Dvt Flashcards
Best day to do fsh
Day 3
Normal value less thsn 10 iu
Most common test for ovarian reserve
Best test for ovarian reserve
AMH(antimullerian hormone) 1 ng
Primary amenorrhea and raised fsh
Gonadal dysgenesis
Primary amenorrhea and low fsh
Kall mann syndrome
Normal level of fsh in
Pcos
Asherman symd
Mullerian agenesis
Amin role of fsh
Seection and growth of dominant follocle
It peaks before ovulation by progestrone
Estrogen
Under the influence of fsh ovary produce estrogen
C18 component
Endometrium proliferation
estrogen peak required for LH surge -200 pg X 48 hr
Decreased estrogem causes
Hot flishes vaginal atrophy
Estrogen levels in pcos
E1 increased
E2 normal
E2 :E1 ratio reversed
LH surge occurs
36 hrs before ovulation
Lh peak 12 hrs before ovulation
Which hormone maintain corpoluteum
Lh
Lh in pcos
Persiaitemly increasing
Persiatance of corpus liteum without pregnancy
Halbans disease
Androgen produced by
Theca cells under lh surge
Leutinized granulosa cells produce progesterone
Progesterone
C 21 compound
Causes secretory changes in endometrium
Cx mucus thick scantvagina intermediate cella
Increased basal body temp
Best test of ovulation
Serum progesterone done at day 21
Progesterone challenge test negative menas
Estrogen deficiency/endometrium/ hpo axis damage
Pct positive means
Anovulation
Peak level of progesterone seen at
8 days post ovulation
Day of ovulat
Length of menstrual cycle -14
Treatment of choice for hyperplasia without atypia
Estrogen
Androgens
C19 compounds
Produced by theca cells
Androgens not produced by ovary
DHT and DHEAS
Androgens priduced in max amount
Androstenidione
Pubic and axilliary hair produced by
Androgens or testosterones
In pcos androgens are
Mildly reduced(<200ng)
GnrH
Decapeptide
Realesed as pulsatile manner at puberty(night by decreased GABA increase in kiss petin)
Ansent gnrh in
Kallman ,anosmia
Low frequency pilses of gnrh causes
Fsh release
High frequency pilse sof gnrh causes
LH release
In case of aub isg shows a growth in uterinecavity what will you do next
Hysteroacooy(best)
Any women with more than 45 years with c/o aub ir how willk you proceed
Irrepective of usg finding do endometrial biopsy or endometrial aspiration cytology (EAC)-karmans cannula
If patient had post coital bleeding and u see visible growth or abnormal cervix
Punch biopsy
Abnormal paps
Colpobiopsy
Treatment of choice for preinvasive lesions
LEEP
If atypical galdular cells on pap smear
Colpobiopsy+endeocerbical curettage+endometrial sampling
Fibroid uterus
Enlarged uterus Heavy bleeding More than equal to 35 yrs Non tender uterus(tender if degeneration or Irregular uterus IOC- USG Treatment Asymptomatic fibroid no treatment Fibroud perimenopausal no treatment Asymptomatic fibroid desire pregnancy-no treatment (except submicosal -hysteroacopic myomevtomy Intramural blocking b/l cornu myomectomy
Investigationof choice for submicosal fibroid
Hysteroacopy
Polyp
Increased size of uterus Irregular bleding Peri and post menopausal Non tender Usg- feeder vessel sign
Ioc-hysteroscopy
Treatment of choice - polypectomy
Adenomyosis
Enlarged uterus Heavy bleeding(dysmenorrhea 40-45 Tender Globular Ioc- mri Diagnosis- HPE
Usg findings in adenomyosis
Venetian blind
Saalt pepper
Poorly defined junctional zone
Treatment of choice-hysterectomy
Treatment of symptomatic traetment
Pressure symptoms-given gnrh analogs and antagonist
If menorrhagia- intramural-ocp
Submicosal- hysteroacopic myomectomy
Endometrial hyperplasia without atypia toc
Progesterone (mirena)
Hyperplasia with atypia
Next step-
D and c with hysteroacopy
If asked toc- hysterectomy
Most common cause of post menopausal bleeding
Atrophic endometritis
Endo ca 10%
Mullerian agenesis
Ansent uterus
Breast >=thanner 2normalnpubic and axillary hair
Absent vagina46 XX
Androgen insensitivity syndrome
Abseent uterus No pibic and axillary hair Braest mor than 2 thanner Blind ending vagina 46XY Gonadectomy after piberty
Most common cause of male pseudohermaphrodite
Androgen insensitivity syndrome
Most comkon cause of female paeudohermaphrodite
CAH- congenital adrenal hyperplasia
Gonadal dusgenesis
Uterus small Abdent breasovary not visible Fsh increased Short statute- turner45 x0 Normal stature -pure gonadal dysgeneses Pure can be xx xy Xy_swyers- gonadectomy as soon as diagnosis made
Kallman
46xX Anosmia Ovaried normal Uterus present Fsh low
Imperforate hymen
Bulging memrane on l/ e
Normal uterua and normal ovaries
46 xx
Breast development presnet
Features of imperforate hymen
Hematocolpos Hematometra Cyclic pain Acute urinary retension Mx- cruciate incision
Imperforate hymen vs vaginal septum
Trnasverse septum there is no bulge
Cough impulse negative in vaginal septum
Cough impulse preseng in imperforate hymen
Investigation of choice for mullerian anomalies
3 D usg
Gold std for mullerian anomalies
Mri
If not
Then laproscopy and hysteroscopy
Dippong fundis and wide angle in
Bicotnuate uterus
Partialfusion 2 uterine horns 1 cervix
Bi cornuate mx
Strass man metroplasty
Complete failure of fusion in which uterine anomaly
Didlephius(2 uterine horns and 2 cervix)
Hoghest riak of vaginal septum
Highest risk of U/l renal agenesis(on the side of septum)
Ectopic pregnancy,evtopic ovary and u/l dysmenorrhea are characteriatic of which uterine anomalu
Unicornuate
High risk of urinary tract anomalies
Endometriosis
Uterine rupture