Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Complex 2 > ECGs > Flashcards

ECGs Flashcards

1
Q

Arrhythmias (dysarrhythmia)

A

Abnormal cardiac rhythm

*Prompt assessment of abnormal cardiac rhythm and patients response is critical

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

12 - Lead ECG

A 
Looks at 12 areas in the heart
Anterior
Lateral sides
Septum
NOT posterior or right ventricle
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

P

A

Atrium depolarization ( SA node fires causing atrium to contract) if patient has a P wave they are in sinus rhythm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

PR interval

  1. What does it measure
  2. What is the normal PR interval
  3. What is the PR interval represent
A
  1. Measures from the beginning of the P wave to the beginning of the of the QRS complex.
  2. Normal PR interval is 0.12-0.20 (5 small squares)
  3. The PR interval represents atrial depolarization
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

QRS Complex

  1. What does it measure
  2. What is the normal QRS complex
  3. What is the QRS complex represent
A
  1. Measure this complex from the beginning of the Q wave until the end of the S wave.
  2. The normal QRS complex is 0.04-0.10 seconds (2 1/2 small squares)
  3. The QRS complex represents ventricular depolarization…… Atrium repolarization
    NB: Remember that not all QRS complexes contain all the QRS waves! Q-first deflection is negative R- Postive deflection after a Q wave S- Negative deflection after an R wave
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

T wave

A

Repolarization of ventricles (relax and resting)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

QT interval

  1. Where to measure it
  2. What does it measure?
  3. Normal?
  4. Prolonged QT interval means?
A
  1. Beginning of QRS till the end of the T wave
  2. Measures the length of the time it takes for the ventricle to contract and relax
  3. Normal QT interval: 0.35-0.45 seconds
  4. Can lead to life-threatening arrythmias ( low magnesium (alcohol can lower))
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

ST segment

  1. below the isoelectric line
  2. Above the isoelectric line
A

Place a ruler under the PR interval
1** If the bottom part of the ST segment line is more than one small square (1mm) below the PR interval, then the patient is having myocardial ischemia
2** If the bottom part of the ST segment is more than small square(1mm) above the PR interval, then the patient is probably having a MI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Small square seconds

A

0.04 second

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Large square seconds

A

0.02 seconds

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Number of large squares in a 6-second strip

A

30

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Phases of Cardiac Action Potential

A

phase 0: Upstroke or rapid depolarization and corresponds with ventricular contraction
: Initial impulse
Phase 1,2,3 Repolarization
1: Overshot
2: plato (little change) opening of the Ca channels helps to sustain contraction… CCB work here
If someone has a low EF (

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

If the rhythm is regular 2 methods to estimate HR
1.
2.

A
  1. Count the number of QRS complexes in a 6 strip x 10
  2. Count number of small squares between two R waves and divide into 1500.
    Count number of large squares between two R waves and divide into 300
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Steps in assessing the Cardiac Rhythm

Steps 1-7

A 
Step 1: Heart rhythm (regular vs irregular) P-P/ R-R intervals identical
Step 2. Heart Rate
Step 3. P waves
Step 4. PR interval
Step 5. QRS Complex
Step 6. ST Segment
Step 7. T wave
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Normal Sinus rhythm ECG characteristics

A 
Rate: 60-100 bpm
P present
P waves precede every QRS complex
P-R ratio is 1:1
Causes: normal
Tx: always be sure your patient has a pulse
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

The absolute refractory period

A

occurs when excitability is zero and the heart tissue cannot be stimulated. Nothing is strong enough to elicit another contraction
Beginning of the QRS complex till middle of the T wave

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

The relative refractory period

A

Heart is almost back to normal but is still unstable
Occurs from the middle of the T wave till the end.
If a strong enough impulse hits this area can cause life-threatening arrythmias… R on T phenomenon

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

ST- elevation

A

MI coronary artery is occluded

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

ST- depression

A

Ischemia or non stemi MI
Partically goes through whole ventricle

Deep and wide q waves (had a previous MI) St will go back to normal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Sinus Bradycardia

A

Rate: less than 60
P present
P waves precedes each QRS complex
P-R ratio is 1:1

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Causes of Sinus Bradycardia

2.S/S

A 
Carotid sinus massage
Hypothermia
Increased vagal tone 
Administration of parasympathomimetic drugs
*Hypothyroidism
* increased ICP
*Obstructive jaundice
* Inferior wall MI
2. Pale, cool skin, hypotension, weakness,angina,dizziness or syncope, confusion,or disorientation, SOB
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Sinus Bradycardia TX

A

Asymptomatic and hemodynamically stable:NONE
Symptomatic: ATROPINE
Temporary pacing
Dopamine/Epinephrine infusion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Sinus Tachycardia

A

Rate: >100/min
P present
P waves precedes every QRS
P-R ratio 1:1

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Sinus Tachycardia Causes

  1. S/S
A 
Exertion
Anxiety
Fever
Anemia
Stimulants
Hyperthyroidism
pain
Drugs
2. S/S: Dizziness, dyspnea, hypotension, angina
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Sinus Tachycardia TX

A

Determined by underlying causes
- B-adrenergic blockers to reduce HR and myocardial oxygen consumption
(Gold Standard in HF and ST elevation MI)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Supraventricular Tachycardia/ Narrow complex tachycardia (normal QRS)

A

Arrhythmias originating in an Ectopic pacemaker site in the atria (not SA node)
Involves enhance automaticity of arterial tissue or conduction of the Ectopic impulse
Rhythm is regular
Ventricular response is greater than 150/min and generally less than 200/min
No P wave
NO PR interval
QRS complex: 0.04-0.10
(If normal QRS -ectopic foci is in atrium

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

Causes Of Supraventricular tachycardia

A 
Unknown etiology
Emotional Stress
Excessive intake of alcohol, caffeine, or tobacco
valvular heart disease (Rheumatic)
Coronary artery disease
Digitalis toxcitiy
28
Q

Tx of SVT

If Hemodynamically stable

A

Stable (hemodynamically)

  • attempt vagal maneuver (slows down heart): Deep cough, Hold breath and bare down, Suction tracheal tubes make them gag stimulates vagus nerve.
  • ADENOSINE Pharmacological cardioversion (stops the heart so somefully the SA node kicks in, short half life less than 6 sec, going to feel bad)
  • If After 3 doses of adenosine, the rhythm continues and the patient is stable, the MD may order a CCB or Beta blocker
29
Q

Tx of SVT

If Hemodynamically unstable

A

Unstable (diaphoretic, cold and clammy)

* synchronized cardioversion

30
Q

Synchronized Cardioversion

A

The “SYNC” button is pressed on the machine so that every R wave is flagged
*If this is not done an R on T phenomenon may occur and the patient will go into a pulseless dysrhythmia
*Patient is sedated prior to the procedure
It stops Heart and hopefully SA node kicks in again

31
Q

Pad placement

A

Not put over the nipple on the right side because it burns skin
Avoid piercings/ pacemakers
Give Versed - sedation blocks short term memory
Im clear your clear were all clear … Discharge
Have to put machine into synchronized mode each use

32
Q

Atrial Flutter

A

Atrial Tachycardia identified by recurring, regular, sawtooth-shaped flutter waves
*May be fast, slow regular or irregular
Eropic foci 300x/min
Av node blocks so many of the impulses
One ectopic foci in the atrium initiating the rhythm
Atrium 300 ( 1 large square) but ventricles 75 ( 4 large squares)
AV node protects the ventricles

33
Q

Atrial flutter occurs with usually:

A 
CAD
Mitral valve disorder
Pulmonary embolus
Chronic lung disease 
Cardiomyopathy
(Atriums not able to empty properly so blood just sits and can form clots) Left artium has a pocket.. blood flows(if clots) goes into left ventricle and out the aorta -> stroke
34
Q

Clinical significance of Atrial Flutter

A

The high ventricular rates (>100bpm) and the loss of the “atrial kick” can decrease CO and cause serious consequences such as Heart failure
Patients wit AFlutter are at increased risk for strokes b/c of the risk of thrombus formation in the atria from stasis of blood.
** Coumadin is used to prevent stroke in pts with atrial flutter of longer than 48 hour duration
NEED ANTICOAGULANTS

35
Q

TX for Atrial Flutter if stable

A

Stable

  • Amidodarone
  • Digoxin
  • Beta blockers
  • CCB
  • After therapeutic anti-coagulation, return for elective synchronized cardioversion
36
Q

TX for Atrial Flutter if unstable

A

Synchronized cardioversion (if pulse)

37
Q

TX for Atrial Flutter

A

Diltiazem, digoxin, beta blockers used to control ventricular rate
Antiarrhythmic drugs used to convert atrial flutter to sinus rhythm or maintain a sinus rhythm
Radiofrequency catheter ablation used a curative therapy ( a catheter is placed in the RA b/w the inferior vena cava and the tricuspid valve, with low voltage, high-frequency form of electrical energy the tissue is ablated(destroyed)

38
Q

Atrial Fibrillation

A 
Arrthymia arsing from Ectopic foci(many) in the atrium discharging impulses at a rate if 400 or more per min
*The atria quiver or fibrillate
* No P Waves
*NO PR interval 
*GRS Complex 0.04-0.10 sec (NORM)
{Irregular HR} ASK
39
Q

A Fibrillation Causes

A 
unknown etiology
Associated with emotional stress or excessive alcohol consumption
Valvular heart disease
Hypertensive or coronary heart disease
Cardiomyopathy
Myocarditis 
Pericarditis
Heart Failure
Hyperthyroidism 
Pulmonary Disease
Following cardiac surgery
40
Q

Clinical significance of a fibrillation

A

Can often result in decreased CO b/C of ineffective atrial contractions or loss of atrial kick, a rapid ventricular response or both
Thrombi may form in the atria as a result of blood stasis. An embolized clot may develop and travel to the brain, causing a stroke.
- Overall risk for stroke increases five-fold with atrial fibrillation
Risk of stroke is even higher in pts with structural heart diseases, HTN, and over the age of 65 yrs old
- Anticoagulation with coumadin used to prevent strokes

41
Q

Atrial fibrillation TX

A

Goals: decrease in ventricular response (to 48 hours before attempt at conversion to sinus rhythm
The cardioversion procedure can cause clots to dislodge which increases the patient’s risk for stroke

If on coumadin (takes 3-5 days to become therapeutic levels)
Measure INR normally want 2-2.5 (if to low- clot.. if to high-bleed… Heparin measure PTT
Newer non-vitamin K antagonist oral anti-coagulants (no tests to monitor.. give based on wt)
Davigatran (pradaxa)
Rivaroxaban (Xarelto)
Apixaban (Eliquis)

42
Q

Ventricular arrhythmias

A

Ventricular arrhythmias originate in the ventricles below the branching portion of the bundle of HIS and includesL
Premature Ventricular Contractions (PVC’s)
Ventricular Tachycardia ( with or without a pulse)
Ventricular fibrillation. Most of these rhythms are or have the potential to be life threatening and demands immediate recognition and treatment

43
Q

Premature Ventricular Contractions (PVC’s)

A

Rhythm: Underlying rhythm usually regular, irregular with PVC
The P wave none associated with PVC however p waves associated with the underlying rhythm
PR interval : not measurable
QRS for PVC wide bizarre, different from the QRS complexes of the underlying rhythm

44
Q

PVC causes

A 
Can be common becoming more frequent with age
Unknown etiology  and can occur in healthy hearts
Anxiety
Excessive caffeine and alcohol intake
Drugs
Hypoxia
Acidosis
Electrolyte imbalance
CHF
MI
Valvular or ischemic heart disease
Reperfusion following thrombolytic therapy and angioplasty, heart surgery or placement of leads or catheters in the ventricles
45
Q

PCV TX

A

Tx is usually guided by the number of PVC’s usually the more concerning if greater then 6 min, couplets, runs of 3, or consecutive PVC’s, R on T phenomena
Tx is also guided by how symptomatic the patient is with the PVC
*reverse possible causes
*Amioodarone, lidocaine, Procainamide

46
Q

Descriptors of PVCs

A

Unifocal- Look the same as coming from the same ectopic focus
Multifocal- look different as coming from various ectopiv foci
Bigeminy: every other complex is a PVC ex normal beat, PVC, normal beat,PVC
Trigeminy- Every third complex us a PVC
Couplet- Two PVC together
Triplet- 3 PVC together (aKA a 3 beat run of ventricular tachycardia)

47
Q

Monomorphic ventricular Tachycardia

A 
Rhythm: regular
Rate: Greater thhan 140/min
P waves: No P waves are associated with ventricular tachycardia. However SA node continues to beat independently and sinus P waves may occasionally be seen
PR interval: not measurable
QRS complex: Wide and bizarre
48
Q

Monomorphic ventricular Tachycardia Causes

A 
usually occurs b/c of some underlying heart disease
Myocardial ischemia or infarction***
Cardiomyopathy
Mitral valve prolapse
CHF
Digitalis toxicity
Antiarrhythmic medications
Electrolyte imbalances
Reperfusion
Mechanical stimulation of the endocardium by a wire or catheter
49
Q

Monomorphic ventricular Tachycardia TX

Pulse and is stable

A 
Pulse and is stable:
**Amidodarone
Lidocaine
Procainamide
*If Rhythm converts, then start and IV maintenance drip with the same antiarrhythmic that converted the rhythm
50
Q

Monomorphic ventricular Tachycardia Pulse and unstable

A

Prepare of synchronized cardioversion

51
Q

Monomorphic ventricular Tachycardia Pulseless and unresponsive

A

CPR
Defibrillation (b/c no pulse only used with pulseless ventricular tachycardia and fibrillation
— The machine is not in SYNC mode
—Before discharging the machine you still need to “clear” everyone

52
Q

Polymonomorphic ventricular Tachycardia

A 
Rhythm: regular or irregular
Rate: greater than 150/min
P wave: Not identifiable
PR interval: not measurable
QRS complex: wide and bizarre with variations in its electrical polarity
T wave: not discernible
53
Q

Polymonomorphic ventricular Tachycardia causes

A

Baseline QT normal: Same etiology as ventricular tachycardia
Baseline QT prolonged:
Look for causes of prolongation of QT interval
Low magnesium levels
Antiarrhythmic medications
Changes in medication
Tricyclic antidepressants
Haldol
Congenital long QT syndrome (esp if young pt)

54
Q

Polymonomorphic ventricular Tachycardia TX

A

Baseline QT interval prolonged:( Torsade de Pointe) Magnesium 1-2 grams IV*

55
Q

Ventricular Fibrillation

A 
Rhythm: No pattern or regularity, Ventricular activity appears as Fibrillatory waves with no recognizable pattern
Rate: Cannot be determine
P waves: none
PR interval: None
QRS complex: cannot be determine
56
Q

Ventricular Fibrillation Causes

A 
CAD
MI and Myocardial ischemia
Untreated ventricular tachycardia
Underlying Heart disease
Acid-base imbalances
Electric shock
Severe hypothermia
Drug toxicity 
Electrolyte imbalances
Artifact: Not true rhythm if see patient and they're up and orientated not true v fib (electrical interference)
57
Q

Ventricular Fibrillation TX

A

Same as pulseless ventricular tachycardia
*CPR
*Debrillation
Iv drug epinephrine ( everyone without a pulse gets it, it is a potent vasoconstrictor to increase blood flow to heart)

58
Q

Implantable Cardiover-defibrillator

A

Programmed to read rhythms

Internal shock, hurts

59
Q

Asytole

A

Rhythm: none
Rate: None
Complexes: none
Heart is stopped

60
Q

Asystole Tx

A

Start CPR
Check a second lead to confirm asystole
When IV established give Epinephrine
may give vasopressin to replace first or second doese of epinephrine
If witnessed or known downtime consider transcutaneous pacing
Search for possible contributing factors

61
Q

Pulseless Electrical Activity

A

You may have a rhythm on the monitor but no detectable pulse
Organized electrical depolarization occurs but no synchronous shortening of myocardial fibers
Pulse comes from contractility (squeeze)

62
Q

Pulseless Electrical Activity causes

A

same as asystole

*advanced cardiac disease, a severe cardiac conduction system disturbances, or end -stage HF

63
Q

Pulseless Electrical Activity Tx

A

Rapid identification and tx of underlying reversible causes is critical for treating PEA
Initiate CPR
Epinephrine
Vasopressin

64
Q

Causes -Think about the H’s and T’s

A

H’s: Hypovolemia, hypoxia, Hydrogen ions(acidosis), Hypo-hyperkalemia, hypoglycemia, Hypothermia

T’s: Toxins/Tablets (drug overdose), Tamponade, Tension Pneumothorax, Trauma (hypovolemia, increased ICP)

65
Q

AV Heart block

A
  • An AV heart block is a disturbance in the atrioventricular conduction of the heart
  • Normally AV node acts as a bridge b/w the atria and the ventricles
  • An AV block is a failure or delay in conduction across the bridge
  • The PR interval measures the time b/w the initial depolarization of the atria and the initial depolarization of the ventricles
66
Q

AV heart block includes

A

First degree AV Block (mild)
Second degree AV block type 1
Seond degree AV block type 11
Third degree AV block or complete Heart block

67
Q

Pacemakers

A 
a battery-powered device that delivers an electrical stimulus to the myocardium resulting in contraction
Reasons to pace a pt:
Symptomatic Bradycardia
Sinus Arrest
Slow atrial fibrillation
Alternating brady and tachy arrythmias
Second degree heart block type 11
Third degree heart block
Asystole

Complex 2 (4 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions