Eating Disorders Flashcards

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1
Q

Perspectives

A
HISTORICAL 
- religious orders
CROSS-CULTURAL
- Western "thin = attractive" ideal
- ie. British TV in Fiji causing eating disorder outbreak and change of concept of "beauty"
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2
Q

Eating Disorder Types

A
ANOREXIA NERVOSA (AN)
BULIMIA NERVOSA (BN)
EATING DISORDER NOT OTHERWISE SPECIFIED (EDNOS)
BINGE EATING DISORDER (BED)
OBESITY (?)
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3
Q

EDT: Anorexia Nervosa

A
  • refusal to maintain body weight (self-starvation)
  • restricting VS binging/purging
  • at least 15% below “normal” weight
  • intense weight gain fear
  • disturbance of body image
  • amenorrhoea (menstruation absence)
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4
Q

EDT: Bulimic Nervosa

A
  • loss of control over eating
  • “normal” weight
  • binging/purging cycles (ie. vomiting/laxative misuse)
  • over-exercising
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5
Q

Management Issues

A
  • major dilemma for clinicians
  • respecting patient choice/autonomy
  • patient refusing insight into self-harm caused
  • enabling patient to reduce health risks
  • treatments vary in invasiveness
  • insufficient contextual/effectiveness research
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6
Q

Common Pathways to Eating Disorders

A
  • many w/AN may develop BN
  • ECKERT et al (1995); some develop BN w/o first having AN
  • both usually develop in adolescence
  • x10 more likely in women than men
  • AN = 1% late adolescent women
  • BN = 1-3% prevalence
  • both probably estimates
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7
Q

Risks

A
  • malnutrition effects (ie. lesser metabolism of vitamins/minerals); brittle bones (bone density reduction))
  • joint disorders
  • dental erosion (particularly in BN via vomit acid)
  • cerebral atrophy (extreme AN)
  • cognitive problems (extreme AN)
  • hypothalamic function abnormalities via neurological differences/weight loss/emotional disturbance in AN
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8
Q

Theories

A
  • adolescent disorder
  • extreme end of “continuum of weight control issues” (60-70% of women on diets)
  • may always be range of factors to various degrees
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9
Q

Causes

A
  • risk factors over time include:
    BIRTH/INFANCY
    CHILDHOOD
    ADOLESCENCE
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10
Q

C: Birth/Infancy Factors

A
  • gender
  • genetics
  • obstetric complications
  • early feeding difficulties
  • high concern parenting
  • neurobiology via impairments to cortical/sub-cortical structures (self-knowledge/reward/food pathways)
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11
Q

C: Childhood Factors

A
  • childhood obesity
  • childhood anxiety disorders
  • sexual abuse/neglect
  • adverse life events
  • OCPD
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12
Q

C: Adolescent Factors

A
  • adolescence in of itself
  • body dysmorphic disorder
  • high exercise levels
  • dieting!!
  • OCD/perfectionism
  • negative self-evaluation
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13
Q

Cognitive/Behavioural Development

A
  • “source of control” in life suggests function to EDs
  • conflict between independence/fear of maturity
  • co-existing/pre-cursive depression magnifies cognitive errors ie. guilt/shame (22-88%)
  • lack of alternative control through transitions; intensified/entrenched via external food control
  • deflects attention from other family issues
  • media’s “aspiration for slimness” (especially AN)
  • dissatisfaction w/body shape/size
  • weight phobia via distorted body image
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14
Q

Body Image Distortion: Self VS Others

A
  • TOVEE (2000); 204 women; 1/6 AN/BN; 5/6 healthy
  • estimate body mass index; shown photos to do the same
  • ED overestimated photos BMI (but control did same, just less); AN were worst
  • ED rated slim photos attractive
  • ED overrated own BMI
  • vicious feedback loop; losing weight = downward shift in attraction perception
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15
Q

Compulsive Exercise

A
  • historical; tenacious drive for physical activity
  • prominent characteristic in AN, even facing severe emaciation
  • BEUMON et al (1994); precedes ED onset
  • CRISP et al (1980); one of last symptoms to pass
  • associated w/
    LENGTHIER HOSPITALISATION
    HIGHER RELAPSE RISK
    POOR LONG-TERM OUTCOME
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16
Q

Social Policy & Treatment

A

1997 MHA TREATMENT GUIDANCE:
- multi-disciplinary intervention emphasis for in/out patients
- avoid involuntary treatment/sectioning always (unless severe risk to self/others)
- client decisions should be MD shared
- compulsion to voluntary acting as soon as possible
- refusing treatment = symptom of it being necessary
- severe measures include:
HOSPITALISATION
FORCED FEEDING
MEDICATION

17
Q

Multi-Disciplinary Approaches

A

PSYCHOTROPIC MEDICATION
- useful for common co-existing affective disorder
- noradrenaline/serotonin/dopamine
NUTRITIONAL COUNSELLING
- education about appropriate nutrition
FAMILY THERAPY
- helpful family changes; tackling issues (ie. EE/scapegoating)
PSYCHOTHERAPY
- ie. CBT for cognitive style/error/food beliefs; picking apart “control” theory; finding what really want to be controlled
SELF-HELP/GROUP WORK

18
Q

Outcomes

A

AN
- 5-20% die (cardiovascular/gastrointestinal/metabolic factors)
- 25% cycle; many develop BN
- remainder gain control
- shift towards targeted brain-based interventions
BN
- good full recovery process; meds = 62% at 10y; psych = 54% at 6y
- CRISP et al (1991); 3 treatments (beh/fam/dynamic); 1y follow = good results; criticised for short follow up
- IMPROVED OVER LAST 20Y

19
Q

Summary

A
  • EDs may have most major negative heath effects of disorders
  • people w/EDs can benefit from treatments (earlier the better)
  • causal/maintenance factors need to be contextually seen w/unhelpful media/transitional demands placed on teens
  • socio-political change/control needed on media to minimise triggering disorder factors