ECG basics Flashcards

1
Q

How many limb leads are there?

Which colour goes where?

A

there are 4 limb leads

Ride Your Green Bike

  • Red- right arm
  • Yellow- left arm
  • Green- left leg
  • Black-Right leg
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2
Q

How many chest leads are there?

what are their names?

where will you find these leads on the chest?

A

6 limb leads

V1: 4th intercostal space right sternal border

  • V2: 4th intercostal space left sternal border
  • V3: Between V2 and V4
  • V4: 5th intercostal space, mid clavicular line
  • V5: Same horizontal line as V4 and V6, anterior axillary line
  • V6: Same horizontal line V4, mid axillary line
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3
Q

value of red (mm/mv)

value of black (mm/s)

value of green one little box (seconds/ms)

valule of blue one big box (secs/ ms)

A

red- 10 mm/mv

black- 25 mm/s

one small box- -.04seconds 40ms

one big box- 0.2 seconds/ 200ms

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4
Q

normal HR

PR interval

QRS duration

normal QRS axis range

where on the ECG is the absolute refractory period

A

HR 60-100bpm

PR interval- 120-200ms

QRS duration- <120ms

QRS axis is between- -30 to 90

absolute refactory period= beginning of the QRS complex to the apex of the t wave

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5
Q

ways to calculate HR on an ECG

A

300/ no. of big squares between R-R

1500/ no. of small squares between R-R

Numbers of QRS complexes per 10 second strip x6 to give bpm

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6
Q

bradycardia

bpm

involves which disorders

A

bradycardia

rate< 60bpm

includes:

  • sinus bradycardia
  • sick sinus syndrome
  • Afib/ flutter with slow ventricular response
  • 2nd and 3rd degree heart AV block
  • escape rhythm (intrinsic ventricular pacemaker backup rhythm of 20-40 bpm)
  • asystole
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7
Q

what is sick sinus syndrome

A

bradycardia with episodes of sinus arrest that may laso present with episodes of tachycardia

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8
Q

First degree AV heart block

A

impulses take longer to reach the AVN (consistently late)

prolonged QT interval of >200ms

Still one P wave for every QRS

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9
Q

Second degree AV block type 1

A

AVN intermittently fails to conduct impulses

not every P wave has a QRS

has 2 types: Mobitz 1 (Wenkebach) and Mobitz 2

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10
Q

2nd degree heart block–Mobitz type 1 (Wenkebach)

A
  • impulse reaches the AVN but doesnt fully conduct the impulse
  • PR interval progressivley elongates
  • eventually a QRS is dropped as AVN is unable to conduct
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11
Q

2nd Degree heart block- Mobitz 2

A

A form of 2nd degree AV block in which there is intermittent non-conducted P waves without progressive prolongation of the PR interval

The PR interval in the conducted beats remains constant

The P waves ‘march through’ at a constant rate

The RR interval surrounding the dropped beat(s) is an exact multiple of the preceding RR interval (e.g. double the preceding RR interval for a single dropped beat, triple for two dropped beats, etc)

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12
Q

Third degree AV block

A

example of a bradycardia

aka complete heart block

the AVN is unable to conduct any impulses from the atria

there is no relationship between P waves and QRS complexes

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13
Q

2 kinds of escape rhythm

A
  • AV junctional
    • QRS morphology will look like a normal narrow QRS
    • Rate 40-60 bpm
  • Ventricular escape
    • Broad QRS
    • Rate 15-40 bpm

Both act as a safety net

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14
Q

Asystole

A

No electrical activity
— Normally not a completely flat line!
— Is a medical emergency and need immediate treatment. BLS/ILS training chest compressions, treat underlying cause.

Non shockable

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15
Q

A fib

A
  • Atrial rate between 300-600 bpm
  • Ventricular rate is irregular and often fast
  • Can be caused by increased BP, heart disease, valve disease (swelling of the atria)
  • 5x risk of stroke (pooling of blood, thrombus formation, sent to brain)

No P waves- fibrillatory waves seen in stead

QRS is irregularly irregular

Can have fast or slow ventricular response

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16
Q

A flutter

A
  • Rentrant circuit in the LA or RA
  • Atrial rate = 300 bpm
  • Ventricular rate can be regular or irregular
  • Causes are that of AFib
  • Risk of clot formation and therefore stroke high

sawtooth baseline

look for a systolic rate divisible by 75

17
Q

Atrial tachycardia

A

Depolarisation of atrial form an ectopic focus

Rate between 120-200 bpm

Can have AV block

18
Q

Atrial ectopics

AKA atrial extrasystoles

A

a P wave earlier than expected usually with a QRS following

delayed PQ interval on the following shoter PQ

can sometimes fall in the AV refractory period (R- T peak) resulting in no QRS

19
Q

Ventricular ectopics

A

An early QRS complex without a preceding P wave

P wave can follow QRS if VA conduction occurs

Can be unifocal or multifocal

Can be single or more frequent

AKA ventricular extrasystole, premature ventricular beats

20
Q

Broad complex tachycardia

A

3 or more successive beats at >120 bpm

  • AV disassociation
  • QRS duration >120ms
  • Ventricular rate of >120 bpm
  • Can be monomorphic or polymorphic
  • Concordance of the chest leads
  • Can be sustained >30secs
  • Potentially life threatening arrhythmia
  • Can quickly degenerate into VF
21
Q

ventricualr tachycardia

A

Broad complex tachycardia originating in the ventricles

VT may impair CO with consequent

Hypotension

Collapse

Acute cardiac failure

This is due to extreme heart rates and lack of coordinated atrial contraction

22
Q

ventricular fibrillation

A
  • Most important shockable rhythm
  • Ventricles suddenly try to contract at rates up tp 500bpm
  • Rapid and irregular electrical activity renders the ventricles unable to contract in a synchronised manner, results in immediate loss of cardiac output
  • Heart is no longer an effective pump and is reduced to a quivering mess
23
Q

how can broad complex tachycardias be terminated

A

cardioversion- drugs or DC

pacing

24
Q

left bundle branch block

sequence of conduction

A
  • QRS duration > 120ms
  • Dominant S wave in V1 (W of William)
  • Broad monophasic R wave in lateral leads (I, aVL, V5-6)
  • Absence of Q waves in lateral leads
  • Prolonged R wave peak time > 60ms in leads V5-6 (M or William)

Conduction delay means impulses travel first via the right bundle branch (black arrow)

2) Septum is activated from right-to-left (yellow arrows)
3) Overall depolarisation vector is directed towards lateral leads (red arrow)

25
Q

Right bundle branch block

A

QRS duration > 120ms

RSR’ pattern in V1-3 (“M-shaped” QRS complex)

Wide, slurred S wave in lateral leads (I, aVL, V5-6)

26
Q

out of T wave inversion and ST elevation, which is a long and which is a short term sign of a STEMI?

A

ST elevation- short term within 12 hours

T wave inversion- longer term

27
Q

Effect on ST segment and T waves in NSTEMI and general ischaemia

A

ST depression

T wave inversion