EENT Emergencies Flashcards

1
Q

Corneal Abrasion

A

*Most common eye injury
*Corneal epithelium is scraped or
compromised

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2
Q

Corneal Abrasion presentation

A

*Symptoms unilateral
*Foreign body sensation
*Eye pain
*Inability to open eye
*Photophobia
*Excessive tearing

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3
Q

Corneal Abrasion diagnosis

A

*No obvious laceration on initial exam
*Assess visual acuity
*Instill topical anesthetic
*Fluorescein & Wood’s lamp
*Slit lamp examination if no obvious
abrasion
*If penetrating injury suspected
*CT or MRI
*If ulcer suspected, obtain cultures
prior to antibiotics

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4
Q

Corneal Abrasion treatment

A

*Prophylactic topical antibiotics until patient is
asymptomatic
*Ciprofloxacin, tobramycin, trimethoprim/polymyxin B
*Pain relief
*Oral narcotic
*Topical NSAID
*Diclofenac (Voltaren) or ketorolac (Acular)
*Long-acting cycloplegic agent can provide significant relief for
photophobia and blepharospasm
* Cyclopentolate
* Contraindicated in narrow-angle glaucoma

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5
Q

T/F Small abrasions can be managed
outpatient

A

T

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6
Q

Abrasions from contacts=_____

A

pseudomonas

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7
Q

Abrasions from _____ r=fungal

A

vegetable matte

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8
Q

Corneal Foreign Body presentation

A

*Red eye
*Pain
*Foreign body sensation
*Photophobia
*Tearing

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9
Q

Corneal Foreign Body exam

A

*Visual acuity may be decreased
*Conjunctival injection
*Visible foreign body
*Epithelial defect that stains with
fluorescein
*Rust ring
*Anterior chamber cell/flare

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10
Q

Corneal Foreign Body diagnosis

A

*Exclude intraocular foreign body
*Orbital CT
*B-scan ultrasound
*Ultrasound biomicroscopy (UBM)
*If metallic object suspected,
consider x-ray as initial study
Slit lamp
*Full-dilatation examination by an
ophthalmologist

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11
Q

Corneal Foreign Body treatment

A

*Remove foreign body
*Ophthalmologist consult
*Infectious corneal infiltrates/ulcers generally require
scrapings for smears and cultures
*Topical antibiotics
*Polymyxin B sulfate-trimethoprim, ofloxacin, tobramycin,
ciprofloxacin
*Antibiotics containing steroids are contraindicated
*Topical cycloplegic (cyclopentolate) for pain and
photophobia

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12
Q

*Do NOT patch if any of the following are present:

A

*A chance of a perforation of the globe exists
*A corneal infiltrate is present
*A chance of a retained intraocular foreign body is possible

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13
Q

Acute Angle-Closure Glaucoma risk factors

A
  • Family history
    *Age over 60 years
    *Hyperopia
  • Female
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14
Q

Acute Angle-Closure Glaucoma presentation

A

*Rapid diagnosis, immediate intervention, and referral can have
profound effects on patient outcome and morbidity
*At least 2 of the following:
*Ocular pain
*Nausea/vomiting
*History of intermittent blurring of vision with halos
*AND at least 3 of the following:
*IOP greater than 21 mm Hg
*Conjunctival injection
*Corneal epithelial edema
*Mid-dilated nonreactive pupil
*Shallower chamber in the presence of occlusion
*Periorbital pain
*Ipsilateral headache with boring pain
*Nausea and vomiting
*Visual deficits
*Halos around objects

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15
Q

Acute Angle-Closure Glaucoma examination

A

*Cornea and scleral injection, ciliary flush
*Visual acuity, visual fields, fundoscopic
exam, ocular motility
*Pain on eye movement, a mid-dilated fixed
pupil, and a firm globe

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16
Q

Acute Angle-Closure Glaucoma diagnosis

A

*IOP via tonometry
*Slit-lamp
*Corneal edema, irregular pupil shape,
synechiae(adhesions), segmental iris
atrophy
*Gonioscopy
*Allows visualization of the angle

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17
Q

*IOP should be checked ____ minutes
after administering drops for Acute Angle-Closure Glaucoma

A

30-60

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18
Q

Orbital Cellulitis

A

*Infection of the soft tissues of the
orbit posterior to the orbital
septum
*10% of cases result in vision loss
*Recent facial trauma or surgery,
dental work, or infection
elsewhere in the body are risk
factors
Periorbital cellulitis- skin of the
eyelid and tissue around the eye

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19
Q

Orbital Cellulitis presentation

A

*Fever, malaise, and a history of
recent sinusitis or URI
*Conjunctival chemosis
*Decreased vision
*Pain on eye movement
*Headache
*Lid edema
*Rhinorrhea

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20
Q

Orbital Cellulitis exam

A

*Ophthalmoplegia
*Dyschromatopsia and relative afferent pupillary defect
*Slit Lamp, elevated IOP
*Orbital pain and tenderness (present early)
*Dark red discoloration of the eyelids, chemosis, hyperemia of the
conjunctiva, and resistance to retropulsion of the globe may be present
*Purulent nasal discharge may be present

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21
Q

Orbital Cellulitis diagnosis

A

*CBC c diff, blood cultures prior to
antibiotics, cultures
*High-resolution contrast CT scan of the
orbit with axial and coronal views
*Rule out peridural and parenchymal brain
abscess formation
*MRI
*Lumbar puncture if cerebral or
meningeal signs develop

22
Q

Orbital Cellulitis treatment

A

*Admit for inpatient care until afebrile x 48 hours and clearly improved
* Broad-spectrum IV antibiotics should be started immediately
* Vancomycin + Ceftriaxone and Metronidazole
*Canthotomy and cantholysis- orbital compartment syndrome
*Surgical drainage
*Periorbital maybe more mild, and oral antibiotics could be used
*Fungal infection requires IV antifungal therapy along with surgery
*Ophthalmology and ENT consults, possibly infectious disease

23
Q

Orbital Blowout Fracture

A

*Force of blow → backward
displacement of eyeball → intraorbital
hydraulic pressure increases
→fracture in the weakest point of the
orbital wall
* A high-velocity object that impacts
the globe and upper eyelid transmits
kinetic energy to the periocular
structures

24
Q

Orbital Blowout Fracture presentation

A

*Facial trauma
*Decreased visual acuity
*Periorbital ecchymosis and edema
*Hypesthesia extending through the
region of the maxillary nerve
*Diplopia
*Decreased extraocular movement

25
Orbital Blowout Fracture diagnosis
*Maxillofacial CT scan
26
Orbital Blowout Fracture treatment
*Systemic antibiotic and no blowing the nose *Reassess in 1 week *Patients who present without significant enophthalmos (2 mm or more), a lack of marked hypo-ophthalmos, absence of an entrapped muscle or tissue, a fracture less than 50% of the floor, or a lack of diplopia may not need surgery
27
Cardinal sign of ocular inflammation
*Conjunctivitis *Blepharitis *Canaliculitis *Keratitis *Dacryocystitis *Scleritis *Episcleritis *Corneal Injury *Iritis *Dry eye syndrome *Glaucoma *Subconjunctival hemorrhage
28
Red flags with red eye presentation
*Decreased visual acuity *Severe, deep eye pain *Ciliary flush * A pattern of injection in which the redness is most pronounced in a ring at the limbus *Photophobia *Severe foreign body sensation that prevents the eye from staying open *Corneal opacity *Fixed pupil *Severe headache with nausea
29
Peritonsillar Abscess (PTA)
*Form between the tonsil and capsule *Usually progress from tonsillitis → cellulitis → abscess formation *Weber glands *Mean age is 20-30 years
30
Peritonsillar Abscess presentation
* Symptoms usually begin 3-5 days prior to evaluation * Fever * Malaise *Headache *Neck pain * Throat pain markedly more severe on the affected side and occasionally referred to the ipsilateral ear *Dysphagia * Change in voice *Otalgia *Odynophagia
31
Peritonsillar Abscess exam
* Mild/moderate distress * Fever * Tachycardia * Dehydration * Drooling, salivation, trouble handling oral secretions * Trismus resulting from pain from inflammation and spasm of masticator muscles * Hot potato/muffled voice * Rancid or fetor breath * Cervical lymphadenitis in the anterior chain * Asymmetric tonsillar hypertrophy * Localized fluctuance * Inferior and medial displacement of the tonsil * Contralateral deviation of the uvula * Erythema of the tonsil * Exudates on the tonsil
32
Peritonsillar Abscess diagnosis
*Testing is not necessary in straightforward cases *If questionable diagnosis *CT scan
33
Peritonsillar Abscess treatment
*Pay close attention to the patient's airway *If the patient's airway is compromised, immediate endotracheal intubation is indicated *Fluid resuscitation *Pain medication *Oral steroids *Empiric antibiotics *Clindamycin, Omnicef, or Augmentin *Can cause sepsis or carotid artery erosion if left untreated
34
Peritonsillar Abscess I&D prep
* Refer to ENT if needed * The patient should be sitting upright in an examination chair * Spray the tonsil and ipsilateral soft palate with benzocaine (eg, Cetacaine) *Using an 18-gauge needle on a 10-mL syringe, draw up approximately 6-10 mL of lidocaine 1% with epinephrine * Change to a 27-ga needle (preferably a long needle) *Inject the mucosa overlying the fluctuant area with local anesthetic
35
Medications for peritonsillar abscess
Clindamycin TID, Percocet, Medrol, tetracaine lollipops
36
Epiglottitis
*Acute inflammation in the supraglottic region of the oropharynx with inflammation of the epiglottis, vallecula, arytenoids, and aryepiglottic folds *Haemophilus influenzae (25%) *Noninfectious causes *Reaction to chemotherapy *Thermal (crack cocaine, throat burns in bottle fed infants) *Caustic insults
37
Epiglottitis presentation
*Onset and progression of symptoms of epiglottitis is rapid *Sore throat (95%) *Odynophagia/dysphagia (95%) *Muffled voice (54%) - "Hot potato voice" *Adults may have preceding URI symptoms
38
Epiglottitis exam
* Tripod position - Sitting up on hands, with the tongue out and the head forward * Drooling/inability to handle secretions * Stridor: A late finding indicating advanced airway obstruction * Muffled voice (54%) * Cervical adenopathy * Fever * Hypoxia * Respiratory distress * Severe pain on gentle palpation over the larynx or hyoid bone * Mild cough * Irritability * Tachycardia * Toxic appearance of patient
39
Epiglottitis diagnosis
*Direct visualization of the epiglottis *Nasopharyngoscopy/laryngoscopy = ENT *Cherry red epiglottis *The airway should be secured or should be readily securable if endoscopy is performed *Lateral neck soft-tissue radiographs *Avoid radiography for patients in distress until the airway is secure *Swollen epiglottis (thumb sign)
40
Epiglottitis treatment
* DO NOT ATTEMPT DIRECT VISUALIZATION OF THE EPIGLOTTIS * Reflex laryngospasm * Airway management is the most urgent consideration * Signs and symptoms associated with a need for intubation include respiratory distress, airway compromise on examination, stridor, inability to swallow, drooling, sitting erect, and deterioration within 8-12 hours * When in doubt, securing the airway is the safest approach * 18.1% require airway management * Patients without signs of airway compromise may be managed without immediate airway intervention by close monitoring in the intensive care unit (ICU) * Avoid therapy such as sedation, inhalers, or racemic epinephrine * Close contacts of patients in whom Haemophilus influenzae type b is isolated should receive rifampin prophylaxis (20 mg/kg; not to exceed 600 mg/d for 4 d)
41
Dental abscess
*Bacteria from the tooth can extend into the gums, the cheek, the throat, the tissues beneath the tongue, or even into the jaw or facial bones *Pus often collects at the site of the infection as the immune system tries to keep the infection from spreading *Can result in death if it spreads to the brain, causes septicemia, or if swelling obstructs the airway *A gum or gingival abscess is the result of infection or trauma to the surface of the gum tissue *Periodontal abscesses are the result of an infection that has moved deeper into gum areas *Periapical abscess refers to a tooth with an infection of the dental pulp
42
Dental abscess presentation
*Pain, swelling, redness *One or more teeth will be very sensitive to pressure *Nausea/vomiting *Fever/chills *Difficulty swallowing *Difficulty opening mouth *Difficulty breathing *Dehydration
43
Dental abscess exam
*Swelling *Warmth *Erythema *Fluctuant mass *Extends toward the buccal side of the gum and to the gingival-buccal reflection *Parulis *Trismus *Necrotizing fasciitis *Teeth *Most frequently the lower third molar, followed by other lower posterior teeth *Increased mobility *Tenderness with percussion *Extrusion
44
Dental abscess diagnosis
*No labs indicated for uncomplicated abscess *If accompanying cellulitis *Leukocytosis with neutrophil predominance *Aerobic and anaerobic blood cultures prior to IV antibiotics *Panorex dental x-rays- not done in the ER *Lateral and neck x-rays *CT scan *Needle aspiration *1-2 drops
45
Dental abscess treatment
Treatment *Assess the airway and secure if necessary *Cultures *Antibiotic *Oral vs. IV *Hydration *Pain management *I & D *F/u discharge referrals *Uncomplicated → dentist *Complicated → maxillofacial oral surgeon/ENT
46
Epistaxis risk factors
*Rhinotillexis/mucophagy *Dry climate *Supplemental oxygen *Nasal anatomy *Trauma *Coagulopathy *Foreign body *Iatrogenic *Infection
47
Epistaxis presentation
*Anterior * 90% of bleeds occur anteriorly * Capillary or venous bleeds * Patients present with a constant ooze or intermittent bleeding that resolves quickly * Posterior * Often of arterial origin * Branches of the sphenopalatine artery * Associated with atherosclerotic disease and hypertension * Patients present with profuse bleeding
48
Epistaxis treatment
Labs and imaging usually not required Treatment of anterior epistaxis * Direct pressure continuously for 15 minutes * Sitting position * Slightly leaning forward * Topical nasal decongestants * Topical 4% cocaine * Silver nitrate * Surgicel * ENT referral if recurrent or persistent
49
Treatment of posterior epistaxis
* ENT ER consult * Rigid nasal endoscopy * Cauterize * Rapid Rhino *Antibiotic * F/u with ENT 3-4 days later
50
BPPV presentation
*Begins suddenly upon waking *Severe spinning *Episodes are brief, less than 1-2 minutes *Triggered by sudden head movement, often unilateral *Lag period *May have no symptoms between episodes *“Mentally foggy” *Exam usually WNL
51
BPPV diagnosis
*Dix Hallpike *Classic rotatory nystagmus with latency and limited duration *In classic posterior canal BPPV, the top pole of the eyes rotates toward the undermost (affected) ear *Imaging typically not indicated in uncomplicated BPPV that resolves with treatment
52
BPPV treatment
* Rehydration *Observation * Benign and can resolve spontaneously * Medication to suppress the vestibular system * Diazepam, meclizine * Canalith repositioning (Epley maneuver) * Vestibular rehabilitation * Patient education * Most cases resolve within 6 weeks * Recurrence rate of 15-20%