Dysrhythmias Medication Flashcards
Class 1A, 1B, 1C
Na+ channel blockers
(Procainamide,Lidocaine, Proprafenone)
Class II
Beta Blockers
(Propranolol)
Class III
K+ channel blockers
(Amiodarone)
Class IV
Ca2+ channel blockers
(Verapamil)
Digoxin
Use: HF and atrial fibrillation
Mechanism of action: Stimulates the vagus nerve causing it to release more acetylcholine. Also inhibits enzymes found in cardiac cells (potassium-ATPase, sodium)
Digoxin HYC
Narrow therapeutic index
Causes GI disturbances (1st sign of toxicity- anorexia, n/v)
May cause visual disturbances as toxicity worsens (Blurred or yellow vision; halos around objects)
May cause Bradydysrhmias- causes dizziness fainting(syncope), and even death
Amiodarone
Use: many dysrhythmias
Mechanism of action: Blocks potassium channels in the heart. Also sodium channels, beta receptors, and calcium channels.
Amiodraone Toxicities (High Yield Concepts)
Lung (pulmonary fibrosis), Liver (Hepatotoxicity), Thyriod(hypo/hyperthyroidism), and Eye problems (optic neuropathy and corneal deposits)
** Can sometimes worsen heart rhythm problems or create new ones»> applies to all dysrhythmia meds
Adenosine
Uses: Resets the heart, administered for SVT
Mechanism of Action: Slows down electrical activity in the SA and AV nodes.
Adenosine High Yield Concept
Impending sense of doom and SOB
In your own words, what is atrial fibrillation?
Irregular & rapid heart rhythm.
What does adenosine do to a person’s cardiac rhythm?
Slows electrical activity in the SA & AV Nodes
How do you administer adenosine?
It must be infused very quickly via an IV line. Push it very rapidly (1-2 seconds) and immediately flush the line with 20 mL of saline
What condition does adenosine treat?
Supraventricular Tachycardia (SVT)
Amiodarone is toxic to which organs?
Lung, liver, thyroid, and eyes
