Dyslexia Flashcards

1
Q

how does the DSM-5 describe Dyslexia?

A

an alternative term used to refer to a pattern of learning difficulties characterised by problems with fluent or accurate word recognition, poor decoding and poor spelling abilities (Lyton, 2003)

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2
Q

how many people is dyslexia thought to affect

A

3-6 % with more boys than girls being affected (Rutter et al., 2004)

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3
Q

What is the prevalence of Dyslexia like worldwide?

A

there is a lot of vairance worldwide:
- Countries use different cut offs
- the structure of language can determine, with languages containing shallower orthography such as Spaish or Fnnish being easier to learn (Seymour et al., 2003).

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4
Q

What are the pre-school symptoms that define dyslexia?

A

delayed speech development
speech problems
little appreciation of ryming words
difficulty learning the alphabet

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5
Q

What are the school age symptoms of dyslexia

A

Problems learning names and sounds of letters
unpredictable and inconsistent spelling
confusing the order of letters in words
making errors when reading out loud
struggling to learn sequences
poor handwriting
taking longer to complete written work

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6
Q

Why do other diagnosis have to be ruled out when diagnosising someone with Dyslexia?

A

because issues such as vision and hearing may impact learning
also there is a high comorbidity with ADHD (Boada et al., 2012) and language impairment and speech disorder which is thought to be mediated by shared etiologic and cognitive factors (Peterson and Pennington, 2015)
COuld impact child’s ability to learn and so would show similar symptoms

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7
Q

what are the steps involded in dyslexia diagnosis?

A

parents and teachers are asked to provide general information
children are asked to perform a set of cognitive tasks

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8
Q

overall what is a key characteristic for the diagnosis of dyslexia?

A

If reading and writning age is lower compared to what is expected for chronological age, education and IQ

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9
Q

What is the active gene-enviroment correlation?

A

It explains how genetic differences change sensitivity to particular environmental effects.
e.g. in dyslexia children choose an environment based off their genotype (avoid reading)
Research has shown the heritability of dyslexia increases as parents education level and socioeconomic status increases.

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10
Q

describe twin studies for dyslexia

A

Dyslexia has been shown to run in families
Twin studies show concordance rates with MZ twins higher than DZ showing a genetic basis (De Fries et al ., 1987)

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11
Q

What has linkage anaysis found for dyslexia?

A

Multitudes of genes that may be related to dyslexia
although we cannot be sure if these genes interact with the environment.
Genetic differences have shown to contribute more to high IQ dyslexia withthis occurring in children from higher economic status families (Peterson and Pennington, 2015)

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12
Q

What is the brain phenotype seen in dyslexia?

A

Darki et al (2012) found that reductions in the left hemisphere white matter volume was significant for variation for three of the risk genes identified in the linage analysis.
These findings being in a typical population were also consistent with dyslexia as a continuous not categorical disorder.

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13
Q

What are the risk genes for dyslexia thought to do?

A

alter brain development in the left hemisphere and therefore alter speech development and language development to make acquisition of written language more difficult.

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14
Q

Explainthe brain structure of individuals with dyslexia

A

they have been found to show less structural a symmetry than typically developing brains

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15
Q

What did Grigorenko et al (2001) study find about a symmetry in dyslexic brains

A

studied 20 dyslexics and 20 controls
Structural MRI measuring brain size
the planum temporal (used in language processing) was a lot smaller

(there is a large range of variation in this area and and the study did not control for other factors such as different levels of exposure)

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16
Q

Explain Brunswick (1999) study on brain imaging tasks

A

Found less activation in the left inferior and medial temporal lobe, left fronal operculum and cerebellum in those who are dyslexic

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17
Q

Why is dyslexia higher in low socioeconomic backgrounds?

A

could be due to:
less practise of literacy based activities at him
Higher status have been shown to read more at home.
schooling can also have a substantial impact on reading and spelling ability

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18
Q

What is phonological processing?

A

The ability to break down words into their individual sounds

19
Q

Outline the theory of phonological deficits?

A

Dyslexic people have a specific impairment in the representation, storage, and / or retrieval of speech sounds
where speech sounds are poorly represented

20
Q

What did Hulme and Snowling (2009) say about phonological deficit theory

A

Phonological deficits are a difficulty in the representation of speech sounds
The more severe the deficit the more marked symptoms of dyslexia.
Dyslexic children struggle to become fluent in matching knowledge of word sounds to written words.

21
Q

What did SNowling et al (2019) say to define phonological deficit theory?

A

Problems with phonology lead to diffiultys in learning mappings between orthography and phonology and other difficulties which include problems learning new spoken words, poor verbal STM and problems with word retrieval and picture naming.

22
Q

What are the supporting points for phonological deficit theory?

A

Mostly comes from evidence that people process speech sounds slowly:
- Baddley Bryant (1983): phonological skills age four predict reading age 8
- Snowling and Hulme (1991): poor at phonological tasks
- Hulme (1981) rules out other deficits

23
Q

Elaborate on why Baddley and Bryant (1983) supports phonological deficit theory of dyslexia

A

got children to complete phonological tasks at age 4
Did not require explicit attention, or manipulation of phenomes, including categorical speech perception etc.
FOund that phonological skills at age four could predict reading age at age 8 even when controlling for IQ and social status.

24
Q

Explain in more detail why Snowling and Hulme (1991) study supports phonological deficit theory of dyslexia

A

48 dyslexics, 48 conrols
Given a battery of phonological tasks (rhym detection, phenome segmentation, phenome blending)
CHildren with dyslexia made more errors and took more time to complete the task.

25
Q

Explain why Hulme (1981) study provides evidence for phonological deficit theory

A

THey are able to rule othe other factors as through a number of tasks showed that dyslexic individuals only had deficits in verbal STM but not visual STM showing the issue is phonological in nature.

26
Q

What are an overview of the criticisms of phonological deficit theory of dyslexia?

A
  • Dickiee et al (2013) dyslexic adults perform normally on phonological tasks
  • Boots and Colliugues (2013) brain imaging study
  • not nessarry or sufficient to account for dyslexia
  • Sakida et al (2016) people don’t always sucom to dyslexia
27
Q

Explain Boots and colligues (2013) study to criticise phonological deficits

A

nuroimmaging sudy on brain resopsonses to phonetic contrasts in the primary and secondary cortex
found responses were similar in dyslexics and non dyslexics.
found structural and functional connectivity issues in individuals with dyslexia arguing these findings are consistent with “intact but less acessable phonetic representations”
Meaning phonological deficits may not be as the theory explains

28
Q

Explain the criticism that phonological deficits is not sufficient to explain dyslexia

A

Phonological deficits are neither necessary or sufficient to account for dyslexia (Pennington, 2006)
Critics don’t deny phonological deficits instead explaining that it is much more extended in its roots in general sensory motor and learning processes Ramus et al (2003)

29
Q

what was Saksada et al ., 2016 criticism of phonological deficit theory

A

Many childre at a family risk of dyslexia do not succumb to reading difficulties but also have problems with phonological awareness as ell as this many individuals have deficits outside of the phonological realm.

30
Q

Explain peterson et al 2014 criticism of phonological theories of dyslexia.

A

Most poor readers demonstrate poor phonological skills but this is not very explanatory as many dyslexic people do not display phonological problems so it is hard to distinguish from people who complain of visual problems.

31
Q

What are magnocellular pathways?

A

Visual pathways which comprise of large cells responsible for detecting contrast motion and rapid changes in the visual field.
The system rapidly focuses ventral steam attention on the letter identified an sequences them by logging the amplitudeand order of shifts of attention and eye movements made during each word.

32
Q

Explain why magnocellular thoeory causes deficits in dyslexia

A

there is a deficit in magnocellular system reduced sensitivity creates difficulty suppressing visual information.
retaal images persist longer than is appropriate resulting in an excess of visual information that creates a masking effect and reduction in visual activity (Stein 2019)

IT explains why children are slower at recognising individual letters and sequencing them correctly compared to good readers

33
Q

What are the supporting points for MAgnocellualar theory

A
  • it is able to describe all of the deficits through one biological cause
  • Stein (2019) flicker fusion task
  • Jarabokva et al (1995) can explain comorbidity
  • Crildo-Chika et al., (2015) empirical evidence
34
Q

Explain Stein (2019) support for magnoellular theory

A

dyslexic people show a significantly slower response in the flicker fusion task
Torch is shone on and off until a continuous image
Dyslexia people show reduced sensitivity in this task and therefore have a higher threshold (doesn’t need to blink at as fast rates)

35
Q

Explain why Jarabkova et al (1995) supports magnocellular theory

A

Magnocellular neurones are found throughout the CNS. They are all characterised by their expression of specific surface signature molecules recognised by antibodies suh as CAT301. This explains why there is a strong association between dyslexia and autoimmune conditions such as allergies, asthma and eczema

36
Q

Explain why Galabrurda et al (1985) study provides support for magnocellular theory

A

Studied post mortem brains and found lear abnormalities affecting the language area in the left hemisphere.
Also found magnocellular layers to be about 40% smaller and more disorganised

37
Q

how did Grildo-CHika et al (2015) provide support for magnocellular theory

A

in 16 dyslexics found magnocellular layers were significantly thinner than age matched conrols

38
Q

what are some of the criticisms of magnocellular theory

A
  • highly controversial Stein (2012)
  • cannot be used in treatment due to lack of evidence
  • correlation doesn’t equal causation
39
Q

explain why magnocellular theory is highly controversial

A

Stein (2012) suggested that 90% of studies posted since 2000 have found a link in support however this has been disputed due to publication bias

40
Q

Why can magnocellulalr theory not be useful in interventions

A

The aerican acaemy of pediatrics (2009) concluded that “given the contrasting findings in literature possible magnicellular deficit has insufficient evidence on what to base treatment” so although the theory is there it cannot help treatment options

41
Q

explain the correlation does not equal causation part of magnocellular theory

A

REduced M cell activity coul be irrelevant to reading ability we do not know yet if the two are related

42
Q

Provide an overview of interventions for individuals with dyslexia

A

most interventions offer extra support and instruction on areas such as phonemic awareness, phonics, spelling writhing and fluency, vocabulary and comprehension.

Coloured filters have also been found to be useful thought to enhance M cell functioning

43
Q

Explain Galuschka et al (2020) study on interventions for dysleixa

A

investigated 34 control trials that evaluated spelling interventions in dyslexic people in a range of ages.
the most successful treatment approaches were the ones that use phonics, orthographic and morphological instruction which was able to have a intermediate to high impact on spelling performance

44
Q

Explain Friths (1985) research on interventions for dyslexia

A

theorised people gain an ability to spell and read when they realise graphines become phenomesand begin self teaching.
Interventions primarily focused on helping this acquisition called ‘phonics intervention’ and a large body of evidence supports this (Galuschka et al., 2014)

(however it doesn’t work for every type of work and is not very applicable to other languages)