Electronic Fetal Monitor Flashcards
Category I or Normal FHR
Rules out fetal metabolic acidemia
Fetal Oxygen Pathway: Extrinsic
- Maternal oxygenation
- Uterine blood flow
- Placental exchange
- Umbilical blood flow
Fetal Oxygen Pathway: Intrinsic
- Fetal circulation
- Oxygenation of tissues
- FHR reguation
What is well-oxygenated blood dependent upon?
- Adequate hemoglobin concentration and saturation (O2 carrying capacity)
- Adequate arterial oxygen tension (PaO2) (immediately available O2 for exchange.
What is intervillous space perfusion dependent upon?
Adequate uterine blood flow
What substances are transferred across the placenta by simple diffusion?
O2
CO2
Narcotics
What substances are transferred across the placenta by facilitated diffusion?
Glucose
What substances are transferred across the placenta by active transport?
Amino acids Ca P Fe I
What substances are transferred across the placenta by pinocytosis?
IgG
phospholipids
What substances are transferred across the placenta by bulk flow?
H2O
Electrolytes
What substances are transferred across the placenta by breaks or leaks?
Intact blood cells
What substances are transferred across the placenta by independent movement?
Treponema pallidum
What substances are transferred across the placenta by infection?
Toxoplasma gondii
What uteroplacental perfusion factors may decrease?
- Excessive uterine activity (tachysystole, hypertonus, abruptio placenta)
- Maternal hypotension (supine, analgesia)
- Maternal hypertension
- Placental changes (decreased surface area, edema, degenerative, calcifications, infarcts, infection
- Vasoconstriction (exogenous – most sympathomimetics, except ephedrine)
How does the fetus survive in an environment with a pO2 value equal to adult venous blood (fetal oxygenation)?
- Fetal hemoglobin concentration is higher than the adult - Ave Hct at term 51-56%
- Fetal hemoglobin has increased O2 affinity than adult - oxyhemoglobin dissociation curve
- Fetus has increased CO and HR than the adult resulting in rapid circulation
How does fetal oxygenation compensate with abrupt decrease in pO2 normoxic fetus?
- Redistribution of blood to vital organs
- 2-3x increase to heart, brain, adrenal glands
- decrease to gut, spleen, kidneys, limbs - with severe acidemia
- decrease CO, BP, blood flow to brain and heart
- tissue damage or fetal death
Sympathetic Innervation Action and Effect
Action: Releases norepinephrine and epinephrine
Effect: Increase HR
Parasympathetic innervation Action and Effect
Action: Releases acetylcholine
Effect: Decrease HR and transmits variability
Baroreceptors (peripheral) Action and Effect
Action: Regulate BP
Effect: Decrease FHR, BP, and CO
Fetal chemoreceptors action and effect
Action: Increase PCO2, Decrease PO2, pH
Effect: Bradycardia, hypertension
Does fetal stroke volume fluctuation significantly?
No, CO = HR
FHR and Fetal Sleep-Wake Cycles: State 2F Active REM Sleep
Active body movement: frequent
Associated FHR: Moderate variability, accelerations with FM, Reactive NST
FHR and Fetal Sleep-Wake Cycles: State 1F Quiet Sleep
Active body movement: Infrequent
Associated FHR: minimal variability, non-reactive NST
What is required for normal fetal acid base status on EFM Category I?
Moderate variability Baseline rate: 110-160 bpm Late or variable decels: absent Early decels: present or absent Accels: present or absent
What is required for normal fetal acid base status on EFM Category III? ( options)
- Absent variability with recurrent late decels, or recurrent variable decels, or bradycardia
- Sinusoidal pattern
What is normal FHR?
110-160 bpm
What is FHR tachycardia?
> 160 bpm
What is FHR bradycardia?
<110 bpm
What is moderate variability
6-25 bpm
What is minimal variability
> 0 and = 5 bpm
What is marked variability (saltatory)?
> 25 bpm