tissues of the teeth (enamel, dentin and cementum).
- Caries are the result of the production of acid by bacterial fermentation of food debris accumulated on the tooth surface.
Caries are formed If ______ exceeds saliva and other_______ factors
demineralization
remineralizing
caries are likely a result of the acidic secretions of what bacteria
strep mutans
name some other bacterial agents implicated w/ caries
populations at risk for periodonatal dz
diabetics
elderly
pregnant women - preg gingivitis due to hormonal changes promoting increase in alterations in types and amounts of pathogens
•Pyogenic Granuloma- Occur in 1% of women, Exaggerated response to irritation
Jaw pain can be [an] ______ equivalent
anginal
postmenopausal women / long-term diabetic patients
and especially lower-left portion of the jaw
Si/ Sx of dental caries
- Sensitivity to hot or cold stimuli
- Pain on biting (trigeminal nerve)
children < 4 y.o. with stiff neck, sore throat and dysphagia should be worked up for ______ ______
retropharyngeal abscess secondary to molar infection
fils mordered for suspected infection
•Panoramic film of the teeth and jaw for evaluation of the extent of the infection
CT w/o contrast determine the extent and density of the swelling, locating the abscess within the soft tissue and bone (aids in determining tx)
first line tx for dental infection
Pen VK
Amox
If PCN allergic:
clinda or erythro
second line dental inf tx
•If long-standing infection or previously treated infection that does not respond to first line treatment:
oral clinda
IF SEVERE consider clinda + double coverage with metronidazole (B. fragilis and C. diff)
admission criteria for pts w/ dental infections
- swelling involving deep spaces (pre fascial planes) of the neck
- unstable vital signs, fever, chills, confusion or delirium
- evidence of invasive infection
complications of dental infections
- Ludwig’s angina (sublingual cellulitis, +/- tracking abscess inferiorly; potential for airway issue)
- Vincent’s angina, aka ANUG (acute necrotizing ulcerative gingivitis), aka ‘trench mouth’
- Smells HORRIBLE, “worst breath you have ever smelt
- Retropharyngeal infection (possibility of retropharyngeal abscess) and mediastinal infection
- Child w/ fever, dysphagia, neck stiffness think retropharyngeal abscess
acute vs chronic rhinosinusitis timeframe
sublingual cellulitis is
Ludwig Angina
•Note the diffuse submandibular swelling and fullness.
airway compromise is a major concern
most common pathpgens responsible for viral sinustitis
most common bacterial pathogens responsible for rhinosinusitis
- S. pneumoniae
- H. flu
- M. catarrhalis
- S. aureus
- S. pyogenes
dx criterial for acute viral vs bacterial sinusitis
<•10 days nonworsening sx - viral
>10 days or biphasicor worsening ® bacterial
tx for bacterial sinusitis
- Amoxicillin
- Augmentin
- Doxycycline
- Levofloxacin
- Moxifloxacin
•
•Macrolides no longer recommended due to resistant so S. pnuemoniae
•Nasal mucopurulent drainage (“post-nasal drip”) is seen with?
chronic sinusitis
cough in children is a sx of
chronic sinusitis
3 types of chronic sinusitis
- Chronic w/ nasal polyposis (20%)
- Allergic fungal rhinosinusitis (8-10%)
- Chronic w/o nasal polyps (60%)
dx criteria symptom wise for chronic sinusitis
imaging dx?
– 2 of 4
- Anterior/posterior mucopurulent drainage
- nasal obstruction/blockage/congestion
- Facial pain, pressure, fullness
- Reduction or loss of sense of smell
Objective evidence w/ one or more using nasal endoscopy or CT
- Purulent (not clear) mucus or edema in middle meatus or ethmoid
- Polyps in nasal cavity or middle meatus
- Imaging showing mucosal thickening, partial or complete opacification of paranasal sinuses
tx chronic sinusitis w/ Nasal polyps present
- Oral glucocorticoids
- Dupilumab (Dupixent)
- Abx if infection
- Allergy/immune eval
- Endoscopic surgery
tx chronic sinusitis w/o nasal polyps
- Intranasal saline irrigation
- Intranasal glucocorticoids
- Oral abx/GCs – if no improvement in 2-4 wks
preferred imaging for chronic sinusitis
•CT – preferred imaging modality - sinus mucosal thickening, polyps, sinus opacification
•Palatal click – when scratching palate w/ tongue is assoc w/
allergic rhinitis
si/sx of allergic rhinitis
- Infraorbital edema & darkening (“allergic shiners”)
- Accentuated lines below eyes (Dennie-Morgan lines)
- Transverse nasal crease (“nasal salute”)
- Hyperplastic lymphoid tissue lining post. Pharynx (“cobblestoning”)
- Retracted TM
- Nasal mucosa pallor
- Prick skin test
- IgE immunoassays (RAST)
used to dx?
allergies
patch better
RAST better w/ severe allergies
common source of bleeding in anterior vs posterior nose bleeed
- Anterior ® Keisselbachs plexus (most common) involves branches of anterior ethmoid artery, sphenoplantine and facial a.
- Posterior ® sphenopalatine a. or branches of carotid a. (significant)
tx minor epistaxis
- Tamponade
- Silver nitrate
- Electrocautery w/ anesthetization
tx of major epistaxis
- Nasal packing – contralateral nare if bleeding persists for tamponade
- ENT consultation
what risk is assoc w/ packing a nose bleed
TSS - prophylaxis is augemntin or cephalexin
positive pressure techniques such as a mothers kiss can help tx
FB impaction
common FB impactions
- Button batteries – at neg poly electrolysis generates hydroxide ions that cause alkaline tissue necrosis or septal perf
- Paired disc magnets – perf from chronic compression
complciations fo FB
- Septal perforation w/ saddle nose deformity
- Nasal meatal stenosis
- Inferior turbinate necrosis
- Cartilage collapse
- Epistaxis
fucntions of ET (3)
- Equalizing pressure across TM
- Protecting middle ear from infection and reflux of nasopharyngeal contents
- Clearance of middle ear secretions
ET tube dysfucntion can be caused by (3)
- Pressure dysregulation
- Impaired protective function – reflux into ET
- Diminished clearance