Emergency Flashcards

1
Q

Signs of potential airway obstruction

A
  • agitation, restlessness, panic & confusion, progressing to lots of consciousness
  • gasping for air & chocking
  • clutching upper chest or throat
  • progressive cyanosis (cyanosis is a late sign of airway obstruction)
  • high pitched stridor, wheezing crowing or whistling on inspiration
  • noisy or gurgling inspiration
  • snoring respirations
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2
Q

what do the aplha receptors do?

A
  • alpha causes vasoconstriction, oxygenated blood from least important to most important (least important is the periphery- that’s why important to check cap refills (will cause ulceration & necrosis)) then GI tract that is not getting enough blood empties, vomits & incontinent of stool, will not have bowel sounds), also liver is part of digestive systtem
  • Next the kidneys (decreased urine output, if less than 30ml/hr kidneys may not be adequately perfused)
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3
Q

what do the aplha receptors do? beta 1 receptors? and Beta 2 recpetors?

A
  • alpha receptors cause vasoconstriction
  • B1 heart increases HR & contractility
  • B2 lungs (bronchodilation to get as much oxygen as can)
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4
Q

what are interventions for an airway problem?

A
  • Open airway (techniques are head tilt chin lift-which manipulates spine a lot) (modified jaw thrust maneuver-then bag valve mask)
  • Oral airway insertion- holds tongue up from back of throat, but oral airway increases risk of aspiration
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5
Q

what are the three causes of hypoxia?

A

problems with:

  • ventilation
  • diffusion
  • perfusion
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6
Q

what are some interventions for breathing problems?

A
  • nasal prongs (oxymizer)
  • face mask (turn up till not fogging, fogging means re breathing carbon dioxide)
  • BVM for ventilating someone
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7
Q

In the primary survey how do you check if there C- circulation is good?

A
  • do they have adequate cardiac output
  • palpable radial pulse is cardiac output
  • Brisk capillary refill?
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8
Q

what are three things that cause circulation problems?

A

problems with:

  • Heart
  • Vessels
  • Volume
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9
Q

what are some interventions for circulation problems

A

-volume resuscitation (isotonic solution - NS or LR)

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10
Q

what does D stand for in ABCD?

A

disability assessment: what is there neurological status (AVPU- awake, verbal stimulus, physical stimulus, unresponsive even to pain)

discomfort assessment:

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11
Q

what is wrong with a patient that has a decreased LOC, what are you worried about with someone that has a GCS of 8 or lower

A

Airway

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12
Q

What are some airway findings of concern?

A
  • absence of breathing
  • trauma to the face, mouth, pharynx, neck or chest
  • inability to speak (age appropriate)
  • substernal or intercostal retractions
  • depressed level of cnsiousness
  • inspiratory or expiatory stridor
  • pale, cyanotic or dusky-gray skin color or ruddy or bright purple colouring
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13
Q

what is trauma?

A

-trauma is an injury to human tissues & organs resulting form the transfer of energy from the environment

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14
Q

trauma is _________ potential problems may become actual problems

A

insidious

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15
Q

With Trauma if ABC’s aren’t kept stable than body’s cells being to metabolize anaerobically which can lead to

A

Multi-organ failure (Kidneys, liver, pancreas)

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16
Q

what is the priority if a patient is burned?

A

-you need to put the fire out stop the burning (why do we run burns under water stop the burn, chemical burns need to be flushed)

17
Q

what is mannatol?

A

used as a diuretic to decrease volume of brain (decrease water in brain) with increasing ICP
it is a high molecular weight sugar that sucks water from interstitial to intravascular able to pee out,
mannatol doesn’t work with leaking capillaries

18
Q

what are the first sign s of increasing ICP?

A

agitation & restlessness, which then leads to a decrease in LOC

19
Q

when do vital signs change with ICP

A

-vital signs change (cushings traid) right before almost dead

20
Q

what is shock

A

-inadequate perfusion of body tissues with oxygenated blood

21
Q

If mean arterial pressure is 70 or above that means that?

A

all organs are perfusing

22
Q

what does a person look like that is adequately perfused (not experiencing shock)

A
NS: awake & alert
RS: spo2 greater than 94% on RA
CVS: palpable radial pulse, brisk cap refill 
GI: BS
GU: urine output greater than 30ml, hr
23
Q

what does a person that is inadequately perfused look like? (experiencing shock)

A

NS: agitation & restlessness (always assume cerebral hypoixa until proven otherwise)
RS: cannot maintain spo2 greater than 94%, may need rebreather mask
CVS: delayed cap refill, no radial pulse,then go to carotid
-GI: no BS, N&V& incontinence
-GU: urine output is less than 30 ml/hr

24
Q

pulse pressure is what? and what number should it be above? and if it is not what does that indicate?

A
  • systolic-diastolic
  • should be above 40
  • if not above 40,
  • loosing stroke volume or volume, therefore not adequately perfused
25
Q

what are the 3 types of shock

A
  • hypovolemic
  • cardiogenic
  • distributive
26
Q

what are the causes of hypovolemic shock?

A

-external losses (a break, loose through vascular)
-internal losses (bleeding into thoracic cage, abdomen, retro-peritoneal space)
-blacked venous return (not important)
could be whole blood loss (hemorrhaging) fluid volume loss (vomiting & diarrhea) plasma loss (burns)

27
Q

what are the signs & symptoms of Hypovolemic shock?

A
  • evidence of internal or external losses
  • organ hypoperfusion
  • stages of compensation
28
Q

what is the management of hypovolemeic shock

A
  • early recognition (notice changes, & notice when body is compensating)
  • stop the losses
  • replace the losses if indicated (fluid resuscitation)
29
Q

with cardiogenic shock what is the problem?

A

the pump is the problem (usually the left side of the pump)

30
Q

what are the S&S of cardiogenic shock

A
  • foward flow, so backward pressure ( heart cannot pump, blood backs up into lungs, so patient will have pulmonary edema, crackles
  • the stages of compensation are the same
31
Q

what is the management of cardiogenic shock?

A
  • early recognition
  • increase myocardial contractility (whole bunch of drugs do this POSITIVE INOTROPES)
  • reduce myocardial oxygen demands
32
Q

what are three types of distributive shock?

A
  • septic shock
  • anaphylactic shock
  • neurogenic shock
33
Q

what is the management of cardiogenic shock?

A
  • early recognition
  • increase myocardial contractility (whole bunch of drugs do this POSITIVE INOTROPES - increase contractility of the heart)
  • reduce myocardial oxygen demands
34
Q

what is the problem with distributive shock

A
  • vasodilation (will have low diastolic pressure)

- dilated so cannot fill properly (can vasoconstrict, but treating symptom not cause)

35
Q

septic shock S&S

A
  • evidence of infection
  • organ hypoperfusion
  • tx: give antiboitic
36
Q

anaphylactic shock S&S

A
  • evidence of exposure
  • airway reactivity
  • organ hypoperfusion
  • no alpha SNS compensation
  • histamine released causing vasodilation
  • need antihistamine and may need epinephrine for vasoconstriciton
37
Q

Neurogenic shock S&S

A
  • evidence of injury
  • no SNS compensation
  • organ hypoperfusion