ECG LP 05 Flashcards

1
Q

ECG IN CONDUCTION ABNOR

A

Delaying/blocking the stimulus in a certain territory (situated in the specific conduction tissue or in the myocardial muscle).

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2
Q

CLASSIFICATION OF THE CONDUCTION ABNORMALITIES (I) - LOCATION

A

Sino-atrial block = the stimulus is delayed/blocked between SAN and the atrial muscle.
Interatrial block = the stimulus conduction between the two atria is perturbed.
AV blocks = the stimulus is delayed/blocked between atria and ventricles, in AV node.
Intra-ventricular conduction abnormalities = the stimulus way is perturbed in one/more of the branches/fascicules/terminal ramifications of HIS bundle

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3
Q

CLASSIFICATION OF THE CONDUCTION ABNORMALITIES (II) - SEVERITY

A
  1. First degree blocks - just a delay of the stimulus.
  2. Second degree blocks - intermittent blocking of the stimulus.
  3. Third degree blocks - complete and permanent blocking of the stimulus
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4
Q

ETIOLOGY OF THE CONDUCTION ABNORMALITIES

A

Ischemic heart disease
Endocarditis, myocarditis, collagen diseases,
Degenerative diseases of the conduction tissue (e.g. Lenegre disease)
Congenital defects of the conduction tissue (AVN agenesy)
Iatrogenic:
- coronarography
- cardiac surgery
- drugs that induce depression of conduction, especially in AV node: digoxin, beta (- sympathetic) blockers, calcium-blockers (verapamil), other anti-arrhythmic drugs

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5
Q

ATRIO-VENTRICULAR BLOCKS (AVB)

A

A delay/ a blocking of transmission of the electrical stimulus between atria and ventricles.

The delaying/blockage may be situated in the AV node or in the His bundle.
There are 3 degrees of AVB:

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6
Q

FIRST DEGREE AVB

A

 just a delay of the stimulus
all P waves are followed by QRS complex
PR interval is longer than 0.20 s

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7
Q

SECOND DEGREE AV BLOCK

A
Intermittent blockage: some impulses from the atria go through to the ventricles, 
but some are blocked.
Some P waves are followed by a QRS complex and some are not.
There are 4 types of second degree AVB:
Mobitz I = Wenckebach 
Mobitz II
Advanced or high degree 
Second degree AVB 2/1 ratio
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8
Q

MOBITZ I = WENCKEBACH

A

PR intervals are prolonged more and more until one atrial impulse is blocked. Then the cycle restarts.
PP interval is constant.
P/QRS ratio = n/n-1.
This cycle is called a ‘Wenckebac period’

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9
Q

MOBITZ II

A

PR interval is constant in duration throughout the entire ECG tracing.
The PR interval may have a normal duration or it may be > 0.20 sec (association with
first degree AVB)
P/QRS ratio = n/n-1
Because not all P waves are followed by QRS complex, the ventricular rhythm is irregular in second degree AVB type Mobitz I and II (RR intervals are not constant)

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10
Q

ADVANCED OR HIGH DEGREE

A

More P waves are blocked than transmitted.
PP interval is constant; RR interval is constant
P/QRS ratio = n/1

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11
Q

SECOND DEGREE AVB WITH 2/1 RATIO

A

It can’t be classified as a Mobitz I or II type.
There are no two consecutive P that are transmitted.
PP interval is constant; RR interval is constant (and double the PP).
One P wave is transmitted, one P wave is blocked.

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12
Q

THIRD DEGREE AVB OR ‘COMPLETE’ AVB

A

No stimulus from the atria is transmitted to the ventricles.
The atria have their pace (PP is interval constant) while the ventricles have theirs.
This is an ‘atrio-ventricular dissociation’.
The ventricles are controlled by a ventricular pace-maker situated after the blockage: in the inferior portion of AV node, in the His bundle, in a branch or in the Purkinje network.
In this situation, on ECG tracing, the relationship between the P waves and the QRS complexes is totally random.
The ventricular rhythm is usually a regular rhythm = the RR interval is constant

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13
Q

NODO-HISIAN RHYTHM

A
The ventricular pace-maker is situated in the inferior portion of AV node or in the  Hiss bundle.
The frequency is 50 – 40 bpm
The QRS complex is narrow (normal).
(red arrows = P wave)
IDIO-VENTRICULAR RHYTH
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14
Q

IDIO-VENTRICULAR RHYTHM

A

The pace-maker is situated in one of the His bundle branches or in the Purkinje network.
The ventricular rate (and the heart rate) = 35 – 25 bpm.
The QRS complex is wide and it has a modified morphology (mimicking LBBB or RBBB)

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15
Q

BUNDLE BRANCH BLOCKS

A

Intraventricular conduction abnormalities: the impulse is delayed/ blocked in one of the branches/ fascicle.
The impulse is transmitted through nonspecific myocardial cells, with a slower speed = the activation is delayed in that territory.
Secondary repolarization anomalies (ST/T changes) are present.
On ECG we can evaluate intraventricular blocks only if there is a supra-ventricular rhythm (= a rhythm that is generated by an atrial or junctional pace-maker).

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16
Q

RIGHT BUNDLE BRANCH BLOCK

A

Complete blockage of the stimulus in the RBB.
Ventricular depolarization is made just with the help of LBB.
Septum depolarization is normal and it is followed by the left ventricle depolarization.
Then, the impulse is transmitted slowly toward the right ventricle (which, in the absence of a viable right branch, has not yet been depolarized).
RBBB ON ECG
QRS complex duration ≥ 0.12 sec.(‘wide’ QRS)
Frontal leads – the aspect may vary with the axis deviatio

17
Q

RBBB ON ECG

A

QRS complex duration ≥ 0.12 sec.(‘wide’ QRS)
Frontal leads – the aspect may vary with the axis deviation
right axis deviation (120 -150 degree)
DIII, aVF: positive QRS : RsR’, RR’, indented R
DI, aVL: negative QRS : rS , wide S wave
Horizontal leads
V1, V2: the most important aspect
- positive QRS : RsR’, RR’, indented R,  depressed ST and negative T
- R pick time > 0.07 sec.
- V5, V6: normal R followed by wide, deep S wave

18
Q

LEFT BUNDLE BRANCH BLOCK (LBBB)

A

Ventricular depolarization:
 The septal depolarization is abnormal, from right to left, followed the right ventricle depolarization, and then the left ventricular activation takes place (slowly).
The repolarization is also modified: the right ventricle that was first and normally activated will be the first to repolarize. So the repolarization vector is oriented towards right, down and anterior.

19
Q

LBBB ON ECG

A

QRS complex duration ≥ 0.12 sec.(‘wide’ QRS)
Frontal leads
left axis deviation (- 30 degree).
DI, aVL: positive QRS : wide R wave with plateau
DIII, aVF: negative QRS : rS , wide S wave
Horizontal leads
V5, V6: the most important aspect  positive QRS : wide R wave with plateau  depressed ST and negative T  R peak time > 0.07 sec.
V1, V2: small r followed by wide, deep S wave; sometimes QS wave

20
Q

LEFT ANTERIOR FASCICULAR BLOCK (LAFB)

A

The left ventricle is activated only through the posterior fascicle: the postero-inferior part of the LV is rapidly depolarized and becomes electro (-) and the antero-superior part is depolarized later (through nonspecific myocardial cells), remaining more time electro (+).
The depolarization vector is oriented from inferior to superior in the frontal plane
The electrical axis of the ventricular complex is left deviated, between -60° and -90°.

21
Q

LEFT POSTERIOR FASCICULAR (LPFB)

A

LV depolarization is slow in the postero-inferior part and this region remains longer electro (+) during ventricular depolarization.
The resultant ventricular depolarization vector is oriented towards inferior and right in the frontal plane
Right axis deviation, usually between +120° and +160°