E1 Flashcards

1
Q

Symptom exacerbation: C Spine flexion

A

Flexion
+disc herniation
+posterior muscle injury
+hypertonicity

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2
Q

Symptom exacerbation: T Spine flexion

A

Flexion
+posterior paraspinal muscles
+shoulder girdle muscles
+disc herniation

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3
Q

Symptom exacerbation: L Spine flexion

A

Flexion
+disc herniation
+lumbar pvm
+lumbosacral ligaments

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4
Q

Sequence of Examination

A
  1. Screen
    +asymmetry
    +spinal curve abnormalities
    +regional ROM abnormalities
  2. Scan
    +layer by layer palpating
  3. Segmental Definition
    +segmental (spine) motion testing
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5
Q

Motion terminology (planes)

A
  1. Sagittal: flexion/extension (transverse coronal axis)
  2. Coronal: sidebending (anterior-posterior axis)
  3. Horizontal: rotation (vertical axis)
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6
Q

Symptom exacerbation: C spine extension

A

Extension
+facet joint disease
+anterior muscle injury
+hypertonicity

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7
Q

Symptom exacerbation: C spine rotation

A

Rotation
+splenius capitis and cervicis
+sternocleidomastoid

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8
Q

Symptom exacerbation: C spine sidebending

A

Sidebending
+trapezius
+levator scapulae

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9
Q

Symptom exacerbation: T spine extension

A

Extension

+facet joint disease

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10
Q

Symptom exacerbation: T spine sidebending

A

Sidebending
+intercostal muscles
+serratus anterior

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11
Q

Symptom exacerbation: T spine rotation

A

Rotation

+abdominal obliques

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12
Q

Symptom exacerbation: L spine extension

A
Extension
\+spondylolisthesis 
\+facet syndrome
\+spinal stenosis
\+psoas
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13
Q

Symptom exacerbation: L spine sidebending

A

Sidebending
+lateral abdominal wall
+IT band

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14
Q

Symptom exacerbation: L spine rotation

A
Rotation
\+discogenic pain
\+abdominal obliques
\+iliolumbar ligaments
\+piriformis syndrome
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15
Q

Mid-gravity line

A

EAM–>Odontoid Process of C2–>Greater Tuberosity of Humerus–>Middle of L3–>Sacral Promontory–>Greater Trochanter of Femur–>Just behind patella–>Just anterior to lateral malleolus

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16
Q

Direct techniques

A

Engage motion barriers

e.g. Muscle energy, soft tissue, direct myofascial release, articulatory, HVLA

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17
Q

Name the barriers

A

Anatomic, physiologic, elastic, restrictive, pathologic

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18
Q

Anatomic barrier

A

Final limit to motion limited by bone and ligaments

“The point past which disruption occurs”

Limited especially by ligaments and bone contour

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19
Q

Physiologic barrier

A

Soft tissue tension that limits voluntary/active motion; further motion toward the anatomic barrier can be induced passively

“As far as you can go by yourself”

Maintained by Golgi receptors, muscle spindles, and Pacinian receptors

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20
Q

Elastic barrier

A

Range between the physiologic and anatomic barriers in which passive stretching occurs before tissue disruption

Determined by the capsules and ligaments around a joint

**determines passive ROM

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21
Q

Restrictive barrier

A

Functional limit that decreases the physiologic range; OMM is okay

22
Q

Techniques for pain

A

Counterstrain
Soft tissue
Indirect technique

23
Q

Techniques for edema

A

Muscle energy
Myofascial release
Lymphatic techniques

24
Q

Techniques for muscle spasm

A

Muscle energy

Counterstrain

25
Q

Techniques for fascia

A

Myofascial release (direct or indirect)

26
Q

Techniques for joint surface

A

HVLA

27
Q

Somato-somatic dysfunction

A

Dysfunction in one somatic structure provokes muscle hypertonicity in a related location

Mode: segmental facilitation

Example: quadriceps strain causing L2, L3, L4 group dysfunction via reflex paraspinal muscle hypertonicity

28
Q

Somato-visceral dysfunction

A

Dysfunction in one somatic structure reflexly provokes hypersympathetic/parasympathetic response in segmentally related viscera

Mode: segmental facilitation

Example: occipito-Atlantal dysfunction causing bradycardia via a reflex vagal hyperparasympathecotonia to the conduction system of the heart

29
Q

Viscero-somatic dysfunction

A

Organ causing reflex hypertonicity of musculature

Mode: sympathetic visceral afferents

Example: ulcerative colitis causing hypertonicity of paravertebral musculature at T10-L2 spinal levels

30
Q

Viscero-visceral dysfunction

A

Reflex effects that one viscus (organ) has on another

Mode: spinal cord modulation of ANS reflexes

Example: increased intraocular pressure provoking slowing of the heart rate via trigeminal parasympathetic afferents

31
Q

Types of muscle contraction

A

Isotometric, concentric isotonic, eccentric isotonic, isolytic

32
Q

Isometric contraction

A

Muscle contracts but origin/insertion distance remains static

**most common contraction in muscle energy techniques

33
Q

Concentric isotonic contraction

A

Muscle contractile force remains constant while proximal and distal attachments approximate

**used to increase strength in muscles that are reflexly weakened by somatic dysfunction

34
Q

Eccentric isotonic contraction

A

Muscle contraction remains constant as proximal and distal attachments are permitted to separate

**used to strengthen weak muscle

35
Q

Isolytic contraction

A

Patient contracts muscle against resistance, but physician overcomes force

**lengthens muscles

36
Q

First part of neuron table

A

H 1-5
S 5-9
L/G 6-9
P 5-11

37
Q

Second part of neuron table

A

SI 9-11
C/R 8-2
K/U 10-1
B 10-1

38
Q

Third part of neuron table

A
O     9-10
T     9-10, 1-2
U    10-1
C
P     1-2
39
Q

Organs with preganglionic neurons in sacrum

A

C, B, T, C

Colon, bladder, testicle, cervix

S2-S4

40
Q

A.T. Still lifetime

A

1828-1917 (died at 89)

41
Q

Year Still broke with allopathic medicine

A

1874 – “flew the osteopathic banner to the breeze”

42
Q

When did Still found the American School of Osteopathy? When are DMU and PCOM founded? GA PCOM?

A

October 3, 1892 – Kirksville, MO
1898
1899
2005

43
Q

First four principles of osteopathy

A
  1. The body is a unit
  2. Structure and function are reciprocally related
  3. The body possesses self-regulatory mechanisms
  4. The body has the inherent capacity to defend and repair itself

(1953 in Kirksville)

44
Q

Steps of layer by layer palpating

A

Observation (skin color, temperature, etc)
Superficial fascia (elasticity of skin, turgor [swelling], tension, thickness, mobility, quality)
Deep palpation (contact periaxial tissues of spinal column – muscle turgor, tension, thickness, shape, irritability)
Bone (contour and motion)

45
Q

Tissue texture changes–acute

A

Skin-warm, moist, erythematous
Soft tissues-boggy edema, acute congestion
Muscles-increased tone, spasm ropiness
Mobility-ROM may be normal, but sluggish

46
Q

Tissue texture changes–chronic

A

Skin-cool, pale
Soft tissues-doughy, stringy, fibrotic, thickened, contracted
Muscles-decreased tone, flaccid, mushy, decreased ROM
Mobility-ROM decreased, but quality is normal

47
Q

Date and founder of first chiropractic school

A

1898 – D.D. Palmer (former student of Still’s)

48
Q

What happened in 1941?

A

Penicillin introduced by Fleming

Casualties greatly reduced in WWII

49
Q

California debacle

A

1962-many DOs give up licenses to become MDs

1974-Supreme Court rules against allopathic takeover; DOs reinstated with full licensure

50
Q

Where are the angles of the ribs?

A

In a line parallel to the medial scapular border

51
Q

What ligament attaches to the ILA of the sacrum?

A

Sacrotuberous ligament

52
Q

What ligament is superior to the PSIS?

A

Iliolumbar