ENT Flashcards

1
Q

What are 5 common causes of epistaxis?

What’s 2 congenital causes?

Whats a drug cause?

A

dry weather or low humidity, barotrauma, nasal or facial fracture/trauma eg. nosepicking, nasal polyp, nasal irritants eg. dust/smoke

Familial Hereditary Haemorrhagic Telangiectasia, septal deviation

Cocaine, topical nasal drugs eg. nasal corticosteroids

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are the 2 types of epistaxis?

A

90% are Anterior:
- originates from Kiesselbach’s plexus (vascular anastamosis in Little’s area)

Posterior:
- produce more active bleeding

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

How would you investigate epistaxis?

A

FBC, Coagulation studies and INR and prothrombin time

Nasal endoscopy and nasopharyngoscopy to identify source

nasal or sinus Xray to show fracture

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

How would you initially manage epistaxis? (active bleeding)

A
  • apply pressure to anterior bleeding sites by pinching the lower compressible cartilage of the nose
  • applying a vasoconstrictor/decongestant ( eg. oxymetazoline) helps encourage haemaostasis
  • topical anaesthetic (eg. lidocaine) can help
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Describe the steps you take once the bleeding source has been visualised

A
  • anterior epistaxis = silver nitrate cautery
  • ** avoid cautery on same area on either side of septum as it can result in deprivation of blood supply and septal perforation ***
  • petroleum jelly after
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Describe the steps you take if the cautery fails?

A
  • anterior nasal packing
  • give co-amoxi antibiotics as there is impaired sinus drainage = infection risk

if the bleeding persists:

  • endoscopy = surgical clip ligation of sphenopalatine artery
  • angiography + embolisation with interventional radiology
  • open surgery ligation
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What if you suspect a posterior bleed?

A
  • posterior packing
  • double balloon epistaxis device OR traditional gauze anterior pack with Foley urinary catheter placed posteriorly
  • IV analgesia and antiemetics
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

A man comes in with acute bacterial sinusitis. What part of the history makes you think this?

How would an acute viral sinusitis present?

A

4 weeks> Symptoms >10 days
** viral sinusitis progresses to bacterial so viral sinusitis will present with <10 days symptoms. Then initial improvement, then secondary bacterial infection **

Symptoms: purulent nasal mucus, nasal congestion, facial pain eg. headache

Virus specific symptoms: myalgia, sore throat, fever

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

How would you manage acute viral sinusitis?

A

Analgesia/antipyretic = Paracetamol/ibuprofen

Decongestant = topical Oxymetazoline nasal

saline spray OR Corticosteroids for congestion = Mometasone nasal

Topical anticholinergics for rhinorrhoea = Ipratropium nasal

Mucolytics = Guaifenesin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

How would you treat acute bacterial sinusitis?

A

wait 10 days before antibiotics!!!

  • amoxi or co-amoxi or phenomethylpenicillin
  • analgesia, decongestant, mucolytics, ipratropium, saline spray, etc as needed.

If immunocompromised:

  • high dose co-amoxi
  • for penicillin allergy, give clindamycin + cefixime
  • for beta lactam allergy, give doxycycline
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

When do you refer sinusitis to ENT?

A

Over 4 episodes per year

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is chronic sinusitis and how is it differentiated?

A

Paranasal sinus inflammation lasting >12 weeks.

There is with and without polyps

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

How does chronic sinusitis happen?

A

Usually due to an anatomical obstruction of the osteomeatal complex, resulting in inadequate sinus drainage of mucus.

Acute exacerbation:
1) Inflammation of the sinonasal mucosa occurs due to allergies, viral infections, pollutants.

2) Results in additional swelling in narrow osteomeatal channels.
3) Pooling of mucus means resp cilia cannot clear it. It thickens and microorganisms grow.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

How would you manage acute exacerbations?

A

1st line = co-amoxi or cefuroxime

Nasal saline irrigations

Intranasal corticosteroids eg. budesonide, mometasone, etc
(ensure proper spraying technique or epistaxis risk!)

Decongestants eg. ometazoline nasal spray

Prednisoline oral to shrink polyps

Antihistamines eg. Loratadine, cetrizine, chlorphenamine for allergic rhinitis

Endoscopic sinus surgery last resort

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are common bacterial organisms that cause acute sinusitis?

A

strep pneumoniae

haem influenzae

moraxella catarrhalis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

How would otitis externa present?

What would a diabetic be at risk for?

A
  • Acute onset ear pain, tender on tragus manipulation
  • ear canal swelling and erythema
  • ottorhoea
  • feeling of fullness
  • decreased hearing due to swelling
  • erythematous tympanic membrane

Malignant or necrotising otitis externa

17
Q

How would you manage OE

A
  • clean the ear from debris/wax
  • Antibacterial otic drops (ciprofloxacin/dexmethasone can be used even in perforated tymp membranes)
  • paracetamol/ibuprofen for pain
  • add systemic antibiotics for immunocompromised (systemic ciprofloxacin OR amoxi OR co-amoxi)
18
Q

Explain the pathophysiology of otitis media

What are 3 common bacteria?

A

Upper resp infections can infect nasal passage –> E tube –> Middle ear, and impair the Eustachian tube’s mucociliary action and ventilator function.

Middle ear effusion occurs and nasopharyngeal bacterial grow.

Pressure against tympanic membrane = perforation = purulent ottorhoea

Common bacteria = strep pneumonia, H influenzae, moraxella catarrhalis

19
Q

How would otitis media present?

A
  • Hx of exposure to resp viruses eg. healthcare/ daycare worker
  • Symptoms of an upper resp infection
  • bulging tympanic membrane
  • myringitis (erythema of tympanic membrane)
  • fever
20
Q

How would you manage otitis media?

A
  • Analgesia eg. paracetamol/NSAIDs

Antibiotics = amoxicillin OR co-amoxi

Tympanocentesis can relieve pressure in middle ear

21
Q

How do cholesteatomas occur?

A
  • a retraction pocket is an area of invagination of the tympanic membrane
  • gets pulled into the middle ear due to -ve pressure caused by eustachian tube dysfunction
  • the pocket can trap squamous cells with keratin accumulation and retentino.
  • These cells proliferate
  • Bacterial infection of the trapped cells can form a biofilm (commonly pseudomonas aeruginosa)
22
Q

What damage can result from the bacteria accumulating in the cholesteatoma?

A

cytokine induced inflammatory changes:

  • activates osteoclasts and lysozymes
  • bony ossicle destruction = conductive hearing loss
  • destruction of semicircular canals = vertigo
  • destruction of cochlea = sensorineural hearing loss
  • erosion into facial canal = facial palsy
  • acute infection of temporal bone = mastoiditis
23
Q

How do cholesteatomas typically present?

A
  • ear discharge unresponsive to antibiotics
  • hearing loss (usually conductive)
    attic crust in retraction pocket
  • white mass behind intact tympanic membrane (congenital)
  • tinnitus
  • otalgia
  • altered taste (facial nerve involvement)
  • dizziness (erosion into semicircular canal)
24
Q

How would you manage a cholesteatoma?

A

surgery!!! eg. mastoidectomy
- 8mg dexamethasone for post-op nausea

  • Topical antibiotics containing quinolone for the discharge (ciprofloxacin, ofloxacin)
  • ** can use a wick to deliver ear drops into a really swollen canal ***
  • aural cleaning to reduce discharge
25
Q

How would Benign Paroxysmal Positional Vertigo present?

A
  • Brief (<30second) episodic vertigo provoked by head movements !!!
  • Episodes = repeated attacks over weeks/months in posterior canal, repeated attacks over days to weeks in lateral/horizontal canal
  • Sudden onset and severe vertigo
  • Nausea, imbalance, lightheadedness
  • NO associated neuro/otological symptoms eg hearing loss, tinnitus, aural fullness, etc
  • Dix-Hallpike manouevre positive !!!
  • Supine lateral head turn positive
26
Q

What is the mechanism behind BPPV?

A

Canalithiasis and Cupolithiasis.

Canalithiasis = free floating particles called “canaliths” that migrate into the semicircular canals via natural head movements.
Usually accumulate in posterior semicircular canal until they manage to displace the cupula. Results in vestibule-ocular reflex and triggers nystagmus.
The canaliths in the semicircular canals also result in abnormal signals and misperception of movement resulting in vertigo.
Vertigo resolves when head movement stops, canalith particles stop moving, cupula returns to normal position.

Cupulolithiasis =
Dense canaliths adhere to cupula. Cannot be fixed by repositioning manouvres so this is more chronic BPPV.

27
Q

How would you manage BPPV?

A

Posterior canal:

  • particle repositioning manoeuvres (can be uncomfortable)
  • Semont manoeuvre
  • Surgery

Refer to tertiary specialist dizziness clinic if horizontal/anterior bppv suspected, neuro symptoms, non-responders, multiple recurrences.

28
Q

How does labrynthitis present?

A
  • Few seconds to minutes long episodes
  • Vertigo and dizziness provoked by head movements
  • nausea/ vomiting
  • hearing loss
  • otorrhoea
  • Nystagmus
  • Tinnitus !!
  • Influenza like symptoms
  • Otalgia
  • Rinne’s and Webers show sensorineural hearing loss
29
Q

What is the pathophysiology of labrynthitis?

A
  • Infections in the middle ear can spread to the inner ear through the oval/round window.
  • Meningitis can spread from inner ear through cochlear aqueduct or cochlear modiolus
  • viral labrynthitis is associated with preceding upper resp infection eg. varicella zoster, cytomegalovirus, mumps, measles, rubella, HIV
  • Bacterial labrynthitis is associated with acute/chronic otitis media, meningitis, cholesteatoma (commonly Treponema pallidium, H Influenza, strep, staph, N meningitidis)
30
Q

How would you manage Labrynthitis?

A

Viral

  • vertigo = vestibular suppressants (eg. diazepam, lorazepam), benzodiazepines, Meclozine (anticholinergic and antihistamine)
  • Nausea = antiemetics eg. metocloperamide, promethazine

Bacterial labrynthitis

  • Acute/chronic otitis media = on previous cards
  • Bacterial meningitis = IV antibiotics eg. cefotaxime, ceftriaxone, meropenem

Vestibular rehabilitation if symptoms persist

31
Q

How would menieres present?

Try and think how it would present differently to BPPV, Labrynthitis, Vestibular Neuronitis

A
  • episodes of vertigo lasting up to hours
  • Unilateral hearing loss (usually low freq hearing loss)
  • Unilateral roaring tinnitus
  • Aural fullness (sometimes precipitates attack)
  • sudden Loss of balance without other symptoms
  • Positive Rombergs test
  • Rinne and Webers show unilateral sensorineural hearing loss
32
Q

How would you manage Menieres?

A
  • Low salt diet and diuretics
  • Anti-emetics eg. Meclizine, Promethazine
  • Intratympanic corticosteroid injections (Dexmethasone sodium phosphate)
  • Meniett device
  • Vestibular and balance rehabilitation
  • Tinnitus retraining therapy, hearing aids, tinnitus maskers, etc
  • Hearing aids or assistive listening devices for hearing loss
  • Labrinthectomy surgery as last resort!