Endodontics Flashcards

Pathological Resorption: 52 - 94 Management of complex cases: 95 - 140 Retreatment Procedures: 141 -175

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1
Q

Endodontic material

A
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2
Q

Types of stainless-steel files

A

Files:
1. K file – have a square cross section
2. Flexofile – have a triangular cross section
3. Hedstroem file
4. Barbed broaches – only used for removing pulpal tissue when present, not suitable for use in narrow, curved canals

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3
Q

Most canal treatments are started with hand instruments followed by the completion by rotary instruments.

A
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4
Q

Hand instruments are especially useful at the early phases of instrumentation to establish a glide path, prior to using rotary instruments.

A
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5
Q

In anatomically challenging cases an in treating instrumentation complications, hand instruments may be the only solution.

A
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6
Q

Flexofiles
Used for most instrumentation:

preparation of a glide path – defined as a smooth, patent passage from the coronal orifice of the canal to the radiographic terminus (apical constriction). A successful glide path is an uninterrupted passage that can be reproduced when small-size files are used in sequence in the canal.

A
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7
Q

Apical gauging – apical gauging implies measuring/assessing the apical diameter of the canal, where the instrument fits snuggly and resist further apical movements. This ensures apical terminus of the prepared canal. Ni-Ti instruments are preferred for gauging because of their flexibility. They are inserted straight in and straight out, without any rotation. This is done after preparation of root canal. They have a nonaggressive tip (batt tip)

A
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8
Q

Hedstroem files – very stiff files, can only be used in up and down motion, file cuts when moved in the coronal direction. Today only used in retreatment cases to help remove gutta-percha or an overfilling of the root canal.

A
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9
Q

Interappointment medicaments:
Use of non-setting calcium hydroxide paste in endodontics, non-setting paste, powder mixed with sterile water.

A

Aims –
1. To reduce and prevent multiplication of microorganisms that remain following careful cleaning and shaping.
2. Prevent reinfection through coronal or apical leakage
Calcium hydroxide uses –
1. Inter-appointment intracanal medicament
2. Pulp capping
3. Apexification
4. During treatment of root perforations, root fractures, root resorption and dental trauma.
5. Root canal sealer

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10
Q

The European society of endodontology’s quality guidelines suggest that when we carry out root canal treatment, we are obliged to do a clinical and radiographic follow-up at least:

A
  • 1 year after treatment
  • Further follow-up for up to 4 years – if bony healing is not complete according to Strinberg Acta Odontol Scand 1956: 14: 1-175
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11
Q

When is root canal retreatment indicated:

A
  1. Persistent periapical pathology following root canal treatment
    No radiographic signs of bony healing after 4 years
  2. New periapical pathology associated with a root filled tooth.
    Initial healing but a new radiolucency develops some time later.
    Root canal system has become infected after previous treatment.
  3. A new restoration is planned for a tooth and radiographic assessment shows an inadequate root canal filling and/or a periapical radiolucency.
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12
Q

The Toronto study is a publication on the treatment outcome in endodontics shows the following for initial treatment.
Primary treatment
- Without periradicular periodontitis = 92%
- With periradicular periodontitis = 74%
- Overall = 81%
The Toronto study then later went to do a publication: treatment outcome in endodontics: the Toronto study—phases 3 and 4: orthograde retreatment.
Retreatment
- Without periradicular periodontitis = 89-100%
- With apical periodontitis = 56-84%

A
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13
Q

A study done by Ng et al. 2011: a prospective study of the factors affecting outcomes of nonsurgical root canal treatment: part 1: periapical health.
The conclusion from this study was that success was based on periapical health
- Primary RCT 83%
- Secondary RCT 80%

A
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14
Q

A systematic review done by Ng: outcome of secondary root canal treatment: a systematic review of the literature.
17 studies were included ranging from 1961-2005, the overall success rate was found to be 77%
The studies had three prognostic factors; these three factors all determine the success rate of secondary root canal treatment.

A
  • Pre-operative periapical lesions
  • Apical extent of root canal filling
  • Quality of coronal restoration
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15
Q

The terms to describe the outcome of RCT is no longer success or failure, they are the following.

A

Healed
- Clinically – no signs/symptoms
- Radiological – no residual radiolucency, or scarring after surgery

Healing
- Clinical – no signs or symptoms
- Radiological – reduced radiolucency in follow-up < 4 years

Asymptomatic function
- Clinical – no signs or symptoms combined with no or persistent radiolucency, reduced in size or unchanged.

Persistent/recurrent/emerged disease
- Clinical – with or without symptoms
- Radiological – new, increased, unchanged or reduced after >4 years

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16
Q

Prevention of post-treatment disease
Guidelines on the quality of root canal treatment

A
  1. Rubber dam isolation
  2. Proximity of preparation to apical constriction
  3. Sufficient taper of preparation – allowing for adequate irrigation and disinfection of the root canal. This will also allow enough space for interappointment medicament if required (non-setting calcium hydroxide)
  4. Correct extension of root canal obturation without extrusion
  5. Adequate coronal seal to prevent re-infection
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17
Q

Indications for root canal retreatment

A
  • Previous treatment has failed
    Signs of inflammation or infection
  • Persistent symptoms, sinus tract (chronic abscess), swelling, pain.
  • Failure of previous treatment because of technical reasons
  • Existing pathology and new restoration planned for tooth.
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18
Q
A

A = radiograph of mandibular with inadequate root filling and asymptomatic apical periodontitis.
B = the tooth has been retreated conservatively through the crown.
C = complete periapical repair is evident at the 6-month control.

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19
Q

This image shows 4 potential areas where microbes can be post treatment.

A
  1. Intraradicular microbes: if the preparation of the canal is not at its correct length, then microbes will persist at the constriction and spread.
  2. Extraradicular microbes: this is when microbes have invaded the host response system and established themselves in the periapical tissues.
  3. Foreign body reaction to extruded gutta- percha.
  4. True cyst which is cavity that has walled itself off from the root canal system.
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20
Q

Causes of post-treatment disease

A

Microbial causes
- Intraradicular microbes: Intraradicular infections, either persistent or secondary are the major causes of endodontic treatment failure.

Persistent = where the microbes were not removed during the initial treatment

Secondary = where the microbes have entered the root canal system via coronal leakage
- Extraradicular microbes
- Radicular cyst
- Cracked teeth, vertical root facture = allowing microbes to enter the root canal system
- Coronal leakage

Non-microbial causes
- Cholesterol crystals in the periapical tissues
- Foreign body reactions in the periapical tissues

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21
Q

Intraocular infections in root canal treated teeth can be classified in two groups

A
  1. Persisting infection: inadequate isolation/disinfection during treatment
  2. New secondary infection through leakage
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22
Q

Radiographically you can see if a poor root canal filling has been placed, and often associated with periapical radiolucency, it is worth to note that radiographs do not indicate the biological status of the root canal.

A

Persistent bacteria
- Those that remain in the root canal system after root canal disinfection and interappointment dressing gram positive bacteria appear to be more resistant to antimicrobial treatment and can adapt to harsh environmental conditions in instrumented and medicated root canals.
However, they do not always maintain an infectious process
- Residual bacteria may die after obturation
- Residual bacteria may be present in insufficient numbers and virulence
- They may be located in areas where they have no access to periapical tissues
In canals that are apparently well treated tend to have – 1-5 species
In canals with inadequate treatment tend to have 10-20 species like untreated canals.
In retreatment cases nine times more likely to harbour Enterococcus Faecalis, candida in 18%

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23
Q

Common microbes associated with retreatment cases:

A
  1. Enterococcus faecalis
  2. Streptococcus
  3. Lactobacillus
  4. Actinomyces
  5. Propionibacterium
  6. Candida albicans
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24
Q

The possible origins of these microbes:

A
  1. Contamination during initial treatment
  2. Leaving a tooth on open drainage
  3. Coronal leakage post-treatment
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25
Q

In most cases apical periodontitis inflammatory lesions succeed in preventing microorganisms from invading the periapical tissues.

The inflammatory response would be the bodies response to supress this infection from reaching the periapical tissues.
Occasionally the microorganisms can overcome the inflammatory defence barrier and establish an?

A

Extraradicular infection e.g., acute apical abscess.

Bacterial colonies can also form biofilms on the external root surface and bacterial colonies can exist inside periapical granulomas.

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26
Q

What is a radicular cyst?

A

Is the most common odontogenic cyst of inflammatory origin, arising from epithelial cell rests in periodontal ligament.

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27
Q

A radicular cyst can develop into a true cyst or a pocket cyst. what are the differences?

A

True cyst: lesion enclosed by epithelial lining.

Pocket cyst: epithelial sac communicates with root canal system.
- A pocket cyst will normally heal following endodontic treatment whereas a true cyst wont.

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28
Q

Endodontic failure caused by inadequate restorative procedures: review and treatment recommendations, Healing et al. J prosthet dent; 87: 674-8 – reviewed literature 1969-99 41 papers concluding:

A

Prognosis can be improved by sealing the canal and minimizing leakage of oral fluids and bacteria – do it as soon after root canal treatment as possible.

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29
Q

Cholesterol crystals are from ?

A

dying cells during chronic inflammation

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30
Q

Foreign body reaction include:

A
  1. Gutta-percha
  2. Sealers
  3. Paper points
  4. Cotton pellets – contain cellulose and the body has no way of breaking it.
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31
Q

A patient coming in for retreatment for root canal, a clinical assessment has to be made, this includes the following.

A
  1. Patient history
  2. Examination
  3. Special investigations as required
  4. Radiographic examination
  5. CBCT
  6. Diagnosis
  7. Treatment plan

Do nothing
Extract tooth
Retreatment
Surgical retreatment

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32
Q

When we look at a radiograph and CBCT for evaluation of retreatment the following is looked for:

A
  1. Caries
  2. Defective restorations
  3. Periodontal health
  4. Quality of obturation
  5. Existence of missed canals or procedural errors
  6. Periapical pathology
  7. Perforations
  8. Fractures
  9. Resorptions
  10. Canal anatomy
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33
Q

when Planning retreatment what should we be considering?

A
  1. Is it a strategic tooth? If it is a lone standing molar, then maybe it’s not worth the time and cost to retreat.
  2. How much remaining coronal tooth structure is remaining? So that tooth can be restored once the complex procedure is complete.
  3. What is periodontal support?
  4. Is periapical disease present?
  5. Are there aesthetics issues
  6. Are there technical difficulties as shown radiographically?
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34
Q

Techniques to retreating root canals
1. Gain access to root canal system
Crown removal
Post removal
2. Removal of root canal filling material
Gutta percha
Carrier based systems
Silver points
Pastes

A
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35
Q

Treatment with direct restorations, what are the stages to get access to the canals?

A
  1. Remove any existing caries
  2. Reduce any unsupported cusps
  3. Ensure sufficient remaining tooth structure to place definitive restoration
  4. Ensure tooth can be isolated under rubber dam
    Orthodontic band/GI
    Electrocautery, crown lengthening procedures
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36
Q

Teeth with indirect restorations, assess quality of restoration
1. Crowns, bridges, inlays, onlays, post cores
2. Integrity of coronal seal – no recurrent caries or marginal deficiencies

A
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37
Q

Access through existing crowns pose a risk, why?

A
  1. A higher risk of perforation
  2. Visibility reduced
  3. Tooth alignment may be altered by the crown

Important to carefully assess the pre-treatment radiograph, if any potential difficulties remove the crown.

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38
Q

Removing crowns, what are the benefits when trying to gain access to the canals?

A
  1. When the crown is defective/caries
  2. Allows assessment of remaining tooth structure
  3. Visibility and access to root canals improved
  4. Avoids risk of perforation when searching for canals – especially if tooth alignment has been altered by the crown
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39
Q

Removing a crown by sectioning, what are the procedures for this?

A
  1. Take a sectional impression first in putty
  2. Section the crown into two halves taking care not to cut through the core, then remove with an excavator
  3. Use a diamond bur for porcelain and tungsten carbide for metal
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40
Q

what is a wamkey used for?

A

a technique used to remove a crown.

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41
Q

Post removal can be complicated, the technique is dependent on the type of post, what should we ensure we do before we remove any posts ?

what are the types of posts/core/cement used?

what must you assess on a radiograph when undergoing post removal?

what techniques are available to remove posts?

A

Technique should minimize risk of root fracture: always important to advise patient that root fracture may be a complication of attempted post removal

Depends on type of post and core and cement: parallel/tapered. Active/passive, metal/quartz fibre

Always assess post type and length/width on a pre-treatment radiograph

2 main techniques:
- Post removal with ultrasonic energy
- Post removal with post pulling devices

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42
Q

Removing cast post core
1. Usually involves removal of coronal restoration
2. May require cut back of core using tungsten carbide bur prior to using ultrasonics
3. If post is extremely well fitted removal can be very difficult especially in oval shaped canals.
Techniques used to remove gutta percha
- Rotary endodontic files
Protaper D files for retreatment – used at 600 rpm 4Ncm
PTG endo instruments
- Ultrasonics
- Heat
- Solvents

A

Removal of GP using PTG
1. Measure estimated WL for radiograph
2. Depending on canal diameter select either an F2 or F3 finishing file, length 21mm
3. Set rpm to 600
4. Use in coronal 2/3s of canal – work from F3-F2-F1 if required
5. Then go down 1mm increments checking with EAL until WL and patency have been achieved
6. If apical part of the canal is underprepared/not obturated, negotiate with size 10 file, establish WL and complete preparation using normal PTG sequence at 300 rpm

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43
Q
A
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44
Q

Types of stainless-steel files
Files:

A
  1. K file – have a square cross section
  2. Flexofile – have a triangular cross section
  3. Hedstroem file
  4. Barbed broaches – only used for removing pulpal tissue when present, not suitable for use in narrow, curved canals
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45
Q

Most canal treatments are started with hand instruments followed by the completion by rotary instruments.

A
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46
Q

Hand instruments are especially useful at the early phases of instrumentation to establish a glide path, prior to using rotary instruments.

A
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47
Q

In anatomically challenging cases an in treating instrumentation complications, hand instruments may be the only solution.

A
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48
Q

Flexofiles

Used for most instrumentation:
preparation of a glide path – defined as a smooth, patent passage from the coronal orifice of the canal to the radiographic terminus (apical constriction). A successful glide path is an uninterrupted passage that can be reproduced when small-size files are used in sequence in the canal.

A
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49
Q

Apical gauging – apical gauging implies measuring/assessing the apical diameter of the canal, where the instrument fits snuggly and resist further apical movements. This ensures apical terminus of the prepared canal. Ni-Ti instruments are preferred for gauging because of their flexibility. They are inserted straight in and straight out, without any rotation. This is done after preparation of root canal. They have a nonaggressive tip (batt tip)

A
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50
Q

Hedstroem files – very stiff files, can only be used in up and down motion, file cuts when moved in the coronal direction. Today only used in retreatment cases to help remove gutta-percha or an overfilling of the root canal.

A
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51
Q

Interappointment medicaments:

Use of non-setting calcium hydroxide paste in endodontics, non-setting paste, powder mixed with sterile water.

A

Aims –
1. To reduce and prevent multiplication of microorganisms that remain following careful cleaning and shaping.
2. Prevent reinfection through coronal or apical leakage

Calcium hydroxide uses –

  1. Inter-appointment intracanal medicament
  2. Pulp capping
  3. Apexification
  4. During treatment of root perforations, root fractures, root resorption and dental trauma.
  5. Root canal sealer
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52
Q

A physiological oresr pathological event mainly occurring due to the action of activated clast cells.

Characterised by the transitory or progressive loss of cementum or cementum/dentine. Onset is associated with significant necrosis of cementoblasts and/or injury to the periodontal ligament – this is the definition of what?

A

Resorption

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53
Q

Resorption occurs mainly due to the activation of which cells?

A

Clasts cells

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54
Q

The onset of resorption usually begins with what?

A

Significant necrosis of cementoblasts and/or injury to the periodontal ligament.

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55
Q

Two phases are required for resorption what are they?

A

Injury and stimulation

  • Injury is related to non-mineralized precemtum or predentine
  • Cementoblasts are destroyed directly or become necrotic as a result of compromised blood supply to PDL or Pulp
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56
Q

There are three main causes of injuries that result in resorption what are they?

A
  1. Mechanical
    - Trauma
    - Surgical procedures
    - Excessive pressure (impacted teeth, cysts tumours, orthodontic treatment)
  2. Infections of root canal or PDL
  3. Chemical – bleaching agents (30% hydrogen peroxide)
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57
Q

What type of stimulation results in resorption?

A
  1. Infection
  2. Pressure
  3. Without a constant stimulus the process is self-limiting (transient resorption)
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58
Q

There are some systemic and endocrine diseases that may result in tooth resorption, what are they?

A
  1. Hypo and hyperthyroidism
  2. Calcinosis - a condition in which calcium salts are deposited in the skin and subcutaneous tissue.
  3. Gaucher’s syndrome - the result of a build-up of certain fatty substances in certain organs, particularly your spleen and liver.
  4. Turner syndrome – female only, developmental problems
  5. Pagets disease – disrupts normal cycle of bone renewal, causing bone to become weak and deformed.
  6. Herpes zoster – shingles
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59
Q

What is the mechanism for resorption?

A
  1. Damage causes chemotactic process
  2. Area of damage attracts activated cells
  3. Activated cells odontoclasts/osteoclasts
  4. Activated cells colonise the damaged surfaces and initiate the resorptive process
  5. The cells are located in depressions known as howship lacunae
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60
Q

When an osteoblasts detects some form of damage what does it release?

A

They release RANKL

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61
Q

What to the RANKL bind on to?

A

They bind on to the RANKL receptors on the nearby monocytes (precursor osteoclast) on the surface

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62
Q

What does the binding of the RANKL do the monocyte?

A

RANKL induces the monocytes to fuse together to form a multinucleated osteoclast cell. RANKL helps the osteoclast cell to mature and activate so they can start resorbing

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63
Q

What enzymes do osteoclasts cells release to resorb the bone?

A

Secrete lysosomal enzymes (collagenase)

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64
Q

The osteoclasts drills pits in to the bone known as what?

A

Howship’s Lacunae

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65
Q

Osteoclast also produce hydrochloric acid which dissolves what to produce what?

A

Hydrochloric acid dissolves to hydroxyapatite to produce calcium and phosphate ions

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66
Q

o keep the bone resorption under control, the osteoblasts also secrete?

A

Osteoprotegrin

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67
Q

What is the purpose of osteoprotegrin?

A

Osteoprotegrin binds to RANKL and prevents its from activating the RANK receptors on the monocyte. Slowing down the activation of osteoclasts

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68
Q

Once osteoclasts have completed there job they commit suicide by?

A

Apoptosis

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69
Q

Following bone resorption osteoblasts migrate to site of resorption (lacunae) and secrete?

A

Osteoid seam, which is made of collagen to fill in the lacunae created by the osteoclasts.

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70
Q

Calcium and phosphate deposit on the seam creating what?

A

Hydroxyapatite

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71
Q

Fuss et al. dent traumatol 2003 19(4): 175-82 based stimulating factors that cause resorption to be the following:

A
  1. Pulp infection
  2. Periodontal infection
  3. Orthodontic pressure
  4. Ankyloses
  5. Tumour
  6. Impacted pressure
  7. Conclusion most common cause of resorption is pulp infection
72
Q

External inflammatory root resorption is usually a result of?

A

Intrusion, lateral luxation and avulsion

73
Q

External resorption damages the non-mineralised precementum which then allows exposure of dentine to what?

A

Odontoclastic activity

74
Q

Once the dentine has been exposed to odontoclastic activity it progresses due to what reason?

A

Due to microbial stimulation from the infected, necrotic pulp.

75
Q

How can you diagnose external inflammatory root resorption?

A
  1. Based on radiographic and CBCT interpretation
76
Q

What are the treatment for external inflammatory root resorption?

A
  1. Removal of necrotic pulp as soon as signs of EIR
  2. Use calcium hydroxide as an interappointment dressing
  3. In many cases the resorption is too advanced to treat
77
Q

When does invasive cervical resorption occur?

A

Occurs when loss of protective non-mineralised layer at CEJ – due to developmental, physical/chemical trauma/microbial stimulation from gingival sulcus

78
Q

What are the predisposing factors for invasive cervical resorption?

A
  1. Orthodontics
  2. Trauma
  3. Surgery
  4. Intracoronal bleaching
79
Q

What can invasive cervical resorption be misdiagnosed as?

A

Internal resorption

80
Q

What are the clinical features of invasive cervical resorption?

A
  1. Asymptomatic
  2. Tooth may look pink
  3. Positive sensibility test
  4. Tooth will be vital as pulp is protected until late in the process by a layer of dentine and predentine
  5. Eventually the lesion will perforate the canal wall resulting in canal infection and necrosis
81
Q

this image shows the Heithersay 1999 clinical classification of cervical resorption. What do each class indicate.

A

Class 1 - small with shadow penetration

Class 2 – close to coronal pulp, no radicular extensions

Class 3 – deeper but not beyond coronal third

Class 4- extensive beyond coronal third

82
Q

What would you see in a radiographic feature that discriminates internal root resorption.

A

With internal root resorption on a radiograph you would not expect to see the outlines of the pulp canal. With invasive cervical resorption, you would see resorption inside, but because you can see the outline of the pulp, this determines that it is invasive cervical resorption.

83
Q

What is the treatment plan for invasive cervical resorption?

A
  1. Remove granulation tissue from defect with caustic acid (90% trichloroacetic acid)
  2. Restore with GI, composite or Biodentine
  3. RCT if communication with pulp canal
84
Q

Internal root resorption originates in and affects the root canal wall, follows damage to odontoblastic layer and predentine, the aetiology still unknown, but most likely as a result of?

A

Trauma

85
Q

To continue the pulp tissue apical to the lesion must have a viable what?

A

Blood supply

86
Q

Clinically extensive internal root resorption can result in pink discolouration of the crown which by confused with?

A

Invasive cervical resorption

87
Q

Oval, round lesions any site along root canal usually symmetrical, what type of resorption is going on?

A

Internal root resorption

88
Q

What is the treatment of internal root resorption?

A
  1. Root canal treatment if tooth can be saved
  2. Lesion difficult to clean (bleeding) and obturate (shape)
  3. Thermoplastic techniques – required to fill in the canal
  4. If untreated will cause perforation and clast cells can obtain nutrients from surrounding tissues
89
Q

this radiograph is an example of what type of resorption?

A

Orthodontic pressure root resorption

90
Q

Ankylotic root resorption occurs when there is?

A

Severe traumatic injuries (intrusive, luxation, avulsion with delayed reimplantation) injury to the root canal is so large that healing with cementum is not possible and the bone comes into contact with the root surface

91
Q

What would you clinically see with ankylotic root resorption?

A

The teeth lack physiological mobility and sound metallic to percussion

92
Q

What would you see radiographically at an ankylotic root resorption?

A

Bone fills the resorption lacuna and there is no radiolucency

93
Q

What is the treatment plan for ankylotic root resorption?

A

There is no stimulation factor to remove and no predictable treatment. Prevention is better by minimising damage to periodontal ligament.

A functional splint should be placed for 7-10 days and root canal treatment to prevent pulpal infect root resorption.

94
Q

In replacement resorption what is replaced?

A

Dentine and cementum are lost and replaced with bone

95
Q

What are the procedural errors that cause complications during root canal treatment?

A
  1. Fractured instrument
  2. Ledges
  3. Canal blockages
  4. Canal transportations
96
Q

What are the two different ways that rotary files can fracture?

A
  1. Torsional stress – happens when a tip binds against canal wall and the coronal part of the file roates; where the elastic limit of the metal is exceeded causing plastic deformation and then fracture.
  2. Cyclic fatigue when repeated cycles of tension and compression happened during bending
  3. Both factors combined together.
97
Q

There are many situations that can contribute to file fracture what are they?

A
  1. File size and taper – fine, more flexible files are more vulnerable to torsional stress (high torque) but more resistant to cyclic fatigue and vice versa
  2. Type of alloy – SS vs NiTi
  3. Manufacturing of NiTi Files
  4. Less experienced operator
  5. Inadequate access and glide path
  6. Anatomy – canals merging or dividing, abrupt curvature and radius, s-shaped, isthmuses, fins
  7. Apical pressure
  8. High speed
  9. Repeated use – UK single use only
98
Q

Gary chung back in 2009 published a paper on incidence of file fracture

What are the favourable conditions when a file has been separated?

A
  1. No periapical periodontitis
  2. File in the apical 1/3 of the root
  3. Able to retrieve non-surgically or surgically if periapical pathology is present
  4. Defect correctable with apical surgery
99
Q

What are the questionable conditions when a file has been separated?

A
  1. Instruments fractured in coronal or mid-root portion of the canal and cannot be retrieved
  2. Patient asymptomatic
  3. No periapical periodontitis
100
Q

What are the unfavourable conditions when a file has been separated?

A
  1. Patient is symptomatic
  2. Lesion present
  3. Extensive procedure to retrieve instrument – compromising long term survival of tooth
  4. Surgical treatment not an option
101
Q

If a file has been separated in the root canal and the patient has irreversible pulpitis what should we be thinking to manage this?

A
  1. Canal will be minimally infected – disease has not progressed to roots yet
  2. No pre-existing apical pathology
  3. If possible, remove or by-pass
  4. If not possible the retained fragment should not influence prognosis
102
Q

If a file has been separated in the root canal and the patient has infected canal what should we be thinking?

A
  1. What stage of the treatment did the file separate?
  • End of instrumentation, canals disinfected -> embedded fragment in filling material if cannot be removed.
  • Early in treatment, canal beyond instrument cannot be cleaned and this may be directly responsible for failure -> attempt removal or by-pass if possible
103
Q

Who proposed this algorithm to manage the fracture of NiTi instruments?

A

Washes et al

104
Q

How can we determine if a fractured can be bypassed?

A

Inserting a small size 10 file alongside the fractured instrument and reaching working length. If this possible we continue canal preparation and obturation.

105
Q

If we can bypass a fractured instrument in the canal, should we later remove the file?

A

No we should not actively attempt removal

106
Q

If we cannot bypass a fractured instrument, what do we have to determine?

A

Where the file is located

107
Q

If the file is in the apical third, should we attempt removal?

A

Removal not practical without risk of damage

108
Q

If the file is located in the middle/coronal third and straight-line access is possible, what should we consider?

A

Consider attempt at removal

109
Q

If the file is located in the middle/coronal third and straight-line access is not possible, what should we consider?

A

do not attempt removal

110
Q

What are the factors affecting prognosis of a tooth that has a file separated into the canal?

A
  1. Periapical lesion
  2. Stage of canal preparation
  3. Potential weaking of root
  4. Perforation/procedural risks
111
Q

What are the issues we have to think about when we have to remove a fractured file?

A
  1. Root length
  2. Curvature
  3. Dentin thickness
  4. Technique of removal
  5. Length of fragment
  6. Presence/absence of periapical radiolucency
  7. Stages of canal preparation when fracture occurred
112
Q

B. Suter published a paper on the probability of removing fractured instruments from root canals, he found that what percentage of files were successfully removed?

A

87%

113
Q

B. Suter published a paper on the probability of removing fractured instruments from root canals, he found that what percentage of files were unsuccessfully removed and due to what reason?

A

13% failure to remove due to perforation (mostly in the apical 1/3), incomplete removal

114
Q

Which author determined that the position of the file in relation to the root curvature influences the successful removal of the file?

A

Bobby Patel.

115
Q

What are the risks of removal of fractured instruments?

A
  1. Excessive removal of radicular dentine which may predispose the root to fracture
  2. Ledging
  3. Perforation
  4. Limited application in narrow and curved canals
  5. Possibility of extrusion of the fractured file
  6. The more apically positioned the higher the risk of iatrogenic damage
116
Q

What are the 4 techniques for the removal of the fractured fie?

A
  1. Mechanical
  2. Ultrasonic
  3. Tube techniques
  4. Other
117
Q

What are the mechanical techniques to remove the fractured file?

A
  1. H file (s)
  2. Gripping devices: fine haemostat or Stieglitz forceps
  3. Excavators
118
Q

The removal of a fractured file using the hedstroem file technique is only possible if?

A

The file is located coronally with a wide canal

119
Q

What are these?

A

Ultrasonics

120
Q

What is the technique used to remove a fractured file?

A
  1. Create a straight-line access
  2. Trephine around the file (anti-clockwise) to expose its coronal part and loosen it off
121
Q

How does the tube system work?

A

Straight line access is required for this system to work

  1. Trephine with ultrasonic
  2. Position the microtube
  3. Engage and remove
122
Q
A

broken tool remover uses lasso mechanism to get file out

123
Q

What if the instrument cannot be removed?

A
  1. More favourable prognosis:
    - If pulp vital and not infected
    - If instrument fractured during advanced stages of preparation
124
Q

Prognosis of tooth depends on? Depends on the presence of preoperative periradicular periodontitis (grossman 1968, crump 1972, fox 1972, molyvdas 2001)

A
125
Q

If you have fractured a file, what must you do for patient management?

A

Legally the patient has to be informed about the complication, as if found later by the patient, they have grounds to sue for negligence.

126
Q

What information must you give when you have fractured a file in a patient?

A
  1. Possible consequences
    - A broken instrument is not always direct cause of treatment failure
  2. Influence on success rates
  3. Complications which might occur
  4. Further treatment
127
Q

What is a ledge?

A

An iatrogenically created irregularity (platform = very firm stop) in the canal, that impedes access of the instruments to the apex.

128
Q

Where is the most common place for ledges to form?

A

Common on outer side of curved canals.

129
Q

What are the causes of ledge formation?

A
  1. Inadequate access cavity
  2. Incorrect assessment of canal curvature
  3. Failure to pre-bend ss files
  4. Using larger, stiffer ss instruments
  5. Failure to use instruments in a sequential manner
  6. Cutting on inward rather than outward stroke
  7. By-passing a fractured instrument
  8. Negotiation of calcified canal
130
Q

What are the management of ledges?

A
  1. Establish the depth at which the ledge is present
  2. Coronal flaring up to f2-f3 working 1-2 mm shorter, use a small irrigation needle (gauge 30-purple) to irrigate between files + recapitulation
  3. PUI with chelator and NaOCL
  4. Probe with a pre-bent 08 ss file (coated in a chelator paste).
  5. Use the shortest file available which the level of the ledge in watch-winding and a gentle picking motion.
  6. If possible to bypass do not remove the file but use short vertical strokes with a gentle anti-curvature pressure to change the shape of the ledge.
  7. Keep the tip of the file apical to the ledge at all times.
  8. Repeat with size 10, 15 and 20 files until ledge removed
  9. Use pre-bent hand ProTaper files to complete preparation
131
Q

If it is not possible to by-pass the ledge, what do you do?

A
  1. If the ledge cannot be by-passed AND the patient has no symptoms prepare the ledge, copious irrigation, dress with non-setting Ca(OH)2, then obturate using a thermoplastic technique
  2. Inform patient of guarded prognosis and review clinical symptoms and bony healing.
132
Q

How do you prevent ledges?

A
  1. Creating a reproducible glide path
  2. Pre-flaring may be necessary (up to s2) before the full working length can be used.
  3. Copious irrigation using gauge 30 needle + recapitulation is essential
133
Q

What is a canal blockage?

A

It is a blockage of the canal in a previously patent canal that prevents access and complete disinfection of the most apical part of the root canal

134
Q

The blocked canal may contain?

A
  1. Compacted dentinal mud, most likely infected; and/or
  2. Residual pulp tissue; and/or
  3. Remnants of filling materials
135
Q

What are the causes of blocked canals?

A
  1. Apical patency is not confirmed and secured when WL is measured with EAL
  2. During instrumentation pulpal tissue is packed and solidified in the apical constriction by the use of instruments
  3. Instrumentation is not accompanied by copious irrigation and recapitulation
  4. Instruments are not cleaned before their insertion in the canal
136
Q

How do you know the difference between canal blockage and ledge?

A

When a canal is blocked, there is a characteristic tactile sensation when a very small file is reaching an almost solid but penetrable ‘wall’. When a ledge is present, the instrument hits a completely solid ‘wall’

137
Q

What determines the prognosis of a tooth with a blockage?

A
  1. If a blockage is recognised and corrected, there is no effect of prognosis
  2. When it cant be corrected, it may have negative effect of Tx outcome, in particularly in infected cases.
    - If a pre-existing AP is present or developed after Tx is completed, P/R surgery may be considered
    - In cases with no symptoms or AP – regular monitoring is indicated
138
Q

How do prevent blockages in narrow and curved canals?

A

Coronal pre-flaring

139
Q

What is canal transformation?

A

Removal of canal wall structure on the outside curve in the apical half of the canal due to the tendency of ss files to restore themselves to their original linear shape during canal preparation may lead to ledge formation and possible perforation by AAE

140
Q

What are the consequences of canal transformation?

A
  1. Damage to the apical constriction – risk of debris extrusion
  2. Zip formation (elliptical shape at the apical endpoint) – negative impact on the apical seal
  3. Elbow formation (narrow point at the maximum curvature) - insufficient taper of preparation
  4. Perforation – on the apical 1/3
  5. Strip perforation – along the inner side of the curvature in the mid and coronal 1/3
  6. Ledging
141
Q

The European society of endodontology’s quality guidelines suggest that when we carry out root canal treatment, we are obliged to do a clinical and radiographic follow-up at least:

A
  • 1 year after treatment
  • Further follow-up for up to 4 years – if bony healing is not complete according to Strinberg Acta Odontol Scand 1956: 14: 1-175
142
Q

When is root canal re-treatment indicated:

A
  1. Persistent periapical pathology following root canal treatment
    No radiographic signs of bony healing after 4 years
  2. New periapical pathology associated with a root filled tooth.
    Initial healing but a new radiolucency develops some time later.
    Root canal system has become infected after previous treatment.
  3. A new restoration is planned for a tooth and radiographic assessment shows an inadequate root canal filling and/or a periapical radiolucency.
143
Q

The Toronto study is a publication on the treatment outcome in endodontics shows the following for initial treatment.
Primary treatment
- Without periradicular periodontitis = 92%
- With periradicular periodontitis = 74%
- Overall = 81%

A

The Toronto study then later went to do a publication: treatment outcome in endodontics: the Toronto study—phases 3 and 4: orthograde retreatment.
Retreatment
- Without periradicular periodontitis = 89-100%
- With apical periodontitis = 56-84%

144
Q

A study done by Ng et al. 2011: a prospective study of the factors affecting outcomes of nonsurgical root canal treatment: part 1: periapical health.
The conclusion from this study was that success was based on periapical health
- Primary RCT 83%
- Secondary RCT 80%

A
145
Q

A systematic review done by Ng: outcome of secondary root canal treatment: a systematic review of the literature.
17 studies were included ranging from 1961-2005, the overall success rate was found to be 77%

The studies had three prognostic factors; these three factors all determine the success rate of secondary root canal treatment.
- Pre-operative periapical lesions
- Apical extent of root canal filling
- Quality of coronal restoration

A
146
Q

The terms to describe the outcome of RCT is no longer success or failure, they are the following.

Healed
- Clinically – no signs/symptoms
- Radiological – no residual radiolucency, or scarring after surgery

Healing
- Clinical – no signs or symptoms
- Radiological – reduced radiolucency in follow-up < 4 years

Asymptomatic function
- Clinical – no signs or symptoms combined with no or persistent radiolucency, reduced in size or unchanged.

Persistent/recurrent/emerged disease
- Clinical – with or without symptoms
- Radiological – new, increased, unchanged or reduced after >4 years

A
147
Q

Prevention of post-treatment disease

Guidelines on the quality of root canal treatment are?

A
  1. Rubber dam isolation
  2. Proximity of preparation to apical constriction
  3. Sufficient taper of preparation – allowing for adequate irrigation and disinfection of the root canal. This will also allow enough space for interappointment medicament if required (non-setting calcium hydroxide)
  4. Correct extension of root canal obturation without extrusion
  5. Adequate coronal seal to prevent re-infection
148
Q

Indications for root canal retreatment

A
  • Previous treatment has failed
    Signs of inflammation or infection
  • Persistent symptoms, sinus tract (chronic abscess), swelling, pain.
  • Failure of previous treatment because of technical reasons
  • Existing pathology and new restoration planned for tooth.
149
Q

A = radiograph of mandibular with inadequate root filling and asymptomatic apical periodontitis.
B = the tooth has been retreated conservatively through the crown.
C = complete periapical repair is evident at the 6-month control.

A
150
Q

This image shows 4 potential areas where microbes can be post treatment.

A
  1. Intraradicular microbes: if the preparation of the canal is not at its correct length, then microbes will persist at the constriction and spread.
  2. Extraradicular microbes: this is when microbes have invaded the host response system and established themselves in the periapical tissues.
  3. Foreign body reaction to extruded gutta- percha.
  4. True cyst which is cavity that has walled itself off from the root canal system.
151
Q

Causes of post-treatment disease

Microbial causes

A
  • Intraradicular microbes: Intraradicular infections, either persistent or secondary are the major causes of endodontic treatment failure.

Persistent = where the microbes were not removed during the initial treatment

Secondary = where the microbes have entered the root canal system via coronal leakage

  • Extraradicular microbes
  • Radicular cyst
  • Cracked teeth, vertical root facture = allowing microbes to enter the root canal system
  • Coronal leakage
152
Q

Causes of post-treatment disease

Non-microbial causes

A
  • Cholesterol crystals in the periapical tissues
  • Foreign body reactions in the periapical tissues
153
Q

Intraocular infections in root canal treated teeth can be classified in two groups

A
  1. Persisting infection: inadequate isolation/disinfection during treatment
  2. New secondary infection through leakage
154
Q

Radiographically you can see if a poor root canal filling has been placed, and often associated with periapical radiolucency, it is worth to note that radiographs do not indicate the biological status of the root canal.

A

Persistent bacteria
- Those that remain in the root canal system after root canal disinfection and interappointment dressing gram positive bacteria appear to be more resistant to antimicrobial treatment and can adapt to harsh environmental conditions in instrumented and medicated root canals.

However, they do not always maintain an infectious process

  • Residual bacteria may die after obturation
  • Residual bacteria may be present in insufficient numbers and virulence
  • They may be located in areas where they have no access to periapical tissues

In canals that are apparently well treated tend to have – 1-5 species

In canals with inadequate treatment tend to have 10-20 species like untreated canals.

In retreatment cases nine times more likely to harbour Enterococcus Faecalis, candida in 18%

155
Q

Common microbes associated with retreatment cases:

A
  1. Enterococcus faecalis
  2. Streptococcus
  3. Lactobacillus
  4. Actinomyces
  5. Propionibacterium
  6. Candida albicans
156
Q

The possible origins of these microbes:

A
  1. Contamination during initial treatment
  2. Leaving a tooth on open drainage
  3. Coronal leakage post-treatment
157
Q

In most cases apical periodontitis inflammatory lesions succeed in preventing microorganisms. The inflammatory response would be the bodies response to supress this infection from reaching the periapical tissues.

Occasionally the microorganisms can overcome the inflammatory defence barrier and establish an Extraradicular infection e.g., acute apical abscess.

Bacterial colonies can also form biofilms on the external root surface and bacterial colonies can exist inside periapical granulomas.

A
158
Q

What is a radicular cyst?

A

Is the most common odontogenic cyst of inflammatory origin, arising from epithelial cell rests in periodontal ligament.

159
Q

A radicular cyst can develop into a true cyst or a pocket cyst?

A

True cyst: lesion enclosed by epithelial lining.
Pocket cyst: epithelial sac communicates with root canal system.

  • A pocket cyst will normally heal following endodontic treatment whereas a true cyst wont.
160
Q

Endodontic failure caused by inadequate restorative procedures: review and treatment recommendations, Healing et al. J prosthet dent; 87: 674-8 – reviewed literature 1969-99 41 papers concluding:

Prognosis can be improved by sealing the canal and minimizing leakage of oral fluids and bacteria – do it as soon after root canal treatment as possible.

A
161
Q

Cholesterol crystals are from dying cells during chronic inflammation

Foreign body reaction include:

  1. Gutta-percha
  2. Sealers
  3. Paper points
  4. Cotton pellets – contain cellulose and the body has no way of breaking it.
A
162
Q

A patient coming in for retreatment for root canal, a clinical assessment has to be made, this includes the following.

A
  1. Patient history
  2. Examination
  3. Special investigations as required
  4. Radiographic examination
  5. CBCT
  6. Diagnosis
  7. Treatment plan
    Do nothing
    Extract tooth
    Retreatment
    Surgical retreatment
163
Q

When we look at a radiograph and CBCT for evaluation the following is looked for:

A
  1. Caries
  2. Defective restorations
  3. Periodontal health
  4. Quality of obturation
  5. Existence of missed canals or procedural errors
  6. Periapical pathology
  7. Perforations
  8. Fractures
  9. Resorptions
  10. Canal anatomy
164
Q

Planning retreatment

  1. Is it a strategic tooth? If it is a lone standing molar, then maybe it’s not worth the time and cost to retreat.
  2. How much remaining coronal tooth structure is remaining? So that tooth can be restored once the complex procedure is complete.
  3. What is periodontal support?
  4. Is periapical disease present?
  5. Are there aesthetics issues
  6. Are there technical difficulties as shown radiographically?
A
165
Q

Techniques to retreating root canals
1. Gain access to root canal system
Crown removal
Post removal

  1. Removal of root canal filling material
    Gutta percha
    Carrier based systems
    Silver points
    Pastes
A
166
Q

Treatment with direct restorations

  1. Remove any existing caries
  2. Reduce any unsupported cusps
  3. Ensure sufficient remaining tooth structure to place definitive restoration
  4. Ensure tooth can be isolated under rubber dam
    Orthodontic band/GI
    Electrocautery, crown lengthening procedures
A
167
Q

Teeth with indirect restorations, assess quality of restoration
1. Crowns, bridges, inlays, onlays, post cores
2. Integrity of coronal seal – no recurrent caries or marginal deficiencies

A
168
Q

Access through existing crowns

  1. A higher risk of perforation
    Visibility reduced
    Tooth alignment may be altered by the crown
    - Important to carefully assess the pre-treatment radiograph, if any potential difficulties remove the crown.
A
169
Q

Removing crowns

  1. When the crown is defective/caries
  2. Allows assessment of remaining tooth structure
  3. Visibility and access to root canals improved
  4. Avoids risk of perforation when searching for canals – especially if tooth alignment has been altered by the crown
A
170
Q

Removing a crown by sectioning

  1. Take a sectional impression first in putty
  2. Section the crown into two halves taking care not to cut through the core, then remove with an excavator
  3. Use a diamond bur for porcelain and tungsten carbide for metal
A
171
Q

Wamkey - a technique used to remove a crown.
Crown tappers
Kavo coronaflex

A
172
Q

Wamkey - a technique used to remove a crown.
Crown tappers
Kavo coronaflex

A
173
Q

Post removal

  • Technique should minimize risk of root fracture: always important to advise patient that root fracture may be a complication of attempted post removal
  • Depends on type of post and core and cement: parallel/tapered. Active/passive, metal/quartz fibre
  • Always assess post type and length/width on a pre-treatment radiograph

2 main techniques:
- Post removal with ultrasonic energy
- Post removal with post pulling devices

Removing cast post core
1. Usually involves removal of coronal restoration
2. May require cut back of core using tungsten carbide bur prior to using ultrasonics
3. If post is extremely well fitted removal can be very difficult especially in oval shaped canals.

A
174
Q

Techniques used to remove gutta percha

  • Rotary endodontic files
    Protaper D files for retreatment – used at 600 rpm 4Ncm
    PTG endo instruments
  • Ultrasonics
  • Heat
  • Solvents
A
175
Q

Removal of GP using PTG

  1. Measure estimated WL for radiograph
  2. Depending on canal diameter select either an F2 or F3 finishing file, length 21mm
  3. Set rpm to 600
  4. Use in coronal 2/3s of canal – work from F3-F2-F1 if required
  5. Then go down 1mm increments checking with EAL until WL and patency have been achieved
  6. If apical part of the canal is underprepared/not obturated, negotiate with size 10 file, establish WL and complete preparation using normal PTG sequence at 300 rpm
A