Emo Behaviors Flashcards

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1
Q

What are the 3 components of emotion?

A

Cognition, feelings, and actions. (Feelings is considered the most central of the three).

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2
Q

Which system do emotional situations arouse?

A

The autonomic nervous system (ANS).

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3
Q

The ANS consists of how many branches? What are they?

A

2 branches; the sympathetic nervous system (SNS) and the parasympathetic nervous system (PNS).

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4
Q

Who was the researcher responsible for identifying the “fight or flight” response of the SNS?

A

Walter Cannon (1871 - 1945)

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5
Q

Explain the James-Lange Theory.

A

The James-Lange theory consists of the idea that the order of events during a stressful or arousing event is in reverse of standardly held common-sense. That is to say, you would expect to experience an emotive instinct and then your body would act on that sensation. The theory holds that, instead, one would experience a situation, then respond with either and action correspondent with fear or rage, then experience the related emotion (i.e. running away causes the experience of fear, and fighting causes experience of rage).

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6
Q

What occurs during pure autonomic failure?

A

output from the autonomic nervous system to the body fails, either completely or almost completely. Heartbeat and other organ activities continue, but the nervous system no longer regulates them. Someone with this condition does not react to stressful experiences with changes in heart rate, blood pressure, or sweating.

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7
Q

According to the James-Lange theory, what kind of person should feel no emotion?

A

Someone who experiences a condition called pure autonomic failure would be expected to report no emotion. With this condition organ activities and physiological changes regulated by the autonomic system would remain detectably unaffected by any arousing stimuli [i.e. in a fearful situation they would likely not experience sweating, quickened heartbeat, or decrease in hunger, etc.]. (however, the person may have a cognitive understanding of the appropriate related emotion).

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8
Q

How might researchers get people to smile or frown without using these words?

A

Researchers may ask participants to hold a pen, either with their teeth or their lips/mouth—this may result in a smile or frown. Also, another study attached golf tees to a persons eyebrows and required the tips to touch one another forcing a frown.

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9
Q

What is the limbic system?

A

the forebrain areas surrounding the thalamus. (critical for emotion). (is considered to be a part of the amygdala).

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10
Q

Which structures comprise the limbic system?

A

cingulate gyrus, anterior thalamic nuclei, septal nuclei, frontal lobe, olfactory bulb, fornix, hippocampus, parahippocampal gyrus, amygdala, mamillary bodies.

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11
Q

What is the primary taste cortex?

A

The insula

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12
Q

The insula is important for which kind of emotion and which kind of sensation?

A

The emotion of disgust, and the sensation of taste.

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13
Q

What is the behavioral activation system (BAS) marked by?

A

Activity of the left hemisphere (particularly the frontal and temporal lobes) w/ low to moderate autonomic arousal and a tendency to approach (characterizing either happiness or anger).

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14
Q

Describe the Behavioral Inhibition System.

A

Associated with increased activity of the frontal and temporal lobes of the right hemisphere. Increases attention and arousal, inhibits action, and stimulates emotions (e.g. fear and disgust).

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15
Q

Which hemisphere appears to be more responsive to emotional stimuli?

A

The right hemisphere.

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16
Q

Explain the “Wada procedure”

A

Injection of a drug into the carotid artery in an effort to anesthetize and isolate each hemisphere of the brain.

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17
Q

What are the contributions of the right hemisphere to emotional behaviors and interpreting other people’s emotions?

A

Activation of the right hemisphere is associated with withdrawal from events and social contact. The right hemisphere is also more specialized than the left for interpreting other people’s expressions of emotions.

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18
Q

Damage to which part of the PFC results in deficient guilt sensations?

A

The ventromedial prefrontal cortex (PFC).

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19
Q

If brain damage impairs someone’s emotions, what happens to the person’s decision making?

A

After brain damage that impairs emotion, people make impulsive decisions, evidently because they do not quickly imagine how bad a poor decision might make them feel.

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20
Q

Name each of the identified structures in this photo.

A
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21
Q

What is a method to decrease anger other than “counting to 10”?

A

Lying on your back (e.g. posturing yourself in a more helpless position)

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22
Q

What is MAOA?

A

Monoamine oxidase. An enzyme which breaks down the neurotransmitters dopamine, norepinephrine, and seratonin.

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23
Q

What relationship did Caspi et al. (2002) report between the enzyme MAOA and antisocial behavior?

A

Overal, people with genese for high or low production of MAOA do not differ significantly in their probability of antisocial behavior. However, among those who suffered serious maltreatment during childhood, people with lower levels of the enzyme showed higher rates of antisocial behavior.

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24
Q

What does the “triple imbalance hypothesis” state?

A

Violence depends on 3 chemicals. Serotonin, testosterone, and cortisol.

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25
Q

How would the “triple imbalance hypothesis” explain increased aggression?

A

A person would be considered to have a low level of cortisol (lowering inhibitions) and a higher level of testosterone (increasing natural aggressive tendencies). This person is also likely to have low serotonin levels (high levels are shows to inhibit violent impulses).

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26
Q

How does testosterone influence emotional and cognitive responses to a facial expression fo anger?

A

It decreases teh ability to recognize the expression consciously but increases the responses in emotion-related areas of the brain.

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27
Q

Explain serotonin turnover.

A

The amount of serotonin that neurons release and replace.

When neurons release serotonin, they reabsorb most of it and then synthesize enough to replace the amount that washed away.

28
Q

How do researchers estimate serotonin turnover?

A

Form the concentration of 5-hydroxyindoleacetic acid (5-HIAA) [serotonin’s main metabolite] in the cerebrospinal fuid (CSF).

29
Q

Given that monkeys with low serotonin turnover pick many fights and in most caes die young, what keeps natural selection from eliminating the genes for low serotonin turnover?

A

Although most monkeys with low serotonin turnover die young, many of the survivors achieve a dominant status that enables them to get more of the food and to reproduce more frequently. Monkeys with high serotonin turnover survive, but at the cost of accepting a low status.

30
Q

Is it possible to alter serotonin sythesis via the diet? If so, how would this be possible?

A

Neurons synthesize serotonin from tryptophan, an amino acid found in small amounts in proteins. Tryptophan crosses the blood–brain barrier by an active transport channel that it shares with phenylalanine and other large amino acids. Thus, a diet high in other amino acids impairs the brain’s ability to synthesize serotonin.

31
Q

How could nutrasweet or maize affect a persons aggression?

A

If a person has a diet which is too high in certain amino acids (phenylalanine) these foods could increase this acid and spike aggressive behaviors. Aspartame (Nutrasweet) is about 50% phenylalanine, and maize (Amer. Corn) is high in phylalanine and low in tryptophan.

32
Q

What is the name of the enzyme that converts tryptophan into serotonin?

A

Tryptophan hydroxylase

33
Q

Why is the serotonin-aggression relationship considered to be complex?

A

Studies imply that serotonin inhibits aggressive behavior, thereby increasing the likelihood of aggression when serotonin levels are low. However, the brain is shown to release serotonin during aggressive behavior. Apparently, if serotonin levels are low prior to aggravation, the aggressive behavior is magnified when the additional serotonin is released at the outset of the aggressive encounter.

34
Q

Startle Reflex

A

the response to an unexpected loud noise

35
Q

What occurs during the startle response?

A

Auditory information goes first to the cochlear nucleus in the medulla and from there directly to an area in the pons that commands tensing the muscles (particularly the neck muscles).

36
Q

How long does it take for the full startle response to occur?

A

Less than 2/10 of a second. Information reaches the pons within 3 to 8 ms after a loud noise.

37
Q

Long term, generalized emotional arousal depends on a brain area called the ___________.

A

bed nucleus of the stria terminalis (critical for long-term adjustments of anxiety).

38
Q

Label the following areas of the stria terminalis:

  1. Bed nucleus of the stria terminalis
  2. Amygdala
  3. Fornix
  4. Stria Terminalis
  5. Corpus Callosum
  6. Hippocampus
A
39
Q

Explain the Kluver-Bucy syndrome.

A

The result of damage to the amygdala causing those afflicted to display less fear response. (The syndrome was explain in relation to monkeys)

40
Q

What is Urbach-Wiethe disease?

A

A rare genetic disorder which causes skin lesions and calcification of the amygdala until it wastes away. Therefore those afflicted experience damage isolated almost solely to the amygdala.

41
Q

Why do eople with amygdala damage have trouble recognizing expressions of fear?

A

They focus their vision on the nose and mouth. Expressions of fear depend almost entirely on the eyes.

42
Q

How is a panic disorder characterized?

A

by frequent periods of anxiety and occasional attacks of rapid breathing, increased heart rate, sweating, and trembling (an extreme arousal of the SNS).

43
Q

Panic disorder occurs in about 15% of people with _______.

A

joint laxity (double jointedness)

44
Q

Panic disorder is associated with decreased activity of _____________.

A

neurotransmitter GABA (gamma-aminobutyric acid) and increased levels of orexin (associated with wakefulness and activity) [drugs which block orexin receptors block panic responses].

45
Q

The common class of anti-anxiety drugs

A

benzodiazepines (i.e. diazepam [valium], chlordiazepoxide [librium], and alprazolam [xanax])

46
Q

How do benzo’s take effect?

A

they bind to the GABAA receptor, including a site which binds GABA as well as sites that modify the sensitivity of the GABA site. The center of the GABAA receptor is a chloride (Cl) channel, when open is permits chloride ions (Cl-) to cross the membrane into the neuron, hyperpolarizing the cell. The chloride channel is surrounded by four units which are sensitive to GABA, benzo’s bind to three of the four sites. The benzo neither opens nor closes the Cl- channel _ it twists the receptor so that the GABA binds more easily. Benzo’s facilitate the effects of GABA.

47
Q

Name three areas where benzodiazepines exert their anti-anxiety effects?

A

The amygdala, hypothalamus, midbrain, and several other areas.

48
Q

What is the name of the experiental drug which has been shown to block the effects of alcohol?

A

Ro15-4513

49
Q

What would be the effect of benzodiazepines on someone who had no GABA?

A

Benzodiazepines faciliate the effects of GABA, so a person without GABA would have no response to benzo’s.

50
Q

Why is extinction more effective a few minutes after a brief reminder of the original learning?

A

The reminder brings the representation of the learning into a labile state from which it can be reconsolidated or extinguished.

51
Q

Explain general adaption syndrom.

A

Any threat to the body (in addition to its specific effects) activates a generalized response to the stress. This response involves 3 stages (alarm, resistance, and exhaustion).

52
Q

What are the three stages of general adaption syndrome?

A

Alarm (characterized by increased activitity of the SNS - readying the body for brief emergent activity)

Resistance (characterized by a decline of the sympathetic response - the adrenal cortex secretes cortisol and other hormones allowing prolonged alertness, immne response, and healing)

exhaustion (marked by fatigue of the body the nervous system and the immune systems)

53
Q

Explain the HPA axis function.

A

the hypothalamus, pituitary gland, and adrenal cortex. Activation of the hypothalamus induces the anterior pituitary gland to secrete adrenocorticotropic hormone (ACTH), which in turn stimulates the human adrenal cortex to secrete cortisol, which enhances metabolic activity and elevates blood levels of sugar and other nutrients

54
Q

What does the following image depict?

A

The HPA (hypothalamus-pituitary-adrenal) cortex axis

55
Q

What is an autoimmune disease?

A

The immune system consists of cells which protect the body against viruses, bacteria, and other. In the event the body’s immune system is over-active it may begin to inappropriately attack it’s own cells.

56
Q

What are 2 examples of autoimmune diseases?

A

Myasthenia gravis

rheumatoid arthritis

57
Q

The most important elements of the immune system.

A

Leukocytes (white blood cells). These leukocyte cells produce cytokines and prostaglandins which are responsible for the “general adaptation syndrome” proposed by Selye.

58
Q

What are the three types of leukocytes?

A

B Cells, T cells, and Natural killer cells

59
Q

Explain B cells.

A

mature mostly in bone marrow, secrete antibodes. These cells recognize the ‘self’ antigens and attack unrecognized cells.

60
Q

Explain T cells

A

mature in the thymus gland, some attack intruders directly without secreting antibodies, other help T and B cells to multiply.

61
Q

Explain natural killer cells

A

attack tumor cells, and virus infected cells. Unlike B and T cells, natural killer cells attak all intruders indiscriminately.

62
Q

What are cytokines?

A

Small proteins produced by leukocytes and other calls which combat infections and communicate with the brain to elicit appropriate behaviors. They trigger the hypothalamus to produce fever, sleepiness, fatigue, loss of appetite, and loss of sex drive.

63
Q

Psychoneuroimmunology

A

The study of the relationship between the nervous system and the immune system.

64
Q

How do the effects of prolonged stress mimic the effects of ilness?

A

Prolonged stress increases release of cytokines. Cytokines tell the brain to initiate reponses to combat ilness, such as fever, inactivity, and loss of appetite.

65
Q

How does prolonged stress damage the hippocampus?

A

Stress incrases the release of cortisol, which inhances metabolic activity throughout the body. When neurons in the hippocampus have high metabolic activity, they becoe more vulnerable to damage by toxins of overstimulation.

66
Q

How do the cortisol levels of PTSD victims compare to those of other people?

A

People with PTSD have lower than normal cortisol levels in contrast to most people, who show elevated cortisol levels in response to stress.

67
Q

What evidence indicates that small than average hippocampus makes people more vulnerable to PTSD?

A

On average, PTSD victims have a smaller than average hippocampus. For those who have an idential twin, the twin also has a smaller than average hippocampus, even if he or she does not have PTSD.