EKG Flashcards

1
Q

what is conductivity?

A

ability for cardiac cells to transmit action potential from one cell to another

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2
Q

what is rhythmicity?

A

ability for cardiac cells to spontaneously generate an action potential at a regular rate

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3
Q

what is a normal SA node rate?

A

60-100 BPM

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4
Q

if the SA node is blocked or damaged, ____ will step up to be the pacemaker of the heart

A

AV node

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5
Q

if the AV node is damaged, then ________ will take over to maintain cardiac output to keep the patient alive

A

the Purkinje fibers

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6
Q

what is the normal rate for the purkinje fibers?

A

15-40 BPM

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7
Q

what does a P wave show?

A

atrial depolarization atrial contraction & shows when the SA node is firing

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8
Q

what does the PR interval show?

A

time in seconds from onset of atrial depolarization to onset of ventricular depolarization

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9
Q

what does the QRS interval show?

A

ventricular depolarization & atrial repolarization; time it takes for the impulse to travel through the bundle branches & purkinje fibers

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10
Q

what is the J point?

A

point at which the QRS returns to the isoelectric line; marks beginning of ST segment

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11
Q

what does a normal ST segment look like?

A

flat & same level as the isoelectric baseline

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12
Q

what may an elevated or depressed ST segment indicate?

A

an MI!

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13
Q

what does a T wave show?

A

ventricular repolarization (ability of the heart to have another action potential)

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14
Q

what can peaked T waves be caused by?

A

high potassium levels

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15
Q

what can flat T waves be caused by?

A

low potassium levels

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16
Q

what does a QT interval show?

A

time in seconds from beginning of ventricular depolarization to the end of ventricular repolarization

17
Q

what is another way to calculate heart rate in addition to counting each R complex?

A

counting the number of small boxes between the R to R complexes and dividing 1500 by that number

18
Q

what is a normal PR interval?

A

0.12-0.20 or 3-5 little boxes

19
Q

what is a normal QRS interval?

A

< 0.12 or less than 3 little boxes

20
Q

what is considered a normal QT interval?

A

“normal” determined according to heart rate; less than half of the R-R interval

21
Q

which med may cause a prolonged QT?

A

Amiodarone

22
Q

if a PR interval is prolonged, what does this mean & what is this considered?

A

it is taking too long for the electrical impulse to leave the AV node; it’s considered a First-Degree AV block

23
Q

if a QRS interval is prolonged, what does this mean & what is this considered?

A

it is taking too long for the electrical impulse to travel down the bundle branches; it’s called a bundle branch block (patients w this may have had previous MIs & there is damage to that bundle branch)

24
Q

what does depression or elevation of an ST segment determine?

A

determines whether there is an acute MI, STEMI or N-stemi

25
Q

what do absence of P waves determine?

A

atrial dysrhymias, afib & aflutter

26
Q

what do you do if a patient has an artifact EKG reading?

A

reposition their leads

27
Q

what can bradycardia be caused by or associated with?

A

sleep, severe pain, inferior wall myocardial ischemia & spinal cord injuries
meds: Digitalis, beta-blockers & Ca channel blockers (cardizem)

28
Q

how do you treat a patient for bradycardia?

A

assess them first! if they are asymptomatic, no treatment is needed. if they are symptomatic, administer atropine 1.0 mg IV every 3-5 min (3 mg max)

29
Q

if Atropine is ineffective, what is done?

A

transcutaneous pacing (TCP); pacing pads are placed over the skin & pacer machine is applied to increase the patient’s cardiac rate

30
Q

which med infusions may be used as an alternative to TCP?

A

Dopamine (5-20 mcg/kg/min) & epinephrine (2-10mcg/min)

31
Q

what are potential causes of sinus tachy R/T increased sympathetic tone?

A

stress, exercise, stimulants (caffeine & nicotine)

32
Q

what are potential causes of sinus tachy associated w clinical problems?

A

fever, anemia, hyperthyroidism, hypoxemia, heart failure & shock

33
Q

which drugs can be a cause of sinus tachy?

A

Atropine (blocks vagal tone allowing sympathetic tone to take over), epinephrine & dopamine

34
Q

what is the first line treatment for sinus tachy?

A

treat the cause! (fix underlying issues e.g, oxygen, sedation, fluids & blood)

35
Q

which meds are used to treat sinus tachy?

A

beta-blockers or Ca channel blockers ( both decrease HR)

36
Q

which meds can cause sinus dysrhymias / arrhymias?

A

digitalis or morphine

37
Q

how can you tell when a dysrhymia is present on an EKG strip?

A

if the RR intervals are irregular by more than 0.12 seconds; the rate gradually increases w inspiration & gradually decreases w expiration

38
Q

do sinus dysrhymias / arrhymias require treatment?

A

generally do not unless there are long pauses!