Dumb crap I cannot remember Flashcards
normal FBS
diabetes FBS
<100
> 126
normal GTT
diabetes GTT
<140
> 200
normal HbA1C
diabetes HbA1C
<5.7%
> 6.5%
3rd nerve palsy: think ______
diabetes
wet keratin: think _________
craniopharyngioma
- clear lipid containing cells
- low nuclear/cytoplasmic ratio
- rare/absent mitoses
adrenal cortical adenoma
-diffusely thickened cortex with streaks
Adrenal hyperplasia
variegated surface
adrenal cortical carcinoma
Zellballen
pheochromocytoma
insulitis
pathologic trademark of T1DM
lymphocytic infilitrate in glomerulus
N/V
Weight loss
fatigue
HYPOcortisolism
acromegaly –> increased risk of dying of ___ & ____
heart disease
colon cancer
most common cause of Cushings
pituitary tumor
diabetes
DVT
rash
glucagonoma
Albright’s Hereditary Osteodystrophy
- pesudohypoparathyroidism
- AD
- short stature
- round face
- developmental delay
- short 4th/5th metacarpals
PTH INDEPENDENT
unresponsiveness of kidney
high PTH
low Ca
high alk phos
best way to assess for adequate production of GH?
insulin tolerance test
insulin –> hypoglycemia –> incr GH
glucocorticoid effect on:
- gluconeogenesis
- protein synthesis
- immune response
- osteoclasts
- incr gluconeogen
- decr protein synth
- decr immune response
- incr osteoclast formation
administration of Cortrosyn causes what?
increased cortisol (in normal adrenals)
if no response –> adrenal atrophy
adrenal cortical adenoma: first steps
watchful waiting
usually non-functional adrenal incidentalomas
mature-appearing neurons in a background of Schwann cells
ganglioneuroma
paraganglioma (or pheo) is usually due to lack of what enzyme?
succinate DH
Conn Syndrome =
primary hyperaldo
Tx for bilateral adrenal hyperplasia
spironolactone
elevated catecholamine metabolites
also: what age, histology?
neuroblastoma
children
homer-wright rosettes
which organs do NOT increase metabolic rate with T3
brain, spleen, gonads
why does rT3 increase?
starvation
do not want metabolic rate to increase; need to conserve energy
increased sedimentation rate
subacute thyroiditis
PTH acts on osteo Blasts or Clasts?
Blasts
–> crosstalk to activate osteoclasts
RankL
recruits osteoclasts to bone surface to increase resorption
actions of Vitamin D
increased serum Ca
increased serum phos
increases Ca/Phos reabs in gut, DCT
mobilize Ca out of bone (?)
other name for activated vit D
calcitriol
receptor type for calcitriol
nuclear hormone receptor
FBHH inheritance
AD
PTHrP
released from small cell lung cancer
incr osteoclast activity
paradoxical inhibition of Ca reabs in kidney
glucagon release stimulated by
low blood glucose
high AA
decrease CV events (diabetes med)
empagaflozin
liraglutide
semaglutide
don’t use with heart failure (diabetes med)
-glitazones