EKG and Heart Rhythms Flashcards
angina is obstruction of what blood vessel and who is at risk of death
LAD
younger pts bc no collateral vessels
P wave
depolarization of atria
QRS
depolarization of AV node
T wave
repolarization of ventricles
BNP
heart failure
troponin
muscle damage (MI)
series of 3 q8h
point of maximal impulse
the apex
midclav, 5th intercostal space
CTA
checks for hard calcium deposits, not soft
Normal PR
3-5 small boxes
Normal QRS
3 small boxes or less
sinus tach
100-160
SVT
160+
associated with overexertion, stress, deep inspiration, stimulants, disease, digitalis toxicity
PAC
ectopic site in atrium
premature P and QRS followed by pause
aflutter
saw tooth!
atrial 250-400
Typically associated with disease
Symptoms result from high ventricular rate and loss of atrial “kick” → decreased CO → risk of HF
afib
atrial >400
paroxysmal or persistent
in older ppl
in pts with underlying heart disease or other disease states
sinus brady mani
Hypotension
Pale, cool skin
Weakness
Angina
Dizziness or syncope
Confusion or disorientation
SOB
Can be normal during sleep
sinus tach causes
vagal inhibition (valsalva maneuver or bearing down stimulates vagus nerve, fixes sinus tach) or sympathetic stimulation
stress
drugs like albuterol
mani of sinus tach
dizziness
dyspnea
hypotension
angina
PAC causes
Stress
Fatigue
Caffeine
Tobacco
Alcohol (depletes electrolytes)
Hypoxia
Electrolyte imbalance
Disease states
mani of PAC
palpitations
heart “skips a beat”
treatment of PAC
Monitor for more serious dysrhythmias
Withhold sources of stimulation
Β-adrenergic blockers
aflutter treatment
Pharmacologic agent
Electrical cardioversion
Radiofrequency ablation
afib treatment
Drugs to convert ventricular rate and/or convert to sinus rhythm (amiodarone and ibutilide most common)
Electrical cardioversion
Anticoagulation (work on platelet aggregation, careful with ppl with high or low platelets)
Radiofrequency ablation
Maze procedure (also done thru angiogram, different technique) with cryoablation
mani of SVT
HR 150-220
180+ is decreased CO
hypotension
dyspnea
angina
SVT treatment
Vagal stimulation
IV adenosine
IV β-adrenergic blockers
Calcium channel blockers
Amiodarone
DC cardioversion
PVC treatment
correct cause
antidysrhythmics
torsades
Associated with heart disease, electrolyte imbalances, drugs, CNS disorder (GBS, parkinsons, spinal cord injury)
Can be stable (pt has a pulse) or unstable (no pulse, treat with CPR and defib)
treat cause
vfib
associated with MI, ischemia, diseases, procedures
no pulse or breathing
CPR and ACLS (defib, epi, vasopressin)
PEA
Electrical activity can be observed on the ECG, but no mechanical activity of the ventricles is evident, and the patient has no pulse
Hs of PEA
Hypovolemia
Hypoxia
Hydrogen ion (acidosis)
Hyper/hypokalemia
Hypoglycemia
Hypothermia
Ts of PEA
toxins
tamponade
thrombosis
tension pneumo
trauma
treatment of PEA
CPR, intubation, and epi
correct cause