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Psych 207 > Drugs > Flashcards

Drugs Flashcards

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1
Q

Agonist drugs

A

Binds to a receptor and artificially increases the function of that receptor.

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2
Q

Antagonist drugs

A

Binds to a receptor and blocks the effects of the receptor (inhibits the way it works)

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3
Q

Antagonist Competitive drug

A

Competes for the same binding sites as the neurotransmitter

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4
Q

Antagonist Noncompetitive drug

A

Does not bind to the same receptor as the neurotransmitter

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5
Q

Partial agonists/ antagonists

A

Does not activate/block a receptor that strongly

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6
Q

Allosteric regulators

A

Always bind to the non-competitive sites
Won’t activate or block the receptor on their own but
increases function of NT = positive
decreases ability of the NT to activate the receptor = negative

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7
Q

Other drugs not cited

A

can block the cleaning up of NTs

like Transport blockers, reuptake inhibitor, enzyme inhibitor. agonist or antagonist like effects.

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8
Q

Small molecule NTs

A

Aminoacids
Monoamines
Acetylcholine
Unconventional neurotransmitters

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9
Q

Large molecule NTs

A

Neuropeptides

we don’t know what they do

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10
Q

Aminoacids

A

Glutamate

GABA

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11
Q

Monoamines

A

Catecholamines: Dopamine, Epinephrine, Norepinephrine
Indolamines: Serotonin, Histamine
(Most receptors metabotropic)

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12
Q

Glutamate

A

Fast EPSP - Primary excitatory NT of the brain

Not very useful as a drug - function too broad, it’s everywhere

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13
Q

Glutamate receptors

A 
Ionotropic:
- AMPAR - standard receptor system
- NMDAR
- Kainate receptor
Metabotropic: mGluR
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14
Q

Glutamate antagonists

A 
All depressants
Barbiturates
Nitrous oxide
Ketamine
Ethanol
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15
Q

Glutamate agonist

A

Would increases anxiety and cause seizure - it would fire too many neurons

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16
Q

GABA

Gamma-aminobutyric acid

A

Primary inhibitory neurotransmitter of the brain

Used throughout the brain

17
Q

GABA receptors

A

Ionotropic receptor causes Cl- to be released metabotropic: GABAbR (K+ out of cell)

18
Q

GABA agonist

A 
All depressants
Benzodiazepines (anti-anxiety medication)
Ethanol
Chloroform 
Ether
19
Q

GABA antagonist

A

Would increases anxiety and cause seizure - it would fire too many neurons

20
Q

Norepinephrine (Noradrenaline)

A

Originates in brain stem region: locus coeruleus
Causes heterosynaptic facilitation (via heteroreceptors)
Enhances memories of stress/emotion
Evolutionarily useful

21
Q

Norepinephrine antagonist

Propranolol

A

Potential PTSD treatment via reconsolidation

Brings back long term memory to working memory and manipulate it

Psych 207 (9 decks)

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