ECGs and arrhythmias Flashcards

1
Q

How would you interpret an ECG?

The steps

A
  1. Determine rate
    • 300/no. of large squares
  2. Determine rhythm
    • Sinus rhythm = each P wave followed by QRS, constant PR interval
  3. Determine cardiac axis
    • Normal = I & II postive
    • Right axis deviation = I negative, III positive
    • Left axis deviation = II & III negative
  4. Assess P waves
    • Peak/tall in right atrial hypertrophy
    • Notched/broad in left atrial hypertrophy
  5. Assess PR interval
    • Time from beginning of the P wave to the beginning of QRS
    • The normal range in 0.12-0.2 sec (3-5 small squares)
    • If longer than 0.22s; first degree heart block
  6. Assess the QRS complex
    • QRS width: wider than 120ms = ventricular origin/bundle branch block
    • QRS height: tall R waves in V1 = Right ventricular hypertrophy, tall R waves in V6 = left ventricular hypertrophy
  7. Assess the ST segment
    • Elevation in MI/pericarditis
    • Depression in ischaemia/digoxin
  8. Assess for pathological T wave inversion
    • T wave inversion in aVR, III and V1/2 can be normal
  9. Assess for Q waves
    • Q waves indicate old infarction - normal in LV leads: I, VL, V5/6
  10. Assess the QT interval
    • From beginning of QRS to end of the T wave
    • Normal is <0.45s, approx. 2 large squares
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3
Q

How should you correctly set up an ECG machine?

A
  • Skin must be clean and dry
  • V1/2 are positioned in the 4th intercostal space either side of the sternum
    • Palpate the angle of Louis, and the 2nd intercostal space is adjacent
  • V4 is positioned in the 5th intercostal space, mid-clavicular line
  • V3 is placed between V2 and V4
  • aVR and aVL go on right and left arms respectively
  • aVF goes on the left ankle
  • There is a neutral lead that is placed on the right ankle
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4
Q

Describe the QRS complex

Abnormalities?

A
  • If the first deflection is downward, then it is a ‘Q’ wave
  • The first upward deflection is always the’R’ wave (regardless of if there has been a Q wave)
  • Any deflection below baseline following the R wave is the S wave
  • QRS is usually <120ms, and if this is prolonged it represents bundle branch block or depolarisation from a ventricular focus
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5
Q

How should the T wave be seen on an ECG?

What are T wave abnormalities and causes?

A
  • The T wave is normally inverted in aVR, III and V1/2
    • Sometimes V3 in black people
  • Otherwise, T wave inversion may represent several pathologies:
    • Ischaemia
      • Occurs in both STEMI and NSTEMI
      • T waves become inverted over 24-48h, often permanent
    • Ventricular hypertrophy
      • In leads looking at the involved ventricles
    • Bilateral bundle branch block
    • Digoxin treatment
      • Also causes classical sloped ST segments
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6
Q

Why is the ECG trace pattern the way it is?

Why are the waves where they are?

How does it work?

A
  • Myocardial contraction causes fibre depolarisation, which is detected by electrodes on the body
  • The ECG trace should represent the normal electrical activity of the heart;
    • SAN ⇒ atrial depolarisation ⇒ AVN delay ⇒ septal depolarisation ⇒ ventricular depolarisation via bundle of His
  • If the electrical activity starts at the SAN, the heart is described as being ‘sinus rhythm’ i.e. a ‘P wave’ is present, representing atrial depolarisation
  • Depolarisation of the larger mass of the ventricles causes the much larger ‘QRS complex
  • The T wave then represents the repolarisation of the ventricular mass back to its resting state
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8
Q

What happens in a Right bundle branch block?

How is it seen on an ECG trace?

A
  • The septum is depolarized from the left side as normal, causing an R wave in V1 and a small Q wave in V6
  • Excitation on the left ventricle causes an S wave in V1 and R wave in V6
  • As it takes longer for excitation to reach the right ventricle, this depolarises after the left, causing a second R wave (R1) in V1, and a deep S wave in V6
  • RBBB is best seen in V1 (RSR1)
    • Ma_rr_oW: ‘M’ shape in V1, ‘W’ shape in V6

https://lifeinthefastlane.com/ecg-library/basics/right-bundle-branch-block/

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9
Q

What happens in a left bundle branch block?

How is shown on an ECG trace?

A
  • The septum will depolarize from right to left, causing Q wave in V1 and an R wave in V6
  • The right ventricle is depolarised before the left ventricle, so there will be a small R wave in V1 and an S wave in V6 (although this often will appear only as a notch)
  • Subsequent depolarization of the left ventricle causes an S wave in V1 and another R wave in V6
  • LBBB is associated with T wave inversion in the lateral leads (I, VI and V5/6 altough not necessarily in all of these)
  • LBBB is best seen in V6 (‘broad ‘M’ complex), and the ‘W’ pattern in V1 is often not fully developed
    • WilliaM’: ‘W’ shape in V1, ‘M’ shape in V6

https://lifeinthefastlane.com/ecg-library/basics/left-bundle-branch-block/

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11
Q

What ST abnormalities in an ECG tracing can occur?

What do they mean?

A
  • The ST segment should be ‘isoelectric’, i.e. at the same level as the part between the T wave and the next P wave
  • ST elevation indicates acute myocardial injury; either a recent infection or due to pericarditis
    • Pericarditis is not localised and thus it causes ST elevation in most leads
  • ST depression is usually a sign of ischaemia rather than MI
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12
Q

What is a branch block?

How is it seen on an ECG trace?

A
  • The depolarization waves reaches the septum normally, so PR interval is normal, yet there is abnormal conduction through left/right bundle branches (of His)
  • Delayed depolarization of the ventricles thus leads to a wide QRS (>120ms)
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13
Q

The ECG is made up of 12 characteristic ‘leads’ that view the heart from different directions. What are the directions of each lead?

A
  • The six limb leads (I, II, III, VR, VL, VF) look at the heart in a vertical plane
    • aVR: right ventricle
    • aVL, I: left heart surface
    • II, III & aVF: inferior surface
  • The six V (chest leads - V1-6) look in a horizontal plane
    • V1/2: right ventricle
    • V3/4: septal area
    • V5/6: left ventricle
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16
Q

What does the PR interval show?

What if it is longer in length?

A
  • Time from the start of P to the start of the QRS complex
  • Represents the time taken for excitation to spread from the SAN to the ventricular muscle
    • Time is mostly made up of AVN delay
  • If >0.2s - 5 squares, there may be heart block
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17
Q

What is first degree heart block?

How is it seen on an ECG?

What can it indicate?

A
  • First-degree atrioventricular block is a disease of the electrical conduction system of the heart in which the PR interval is lengthened beyond 0.22 seconds.
  • PR interval >0.22 seconds
  • First degree heart block is not patholigical in itself but can indicate:
    • Coronary artery disease
    • Acute rheumatic fever
    • Electrolyte disturbances
    • Digoxin toxicity
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18
Q

Where is the ST segment?

What may changes show?

How big must these changes be?

A
  • Interval between the end of the S wave and the beginning of the T wave
    • Changes may represent myocardial ischaemia
  • ST elevation must be >2mm in a chest lead, >1mm in a limb lead, on two leads to be significant
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19
Q

What is complete/third degree heart block?

How is it seen on an ECG trace?

What happens to ventricle contraction?

What can cause 3rd degree heart block?

A
  • Atrial contraction is normal but no beats are conducted to the ventricles
  • P waves will be dissociated from the QRS complexes
  • The ventricles are excited by a ‘slow escape mechanism’ from a depolarising focus within the ventricles, giving a wide QRS
  • Can occur acutely following MI or be a chronic state (the chronic state indicates conducting tissue disease)
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20
Q

Where is the QT interval?

What can cause a prolonged QT interval?

What may this cause?

A
  • Time from the start of Q to the end of T
  • Varies with heart rate but can be prolonged by drugs/electrolyte abnormalities
  • A prolonged QT interval may lead to ventricular tachycardia
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22
Q

What may cardiac axis deviations be a sign of?

A

Axis deviations are not significant in themselves, yet their presence should alert to look for other signs of left or right ventricular hypertropy or other causes such as pulmonary embolus (right axis deviation) or conduction defects (left axis deviation)

23
Q

What are signs of normal axis, right and left axis deviation?

A

Normal = I & II positive

RAD = I negative, III positive

LAD = II & III negative

24
Q

Describe atrial fibrilation?

What is seen on an ECG?

Symptoms of AF?

A
  • Irregular baseline with no P waves
  • The AVN is bombarded and thus will depolarize irregularly, leading to ventricular contraction at an irregular rate;
    • Usually 450-600 atrial contractions per minute
    • Normal QRS as conduction from the AVN is not abnormal
  • Can be asymptomatic, or present with dyspnoea, palpitations, syncope, chest pain or stroke/TIA
25
Q

Describe atrial flutter?

What is seen on an ECG?

Similarities and differences with atrial fibrilation?

A
  • If the atrial rate is above 250/min and no flat rate baseline between P waves exists, atrial flutter is present
    • Normally 300-450 contractions per minute
    • Classic ‘saw-toothed’ baseline
  • It can be thought of similar to atrial fib. in that the normal co-ordination of the atria is lost, however some element of synchronicity still exists
26
Q

Compare atrial fibrilation and atrial flutter on an ECG

A
27
Q

What is Wolff-Parkinson-White (WPW) syndrome?

What is seen on an ECG?

A
  • Wolff–Parkinson–White syndrome (WPW) is a disorder due to a specific type of problem with the electrical system of the heart which has resulted in symptoms.
  • Some people have an accessory conducting bundle alongside the bundle of His, usually in the left hand side of the heart, and unconnected to the AVN
  • As there is no AVN delay, ‘pre-excitation’ occurs with a short PR interval and the QRS shows an early, alurred upstroke called a ‘delta wave’
  • The second part of QRS is normal as the bundle of His conduction catches up
  • The ECG rhythm is sinus, but there is right axis deviation, short PR interval and widened QRS with ‘delta wave’ of pre-excitation
  • The obnly clinical significance of this is that it can cause paroxysmal supraventricular tachycardia
28
Q

What is heart block?

A

Abnormal conduction from the SAN to the ventricles

30
Q

What is second degree heart block?

What are the variations of it?

How is it seen on an ECG?

What can it indicate?

A
  • Second-degree atrioventricular block is a conduction block between the atria and ventricles. Excitation intermittently fails to pass through the AVN or bundle of His
  • There are 3 variations:
    • ‘Mobitz type 2’ phenomenon: constant PR interval yet sometimes there is atrial contraction without ventricular contraction
    • ‘Wenckebach’ phenomenon: progressive PR lengthening until an atrial beat is not concluted, and then this cycle repeats
      • Also known as Mobitz type 1
    • 2:1/3:1 conduction: twice as many P waves as QRS complexes

https: //lifeinthefastlane.com/ecg-library/basics/mobitz-2/
https: //lifeinthefastlane.com/ecg-library/basics/wenckebach/

32
Q

How is sinus bradycardia defined?

How is sinus tachycardia defined?

A

Sinus bradycardia <60 beats per min

Sinus tachycardia >100 beats per min

33
Q

What is an extrasystole?

A
  • If any part of the heart depolarizes quicker than it sound, and this is accompanied by an extra heart beat, this is termed an extrasystole
34
Q

Non-sinus rhythms:

Where can they occur in heart from?

How do they differ on an ECG?

A
  • Abnormal rhythms begin in one of 3 places: atrail muscle, ventricular msucle or AVN (nodal or junctional rhythm)
  • Sinus artial and junctional rhythms are know as ‘superventricular’ and in these the QRS complex is normal as the depolarization spreads to the ventricles in the usual way via the bundles
    • Sinus rhythms give a normal P wave
    • Atrial rhythms give an abnormal P wave
    • Junctional rhythms will not show P waves
  • Ventricular rhythms on the other hand give wide (slower spread of depolarization throigh the ventricles) and abnormal QRS complexes
35
Q

What is supraventricular tachycardia?

4 main types?

Symptoms?

A
  • Supraventricular tachycardia is an abnormally fast heart rhythm (>100) arising from improper electrical activity in the upper part of the heart
  • There are four main types:
    • atrial fibrillation
    • paroxysmal supraventricular tachycardia (PSVT)
    • atrial flutter
    • Wolff-Parkinson-White syndrome
  • Symptoms may include palpitations, feeling faint, sweating, shortness of breath, or chest pain
40
Q

What is ventricular tachycardia?

How is it seen on an ECG?

A
  • Ventricular tachycardia is a type of regular and fast heart rate that arises from improper electrical activity in the ventricles of the heart

ECG:

  • Wide, abnormal QRS seen in all 12 leads
  • Broad complex tachycardia
  • Extreme axis deviation
  • P and QRS at different rates
  • Potential to transform to VF
41
Q

What is ventricular fibrillation?

How is it seen on an ECG?

A
  • Ventricular fibrillation is when the heart quivers instead of pumping due to disorganized electrical activity in the ventricles
  • It is a type of cardiac arrhythmia Ventricular fibrillation results in cardiac arrest with loss of consciousness and no pulse

ECG:

  • No QRS can be identified and the ECG is totally disorganised
  • The patient will have lost consciousness
42
Q

Describe the ECG features of STEMI and the changes over time

A

ST elevation, then Q wave and T wave inversion begin to develop, Q wave grows larger then stays large, T wave inverts a long way and then reverts a slight way (to be less inverted).