Ears, Nose, Throat Flashcards
Why can a haematoma cause AVN?
Perichondrium supplies nutrients to the cartilage. Disruption due to blood accumulation between these two layers causes AVN
What are the common causes of tympanic membrane perforation?
Infection
Barotrauma
Direct trauma
What are features of tympanic membrane perforation?
Pain, possible conductive hearing loss, increased risk of otitis media
Management of tympanic membrane perforation?
Watch and wait - usually heal within 6-8 weeks.
Don’t get water into ear during this time.
Prescribe abx if perforation follows acute otitis media.
If hasn’t healed after 6 months - myringoplasty.
Causes of otitis externa?
Infection - staph aureus, pseudomonas or fungal. Sebrrhoeic dermatitis Contact dermatitis (allergic and irritant)
Features of otitis externa?
Ear pain, itch, discharge
Otoscopy - red, swollen, or eczematous canal.
Initial management of otitis externa?
Topical abx or combined topical abx with steroid.
AVOID aminogylcosides.
If canal debris present, consider removal
If canal extensively swollen then ear wick sometimes inserted.
Second line management of otitis externa?
Consider contact dermatitis secondary to neomycin.
Oral abx (flucloxacillin) if infection is spreading.
Take swab inside ear canal.
Empirical use of anti fungal agent.
If still fails - refer to ENT.
Features of malignant otitis externa?
Found in immunocompromised - eg DM.
Most commonly pseudomonas aeruginosa.
Spreads from soft tissue of external auditory meatus to involve soft tissues and into bony ear canal.
Can progress to osteomyelitis.
Severe, unrelenting, deep-seated otalgia.
Temporal headaches.
Purulent otorrhoea
Dysphagia, hoarseness, and/or facial nerve dysfunction
How to diagnose malignant otitis externa?
CT scan
Treatment of malignant otitis externa?
Non-resolving with worsening pain referred to ENT urgently.
IV abx eg ceftazidime.
What are the muscles of the middle ear that are involved in preventing hyperacusis?
Tensor tympani - branch of mandibular nerve
Stapedius muscle - facial nerve
What are retraction pockets?
Sucked in areas of TM, caused by negative pressure and ET dysfunction.
Most commonly in pars flaccida or attic region.
If deep enough, collects keratinus debris from squamous epithelial cells of TM and ear canal skin - can lead to cholesteatoma.
Features of acute otitis media including causes?
Most common in children
Causes - strep pneumonia, H. influenza, moraxella, viral.
Ear pain, ear pulling, discharge if TM ruptures. Pain settles with discharge.
Fever
Otoscopy - hyperaemia, suppuration and bulging ear drum.
Management of acute otitis media?
Conservative
Oral abx (amoxicillin for 5 days/clarithromycin or erythromycin) if:
- symptoms lasting more than 4 days or not improving
- systemically unwell but not requiring admission
- immunocompromised or high risk of complications secondary to significant heart, lung, kidney, liver or neuromuscular disease.
- <2 years of age with bilateral otitis media
- otitis media with perforation and/or discharge in canal.
Surgical - recurrent AOM helped with grommet insertion.
Features of glue ear?
Otitis media with effusion.
Peaks at 2 years of age.
Conductive hearing loss is usually the presenting feature.
Painless, but if infected leads to acute OM which is painful.
Can have speech delay at school.
Otoscopy - dull grey TM, may look retracted.
Investigations of glue ear?
Tympanogram - flat type B
Pure tone audiogram - conductive hearing loss.
Management of glue ear?
Conservative - settle in 3 months.
Hearing aid.
Surgery - grommets and possible adenoidectomy.
What are the subdivisions of chronic otitis media? And the features of each?
Subdivided into squamous and mucosal disease. And active or inactive depending on if there is discharge or not.
Squamous:
Active - cholesteatoma (pars flaccida perforation.
Inactive - retraction pocket which may develop into cholesteatoma.
Mucosal:
Active - chronic discharge from middle ear through TM perforation.
Inactive - TM perforation but not active infection/discharge
Mucosal develops as consequence of AOM where there is TM rupture and failure to heal.
Management of chronic otitis media
If cholesteatoma present - radical mastoidectomy
If cholesteatoma not present - topical abx and aural toilet. If ineffective, surgery to repair perforation and ensure good ventilation of middle ear and mastoid bone.
What are the risks of mastoid surgery?
Facial nerve palsy
Altered taste from damage to chorda tympani
CSF leak
Tinnitus
Vertigo
Complete loss of hearing in operated ear.
What is cholesteatoma?
Non-cancerous growth of squamous epithelium trapped within skull base causing local destruction.
Peaks at age 10-20.
Being born with cleft palate increases risk 100 fold.
Features of cholesteatoma?
Foul-smelling, non-resolving discharge.
Hearing loss.
Local invasion -> CNS complications -> vertigo, facial nerve palsy, cerebellopontine angle syndrome.
Otoscopy - attic crust.
Investigations of cholesteatoma?
Pure tone audiogram - conductive hearing loss.
Microbiology - often pseudomonas.
Fine-cut CT scan of temporal bones.
MRI