Emergency medicine and trauma Flashcards

1
Q

Mandible injuries

  • signs
  • diagnosis
  • management of TMJ dislocation
  • management of mandibular fractures
A

Signs: tenderness and swelling, jaw malocclusion (misalignment of the teeth)
Tongue may make airway management difficult

Diagnosis: orthopantogram (OPG) X-rays

Management of TMJ dislocation: place thumbs over the back teeth and press downwards while at the same time levering the chin upwards with your fingers (may require midazolam sedation)

Management of mandibular fractures: ORIF with miniplates

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2
Q

What is the complication of a pterion fracture?

A

May rupture the middle meningeal artery and cause and extra-dural haemorrhage which could compress the brain and be life-threatening

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3
Q

Features of a basal skull fracture

A
Raccoon eyes
Panda eye
Ecchymosis
CSF rhinorrhoea
CSF otorrhoea
Haemotympanum
Battle sign
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4
Q

Management of drowning

A

Do not attempt CPR in water
Try to maintain C spine immobilisation throughout
Place the patient in prone position when lifted form water in order to exert pressure on venous return and prevent circulatory collapse
Intubation with high PEEP
Always suspect hypothermia
ALS protocol if cardiac arrest occurs (cardiac arrest secondary to hypoxaemia)

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5
Q

Key concerns in drowning

A

Cardiac arrest
Hypothermia
Pulmonary oedema

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6
Q

Do all drowning patients require admission?

A

Patients who have been in a drowning event need to be observe for 4 hours - monitor for haemoptysis, basal crackles, CXR changes, and hypoxia on room air

If asymptomatic 4 hours later then they can be discharged

Outpatient follow up with CXR 2 weeks later is advised

Prophylactic antibiotics not requires

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7
Q

Factors that increase the severity of electric shock injuries

A
  • Types of current: alternating current (AC) is more dangerous than direct current (DC). DC causes one large muscular contraction and asystole. AC causes repeated muscle spasms making it impossible for the victim to let go of the source, and causes VF.
  • Energy delivered
  • Current pathway through the body: if it enters the head or chest (eg. through one arm and out of the other arm), fatal injury is more likely
  • Resistance encountered: Bone is the most resistant tissue, and thick skin can limit the amount of current passing through. Heart and nerves conduct electricity well so often receive the most damage.
  • Contact duration
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8
Q

Management of electric shock

A

Turn off the source of current
Long, aggressive resuscitation
More fluid replacement is required for electric shock burns than needed for thermal burns, as electrical burns penetrate deeper
Check for rhabdomyolysis
Treat arrhythmias conventionally
Compartment syndrome may occur - treat accordingly

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9
Q

Management of wounds

A

Tetanus prophylaxis is vital. Prophylactic abs often not needed.

  1. LOTS of irrigation with 0.9% saline
  2. Infiltrate with lidocaine 3mg/kg plain (or adrenaline if necessary)
  3. Remove debris, foreign bodies, and necrotic tissue, thorough scrubbing of abrasions, trim ragged skin edges
  4. Use absorbable subcutaneous sutures (vicryl) to bring skin edges together, avoiding skin tension. Then use interrupted monofilament (nylone) on the skin.
  5. Remove sutures at a correct time to minimise risk of unsightly permanent stitch marks (face = 5 days, UL/body = 7-10 days, LL = 14 days)
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10
Q

Primary survey in trauma

A

ABCDE

Airway and cervical spine (jaw thrust to protect C spine)
Breathing and oxygen: 100% O2
Circulation and haemorrhage control: local pressure, cross match
Disability and blood glucose
Exposure and temperature control: totally undress patient and do rewarming techniques, remembering dignity

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11
Q

Adjuncts to the primary survey

A

CT within 30 minutes
Urinary catheter to assess urinary output
NG tube, unless facial injury
ABG can assess oxygenation in cold patients where pulse oximeter fails

Airway adjuncts
Logrolling a patient to examine the back whilst immobilising the C spine

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12
Q

Indications to intubate in trauma

A
GCS <9
Sustained seizure activity
Facial or airway trauma
High aspiration risk
Flail segments or respiratory failure
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13
Q

Life threatening chest injuries in trauma

A

ATOM FC

Airway obstruction
Tension pneumothorax
Open pneumothorax (sucking chest wound)
Massive haemothorax
Flail chest
Traumatic cardiac tamponade
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14
Q

Types of primary head injuries

A

Focal: Contusions, haematomas
Diffuse: diffuse axonal injury

Haematomas may be extradural, subdural or intracerebral

Contusion may be adjacent to the site of impact (coup) or contralateral side (contracoup)

Diffuse axonal injury occurs as a result of mechanical shearing following deceleration, causing disruption and tearing of axons

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15
Q

Types of secondary brain injuries

A

Cerebral oedema, ischaemia, infection, tonsillar or tentorial herniation

Secondary injury exacerbates the primary injury

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16
Q

Cushings reflex

A

Hypertension, bradycardia, abnormal respiration

Due to brainstem compression and raised ICP causing dysfunction of the brains autoregulation
Usually a preterminal event

17
Q

Management of head injuries

A

If there is a life threatening rise in ICP -> use IV mannitol/furosemide whilst waiting for theatre to prep

Diffuse cerebral oedema -> decompressive craniotomy

Depressed open skull fractures -> formal surgical reduction and debridement

GCS 3-8: ICP monitoring is acceptable if CT is normal but MANDATORY if CT is abnormal

Maintain minimum cerebral perfusion pressure of 70mmHg in adults and 40-70mmHg in children

18
Q

Pupillary findings in head injuries

  • CNIII compression secondary to tentorial herniation
  • Bilateral CNIII palsy or poor CNS perfusion
  • Optic nerve injury
  • Opiates, pontine lesions, metabolic encephalopathy
  • Sympathetic pathway disruption
A
  • CNIII compression secondary to tentorial herniation: unilaterally dilated, sluggish or fixed in response to light
  • Bilateral CNIII palsy or poor CNS perfusion: bilaterally dilated, sluggish or fixed in response to light
  • Optic nerve injury: Marcus-Gunn pupil (RAPD) unilaterally dilated or equal, cross-reactive
  • Opiates, pontine lesions, metabolic encephalopathy: bilaterally constricted, may be difficult to assess
  • Sympathetic pathway disruption: unilaterally constricted, preserved reaction to light
19
Q

Indications for immediate CT head following head injury

A
GCS<13 on initial assessment
GCS<15 2hrs later
Suspected open or depressed skull fracture
Basal skull fracture
Post-traumatic seizure
Focal neurological deficit
More than 1 episode of vomiting
20
Q

Indications for CT within 8 hours of head injury

A

Adults who have experiences some loss of consciousness or amnesia since the injury, with one of the following risk factors:

  • 65y+
  • history of bleeding or clotting disorders
  • dangerous mechanism of injury (ejection from motorvehicle, fall from >1m height)
  • more than 30mins retrograde amnesia of events immediately before the head injury
21
Q

Medical management of head injury

A
Keep systolic BP >90
Avoid hypoxia or hypercapnia
Opiates
Mannitol
Seizure control (IV lorazepam/buccal midazolam)
Raise head 30 degrees to improve jugular venous return
Avoid hyperglycaemia and hyperpyrexia
Urgent neurosurgical input
22
Q

Equation for cerebral perfusion pressure, and how to enhance cerebral perfusion pressure

A

CPP = mean arterial pressure - ICP

Enhancing cerebral perfusion pressure: normalise the ICP (<20), and target mean arterial pressure to approx 90mmHg so that CPP remains >60mmHg, limiting the risk of ischaemia to the brain