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Anti Arrythmia > Drugs > Flashcards

Drugs Flashcards

1
Q

What is the mechanism of Class I antiarrhythmics?

A

A: Sodium channel blockers that reduce the speed of depolarization in the heart.

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2
Q

What are the subclasses of Class I antiarrhythmics?

A

Class IA, IB, and IC.

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3
Q

Q: What is the effect of Class IA antiarrhythmics?

A

A: Moderate sodium channel blockade; prolongs action potential and refractory period.

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4
Q

Q: Give two examples of Class IA drugs

A

Quinidine, Procainamide.

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5
Q

Q: What is the mechanism of Class IB antiarrhythmics?

A

A: Weak sodium channel blockade; shortens action potential duration.

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6
Q

Q: Give two examples of Class IB drugs

A

A: Lidocaine, Mexiletine

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7
Q

Q: Give two examples of Class IB drugs

A

A: Lidocaine, Mexiletine.

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8
Q

Q: What is the mechanism of Class IC antiarrhythmics?

A

: Strong sodium channel blockade; minimal effect on action potential duration.

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9
Q

Q: What is the mechanism of Class IC antiarrhythmics?

A

: Strong sodium channel blockade; minimal effect on action potential duration.

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10
Q

Q: Give two examples of Class IC drugs

A

A: Flecainide, Propafenone

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11
Q

Q: What is the mechanism of Class II antiarrhythmics?

A

A: Beta blockers that reduce heart rate and conduction by blocking beta-adrenergic receptors.

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12
Q

Q: Give two examples of Class II drugs

A

A: Metoprolol, Esmolol

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13
Q

Q: What is the mechanism of Class III antiarrhythmics?

A

A: Potassium channel blockers that prolong repolarization and increase the refractory period.

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14
Q

Q: Give two examples of Class III drugs.

A

A: Amiodarone, Sotalol

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15
Q

Q: What is the mechanism of Class IV antiarrhythmics?

A

A: Calcium channel blockers that slow AV node conduction and reduce heart rate.

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16
Q

Q: Give two examples of Class IV drugs

A

A: Verapamil, Diltiazem

17
Q

Q: What is the mechanism of adenosine in antiarrhythmic treatment?

A

A: Slows conduction through the AV node by hyperpolarizing the cell membrane

18
Q

Q: What arrhythmia is adenosine commonly used to treat?

A

A: Supraventricular tachycardia (SVT).

19
Q

Q: What is the mechanism of digoxin in antiarrhythmia?

A

A: Inhibits sodium-potassium ATPase, increasing intracellular calcium and vagal tone to slow AV node conduction.

20
Q

Q: What is magnesium sulfate used to treat in antiarrhythmia?

A

A: Torsades de pointes and magnesium deficiency-related arrhythmias.

21
Q

Q: What is a common side effect of Class IA antiarrhythmics?

A

A: Risk of torsades de pointes due to QT prolongation.

22
Q

: What are the main uses of Class II antiarrhythmics?

A

A: Supraventricular tachycardia (SVT), atrial fibrillation, and post-myocardial infarction prevention of sudden cardiac death.

23
Q

Q: What is the major side effect of amiodarone, a Class III drug?

A

A: Pulmonary fibrosis, thyroid dysfunction, and liver toxicity.

24
Q

Q: What is the major proarrhythmic risk associated with Class III drugs like sotalol?

A

A: QT prolongation and risk of torsades de pointes.

25
Q

Q: Which class of antiarrhythmics primarily affects the AV node?

A

A: Class IV (Calcium channel blockers).

Anti Arrythmia (1 decks)

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