EKG and electrolytes Flashcards
Potassium action
- K+ and NA+ pump intracellularly / extracellularly
- Determines conduction velocity
- Helps to confine pacing activity to the SA node
- Excess or deficiency can alter myocardial muscle function
hyperkalemia EKG changes
(1) Tall, peaked T-waves
(2) Prolonged PRI, loss of P waves, ST segment depression, widening QRS complex
(3) Dysrhythmias – heart block, ventricular fibrillation, cardiac standstill
hyperkalemia mechanism
(1) Decreases rate of ventricular depolarization (slows)
(2) Shortens repolarization (accelerates)
(3) Depresses AV conduction
hyperkalemia causes
(1) Impaired renal excretion
(2) Excess K+ administration
(3) K+ sparing diuretics (spiralactone), ACE inhibitors, ARB drugs
(4) Extensive skeletal muscle destruction
***(K+ leave cells bc muscle crushed, burned patient expects increase bc it releases K+ since cells destroyed and leak out of blood vessels; burn and crushed injuries leads to this)
overall: either consume too much, kidneys don’t work, or drug is sparing too much K)
hyperkalemia management
(1) Stop oral and IV intake of K+
(2) Increase K+ excretion
(3) Force K+ from ECF to ICF (insulin, bicarb, albuterol)
(4) Stabilize cardiac cell membrane
(DICK)
hypokalemia EKG changes
(1) Flattened T wave, depresses ST segment, p waves peak, QRS prolonged, U wave
(2) Risk for heart block, ventricular tachycardia, ventricular fibrillation
hypokalemia mechanisms
- impaires myocardial conduction
- prolongs ventricular replolarization
hypokalemia causes
(1) Increased loss of K+ (loop sparing diuretics, increase renal loss, diarrhea)
(2) Increased shift of K+ from ECF to ICF
(3) Dietary K+ deficiency
(4) Renal losses
hypokalemia management
Oral or IV K+ replacement
(a) High Alert Medication
(b) NEVER IV push, (Give 10 mEq/hour- infusion pump)
(c) Dilute K+ sufficiently & administer slowly
(d) Monitor for phlebitis
magnesium actions
- Essential for enzyme, protein, lipid, and carbohydrate functions in the body
- Extracellular level essential for normal cardiac muscle function
hypermagnesemia (RARE) EKG changes
like hyperkalemia
(1) Tall, peaked T-waves
(2) Prolonged PRI, loss of P waves, ST segment depression, widening QRS complex
(3) Dysrhythmias – heart block, ventricular fibrillation, cardiac standstill
if all electrolytes are abnormally low what do you replace first and why
magnesium since it will overpower K+ and deplete everything. so Mg first, K+ second, and Ca third
hypermagnesemia causes
increased intake
excess IV magnesium administration
hypomagnesemia EKG changes
(1) Prolonged ST segment, Prolonged QT interval
(2) Torsades de pointes, ventricular fibrillation
hypomagnesemia mechanism
(1) Impairs myocardial conduction
(2) Prolongs ventricular repolarization
(3) May lead to coronary artery spasm, hypertension