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NHB MVST 1b > Emotion > Flashcards

Emotion Flashcards

1
Q

What are the main theories of emotion?

A

James-Lange

Schacter and Singer’s two factor theory

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2
Q

Define the James-Lange theory

A

Emotional experience may derive from bodily experience
James: we feel emotion because of what is happening in the body. ‘We don’t run because we’re afraid, we’re afraid because we run’
Lange: Emotion can be experienced as organic symptoms

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3
Q

Define the Schacter and Singer theory

A

Cognitive factors are critical for interpreting bodily changes to result in emotional experience

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4
Q

Define emotional behaviour

A

Behaviour driven by the need to fulfil social and internal goals.
Stems from presence or absence of positive and negative reinforcers.

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5
Q

What are the major responses measured in studies of emotion?

A 
HR
BP
Galvanic skin resistance (sweating)
Muscle tension
Arousal (desynchronisation of the EEG)
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6
Q

What are the 5 primary emotions? Why are they called that?

A 
Fear
Sadness
Anger
Happiness
Disgust
Can detect these in animals
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7
Q

What discredited the James-Lange theory?

A

Injecting adrenaline to produce tachycardia didn’t produce emotional experiences

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8
Q

What did Hohman find?

A

Subjective feelings of anger and fear were diminished in patients with spinal cord transections. Effect greater the higher the transection. Patients could ‘act’ emotions, but without emotional coloring and intensity. Shows that cognitions of emotions intact when brain separated from viscera, but emotional experience greatly reduced

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9
Q

Describe the Schacter-Singer experiment

A
  • Injected adrenaline while manipulating cognitive factors to test this
  • 3 main groups:
    1. Adrenaline ignorant: subjects told while being injected that injection was mild and harmless with no side effects. Patient thus had no externally provided explanation of their bodily state.
    2. Adrenaline misinformed: told to expect totally inaccurate side effects – numbness of feet, itching
    3. Adrenaline informed: told to expect hand tremor, palpitations, flushing
    4. Placebo: given saline, told group 1
  • Divide each group into 2 contexts
    1. Euphoria: actor behaved euphorically
    2. Anger: behaved angrily
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10
Q

What were the results of the Schacter-Singer experiment?

A
  1. Ignorant or misinformed subjects showed more emotion than placebo-treated subjects or subjects receiving Adr and informed of its tachycardia inducing effects (no significant difference between these two)
  2. Quality (euphoria or anger) of emotion determined by subject’s cognitive appraisal of the context in which the bodily changes occurred.
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11
Q

What does the Schacter-Singer experiment tell us?

A
  1. Provides support for James-Lange position that peripheral arousal produces a substrate for emotion
  2. Importance of cognitive factors (attribution or labelling of bodily states) in interpreting this general non-specific arousal as specific emotions
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12
Q

What are the two forms of anxiety?

A

Somatic anxiety

Psychic anxiety

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13
Q

Define positive reinforcer

A

something the subject will do more of to get more

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14
Q

Define negative reinforcer

A

Something the subject will do more of to get less

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15
Q

What are the problems with the Schacter-Singer model?

A

Tiny numbers of subjects

Hasn’t been replicated

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16
Q

Define somatic anxiety

A

Anxiety composed of physical experiences e.g. palpitations, hyperventilation

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17
Q

Treatment for somatic anxiety

A 
Beta blockers (e.g. propranolol)
Benzodiazepines like Valium , diazepam (enhance effect of GABA at GABA A receptors)
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18
Q

Define psychic anxiety

A

Anxiety characterised by the contents of consciousness - the feeling of dread or fear. Due to external stressors.

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19
Q

Treatment psychic anxiety

A

Benzodiazepines

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20
Q

What was Tyrer’s study about?

A

Treatments of somatic and psychic anxiety: diazepam, propranolol, placebo

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21
Q

What did Tyrer’s study show?

A
  • Results: Diazepam treats both, propranolol only effective for somatic.
  • Under propranolol psychic anxiety patients had a greatly reduced pulse rate, but showed no subjective reduction in their anxiety.
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22
Q

Define limbic system

A

The structures from Papez’ circuit along with the amygdala, septum and prefrontal cortex.

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23
Q

What did Broca discover?

A

A rim of old cortex on the medial wall of the hemisphere: the Grande lobe limbique. Now known as cingulate gyrus and uncus

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24
Q

What did Papez do?

A

Combined information from brain damage patients with research on the role of the hypothalamus in the control of emotional reactions in animals to create a theory explaining the flow of information through different structures that underlied emotion: Papez’ circuit

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25
Q

Who was Phineas Gage?

A

A worker where a metre long iron rod damaged prefrontal cortex and orbitofrontal area. Personality dramatically changed – could no longer regulate emotions effectively. Became quick-tempered, foul mouthed, behaved sexually inappropriately.

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26
Q

What is the Cannon-Bard hypothesis?

A

Showed removal of the whole cerebral cortex didn’t eliminate rage, but removal of the hypothalamus fragmented the emotional response.

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27
Q

Damage to cingulate gyrus

A

Apathy, depression, loss of emotional spontaneity

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28
Q

What did Papez include in his circuit which is no longer included, and why?

A

Hippocampus

Rabies damages it, results in emotional disturbance

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29
Q

What is Kluver-Bucy syndrome?

A

Temporal lobe removal in monkeys –> Psychic blindness including tameness, lack of emotional responsiveness, excessive examination of objects often with mouth, eating of previously rejected items, hypersexual behaviour.

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30
Q

What is thought of now as underlying emotion?

A

Hypothalamus, amygdala, orbitofrontal cortex, ventral striatum (nucleus accumbens), cingulate cortex.
Recently ant hippocampus implicated in emotional processing

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31
Q

What does the amygdala do with respect to emotion?

A

Learns about the emotional significance of stimuli and subsequent expression of these emotions

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32
Q

Why is the amygdala well placed to regulate emotion?

A

Connected to brainstem and hypothalamus so well placed to initiate fearful and aggressive responses

33
Q

What is an unconditioned stimulus?

A

A stimulus that is intrinsically appealing or aversive

34
Q

What is a conditioned stimulus?

A

A stimulus you have to condition the subject to have a response to

35
Q

What is Pavlovian fear conditioning?

A

Presenting an animal with a neutral stimulus followed by a mild electric shock
After a while the CS will elicit the same autonomic, endocrine and behavioural responses as the original unconditioned stimulus

36
Q

Effect of lesions of the amygdala?

A

Central nucleus: Lose Pavlovian fear conditioning

Impair fear recognition on faces

37
Q

Evidence that the amygdala is involved in processing faces?

A
  1. Lesions impair fear recognition

2. fMRI showed amygdala activated when presented with emotional faces

38
Q

What does the amygdala stimulate?

A

Laterla hypothalamus –> symp stim
Dorsal motor nucleus of X, Nucleus ambiguous –> para stim
Parabranchial nucleus –> increased resp
VTA/Locus coerulus/Dorsal lateral tegmental nucleus –> activation DA/NA/ACh
Nucleus Reticularis Pontis Caudais –> increased reflexes
Central grey –> cessation behaviour
Trigeminal/Facial MN –> mouth open, jaw movements
Paraventricular nucleus –>ACTH –> corticosteroids

39
Q

Define anxiety disorders

A

Healthy response to, and anticipation of, fear becomes dysfunctional.

40
Q

What are the categories of anxiety disorders?

A
  1. Phobias
  2. Panic disorder
  3. PTSD
  4. Agoraphobia
  5. GAD
41
Q

Define phobia

A

Irrational fear of a specific object or situation

42
Q

Define panic disorder

A

Intense and overwhelming attacks of anxiety

43
Q

Define PTSD

A

Recurrent flashbacks, intrusive thoughts and anxiety after a traumatic experience

44
Q

Define agoraphobia

A

A fear of crowds or public places that often gives rise to panic attacks

45
Q

Define GAD

A

Generalised Anxiety Disorder

Pervasive anxiety or worry lasting for months

46
Q

What are the key characteristics of phobias?

A
  1. Persistent fear of a situation or object that most people find tolerable
  2. Outside the panic situation can acknowledge that the fear is irrational
  3. Patient changes daily routine to avoid contact with the phobic object or situation. Can become debilitating.
47
Q

What are the types of phobia?

A
  1. Specific: to animals, activities or inanimate objects, illness/injury, blood/needles
  2. Social phobia: fear of being observed linking to fear of humiliation or embarrassment
  3. Agoraphobia
48
Q

What happens physiologically in needle phobia?

A

HR/BP decreases

49
Q

Prevalence of phobias in me and women?

A

8% women, 4% men

50
Q

What is the evidence that phobias have a genetic component?

A

First degree relatives of phobic patients 3x more likely to suffer phobias
Twins - 30% heritability

51
Q

Who was Little Albert?

A

Conditioned to fear a white rat (CS) by pairing with a loud gong (US) over his head. After 5 exposures, presentation of rat alone elicited crying and hiding behaviour (UR)

52
Q

Define avoidance.

A

Phobic patients run away when they see the CS, so CR doesn’t extinguish - they see it as a lucky escape. Negative reinforcement as avoidance relieves anxiety.

53
Q

Define Mowrer’s 2 factor model

A

Phobic anxiety is acquired by pavlovian conditioning but maintained by operant conditioning (avoidance)`

54
Q

Define Seligman’s biological preparedness model. Which studies support it?

A

There is an evolutionary bias for phobias to develop in response to objects and situations that were harmful to our ancestors (so lack of gun and knife phobias)

  1. Mineka: rhesus monkeys
  2. Ohman: extinction trials
55
Q

Define vicarious conditioning

A

The development of phobic responses based on observing someone else having a phobic response.

56
Q

Describe the Mineka study

A

Lab reared Rhesus monkeys do not exhibit behavioural avoidance or emotional distress to toy snakes, while wild-reared do. Shows early experience can produce phobic reactions. Also showed that lab-reared adolescent monkeys did develop phobic responses if put into a situation where they could observe their parent’s fearful reactions to the same snake stimulus (vicarious conditioning). Vicarious conditioning occurred more easily to the toy snake than to a flower, so suggests snakes have some innate properties that are recognised by the fear system.

57
Q

Describe the Ohman experiment

A

Presented human subjects either 1 or 5 pairings of a picture CS and shock US. Picture was of a potentially phobic stimulus (snake) for half the subjects, and a neutral stimulus (flower) for the remainder. CR measured in extinction (just CS, not US). Control groups, to check any change in SCR (sweating) was due to the positive temporal correlation of CS and US, received simple exposure either to shock alone or to the picture alone.

58
Q

What did the Ohman experiment show?

A
  • Showed that conditioned group showed larger SCR than controls
  • More conditioning to phobic CS (snake) than to neutral (flowers) – indicating that certain stimuli are more easily established than others as CSs for an aversive US.
59
Q

What did the Ohman experiment modified by instruction that the US would no longer occur show?

A

Instructions reduced SCR responding to the CS when a neutral stimulus was employed, but there was no effect when a phobic CS was used, i.e. the conditioned fear response persisted.

60
Q

Treatments of phobias?

A
  1. Flooding - patient exposed to CS until their anxiety subsides in absence of the US
  2. Modelling therapy: patient observe another person experience the CS in a non-fearful manner. Assumes that conditioning processes can occur vicariously. By watching another person behave according to the stimulus, our own fear reactions are modified
  3. Systematic desensitisation
61
Q

What did Doogan and Thomas observe?

A

Dog phobia can be transmitted to children from parents

62
Q

What are the stages of systematic desensitisation?

A
  1. Train patient in a relaxation technique
  2. Patient and doctor develop a hierarchal list of fear situations, from slightly scary to full exposure
  3. Expose to first situation and implement relaxation to counter-condition their anxiety
  4. Once can handle with no fear reaction, progress to second scenario,
63
Q

Define predisposing factors

A

Biological or psychological factors that increase the risk of developing a phobia e.g. being bitten by a dog in childhood

64
Q

Define precipitating factors

A

Factors that bring the issue to the surface, e.g. moving to a new house where the phobic stimulus is encountered more often

65
Q

Define maintaining factors

A

Factors that interact with the triggering factors to perpetuate the problem, e.g. avoidance, hypervigilance, thought suppression

66
Q

What is the effect of hypervigilance?

A

Constantly checking for the phobic stimulus means the patient detects more of them

67
Q

What is the effect of thought suppression?

A

Patient actually thinks about it more

68
Q

What is the Spider-Stroop task?

A

Spider phobics slower to read the colour of spider-associated words

69
Q

What did Thorpe and Salkovskis show?

A

That phobic patients don’t appreciate their irrationality in the phobic situation

70
Q

Define cognitive behavioural therapy

A

a type of psychotherapy in which negative patterns of thought about the self and the world are challenged in order to alter unwanted behaviour patterns or treat mood disorders such as depression.

71
Q

How is the brain altered in anxiety disorders?

A

Overactive amygdala
Underactive prefrontal cortex - suggests normally prefrontal cortex acts to downreg amygdala responsivity to aversive stimuli

72
Q

What do benzodiazepines do?

A 
E.g. valium
Treat anxiety
Anti-convulsant
Hypnotic
Sleep-inducers
73
Q

What are the advantages and disadvantages of benzodiazepines?

A

Advantage: hard to lethally overdose
Disadvantage: associated with dependence-withdrawal that includes insomnia, loss of appetite, anxiety

74
Q

What is the Geller-Seifter conflict procedure?

A

A mildly thirsty or hungry rat trained to press a lever for water or food, but occasionally a neutral light or tone stimulus is presented terminated with a mild electric footshock. Provides conflict, when the now CS is presented alone, resulting in suppression of behaviour until the anxiety-eliciting CS is switched off.

75
Q

What is the effect of benzodiazepines on the Geller-Seifter conflict?

A

Increases number of responses made during periods signalled by fear CS - ‘anxiolytic’

76
Q

What do benzodiazepines bind to and where?

A

GABA-A in amygala

77
Q

What do SSRIs do?

A

Selective serotonin reuptake inhibitors e.g. Prozac

Only work after a few weeks, probably regionally up or down regulate specific 5-HT receptors

78
Q

What is the genetic basis of the amygdala hypersensitivity to aversive stimuli?

A

Subjects with less effective (short) allele of 5-HT transporter show increased amygdala responses to fearful facial expressions and are at increased risk of anxiety compared to subjects with the more effective long allele.
Serotonin also involved in developing brain circuits, so short allele probably has effects on development, and so may have changes beyond those specific to serotonin.

79
Q

What are the treatments of anxiety?

A
  1. Benzodiazepines e.g. Valium
  2. SSRIs like Prozac
  3. Rarer 5-HT receptor blockers
  4. Propanolol for somatic anxiety

NHB MVST 1b (11 decks)

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