Energy? Flashcards

1
Q

Describe energy storage and demand.

A
  • Organism has energy expenditures: maintenance, repair, growth, activity, thermogenesis
  • Organisms has storage of energy: glycogen in liver and muscle and fat in adipose tissue
  • If expenditures increase, energy can be taken out of storage
  • If activity increases greatly, this may increase the demand
  • Increased demand is supplied by eating fats, carbohydrates, proteins, vitamins and minerals
  • At rest, principal energy users are the heart, liver and brain
  • When active, skeletal muscles are the principle users of energy
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2
Q

How are eating behaviours controlled?

A
  1. Energy reserves generate signals of available energy
  2. Signals to hypothalamus
  3. Behaviour can be modified to either increase/decrease energy in eating or increase/decrease activity, growth, repair and cellular metabolism
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3
Q

How is brown fat distinguishable from white fat?

A

Can turn energy into heat

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4
Q

How is intake regulated?

A
  1. Satiety centre inhibits the appetite centre.
  2. Appetite centre stimulates feeding behaviours.
  3. Modulating input go into both the satiety and appetite regions of the brain.
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5
Q

What stimulates the satiety centre?

A
  • Increased glucose and insulin
  • Increased CCK, which correlates to fat content of meal]increased stomach distension
  • Increased body fat stores
  • Leptin from white fat, proportionate with fat reserves
  • Peptide YY/PYY from epithelium of small and large bowel, with a release proportional to the meal size
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6
Q

What inhibits the satiety centre?

A
  • Smell of desirable food
  • Sight of desirable food
  • Ghrelin from stomach. Levels increase during fasting
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7
Q

What is parabiosis in feeding behaviour?

A

The anatomical joining of 2 individuals, especially artificially in physiological research. The 2 share a bloodstream.

  1. When stable, both animals eat normally.
  2. Electrical lesion of ventromedial nuclei in A
  3. A becomes obese and B becomes anorexic
  4. Unkown substance passes form mouse with lesion/A into the blood stream of the normal mouse/B to reduce feeding behaviour, causing B to starve

Feeding/appetite centre in the lateral nucleus – stimulation causes feeding and lesioning causes disinterest in food.

Satiety centre in ventromedial nucleus – stimulation terminates eating and lesioning causes voracious and constant feeding.

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8
Q

How is output regulated?

A

Eating increases basal metabolic rate and heat production, dietary induced thermogenesis is a process related to secretion, digestion and absorption and uses additional blood supply.

Output/metabolism is measured as oxygen consumption/heat production under eating/basal condition.

With increasing body mass, energy consumption per unit mass and need of food falls. Partly due to heat loss.

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