E3

Question 1

An early indicator of DKA treatment response?

Answer 1

Normalization of anion GAP(indicate blood ketone reduction)

Question 2

F-up of DKA tx?

Answer 2

Glucose every 1 hr

PH/Ketone every 2 hr

Question 3

Why patients with PHA will not have significant hypernatremia and Edema?

Answer 3

Aldosterone escape mechanism

Question 4

Usue of High dose dexametazone supresion test in cushing?

Answer 4

To differentiate type of ACTH dependent(high ACTH) Cu.

1) If due to hyperpitutarism–High dose dexa supress ACTH
2) If Ectopic ACTH(tumor)–No ACTH suppression by high dose DEXA.

Question 5

Symptoms PAI not explained by low cortisol?

Answer 5

Abdominal pain/constipation

Normocytic anemia

Question 6

Lipid abnormality in Hypothyroidism?

Answer 6

1) Hyperchlestrolimia—Due to decrease LDL receptor expression
2) Hypertryglecerimia—Due to decrease LPL activity

Question 7

H.rhythm defect in thyrotoxicosis?

Answer 7

S.Tachycardia

PAC & PAV

Question 8

Hemodynamic effect of thyrotoxicosis?

Answer 8

Systolic HTN
Inc.PP
Dec.SVR
Inc.MOD

Question 9

other?

Answer 9

HOHF and Coronary vasospasm

Question 10

Treatment of subacute(de Quervain thyroiditis)?

Answer 10

BB(control tyrotoxic symptome)
NSAID(Pain controle)
Steroid(p

Question 11

why patients with Shehan syndrome will have hyponatremia?

Answer 11

Normally cortisol inhibits ADH secretion—Low cortisol cause SIADH.

Question 12

approach to acromegaly?

Answer 12

First, do ILGFI
1)if N–R/O acromegaly
2) if elevated–do an oral glucose suppression test
2A)If not suppressed –Diagnose Acromegaly and do MRI
2B) If suppressed—R/O acromegaly