Eating Disorders Flashcards

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1
Q

Anorexia nervosa key points

A

restriction of energy intake leads to significantly low weight that is minimally normal for young people. With an intense fear of gaining weight or becoming fat. Often co-morbid with depression

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2
Q

Bulimia nervosa

A

Recurring episodes of binge eating, easting in a set time a larger amount than what most people would eat in that amount of time in similar circumstances. A sense of lack of control during that eating period, with recurrent behaviours such as vomiting or excessive exercise. Binge eating and compensatory behaviours must last 3 months

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3
Q

Binge eating disorder

A

Recurring episodes of binge eating, eating in a set period of time an amount of food that is larger that what most people would eat in that period, a sense of lack of control in that period. Also have 3 or more of the following: eating more rapidly than normal, eating till too full, eating larger amounts when no longer hungry, eating alone due to shame, feel disgusted or guilty after eating. Once a week for 3 months

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4
Q

Avoidant/Restrictive Food Intake Disorder (ARFID)

A

Eating disturbance such as lack of interest in eating food, avoidance based on sensory aspects of food or concern about adverse consequences of eating. Significant weight loss, not meeting growth or nutritional gains, dependence on supplements, marked interference with psychological functioning

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5
Q

Orthorexia Nervosa: Criteria A

A

Obsessive focus on ‘healthy’ eating defined by dietary theory or set of beliefs which may lead to unhealthy weight loss. Restrictive diet as optimally healthy, violation of self imposed rules cause exaggerated fears.

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6
Q

Orthorexia Nervosa: Criteria B

A

Compulsive behaviour begins to result in malnutrition and weight loss. Intrapersonal distress and weight loss. Intrapersonal distress and reduced social/personal functioning due to beliefs about a healthy diet.

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7
Q

Key statistics on prevalence of eating disorders, NICE 2020

A

Estimates 725,000 people in the UK have an eating disorder. Western countries between 5.5% and 18% of young females but depended on accessibility of seeing a GP. Between 2015/16 and 2020/21 hospital admissions for eating disorders increased by 84%. Binge eating disorder among the most common

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8
Q

Genetic risk factors

A

Strong genetic risk for AN, BN, BED. If have a parent with history of ED makes someone 2x as likely. 11x
more likely to develop AN if have a relative with the disorder. 9.6x more likely to develop BN if have a relative with the disorder and 2.2x more likely to develop BED of have a relative with the disorder.

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9
Q

Gut health risk factors

A

Endocrines produces in the gastrointestinal (GI) tract communicate with the brain to regulate functions of appetite and satiety. Dysregulation of the gut microbiome may be responsible for ED pathology. In C sections babies will be swiped with the endocrines instead as may not get the needed endocrines

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10
Q

Early childhood and adolescence risk factors

A

High in-utero exposure to cortisol through maternal stress is associated with ED. Pre term infants living in an incubator are at risk due to different feeding experiences and structural brain alterations linked to under development. Rules such as the need to clear their plate. If the parent displays characteristics associated with an ED e.g. drive for thinness. Attachment need to make our bodies look good to be accepted. Associating food as a reward.

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11
Q

Childhood weight risk factors

A

Higher weight in childhood leads to increased risk, especially among culturally and linguistically diverse individuals and males. As well as parental perception of their child being overweight.

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12
Q

Abuse and Trauma risk factors

A

Emotional abuse is a significant preditor of purge/binge eating in women, whilst sexual abuse and physical neglect were associated with symptoms in men.

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13
Q

Socioeconomic status risk factors

A

Occurs at similar rates across all income levels. High educated parents increase the risk due to the perfectionism. Not set meal times mean dangerous eating choices can continue. US studies found low food security a preditor for ED behaviours.

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14
Q

Body image and social influence risk

A

Endorsement of thin ideals and drive for thinness are exacerbated in response to exposure to messages which are stigmatising towards individuals who are overweight or obese. Meta-analysis of labatory-based experimental studies found viewing idealised images can result in small increases in body dissatisfaction, but a greater impact on groups with high risk of ED.

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15
Q

Elite sports, female athlete triad and excessive exercise.

A

Engaging in activities that promote strict dieting and low body fat could contribute to ED and maintenance of symptoms. Among 224 athletes 22.8% prevalence of ED symptoms. In non-elite populations recognising excessive exercise among women is important in the risk assessment of ED as they are 2.5x more likely to have an ED diagnosis.

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16
Q

Psychological theories of ED’s: Sexual objectification and Sociocultural theory

A

Women’s life experience and gender socialisation include experiences of sexual objectification reduces women to their bodies, parts or functions, e.g., whistles from cars, inappropriate comments. Routine sexual objectification socialises women and girls to treat themselves like objects to be looked upon.

17
Q

Psychological theories of ED’s: Sexual objectification and Sociocultural theory, Fredrickson et al. (1998)

A

women who tried on swimsuits vs sweaters experienced more body shame and self objectification. Internalised sociocultural standards of beauty mediate eating disorder symptomology

18
Q

Cognitive Models of Bulimia

A

Negative theories about the self (unlovable) are expressed as automatic thoughts. Binging or purging take place to manage distress by provoking distraction. Associated with negative beliefs about the consequences (I’ll get fat). Permissive thoughts make it easier to carry on (“this will be the last time”)

19
Q

Cognitive-interpersonal maintenance model of AN:

A

Predispositions such as OCD especially in connection with relationships increase vulnerability. Maintenance of AN such as fostering pro anorexic beliefs, these traits and symptoms can cause problems interpersonal relationship which in turn maintains the disorder. Links to importance of family therapy.

20
Q

Interpersonal model of BED Theory

A

Difficulties with social functioning precipitate low self-esteem and negative effect, in turn triggers eating to cope with negative feelings.

21
Q

Interventions for ED: MyBody, MyLife: Body Image Program for Adolescent Girls

A

6 Weekly sessions, 90 mins, 4-8 pps , patients read the materials and complete activities fir each session. Discuss body image and eating concerns, to learn strategies to enhance body image and learn good eating behaviours. Discussion board focuses on exploring relationships between teasing, fat talk and low self esteem. Clinical trials suggest good items e.g., improved body confidence and eating disorder self reports.

22
Q

CBT:

A

Self-help group and individual therapy. Begins with enhancing motivation followed by goal setting, formulation, identification and challenging of specific automatic thoughts then working on underlying assumptions assumptions, core beliefs and relapse prevention. Emphasis on motivation, challenging negative beliefs and conducting behavioural experiments e.g., “today i’m going to do X and see what happens.”

23
Q

Treatments: The Maudsley Model of AN treatment for adolescents and young adults, (,MANTRa)

A

Patient workbook that addresses illness maintaining factors inc : avoidance of emotions, rigid and inflexible thinking styles, identity development. Addresses interpersonal maintenance factors e.g. unhelpful behaviour of close other, involving family and loved ones over 20 sessions + 4 monthly booster sessions.

24
Q

Therapies: Eating disorder-specific model of interpersonal psychotherapy (IPT-ED)

A

Attachment theory is a basis, as a persons need for attachment not being met has been linked to ED. Negative social evaluation plays a role in triggering disturbance of the self and causing ED symptoms. So treatment involves creating a healthy relationship between the individual and their social world.

25
Q

Future directions: Body image, social media:

A

Evidence from 50 studies in 17 countries shows indicated social media usage leads to body image concerns, disordered eating and poor mental health, due to comparison, thin/fit ideals and self objectification. Specific exposures such as pro-eating disorder content and appearance focused platforms.

26
Q

Further directions: Orthorexia messages are very commonly pushed through social media.

A

Hight Instagram usage had greatest tendency towards ON no other media having this effect. Twitter showed a small positive association with orthorexia symptoms.

27
Q

Further directions: Gender differences:

A

Males represent 25% odd AN and BN and 50% of BD, yet significantly less studied compared to women. The way EDs are assessed and treated is largely reflective of a female-orientated diagnostic framework. Less than 1% of current peer-reviewed, published articles relate specifically to male presentation of AN, sometimes even assuming female trends would be the same for men.

28
Q

Further Direction: Eating disorders and minority groups

A

Culture plays a role in development and maintenance of an ED. Western culture and ideals about weight and shape impact ED risk and course. Understanding a patients culture improves the treatment. ED prevalence is increasing among non-White individuals, and similar rates are found across White, Latina, Asian and African-American women in the US. Stress associated with idealization of Western beauty standards among Asian, Latina and Black women are associated with ED symptoms. Need better diagnostic tools flexible for non-white patients.