DIT Cardiac Path End session Quiz Flashcards
What is the classic presentation of a patient with temporal arteritis? What lab findings help diagnose temporal arteritis?
Jaw claudication, unilateral headache, blindess (late). Elevated ESR (definitive diagnosis is temporal artery biopsy)
What are the differences b/w acute and subacute bacterial endocarditis?
Acute is caused by staph aureus-> even normal valves can be effected. Rapid onset (days)
Subacute -> classically is strep viridans with pre-existing valvular damage. More insidious onset (weeks to months)
What are the Jones criteria for acute rheumatic fever?
JONES
Joints- Migratory polyarthritis
O = heart- pancarditis
N- nodules (subQ -> contain Aschoff bodies)
E- Erythema marginatum (rash w/ red edges)
S- Syndenham’s chorea (chorea of the tongue, face and upper limbs)
Why do the kidneys retain fluid in CHF pts?
Kidneys are sensing poor perfusion because the heart is not pumping out enough blood (hence the name heart failure). The JG apparatus senses this low perfusion and secretes renin. Renin converts angiotensinogen (from liver) to angiotensin 1 which is then converted by ACE (from lungs) to angiotensin 2. Ang II causes vasoconstriction via AT1 receptors on vessels. But ultimately to answer the question, Ang II causes aldosterone release from the adrenal gland leading to sodium and water reabsorption.
What are the common causes of restrictive cardiomyopathy?
Anything that bulks up heart tissue.
Sarcoid, amyloid, hemochromatosis, Loffler’s syndrome (endomyocarial fibrosis w/ eosinophils), post radiation fibrosis, endocardial fibroelastosis (congenital, thick fibroelastic endocardium in young children).
Crescendo-decrescendo systolic murmur best heard in the 2nd-3rd right interspace close to the sternum
Aortic stenosis
Rumbling late diastolic murmur with an opening snap
Mitral stenosis
Pansystolic (aka holosystolic or uniform) murmur best heard at the 4th-6th left intercostal space
Tricuspid regurgitation or VSD
Continuous machine like murmur in systole and diastole
PDA
What is dressler’s syndrome?
autoimmune phenomenon resulting in fibrinous pericarditis (several weeks post-MI)
What are the two most common complications after an MI?
Arrhythmias (most common lethal = ventricular fibrillation) and Left ventricular failure
Evolution of an MI 0-4 hours- 4-12 hours- 12-24 hours- 1-3 days- 3-14 days- 2 weeks- several months-
Fill this out later 0-4 hours- 4-12 hours- 12-24 hours- 1-3 days- 3-14 days- 2 weeks- several months-
What are the most common locations for atherosclerosis?
- Abdominal aorta (leads to aneurysms)
- Coronary arteries (angina and MI’s)
- Popliteal arteries (claudication and PVD)
- Carotid arteries (TIAs, strokes, multiple infarct dementia)
** An adult patient with a history of hypertension presents with a sudden sharp, tearing pain radiating to the back. What would you expect to see on CXR?
Widening of the mediastinum (caused by aortic dissection)
** On auscultation of a patient, you hear a pansystolic murmur at the apex with radiation to the axilla. What is the most likely cause of this murmur?
Mitral regurgitation