ECGs Flashcards

1
Q

What are the rough steps to reading an ECG (in an OSCE)?

A
  1. Check this is the right ECG
  2. Rate
  3. Rhythm
  4. Cardiac Axis
  5. P waves
  6. P-R interval
  7. QRS complexes
  8. S-T segment
  9. T waves
  10. U waves
  11. Summarise what you have identified and what it might suggest
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2
Q

What should you check for in the first step of reading the ECG?

A
  • patient ID (name and DoB)
  • age
  • presentation (e.g. chest pain)
  • the date and time the ECG was performed
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3
Q

How do you count the heart rate?

A

If REGULAR:

  • Count the number of large squares present within one R-R interval
  • Divide 300 by this number to calculate the heart rate

If IRREGULAR, this won’t work. Instead:

  • Count the R waves in the whole strip (=10 seconds)
  • Multiply by 6
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4
Q

What’s a normal adult heart rate?

A

Normal = 60 – 100 bpm

Tachycardia > 100 bpm

Bradycardia

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5
Q

How long is a small square in an ECG?

A

0.04 seconds

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6
Q

How long should the P-R interval be?

A

0.12 - 0.2 secs (3-5 small squares)

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7
Q

How long should the QRS complex be?

A

0.08 - 0.12 secs (2-3 small squares)

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8
Q

What is the normal:

  1. Width of the QRS?
  2. Height of the QRS?
A
  1. QRS can be described as
  • narrow (<0.12s / 3small squares) - normal
  • wide (<0.12s / 3 small squares) - sign of abnormal depolarisation sequence (e.g. ventricular ectopic) or BBB
  1. QRS can be described as
  • small < 5 small squares in the limb leads or < 10 small sqares in the chest leads
  • tall - imply ventricular hypertrophy (can be due to habitus ie. in tall slim people)
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9
Q

How big does ST elevation have to be to become significant?

A

ST elevation is significant when it is greater than 1 mm (1 small square) in 2 or more contiguous limb leads or >2mm in 2 or more chest leads.

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10
Q

What does ST depression show?

A

ST depression ≥ 0.5 mm in ≥ 2 contiguous leads intidcates myocardial iscaemia

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11
Q

T waves are tall if they are…

A
  • > 5 small squares in the limb leads AND
  • > 10 small squares in the chest leads
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12
Q

What do tall T-waves indicate?

A
  • Hyperkalaemia (“Tall tented T waves”)
  • Hyperacute STEMI
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13
Q

In which leads is T wave inversion normal?

A
  • V1
  • lead III
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14
Q

What do biphasic T waves indicate?

A
  • ischaemia
  • hypokalaemia
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15
Q

Causes of LBBB

A
  • myocardial infarction
    • diagnosing a myocardial infarction for patients with existing LBBB is difficult
  • hypertension
  • aortic stenosis
  • cardiomyopathy
  • rare: idiopathic fibrosis, digoxin toxicity, hyperkalaemia
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16
Q

How do you differentiate LBBB from RBBB?

A

William LBBB and Marrow RBBB

in leads V1 and V6

17
Q

Causes RBBB

18
Q

What is the ECG change on 1st degree heart block?

A

Consistently prolonged PR interval

19
Q

What is the ECG change on second degree heartblock:

  1. Mobitz 1
  2. Mobitz 2
A
  1. Progressively increasing PR interval followed by dropped QRS (long, long, drop)
  2. Regularly dropped QRS complexes in ratio of normal:dropped e.g. 3:1
20
Q

What is the ECG change on 3rd degree heart block

A

No relation between P wave and QRS complexes

AND often very bradycardic

21
Q

What are the ECG changes in hyperkalaemia?

K>5.5

A

Early: Tall tented T-waves

>6.5 K: P wave widening and flattening until disappeared, PR segment lengthens

>7: wide QRS and bradycardia +/- conduction blocks

>9 sine wave

22
Q

What are the ECG features in hypokalaemia?

A
  • Increased P wave amplitude
  • Prolongation of PR interval
  • Widespread ST depression and T wave flattening/inversion
  • Prominent U waves (best seen in the precordial leads V2-V3)
  • Apparent long QT interval due to fusion of T and U waves (= long QU interval)
23
Q

What are the ECG changes with pericarditis?

A

Very similar to STEMIs just EVERYWHERE

  • Widespread concave ST elevation (rather than tombstone) and PR depression throughout most of the limb leads (I, II, III, aVL, aVF) and precordial leads (V2-6)
  • Reciprocal ST depression and PR elevation in lead aVR (± V1)
  • Sinus tachycardia is also common in acute pericarditis due to pain and/or pericardial effusion
24
Q

What does VT look like?

A

Fast, broad complex

25
Q

What does ventricular flutter look like?

A
  • Chaotic irregular deflections of varying amplitude
  • No identifiable P waves, QRS complexes, or T waves
  • Rate 150 to 500 per minute
  • Amplitude decreases with duration (coarse VF –> fine VF)
26
Q

If a patient in VT is haemodynamically STABLE what do you give?

A

Drug therapy

  • amiodarone: ideally administered through a central line
  • lidocaine: use with caution in severe left ventricular impairment
  • procainamide
27
Q

What do you do if someone in VT is haemodynamically unstable?

A

electrical cardioversion

28
Q

What is the management of atrial fibrillation?

A

Rate control:

  • b-blockers
  • CCBs if asthmatic
  • digoxin if they don’t work

Rhythm control:

  • Cardioversion (amiodarone or electrical) if new / indicated

Stroke risk:

CHADSVASC for DOAC use

29
Q

What is the management of STEMI?

A

MONA

+ PCI

: will need clopidogrel + unfractionated heparin