ECG interpretation Flashcards
Ostium Secundum ASD
RBBB + RAD
RBBB + LAD + PRLONGED PR
Dextrocardia
- Inverted P waves in lead 1
- RAD
- Loss of R progression
Torsades de pointes
Polymorphic VT + QTc prolongation
HB follows this pathology
Inferior STEMI - RCA
Complete Heart block
- No association between the P waves and QRS complexes
- Regular bradycardia (30-50 bpm)
- Wide pulse pressure
- JVP: cannon waves in neck
- Variable intensity of S1
Monomorphic VT
- Ventricular tachycardia (VT) is broad-complex tachycardia originating from a ventricular ectopic focus. It has the potential to precipitate ventricular fibrillation and hence requires urgent treatment.
- Regular rhythm
Stokes Adam syndrome
RBBB + inverted T waves + HB
Hypokalaemia
- Prolonged PR + U waves
- +/- flattened T waves and ST depression
Irregular- saw tooth appearance
AF
- HOCM
- left ventricular hypertrophy
- non-specific ST segment and T-wave abnormalities, progressive T wave inversion may be seen
- deep Q waves
- atrial fibrillation may occasionally be seen
Pericarditis
Wide spread ST elevation + PR Depression
Wellens Syndrome is a clinical syndrome characterised by biphasic or deeply inverted T waves in V2-3, plus a history of recent chest pain.
Hypercalcaemia
Shortening of QT interval
Hyperkalaemia
Tall, peaked T waves with a narrow base, best seen in precordial leads ; shortened QT interval; and ST-segment depression.
Anteroseptal STEMI
V1-V4 changes = Left anterior descending pathology
Anterolateral STEMI
V4-6, I, aVL changes
Left anterior descending or left circumflex pathology
Lateral STEMI
I, aVL +/- V5-6 changes
Left circumflex pathology
Posterior MI
Changes in V1-3
Reciprocal changes of STEMI are typically seen:
- horizontal ST depression
- tall, broad R waves
- upright T waves
- dominant R wave in V2
- Posterior infarction is confirmed by ST elevation and Q waves in posterior leads (V7-9)
