ECG Flashcards

1
Q

J point

A

Junction between the QRS complex and beginning of the ST segment

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2
Q

1 heavy box is ___ seconds

A

0.2 seconds

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3
Q

PR interval in first degree AV block

A

Greater than 1 heavy box (longer than 0.2 seconds)

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4
Q

RBBB- which leads to check

A

QRS greater than 0.12 seconds. 2 R waves present in V1 and V2. Also, the last QRS deflection should be pointing upward.

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5
Q

LBBB- which leads to check

A

QRS greater than 0.12 seconds. Two R waves present in V5 and V6. The last QRS deflection pointing downward.

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6
Q

What can you NOT diagnose when BBB present?

A

Infarct pattern, axis, or ventricular hypertrophy

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7
Q

Lead I and lead AVF in Right axis deviation

A

Lead I negative, lead AVF positive

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8
Q

Lead I and lead AVF in extreme right axis deviation

A

Both Lead I and AVF negative

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9
Q

Lead I and lead AVF in left axis deviation

A

Lead I is positive, lead AVF negative

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10
Q

Lead I is positive, and lead AVF is negative. What is your next step

A

Check lead II. If negative, confirm LAD. If positive, axis is normal.

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11
Q

What axis deviation might you find in healthy children or really tall thin adults?

A

RAD

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12
Q

Patient with occlusion of Left anterior descending artery and left circumflex a. Which way does the axis deviate?

A

LAD artery occlusion- infarction of left ventricular anterior areas. Left circumflex occlusion causes necrosis of lateral left ventricle. causes RAD.

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13
Q

Patient with WPW syndrome. Axis deviation…

A

If right sided accessory pathway- causes LAD. If left sided accessory pathway- causes RAD

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14
Q

Patient with pulmonary stenosis (due to pulmonary embolism), causing increased afterload. RVH results- what finding do you look for in ECG?

A

Look at Lead V1- usually small R and large S. In RVH, LARGE R present. (but don’t confuse with posterior infarction)

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15
Q

70 year old man with aortic stenosis due to calcification of valve. Increased afterload, causing LVH. What finding do you look for in ECG?

A

Large S wave in V1, and large R wave in V5. Each peak should be greater than 35 mm. (35 small squares)

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16
Q

Ischemia on ECG signified by

A

Symmetric T wave inversions

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17
Q

Transmural ischemia vs. subendocardial ischemia

A

transmural- st elevation, subendocardial ischemia- st depression.

18
Q

What should you not confuse with ST elevation?

A

J point elevation- concave up T wave in leads V2-V5

19
Q

When is a Q wave pathologic?

A

Greater than 0.04 seconds (1 small square), if depth is at least 1/3 the height of the R wave, and is present in two or more contiguous leads

20
Q

Anterior wall infarction involves leads

A

V1-V4

21
Q

Inferior wall infarction involves leads

A

II, III, AVF

22
Q

Lateral wall infarction involves leads..

A

V5 and V6

23
Q

Posterior wall infarction signified by..

A

Large R wave and ST depression in V1 and V2

24
Q

4 key properties of myocardial cells

A

automaticity, excitability, conductivity, and contractility

25
Q

Normal P wave height and duration

A

0.5-2.5 mm, and 0.06-0.10 seconds in duration

26
Q

Most common cause of atrial tachycardia

A

digitalis toxicity

27
Q

What rhythm is COPD often associated with?

A

Multifocal Atrial tachycardia

28
Q

WPW Tx

A

Ablation. antiarrhythmics

29
Q

What consequence may occur if atrial fibrillation sustains for more than 2 days?

A

Emboli formation in arteries

30
Q

What is holiday heart syndrome associated with, and what rhythm might result?

A

Binge drinking- atrial fibrillation

31
Q

HR of junctional arrhythmia

A

40-60 bpm

32
Q

HR of accelerated junctional arrhythmia

A

60-100 bpm

33
Q

HR of junctional tachycardia

A

100-190 bpm

34
Q

Most common cause of ventricular dysrhythmia

A

ischemia

35
Q

Idioventricular rhythm HR

A

20-40 bpm

36
Q

Accelerated idioventricular HR

A

between 40-100 bpm

37
Q

Ventricular tachycardia HR

A

more than 100 bpm

38
Q

Why should you be very concerned with ventricular tachycardia?

A

can turn into ventricular fibrillation

39
Q

example of polymorphic VT

A

torsades de pointes

40
Q

Tx for congenital prolonged QT syndrome

A

beta blockers with pacemakers

41
Q

Acquired causes of prologed QT syndrome

A

drugs- antiarrhythmic agents, antidepressants, electrolyte imbalances, myocardial ischemia, significant bradycardia