Endocrine Flashcards
Hyperthyroidism increases the risk for atrial fibrillation which additionally increases the risk for ___
thromboembolic stroke
Risk of AF is increased up to (3)_________ in patients with(4) __________ hyperthyroid (low TSH with normal free T4)
Risk of AF is increased up to (3) 3x in patients with(4) subclinical hyperthyroidism hyperthyroid (low TSH with normal free T4).
Two possible places to admit a new onset afib (hyperthyroid) pt.
acute telemetry or ICU
four tx considerations for someone in afib with hyper thyroid - rate v. rhythm
(1) rate control - diltiazem/verapamil (CCB), digoxin, propanolol (BB)
(2) rhythm control - cardioversion (amiodarone or shock or spontaneous) [*amiodarone can cause hyperthyroid]
(3) antiarrhythmics
(4) anticoag (hep)
Before __hrs is it ok to sitll do cardioversion?
onset less than 48 hours ago
Measure TSI - thyrotropin receptor Ab in what autoimmune thyroid disorder?
grave’s
Measure TPOab in what autoimmune thyroid disorder?
Hashimoto
chapman point for?
bilateral 2nd intercostal spaces just lateral to sternum
myocardium
SNS for what? T1-5
H/N/cardiac (increased warmth, mm tension, moisture)
PSNS for what? vagus
OA F RRSL, AA RL
Define: rapid and deep respirations
kussmaul respirations
increased hunger
polyphagia
AG metabolic acidosis
M Methanol U Uremia D Diabetic Ketoacidosis P Paraldehyde I Infection L Lactic Acidosis E Ethylene Glycol S Salicylates
Admit DKA where?
**ICU, always!
Most important treatment for DKA.
**IV Fluids and correct the anion gap.
Why do you want to include potassium in electrolyte replacement in DKA, even it pt’s K is initially elevated?
Insulin/IVF [correction of
pH] will drive K into cells and they usually become hypokalemic)
What is the equation used for?
Na + [(glucose -100) x 0.016]
To correct sodium when glucose is high
Initally, IV fluid tx for DKA is ____. Then it is swtiched to _____. Why?
Initially normal saline, switch to D5 1/2 NS when following protocol with insulin gtt when their
glucose gets to 250 prevent hypoglycemia (can cause hypoosmolarity, herniation, death).
Goal of DKA tx
fix acid base disturbance and NOT until sugar level is normal
why do you not want to tx DKA until sugar is normal?
bc they will go right back to DKA if you stop too soon - better to keep glucose slightly high until anion gap is corrected
Notable findings in a pt with… diffuse abdominal pain, fruity breath, unintentional weight loss, ketonuria, hyperglycemia.
DKA
When can you end DKA tx protocol?
when anion gab closes - Switch to subQ insulin, stop gtt 2 hours after administration of SQ long acting (they will go right back into DKA if you stop too soon. )
What is this the chapman point of?
R 7th intercostal space tissue texture changes
Pancreas
What is this the SNS of?
T5-9 (increased warmth, tension, and moisture)
Pancreas
What is this the PSNS for?
OA E RLSR, AA RR, restricted thoracic inlet
Pancreas
if pt has HF, cannot give what for AFib?
CCBs - verapamil, diltiazem