Endo third year Flashcards
Schilder’s design objectives
continuously tapering funnel shape
maintain apical foramen in original position
keep apical opening as small as possible
why is it important to keep the apical opening as small as possible?
wound healing
less likelihood of trauma
apical control
why is cuspal protection important?
prevent microbial ingress
prevent catastrophic fracture
what does the pre-tx radiograph need to include?
all root
2-3mm of surrounding PR tissue
why should dam be used?
bacterial contamination inhalation protects Sts access and vision can use disinfectants
sizes of SS instruments
21/25/31mm
taper on SS instruments
2%, 0.32mm
cutting flutes on SS instruments
16mm
diameter at D2 = apical size + 0.32mm
ISO colour code - 06
pink
ISO colour code - 08
grey
ISO colour code - 10
purple
ISO colour code - 15
white
ISO colour code - 20
yellow
ISO colour code - 25
red
ISO colour code - 30
blue
ISO colour code - 35
green
ISO colour code - 40
black
ISO colour code - 45
white
ISO colour code - 50
yellow
ISO colour code - 55
red
ISO colour code - 60
blue
ISO colour code - 70
green
ISO colour code - 80
black
ISO colour code - pink
06
ISO colour code - grey
08
ISO colour code - purple
10
ISO colour code - white
15
45
ISO colour code - yellow
20
50
ISO colour code - red
25
55
ISO colour code - blue
30
60
ISO colour code - green
35
70
ISO colour code - black
40
80
objectives of irrigants
disinfect RC dissolve organic debris flush out debris lubricate instruments remove smear layer
what is used to deliver irrigant to RC?
Luer lock syringe with 27 gauge needle
what is recapitulation?
after each file irrigate and use file smaller than MAF
what are the aims of recapitulation?
disturbs debris and lifts into solution
prevents blockages
where should RC preparation end?
at the jct of pulpal and PA tissue - as close as possible to CDJ - usually apical constriction
EWL
estimated length at which instrumentation should be limited
calculating EWL
measure pre-op radiograph from FRP to radiographic apex and -1mm
how does the distance of the apical constriction from the radiographic apex vary?
greater in older teeth with secondary cementum
varying anatomy
RR
can give a false reading of where RC terminates
CWL
length at which instrumentation and subsequent obturation should be limited
when is CWL determined?
after coronal flaring
methods of determining CWL
EAL
WL radiograph
PP length discrimination
EAL
impedence/resistance drops when you touch PDL
unreliable if wide apical foramen
subtract 0.5-1mm
PP length discrimination
wet dry interface
PR tissues wet, RC should be dry
MAF
largest diameter file taken to CWL and therefore represents the final prepared size of the apical portion of the canal at the WL
reasons for early flaring of coronal portion
reservoir for irrigant
avoids hydrostatic pressure in canal
early removal of heavily contaminated contents
improved SL access to apical 1/3
modified double flare technique
uses BF
1 - enlarge/flare coronal part
2 - apical enlargement
3 - apical taper - step back
apical size
small as practicable but large enough to irrigate
ISO 25 or above
mid root prep
step back
increase file size as -1mm each time
until file “falls out”
brush MAF around wall to get rid of steps
what is patency filing?
ISO 10 or smaller 0.5-1mm through apical constriction
passive placement
purpose of patency filing
prevent apical blockage
risk of patency filing
risk of extrusion of infected debris into PA tissues
resin sealers
2 pastes - AH plus
8hr set
good seal and flow
initial toxicity decreases after 24hours
GP components
20% GP
65% ZnO (filler)
10% radiopacifiers
5% plasticisers
is the outcome affected if GP goes through apex?
yes
CLC advantages
good length control
gold standard
removable filling
CLC disadvantages
does not allow good adaptation to canal irregularities
doesn’t produce homogeneous mass of GP
finger spreaders
tapered, smooth-sided
lateral pressure
checking for tug back with master cone
should have slight resistance when tug back
corresponds to size of MAF - good apical seal
apical portion must remain undistorted when at length
altering fit of master cone
trim apically with scalpel
try another
confirm prep
accessory cones
greater taper
corresponding FSs
all interfaces filled with sealer
excess GP removal
heated instruments to sever at ACJ/level of attachment
plug GP to compact
remove excess sealer
RMGI primary seal
upper incisors access cavity
triangular palatal
upper incisors RCs
1 canal
upper canines access cavity
oval palatal
upper canines RCs
1 canal
upper 1st premolar access
oblong
upper 1st premolar RCs
1 - 6%
2 - 93%
3 - 1%
upper second premolar access
oval
upper second premolar RCs
1 - 75%
2 - 24%
3 - 1%
upper 1st molar access
rhomboid
upper 1st molar RCs
4 - 93% (MB2)
3 - 7%
upper 2nd molar access
triangle
upper 2nd molar RCs
4 - 37%
3 - 63%
lower incisors access
palatal similar shape to crown
lower incisors RCs
1 - 59%
2 - 41%
lower canine RCs
1 - 86%
2 - 14%
lower premolars access
oval
Lower first premolar RCs
1 - 73%
2 - 27%
lower second premolar RCs
1 - 85%
2 - 15%
lower first molar access
oval/square
lower first molar RCs
3 - 67%
4 - 33%
lower second molar RCs
2 - 13%
3 - 79%
4 - 8%
radix entomolaris
additional root in mandibular molars - DL
radix paramolaris
additional root in mandibular molars - DB
accessing posterior teeth
not vertically due to bulbosity of crown
need distal inclination
Anatomy of the pulp chamber floor - laws
laws of symmetry law of colour change laws of orifice location law of centrality law of concentricity law of the CEJ
laws of symmetry
1 - except maxillary molars, orifices of canals are equidistant from a line MD direction through floor
2 - except maxillary molars, orifices of canals lie on line perpendicular to a line drawn in a MD direction across centre of floor
law of colour change
colour of floor always darker than walls
laws of orifice location
always at jct of walls and floor
always at angles in floor wall jct
at terminus of root developmental fusion lines
law of centrality
floor always at centre of tooth at level of CEJ
law of concentricity
walls of pulp chamber are always concentric to the external surface of the tooth at the level of the CEJ
law of the CEJ
the CEJ is the most consistent repeatable landmark for locating the position of the pulp chamber
what is the most reliable landmark for locating the pulp chamber floor?
ACJ - pulp chamber floor central and concentric to shape of tooth at ACJ
what may you find when accessing the pulp chamber?
healthy pulp necrotic pulp empty pus GP
straight line access
instrument relatively passive in canal until 1st curvature
protects canal and instrument
what to put in table in notes
canal EWL ref point CWL MAF
pulpal physiology
hydrodynamic theory (AB, Ad, C) generation of movement of tubular fluid leading to activation of the nerve fibres
pulpal physiology - AB and Ad fibres
short sharp pain
pulpal physiology - C-fibres
long dull throbbing pain
which MOs predominate in necrotic untreated cases?
gram - anaerobes
which MOs predominate in failed and persisting infection?
mostly gram + anaerobes
biofilm
protein matrix with bacterial cells embedded
SOCRATES
Site Onset Character Radiation Association Time course Exacerbating/Relieving factors Severity
clinical endo notes for a tooth
buccal soft tissue palatal/lingual mucosa colour palpation restoration TTP sinus mobility EPT ethyl chloride radiograph diagnosis
what do CN5 branches mostly transmit pain in response to?
thermal, mechanical or chemical stimuli
referred pain
perception of pain in one part of body distant from source of pain
difficult to discriminate location of pulpal pain
provoked by intense stimulation of C-fibres - intense, slow, dull pain
radiates to ipsilateral side
rare anteriors
posteriors (esp mandibular) - to opp arch or periauricular area but rarely to anteriors
what does vitality testing mean?
if it has an intact blood supply
sensibility tests
thermal
electric
problems with sensibility tests
subjective
testing nerve not blood
problems with multi-rooted teeth
other sensibility tests
laser doppler flowmetry pulse oximetry bite test test cavity staining and transillumination selective anaesthesia
pulpal diagnoses
normal pulp reversible pulpitis irreversible pulpitis - asymptomatic - symptomatic pulp necrosis prev. treated prev. initiated
normal pulp
symptom free
normally responsive to pulp testing
reversible pulpitis
sharp transient, only lasts a few secs
reactive to stimulus - not spontaneous pain
not TTP
no significant radiographic changes
management of reversible pulpitis
manage aetiology
what is the nature of the pulp in irreversible pulpitis?
vital and inflamed
asymptomatic irreversible pulpitis
usually normal response to thermal testing
no clinical symptoms
management of asymptomatic irreversible pulpitis
RCT
symptomatic irreversible pulpitis
lingering, spontaneous, referred pain
sharp pain on thermal stimulus
OTC analgesics typically ineffective
if inflammation hasn’t reached PA tissues - not TTP
management of symptomatic irreversible pulpitis
RCT
pulp necrosis
non-responsive to pulp testing, asymptomatic
only causes apical periodontitis if canal infected
usually no obvious radiographic changes
other reasons than necrosis for non-responsive to pulp testing
calcification
recent trauma
just not responding
apical diagnoses
normal apical tissues symptomatic apical periodontitis asymptomatic apical periodontitis chronic apical abscess acute apical abscess condensing osteitis
normal apical tissues
not sensitive to percussion/palpation
radiographically LD intact and PDL space uniform
symptomatic apical periodontitis
pain on biting/percussion/palpation
may have radiographic changes - PA radiolucency, widened PDL, thinning LD
severe pain to P/P - degenerating pulp - RCT
apical periodontitis
inflammation and destruction of apical periodontium of pulpal origin
asymptomatic apical periodontitis
no clinical symptoms
apical radiolucency
chronic apical abscess
inflammatory reaction to pulpal infection and necrosis
gradual onset
little/no pain
associated sinus tract - intermittent pus discharge
radiographically signs of osseous destruction e.g. radiolucency
acute apical abscess
inflammatory reaction to pulpal infection and necrosis rapid onset, spontaneous pain extreme tenderness to pressure pus swelling may be no radiographic signs of destruction may be mobile often malaise, fever, lymphadenopathy
condensing osteitis
diffuse radiopaque lesion representing a localised bony reaction to a low grade inflammatory stimulus
usually seen at apex
usually symptom free
tx options
RCT re-RCT extract monitor surgery
purpose of obturation
prevent bacteria that are left from accessing any nutrients
endo-restorative interface - purpose of endo
provide env that allows healing of PR tissues so the tooth is retained as a functional unit in the dental arch
aims of instrumentation
remove infected hard and soft tissue
give disinfecting irritants access to apical canal space
create space for medicaments and obturation
retain integrity of radicular structures
irrigant ideal properties
washing action
lubrication
improve cutting of dentine by the instruments
temp control
dissolution of organic and inorganic matter
good penetration within RC system
killing of planktonic and biofilm microbes
detachment of biofilm
non-toxic to PA tissues
non-allergenic
doesn’t negatively react with other dental materials
does not weaken dentine
NaOCl chemistry
ionises in water into Na+ and OCl- establishes equilibrium with HOCl acid/neutral HOCl predominates pH9 and above OCl- predominates HOCl - antibacterial activity
why NaOCl?
potent antimicrobial activity
dissolves pulp remnants and collagen
only RC irrigant that dissolves necrotic and vital tissue
helps disrupt smear layer by acting on organic component
what is the least effective method of irrigation?
syringe irrigation alone
what is the ideal irrigation technique?
Manual Dynamic Irrigation - GP point - increase efficacy of NaOCl
can also use Endoactivator - mechanical agitation
factors important for NaOCl function
conc 3% vol contact - 10mins mechanical agitation exchange
problems with NaOCl
possible effects on dentine properties
inability to remove smear layer by itself
effect on organic material
when is the smear layer formed?
during prep
smear layer
organic pulpal material and inorganic dentinal debris
superficial 1-5um with packing into tubules
problems with the smear layer
bacterial contamination
substrate
interferes with disinfection
prevents sealer penetration
removal of the smear layer options
17% EDTA
10% citric acid
MTAD
S and US irrigation
EDTA concentration
17%
EDTA function
removes smear layer
what type of agent is EDTA?
chelating agent
EDTA contact time
1min
why shouldn’t EDTA and NaOCl interact?
get ppts which block tubules
irrigant protocol
irrigate/dry with PP between
NaOCl 10mins
EDTA 1min
NaOCl final rinse
NaOCl complications
fabric discolouration
ophthalmic injuries
apical extrusion - tissue necrosis
allergic reactions
common symptoms of NaOCl extrusion
pain swelling ecchymosis - along course of superficial venous vasculature haemorrhage neurological complications airway obstruction
extrusion risk factors
excessive pressure - use index finger not thumb
needle locked within canal
loss of control of WL
larger apical diameters/constriction - RR, immature teeth, developmental anomalies
anatomical factors/proximity to sinus
higher NaOCl conc?
EndoVac
negative pressure system to pull irrigant down canal
management of NaOCl extrusion
LA for pain relief irrigate with saline relax pt nsCaOH cold and warm compresses, analgesics, review in 24hrs, antibiotics?
prognosis of tooth not - affected
advantages of CHX digluconate
antibacterial activity
dentine medicated with CHX acquires antimicrobial substantivity - prevents colonisation for time beyond application
acceptable biocompatibility
how does CHX digluconate have antibacterial activity?
+CHX attracted to -phospholipids in cell wall
binds to cell wall - lysis
disadvantages of CHX digluconate
less antifungal activity
unable to disrupt biofilms
sensitivity possible, risk of anaphylactic reaction
NaOCl and CHX interaction
forms parachloroaniline
cytotoxic and carcinogenic
uncertain bioavailability
brown ppt - discolours tooth
what cases are often done in a single visit?
vital cases
single vs multi visit
case by case
which cases may be done over multi visits?
non-vital cases more complex with greater resistance to endo tx - inter-appt dressing may be important
may not always want to fill teeth on first visit e.g. if huge abscess filled with pus
purpose of intracanal medicaments between appts
destroy MOs
prevent reinfection
decrease inflammation and exudate
control of RR
antimicrobial paste
paste containing corticosteroid and tetracycline
good for hot pulps = when can’t get numb due to extent of inflammation
not gold standard otherwise
can facilitate follow-up treatment
effective for 5-7 days e.g. odontopaste
how long should nsCaOH be left?
7days
properties of nsCaOH
bactericidal and bacteriostatic, antimicrobial
hydrolysis of LPS therefore reducing its inflammatory potential
effective in removing tissue debris
high pH - stimulates fibroblasts for reparative dentine formation
adheres directly to dentine
doesn’t dissolve in biological liquids
neutralises low pH from acidic restorative materials
inter-appt temp dressing
CaOH
cotton wool
Coltosol
3mm GI
file motions
filing reaming WW balanced force envelope of motion
filing motion
up and down
reaming
repeated clockwise rotation
if bind too far breaks
WW
back and forward oscillation 30-60 degrees with light apical pressure
balanced force technique
LAP clockwise anticlockwise a bigger degree of rotation x1-3 then remove safer for canal and file
envelope of motion
evenly strip
what is a glide path?
path along which tip of subsequent instrument will pass, protects that tip
“smooth radicular tunnel from canal orifice to apical constriction”
importance of a glide path
confirm SL access
explore anatomy
creating a glide path
always introduce files 10-25 to resistance only
coronal flare
size 10 with WW establish apex
irrigate and repeat using 15 (WW) and 20 (BF)
what is apical gauging?
technique to best determine the size of the apical constriction and the taper of the apical portion closest to the foramen
how to determine apical gauging
2 sizes bigger than one that originally bound at length
protaper hand use properties
superelasticity
variable taper
protaper hand use properties - superelasticity
NiTi alloy - v flexible
shape memory effect after heat tx
lowers risk of ledges and perforation - doesn’t apply much force at its tip
protaper hand use properties - variable taper
more specific shape
larger taper apically so more control
not whole way up
S shaping files - where are they used?
coronal and mid root
S shaping files
S1
S2
SX
S shaping files - S1
purple
17
coronal flare
2/3 BF technique
S shaping files - S2
white
20
mid-root shaping
S shaping files - SX
red
19
only for short canals
F finishing files - F1
yellow
20
F finishing files - F2
red
25
F finishing files - F3
blue
30
F finishing files - F4
black
40
F finishing files - F5
yellow
50
protaper colours mnemonic
Please Will You Read Books By Yourself
summary of Protaper process
10 - to 2/3 EWL 15 - to 2/3 EWL S1 - no deeper than 15 (SX) 10 - EWL/0 with EAL
find CWL
15 - glide path to CWL S1 S2 F1 F2 ... K25 to length
barbed broach
extirpating only
do not engage dentine
tapered round shaft with portions lifted almost at a right angle
extremely fragile
Hedstrom (H) files
use in filing motion - cuts on withdrawal
can cause iatrogenic damage - no longer used for canal prep
useful for removing GP or fractured instruments for re-tx
K reamers
made by twisting a tapered triangular shaft
must be in contact with walls, but must not bind or may break
C+ files
similar to K-files but has a cutting tip and slightly more rigid
K files
flexible so useful in curved canals
SS
cut when used in rotation
twisting a square shaft
flexible K files (flexofiles)
cross-sectional shape allows greater flexibility
SS/NiTi
filing/rotation motion
complications of SS hand instrumentation
mishaps - ledges, canal blockage, zipping of apical foramen
debris extrusion with filing motion
blockage
dentine debris packed into apical portion of root
when packed tightly can be as hard as dentine
attempts to remove it can lead to false canal and perforation
irrigate
ledges
an internal transportation of the canal
when do ledges occur?
when working short of length
management of a ledge
hard to bypass - need to curve tip of a small file
what happens if curved canals are instrumented as if they were straight?
get ledging and the apical few mm will remain uninstrumented and infected
apical zipping/transportation
occurs due to the tendency of the instrument to straighten inside a curved canal
management of minor apical zipping/transportation
canal can be reshaped to a new level just above the foramen
severe apical zipping/transportation issues
bleeding is a problem and attempting to reshape can weaken/perforate root
consequences of apical zipping/transportation
over enlargement along outer side of curvature
under prep of inner aspect at apical end point
main axis of canal is transported
results in a teardrop/hourglass shape
cases tend to be over-extended and poorly filled = fails to provide resistance for packing of GP
how to avoid apical transportation
always pre-curve initial small sized hand instruments
don’t skip instruments in sequence
never rotate the instruments in curved canals
diagnosis of root perforation
persisting bleeding into canal
multiple radiographs - can do with a file in to see if perforation
EAL
dental operating microscope
what does prognosis of perforation depend on?
location time elapsed size PD irritation material used for repair
MTA for perforation
only works well for small - not strong in CS
dentine fibres and cementum will bind to MTA
importance of superelasticity
can be strained more than others before permanent deformation
can place in curved canals with less lateral forces exerted
- less transportation, zipping and ledging
- more centrally placed prep in harmony with original canal shape
components of an endo rotary instrument
taper flute leading/cutting edge land relief helix angle
taper
diameter change along working surface
flute
groove to collect dentine and ST
leading/cutting edge
forms and deflects dentine chips
land
surface extending between flutes
relief
reduction in surface of land
helix angle
angle cutting axis makes with LA of file
advantages of NiTi vs SS
increased flexibility in larger sizes and tapers
increased cutting efficiency
if used appropriately good safety in use
more user friendly
Protaper gold
NiTi wire plus gold tx
triangular in CS
active length 16mm
impacts the metallurgy in a favourable way to provide a higher flexibility and a higher resistance to cyclic fatigue
disadvantages of NiTi
instrument fracture £ access can be difficult in posteriors unsuitable for complex canal anatomy effect of prion decontamination protocols on NiTi rotary surfaces?
true reciprocation
mimics manual movement
lower risks associated with continuously rotating a file through canal curvatures
decreased cutting efficiency
requires increased inward pressure
limited capacity to auger debris out of a canal
rotary instrumentation guidelines
straight line access CS diameter (ISO 15 or more) RC system anatomy speed and sequencing lubrication and light touch
Rotary NiTi generations
1st - K file type helix 2nd - reamer type helix, Protaper 3rd - metallurgy, safer, Protaper Gold 4th - reciprocation movement, safer 5th - offset design
engine driven systems movement
vertical movement
reciprocation rotation 90/30 degrees
rotation 360 degrees
rotary endo - greater flexibility and taper
torsional stress
extensive instrument surface encounters excessive friction on canal walls
instrument tip is larger than canal section to be shaped, tip may lock, torque exceeds critical level
flexural stress
repeated cyclic metal fatigue
cannot be influenced by clinician
cyclic fatigue
freely rotating in a curvature
get tension/compression cycles
failure
torsional fatigue
instrument binds, if further rotated - stress in torsion, torque
structure of metal can undergo irreversible changes
each time causes torsional fatigue - eventually fracture
how to avoid torsional fatigue in reciprocation
clockwise and anticlockwise angles of rotation should be set lower than the elastic limit
the lower the angles of rotation the safer the procedure, as long as instrument can still cut dentine, advance apically in canal and remove cutting debris in coronal direction
in torsional fatigue what determines if the metal structure changes are reversible or irreversible?
amount of rotation when instrument is binding
what is between the elastic and plastic phases?
elastic limit
protaper size matched cones
complement file size and shape so leave v little space for accessory cones
check for tug back
spreader won’t go as deep as before
what is the outcome if good endo and good restoration?
91%
disadvantages of radiographs
2D
magnification/distortion
radiographic apex could be different from the actual terminus
evaluation of pt - case assessment
medical
psychological
social factors
case selection
pt evaluation
tooth evaluation
self-evaluation of clinician
case assessment - medical findings
no absolute contraindication to endo - if in doubt speak to physician pregnancy CV disease cancer diabetes mellitus bisphosphonate therapy allergies
pregnancy
1st trimester emergency intervention only - when foetus most at risk from env factors
balancing act - if don’t tx infection could make them systemically unwell
pain and infection managed in collaboration with physician
cancer
history
chemo and radio to head and neck can compromise healing
consult with oncologist
CV disease
contraindication - MI in last 6m
consult cardiologist re emergency tx
Stress Reduction Protocol - short appts, sedation, pain and anxiety control
diabetes
endo infection can affect their glycemic control
monitor carefully
schedule appts around insulin and meal schedule
minimise stress
may also impact on their outcome - RCT prognosis worse
bisphosphonates
IV>oral
preventive care - avoid extractions
non-surgical endo tx of teeth that might otherwise be extracted
allergies
latex allergy - use vinyl dam
GP not a risk as non-cross reactive
dental evaluation
PD considerations restorative considerations calcifications, dilacerations, resorption inability to isolate tooth unusual anatomy ledges and perforations posts separated instruments developmental abnormalities
endo perio lesion
do endo first then PD therapy later if necessary
calcifications
usually coronal to apical
isolated/continuous - can make tx vvv difficult
may consider surgery
restorative considerations
sub-osseous caries poor crown/root ratio misalignment pre-existing full coverage restorations restorability - remove all decay so extent of healthy tooth structure can be determined
effect of resorption on RCT
harder to control irrigants
when would you consider CBCT?
only if the additional information from 3D reconstructed images will potentially aid formulating a diagnosis/enhance the management of a tooth with an endo problem
how to decide whether to tx or refer
simple formula - e.g root number/chronic or acute
AAE endo case difficulty assessment form
Restorative dentistry IOTN - complexity assessment
options for tx
no active tx with review extract orthograde RCT surgical endo referral
factors that affect tx decision
pt assessment dental assessment pt motivation pt time cost
AAE endo case difficulty assessment form - categories
minimal
mod
high
AAE endo case difficulty assessment form - minimal
routine complexity
predictable tx outcome should be attainable by competent with limited experience
AAE endo case difficulty assessment form - mod
complicated
challenging for competent, experienced practitioner
AAE endo case difficulty assessment form - high
exceptionally complicated
challenging for even the most experienced practitioner with extensive history of favourable outcomes
AAE endo case difficulty assessment form - considerations
pt
diagnostic tx
additional
AAE endo case difficulty assessment form - pt considerations
MH anaesthesia pt disposition ability to open mouth gag reflex emergency condition
AAE endo case difficulty assessment form - diagnostic tx considerations
diagnosis radiographic difficulties position in arch isolation radiographic appearance of canals resorption crown, canal and root morphology
AAE endo case difficulty assessment form - additional considerations
trauma history
endo tx history
perio-endo condition
NHS IOTN complexity assessment
complexity 1/2/3
modifying factors that are relevant to RCT
MH that significantly affects clinical management
prognosis of orthograde RCT
predictable and usually successful
outcome rates up to 90% over 10 years for teeth with irreversible pulpitis
up to 80% over 10 years for necrotic teeth
many tx modalities yet v little difference in outcome
importance of filling the RC system
prevent passage of MOs and fluid
block apical foramina, dentinal tubules and accessory canals
chemomechanical disinfection can’t get to it all
properties of obturation materials
biocompatible dimensionally stable able to seal insoluble unaffected by tissue fluids non-supportive of bacterial growth radiopaque removable from canal if re-tx needed
disadvantages of CLC
voids
spreader tracts
incomplete fusion of GP cones
lack of surface adaptation
disadvantages of single cone obturation
quality
microleakage
bacterial penetration
similar/lower to others
warm vertical compaction
3D obturation
need continuously tapering funnel and small apical diameter
repeated heating and compaction of GP
yellow Buchanan hand plugger
25
red Buchanan hand plugger
40
blue Buchanan hand plugger
70
continuous wave obturation
downpack, apical pressure cool reactivate separate tip have apical plug - forced into lateral canals backfill uses heat
carrier based obturation
solid (flexible) plastic/cross-linked GP core, coated with molten GP
good for curved canal
but easy to extrude beyond apex
low void incidence
what is a potential disadvantage of thermal obturation techniques?
less apical control?
bio ceramic cements
may be good in some complex canals
root perforations
MTA
comparable filling quality to GP and sealer
how to measure sealing ability
dye penetration isotope penetration EC technique bacterial penetration salivary penetration
resilon
resin based system
dentine bonding technology
“monoblock”
much higher failure than GP
ideal sealer properties
tackiness - good adhesion hermetic seal radiopaque easily mixed no shrinkage on setting non-staining bacteriostatic slow set insoluble in tissue fluids biocompatible soluble on re-tx
ZOE based sealers
e.g. Tubliseal antimicrobial cytotoxic free eugenol - irritant lose vol with time due to dissolution - resins can modify
GI sealers
dentine bonding properties
minimal antimicrobial activity
soluble
difficult to remove upon re-tx
EndoRez
MDMA resin-based sealer hydrophillic good penetration into tubules biocompatible good radiopacity
calcium silicate sealers
high pH initial 24hrs hydrophillic biocompatible doesn't shrink non-resorbable excellent sealing ability quick set 3-4hrs - needs moisture easy to use
sealer placement
file
lentulospiral
US files
master GP cone
sealer functions
seal space between dentinal wall and core
fills voids and irregularities
lubricated
what type of sealers are not recommended?
ones containing organic materials e.g. aldehydes
why is RC culturing not always done?
assays not always reliable/relevant
timing of obturation
S/S - don't want to obturate while patient still has symptoms i.e. swelling pulp status PA status difficulty pt management
is length a prognostic determinant?
yes - >2mm short of apex harboured bacteria
how to asses obturation
post-op radiograph including root apex and at least 2-3mm of PA region
criteria for successful obturation
completely filled (unless space for post)
prepped and filled canal should contain original canal
no spaces
to length
to facial CEJ anteriors and canal orifice in posteriors
what is the most important factor for apical PD health?
coronal seal
orifice closure
ZnO/Eugenol
RMGIC/flowable composite
GP rapidly infected if exposed
future - regenerative endo?
vital pulp therapies with SCs
pulp regenerative therapies
defining success
means different to researchers, clinicians and patients
technical vs biological outcome
ESE success - assessment of outcome
RCT should be assessed at least after 1 year and subsequently as required
successful
uncertain
unfavourable
successful outcome
no pain, swelling, other symptoms
no sinus tract
no loss of fct
radiological evidence of a normal PDL - intact LD
uncertain outcome
radiographic changes same size/smaller
doesn’t have intact PDL - radiolucency, widened etc
clinically and radiographically assess every 12m for 4yrs/until resolved
if persists >4yrs - associated with post-tx disease - unfavourable
exceptions to outcome - scar tissue formation
an extensive radiological lesion may heal but leave a locally visible, irregularly mineralised area
defect may be scar tissue formation rather than a sign of persisting apical periodontitis
should continue to assess tooth
unfavourable outcome
tooth associated with S+S of infection
a radiologically visible lesion has appeared subsequent to tx or a pre-existing lesion has increased in size
lesion remained same size/only diminished in size over 4 year assessment period
signs of continuing RR
advise further tx for tooth
definitions of outcome - how success is defined
strict criteria - ESE guidelines, strict radiographic criteria for success 74.7%
loose criteria 85.2%
retention only 97.1%
pre-op factors affecting success
presence/absence of lesion - PA radiolucency pre-tx - outcome worse
presence of sinus
increased lesion size
operative factors affecting success
fill to within 2mm of radiographic apex but not extruded well-condensed, no voids good quality coronal restoration no perforation patency penultimate rinse with EDTA (re-RCT) avoid mixing CHX and NaOCl no flare up between visits instrument fracture missed canal - failed biological objective - MB2
how does instrument fracture affect outcome?
may do - depends on when fracture occurs - how much disinfection has already occurred
e.g. F3 better than S2
instrument fracture management options
remove bypass obturate up to instrument surgical removal extract
iatrogenic factors leading to failure
poor planning poor access poor length control forcing instruments failure to observe sequence failure to maintain patency
biological reasons for failure
persistent intraradicular infection - canal complexities, biofilm, resistant bacteria
extra-radicular bacteria - actinomycosis, extruded biofilm
cyst formation
cholesterol crystals
foreign body reactions - delayed healing
scar tissue ‘healing’
periapical cysts
form from mature granuloma, inflammatory mediators acting on epithelial cell rests - proliferate
true cyst
epithelial lined cavity that is distinct from the root
pocket cyst
cyst cavity continuous with RC space
how can you tell if it is a cyst?
only histologically
periapical cyst prevalence
about 15%
retreatment options
KUO
orthograde retx - but can you change something?
surgical tx - periradicular surgery
extract
what are 3 key points before retx?
establish cause of failure
assess restorative prognosis
re-tx complexity
when is KUO a risk?
CV disease/diabetes
removing insoluble resins
US
removing soluble pastes
handfiles +/-solvent - Protaper, D/Reciproc
removing poorly condensed GP
generally easier
Hedstroem files
removing well-condensed GP
generally harder
need to create space
removing GP
handfiles
solvent
Protaper D1
removing GP - handfiles
30/40 , few clockwise turns then pull
not on PD compromised teeth
C+ files - wind tip in WW, try to make space
if you can - back to 30 and remove. if can’t - solvent
removing GP - solvents
eucalyptus oil or chloroform
removing GP - Protaper D1
active tip - better initial penetration
but perforates teeth easily
good for coronal GP ONLY
Reciproc blue
“increased resistance to cyclic fatigue, increased flexibility”
good for re-tx
what could PAP be (in order of likelihood)?
granuloma
abscess
true cyst
pocket cyst
what speed should you use for protaper re-tx?
lowest speed that engages obturation material
500-700 rpm
Protaper retx D1
coronal removal
16mm, ISO30, 9%
protaper retx D2
middle 1/3 removal
18mm, ISO25, 8%
protaper retx D3
apical 1/3 removal, stop 2-3mm short of apex
22mm, ISO20, 7%
how to bypass ledges in protaper retx
pre-curved C+ files
PR surgery
surgical shortening of the root apex +/- retrograde sealing
is PR surgery ideal?
no - retx almost always preferable to or at v least better to use in conjunction with root end surgery
reciproc system for retx
reciprocating movement
R25 red
R40 black
R50 yellow
v efficient
remove bulk of GP (US, heat carrier)
brushing
why should you delay the use of solvents for as long as possible?
creates things which get pushed into tubules and into accessory canals - affects ability to get into RC space
indications for PR surgery
1 - failure of prev endo tx - retx not possible/won’t correct problem e.g. can’t regain access
2 - anatomical deviations - prevent complete cleaning and obturation - tortuous, curved roots, pulp stones, calcifications
3 - procedural errors - ledges, perforations etc
4 - exploratory surgery - identify root fractures
contraindications for PR surgery
anatomical factors - proximity to NV bundles
inadequate PD support
unrestorable tooth
medical factors - leukaemia, neutropenia, recent heart/cancer surgery. postpone if recent MI/radio tx
skill and ability of surgeon - refer?
microsurgery
uses “microscopes and miniaturised precision instruments to perform intricate procedures on v small structures”
higher success than contemporary root end surgery
- magnification, instruments, illumination
haemostasis in PR surgery - pre-op
LA - 1:50 000 adrenaline and 2% lidocaine
haemostasis in PR surgery - intra-op
examine bone crypt at high magnification
epinephrine pellets
ferric sulphate (cytotoxic - affects osseous healing)
CaSO4 - mechanically blocks open vessels
haemostasis in PR surgery - post-op
pressure
sulcular full thickness flap or mucogingival flap
mucogingival flap
crowned anteriors
scalloped incision in middle of attached gingiva at 45 degrees
vertical relieving incisions
microsurgical instruments
blades ST elevators curettage instruments US tips inspection mirrors carriers and pluggers needles and holders
osteotomy - clinical possibilities
intact cortical plate, no radiographic lesion
intact cortical plate, periapical lesion
fenestration through cortical plate leading to apex
- send lesion to histology to confirm just endo lesion
PRS - osteotomy
remove cortical plate to expose root end
assess
keep small - healing - microscope
use curettes to remove granulation tissue
clean L/P aspect after root end resection
PRS - root end resection
3mm resected perpendicular to long axis of tooth
PRS - why is 3mm resected?
terminal 3mm - a delta
- intricate design of canals
- can’t disinfect
- removes most lateral canals and ramifications
may be extraradicular biofilm
- could be initiating factor in inflammation
what is the aim of the root end filling in PRS?
seal apex so bacteria/products can’t enter/leave canal
PRS - US root end prep
low magnification US tip at apex coolant 3mm depth inspect with micro mirror at high magnification - remnants of GP, recondense GP
other ways to inspect resected root surface
PP
stroptko device - low pressure 10psi
isthmus
communication between 2 separate canals in one root
isthmus frequency
anteriors 15%
premolars 30%
M roots of L6s - 70%
MB roots of U6s - 45%
clinical significance of isthmus
untreated is one of the main causes of surgical failure
management of isthmus
identify with microscope
US prep - cut hole along isthmus so you can debride, disinfect and seal root properly
properties of ideal root end filling material
well-tolerated by apical tissues bactericidal/static adhere to tooth dimensionally stable easy to handle do not stain non-corrosive non-dissolving promote cementogenesis radiopaque
amalgam as a root end filling material
no longer used - slow set, biocompatibility, leakage, corrosion and staining
will see in pts historically
MTA chemistry
powder of fine hydrophilic particles
tricalcium silicate/oxide/aluminate, silicate oxide, bismuth oxide
mix water - slurry
MTA properties
long setting time antimicrobial alkaline superior sealing ability moisture tolerant radiopaque excellent biocompatibility regeneration of cementum - bioinductive
clinical applications of MTA
pulp capping RR apexification root end filling perforation repair furcal repair
root end filling - MTA placement
protect bone crypt sterile water - putty MTA gun/carrier micropluggers/burnishers to lightly condense material wipe excess with moist cotton pellet
regenerative procedures
quantity of remaining cortical bone influences surgery outcome
GTR
build scaffold on which pts bone can heal
doesn’t seem to have big impact on PR surgery outcome
PRS suturing
interrupted suture
ideally monofilament
remove 72hrs
PRS post op paraesthesia
abnormal sensation or numbness caused by impingement/handling/laceration/severance of nerve
often transient caused by swelling due to inflammation
normal sensation returns in 4wks
outcome of endo surgery
classification of healing - healed - incomplete healing (scar) - uncertain healing - failed follow up 1yr, then up to 4 yrs
preventing lacerations in PRS
vaseline lips
avoid careless flap elevation
PRS prognostic factors
age tooth position (L ants and L7s hard) root end filling material presence of co-existing PDD apical seal coronal seal crypt size
PRS success
1yr post op 96.8%
5-7yrs 91.5%
endodontists higher than oral surgeons - microscopes
1/3 success for repeat surgery
PPV
positive test result (no response) are truly non-vital
NPV
probability teeth with negative response are truly vital
mnemonic for ISO colour code
Please Give Peter Why Yelling Red Because Green Blacked out
most common number of canals - maxilla
1 1 1 2 1 4 3
most common number of canals - mandible
1 1 1 1 1 3 3
reaming
repeated clockwise rotation
what dictates the access cavity shape?
shape of pulp chamber
Endo Z bur
non-cutting tip
make access wider
funnel shape
GG bur
non-cutting tip
brush strokes
remove ledges and widen
SL access
aims of access cavity prep
remove entire roof = can remove all of pulpal tissue
visualisation of RC entrance
smooth-walled, no overhangs
SL access
(leave MR if possible to preserve strength)
why shouldn’t you use hand spreader?
too much force - may fracture tooth
are side vented needles good?
yes they are safer
indications for endo tx
irreversibly damaged/necrotic pulp
overdenture - decoronated teeth retained in arch
crowns - prophylactic tx of pulp before crown to reduce complications
PDD - root resection may merit elective devitalisation
contraindications for endo tx
unrestorable tooth poor OH insufficient PD support root fracture bizarre anatomy internal resorption
why obturate?
seal remaining bacteria - prevent them from accessing any nutrients
provide apical and coronal seal
prevent reinfection