ekg 3 Flashcards

1
Q

atrial enlargement

A

produced by increased atrial work or volume overload

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2
Q

ventricular enlargement

A

is caused by clinical conditions that increase workload of the ventricle or volume overload

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3
Q

right atrial enlargement on ECG

A

tall peaked P wave in inferior leads (II, III, aVF)

tall initial upstroke of the P wave in V1 and terminal negative deflection

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4
Q

causes of right atrial enlargement

A

usually associated with right ventricular enlargement

  • pulmonary disease
    acute: bronchial asthma, PE
    chronic: emphysema, bronchitis

-congenital heart defects
ASD, pulmonic stenosis, ebstein’s anomaly, tetralogy of fallot

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5
Q

left atrial enlargement on ECG

A

notched M shape P wave in leads I, II and aVL

small initial upstroke of the P wave in V1 with deep terminal negative deflection

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6
Q

LAE think

A

mitral valve
p mitrale
P wave may be humped like an M

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7
Q

RAE think

A

p pulmonale
think pulmonary
tall and peaked p waves

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8
Q

right ventricular hypertrophy on EKG

A

right axis deviation
I and aVF -thumb

r wave in lead V1 exceeds the depth of S wave

RV strain pattern:
-T wave inversions with possible ST depression in leads V1 and V2

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9
Q

left ventricular hypertrophy ECG

A

left axis deviation

depth of S wave in V1 or V2 plus the height of R wave in V5 or V6 > 35 mm

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10
Q

right bundle branch block

A

QRS greater than 0.12 sec
V1 rSR pattern with final R wave (Big M wave)

V6 qRS pattern with final wide S wave

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11
Q

left bundle branch block

A

V1 QS pattern greater than 0.12 seconds

V6 wide tall R wave

inverted T waves in leads I aVL, V5 and V6

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12
Q

bundle branch turn signal method*

A

ONLY WORKS IN V1
go to j point at the end of QRS
draw a straight line into the QRS
connect this line with the peak or vally of the QRS complex
OBSERVE the direction the triangle points
down: LBBB
up: RBB

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13
Q

INVERT -causes of T wave inversion

A
I: ischemia and injury
N: normal variant
V: ventricular hypertrophy
E: ectopic foci (calcified plaque)
R: right and left bundle branch block
T: treatment (digitalis)
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14
Q

DEPRESSED ST- causes of ST segment depression

A
D: drooping mitral (prolapse)
E: enlargement of LV
P: potassium loss
R: reciprocal ST depression in  AMI
E: embolism in lungs
S: subendocardial ischemia
S: subendocardial infarct
E: encephalon hemorrhage
D: dilated cardiomyopathy
S: shock
T: treatments
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15
Q

Causes of ST segment elevation

A
E: electrolytes
L: LBBB
E: early repolarization
V: ventricular hypertrophy
A: aneurysm (ventricular)
T: treatment (pericardiocentesis)
I: injury (AMI, cardiac contusion)
O: osborne wave (hypothermia)
N: non-occlusive vasospasm (prinzmetal)
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16
Q

anterior wall MI

A

LEADS V3 -V4- tombstones so ST segment elevations

artery Left anterior descending

17
Q

Septal wall MI

A

leads V1-V2 ST elevation

artery: septal branches of the left anterior descending

18
Q

interior wall MI

A

leads II, III, aVF
ST segments elevation
right coronary artery

19
Q

lateral wall MI

A

Leads I aVL V5 V6 ST elevation

left circumflex artery

20
Q

posterior wall MI

A

large R wave in V1-V2

is the inferior wall subset so you have to do right sided ecg if you have an inferior wall mi

21
Q

posterior wall mi artery

A

posterior descending coronary artery

do not use nitrates

22
Q

prinzmetal angina

A

st elevatins in affected leads with resolution at baseline

23
Q

pericarditis ecg

A

diffuse ST elevation in inferior and anterior leads

24
Q

pericardial effusion

A

low voltage: amplitude of QRS (muted) complexes in each of the 6 extremities

  • sill have varing amplitudes of the QRS because the heart is swinging in the fluid
25
Q

WPW ecg

A

AV nodal bypassing

-delta waves: slurring of the initial portion of the QRS complex

26
Q

can have WPW and a-fib

A

looks like v-tach but look for delta

27
Q

WPW medication to avoid

A

adenosine
belta-blocker
calcium channel blocker
digoxin

28
Q

WPW what meds to use

A

amiodarone

procainamine