EKGs Flashcards
Atrial hypertrophy
Biphasic P wave in II and V1
Initial component = RA, terminal component = LA
Most common cause of RVH
LVH
Causes of RAH
Pulmonary HTN
Tricuspid/pulmonary valve dz
PE
Congenital heart defects
Causes of LAH
HTN
AMI
Mitral/aortic stenosis
LVH
RVH
R wave > S wave in V1
Normally S wave > R wave in V1
LVH EKG findings
LAH + voltage criteria (35 mm+)
Voltage criteria for LVH
Deepest S in V1/V2 + tallest R in V5/V6
Causes of LVH
systemic HTN
Mitral/aortic stenosis
Strain pattern with LVH
Down sloping ST in V5/V6
Ischemia
Inverted T or ST depression in 2+ contiguous leads (V1-V6)
Injury
ST elevation 1+ mm in 2+ contiguous leads
Infarct
Pathological Q waves (>.04 sec, deep –> 1/3 of QRS)
Leads for Septal Infarct
V1, V2
Leads for Anterior Infarct
V3, V4 (also V1, V2)
Leads for Inferior Infarct
II, III, aVF
Leads for Lateral Infarct
I, aVL, V5, V6
Findings for posterior infarct
ST depression and tall R in V1, V2, V3
Right ventricular infarct
inferior infarct + ST elevation in V4
Triad: hypotension, clear lung, JVD
RBBB
Wide QRS with R’ > 0.12 seconds
In V1 and V2
(Broad S in V5 V6)
LBBB
Double QRS in V5, V6, >0.12 sec
Deep S in V1, V2
Effects of Calcium on EKG
Hypercalcemia: shortened QT
Hypocalcemia: prolonged QT, prolonged ST
Effects of Potassium on EKG
Hyperkalemia: wide/flat P, wide QRS, peaked T
Hypokalemia: flat T, U waves, prolonged QT
Effects of digitalis on EKG
Prolonged PR
Scooped out ST depression
Short QT, flat T waves
WPW EKG findings
Shortened PR interval with delta R wave