ECG Basics Flashcards

1
Q

List the Limb leads.

A

I

II

III

AVR (IV)

AVL (V)

AVF (VI)

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2
Q

Which leads are in the frontal plane axis?

A

I

II

III

AVR (IV)

AVL (V)

AVF (VI)

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3
Q

What are the stages of approaching an ECG (in order)?

A
  1. Identity, standardisation
  2. Rate
  3. Rhythm
  4. P-wave
  5. P-R interval
  6. QRS complex
  7. QT interval
  8. ST segment
  9. T wave
  10. Axis
  11. Abnormal component
  12. Formulate and interpret
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4
Q

How do you perform identity and standardisation and what step is this?

A

Step 1 ≈ identity and standardisation ≈


a) Confirm ID: Name, Age and ECG Date



b) Standardisation: 1cm = 1mV; Paper speed 25mm/sec

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5
Q

For a young child, what changes may you have to make to the Standardisation of the ECG?

A

Subject to change e.g. young child will have faster HR thus may alter paper speed or mV

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6
Q

How do you determine rate and what step is this?

A

Rate is the second step ≈ calculation from second lead

300 / R-R boxes

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7
Q

What are the thresholds of rate in an ECG?

A

Normal: 60-100bpm

Bradycardia: < 60bpm

Tachycardia: 100bpm

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8
Q

How would you report a rate which varies?

A

Patient heart rate irregular from X to Y

Use X and Y, reporting range and make an average

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9
Q

How do you determine the rhythm of an ECG and what step is this?

A

Rhythm of ECG is step 3 ≈ determine by criteria + normal rhythm (sinus rhythm) ≈ mark 3 consecutive R waves on another piece of paper then slide across ECG to intervals equal ≈ rhythm is regular

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10
Q

How do you determine a normal P wave and what step is this?

A

P wave is step 4 ≈ present + upright (<0.25mV and upright in II, III and AVF) + precedes QRS complex (1:1) ≈ atrial depolarisation

1) P-wave present: upright in leads II, III and AVF + <0.25mV 


2) P-wave precedes each QRS complex

Absent P wave: 

- Atrial fibrillation


- Nodal (junctional) tachycardia

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11
Q

What would an absent P wave suggest?

A

Absent P wave: 

- Atrial fibrillation


- Nodal (junctional) tachycardia

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12
Q

How do you determine the P-R interval and what step is this?

A

P-R interval is the time form beginning of P to beginning of Q wave ≈ 0.12-0.2s ≈ P-R interval on ECG ≈ AV conduction 
- 120-200ms (0.12-0.2s) (3 to 5 small boxes) ≈ time between atrial and ventricular depolarisation ≈step 5

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13
Q

How do you determine the QRS complex and what step is this?

A

Measure from beginning of Q wave to end of S wave ≈ step 6 ≈ Normal duration of < 0.12s (120ms) + Normal Q wave (<40ms ≈ 0.04s) and <2mm depth ≈ Ventricular depolarisation

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14
Q

What could an abnormal QRS complex suggest?

A

Dependent on time or magnitude

Time: X > 120ms (0.12s) 

- Ventricular conduction defect 
- Bundle branch block (R or L Bundle Branch Block)

Amplitude: 

i) Low voltage < 5mm

- Hypothyroidism 

- COAD 

- Myocarditis 

- Pericarditis

- Pericardial effusion

High voltage 

a) ∑ S wave in V1 + R wave in V5 or V6 > 35mm

- Left Ventricular Hypertrophy 



b) Dominant R wave in V1;
T wave inversion in V1-V3/V4; Deep S wave in V6


- Right ventricular hypertrophy

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15
Q

What is the QT interval and how can it be measured? How can it be calculated?

A

Start QRS complex to end of T wave (varies with rate) ≈ calculated by Corrected QTc interval ≈ step 7 in ECG procedure

Corrected QTc interval ≈ 380-420ms (0.38-0.42s)

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16
Q

What does a planar elevation (> 1mm) or planar depression (> 0.5mm) indicate on an ECG at the QTc?

A

Infarction

17
Q

What is the ST segment?

A

Time from S wave to beginning of T wave ≈ isoelectric ≈ time from end of ventricular depolarisation to start of ventricular repolarisation

18
Q

What is a normal T wave?

A

T wave ≈ ventricular repolarisation

19
Q

What are the normal and abnormal T wave findings and in what leads may you see this?

A
  • Inverted in VR and V1 and V2 in young



- Abnormal if inverted in I, II and V4-V6 ≈ ischaemia/infarction

20
Q

What is a normal axis of an ECG?

A

Normal: -30º to +90º

21
Q

What is the J wave?

A

Camel’s Hump/Osborn wave ≈ QRS-ST junction ≈ deflection occurring at positive defection at J point (negative in aVR and V1) ≈ normally isoelectric

22
Q

Give 3 causes of tachycardia.

A
- Anaemia 

- Anxiety 

- Exercise 

- Pain 

- Temperature / pyrexia 

- Hypovolemia 

- Heart failure 

- PE 

- Pregnancy 

- Thyrotoxicosis 

- BeriBeri 

- CO2 retention 

- Autonomic neuropathy 

- Sympathomimetics (caffeine, adrenaline and nicotine)
23
Q

Give 3 causes of bradycardia.

A
  • Physical fitness

  • Vasovagal attacks 

  • Sick sinus syndrome
    
- Acute MI (especially inferior) 

  • Drugs (ß-blockers, digoxin, amiodarone, verapamil) 
- Hypothyroidism 

  • Hypothermia
    
- Increased intracranial pressure
    
- Cholestasis
24
Q

What is notable with AF? Causes?

A
  • Rhythm: No discernible P waves
  • IHD
  • Thyrotoxicosis
  • Hypertension
25
Q

What is notable in 1st and 2nd degree heart block? Causes?

A

Electrical impulse moves more slowly than normal but conducts via AVN

1st º = PR > 0.2s

2nd º = PR > 0.2s + QRS fails

Causes:

- Normal variant 

- Athletes 

- Sick sinus syndrome 

- IHD 

- Acute carditis 

- Drugs (digoxin, ß-blockers)
26
Q

What is notable in 3rd degree complete heart block?

A

Electrical signal not sent from atria to ventricles

  • P wave present but not related to QRS complex and < 0.12s
27
Q

List some causes of 3rd Degree complete Heart Block.

A
- Idiopathic 

- Congenital 

- IHD 

- Aortic valve calcification 

- Cardiac surgery/trauma 

- Digoxin toxicity 

- Infiltration (abscesses, granulomas, tumours and parasites)
28
Q

What could cause a prolonged QT interval?

A
  • Acute MI 

  • Myocarditis 

  • Bradycardia 

  • Head injury 

  • Hypothermia
    
- U+E imbalance
    
- Congenital
    
- Drugs (Quinidine, Antihistamines, Macrolides, Amiodarone, Phenothiazines)
29
Q

What makes an abnormal T wave and in what leads?

A

Inversion in leads I, II and V4-V6 ≈ ischaemia/infarction

30
Q

What iatrogenic/toxin may cause this change in the ECG and what is the abnormality?

A

[Digoxin] elevated ≈ digitalis effect ≈ T wave inversion + ST segment sloping depression

31
Q

What is left axis deviation? What may left axis deviation be caused by?

A

Negative QRS deflections in II and III ≈ -30º to -90º ≈ LV hypertrophy, MI

32
Q

What is right axis deviation? What may cause right axis deviation?

A

Negative QRS deflections in I ≈ +90º to +180º ≈ RV hypertrophy, PE or LV atrophy

33
Q

What are the ECG changes seen in MI?

A

1) T wave peaking then T wave inversion


2) ST segment elevation

3) Appearance of new Q waves

34
Q

How can the ECG be used to localise an infarct?

A

1) Anterior infarction
- Any precordial leads (V1 to V6)

2) Lateral infarction
- Leads I, AVL, V5 and V6

3) Inferior infarction
- Leads II, III and AVF

4) Posterior infarction
- Reciprocal changes in lead V1 (ST-segment depression, tall R wave)

35
Q

What impact would hyperkalemia have on an ECG?

A

Tall, tented T waves

Widened QRS complex

36
Q

What impact would hypokalemia have on an ECG?

A

Small T waves

Prominent U waves

37
Q

What impact would hypercalcemia have on an ECG?

A
  • Short QT interval
38
Q

What impact would hypocalcemia have on an ECG?

A
  • Long QT interval


- Small T waves