Disorders of Skin integrity and function Flashcards

1
Q

Tinea (fungal infection) and appearance

A
  • Tinea corpus: ringworm
  • Children most prone
  • Transmission is from kittens, puppies, other children

Appearance

  • Circular/oval lesions on trunk, back buttocks
  • Red papule with sharp border
  • Pruritus, mild burning, erythema
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2
Q

Tinea Capitis and appearance

A

-Most common for children
-Usually scalp and shaft of hair
Appearance
-Primary lesions
- gray, round, hairless patches
Inflammatory type
-Delayed hypersensitivity
-Pustular, scaly, round
-Can evolve to bacterial infection

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3
Q

Tinea Pedis and appearance

A

-Athlete’s foot
-Between toes, soles/sides of feet
Risk
-Males > females
-Barefoot in public swimming pools, sauna’s etc.
-Sharing area/clothes with someone with infection
-Recurrance with exercise/sweating
Appearance
-Mild to inflamed lesion
-Possible exudate
-Painful, pruritus
-Foul odor

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4
Q

Candidiasis

A

-Yeast-like fungus: Candida albicans
-Normally in GI tract, mouth, vagina
-Thrives in warm, moist areas of skin
Oral infection can be:
-d/t long-term antibiotic use
initial sign of human immunodeficiency virus (HIV)
Appearance
-Red rash with well-defined border
-Pruritus, burning
-Can form pustules, infection

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5
Q

Impetigo

A

-Common in infant and young children
-Usually d/t staphylococci or streptococci
-Highly contagious
Appearance
-Small vesicle on face (usually)
-Ruptures honey-colored serous that hardens and crusts
-Pruritis
-Multiplies with scratching

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6
Q

Cellulitis and manifestations

A

-Deeper infection of dermis and subcutaneous tissue
-Normal skin flora or exogenous bacteria
-Transmission via previous skin opening/injury
-Handling fish
-Swimming
-Animal bites
Manifestations
-red, edematous, shiny
-Possible fever, pain
-Can result in septicemia if not treated properly

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7
Q

Necrotizing Fasciitis and risks

A
Rare bacterial infection 
Usually streptococcal but can be others
Involves deep skin and facial plane of subcutaneous tissues
Infection that starts on the surface but grows inward
Risks
Immunocompromized
Cancer
Diabetes
Recent major infection
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8
Q

Necrotizing Fasciitis manifestations & Treatment

A

Manifestations
red, swollen, painful area that expands quickly
Signs/symptoms of inflammation/infection
Progresses to sepsis

Treatment needs to be immediate
Antibiotics – high doses
Surgical debridement

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9
Q

Verrucae (warts) Transmission and appearance

A
Benign human papillomavirus (HPV) lesion
Invades keratinocytes
Multiple kinds/shapes/sizes
Transmission
Direct contact via break in skin
Sexual contact for genital warts
Non-genital warts common

Appearance
Small, gray-white to tan flat papules with rough surface

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10
Q

Herpes Simplex (cold sore) & transmission/appearance

A

-Associated with oropharynx infections (Type 1)
Triggers: stress, menses, infection, UV burns
Transmission while active
-Direct contact with infected saliva
-Skin contact via athletics/dentistry/healthcare
-Sexual contact (usually results in Type 2)
Appearance of HS Type 1
-burning, tingling pustules that crust and heal
-Common on face, mouth, nasal septum
-More often and sever if immunosuppressed

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11
Q

Herpes Zoster (shingles) & risks

A
Result of reactivation of latent varicella-zoster virus infection dormant in dorsal root ganglia since primary childhood infection
Travels up dermatome
Transmission when active
Risk
h/o chicken pox varicell –zoster infection
Age
HIV, immunosuppression
Malignancies
Corticosteroid/chemo/radiation therapy
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12
Q

Herpes Zoster (cont) manifestations and complications

A
Manifestations
-Burning pain, pruritis
-Sensitive skin
-Vesicles form, erupt, crust, fall off
-2-6 weeks
Complications
-Neuralgia 1-3 months after skin clears (common)
-Blindness if contact with eyes (permanent, rare)
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13
Q

Acne Vulgaris: non-inflammatory & inflammatory

A

Lesions of face, neck, back (usually) d/t increased testosterone during puberty
85% of teens
Self-esteem issues
Familial connection

Non-inflammatory
Sebaceous glands plug up
Blackheads: melanin moves into gland
Whitehead: pale, fatty acid irritation
Inflammatory
Pustules, nodules, cysts
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14
Q

Rosacea

A

-Chronic inflammatory skin disorder of UKE
-Usually affects middle-aged adults, women
Manifestations
-Repeated “blushing” episodes, eventually remains
-Usually nose, cheeks
-Inflammatory facial pustules, nodules, edema
-Dry itchy burning eyes
-Telangiectasia (spider veins)
-Skin thickens and is sensitive to heat

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15
Q

Psoriasis and manifestations

A

-Chronic skin inflammation
-3rd decade onset
Risk: heredity, age
Manifestations
-Well-defined red plaques with silver scales (flat or raised)
-Elbows, knees, scalp, lumbosacral, intragluteal cleft
Hyperkeratosis: epidermal thickening over time permanent damage to capillaries leads to bleeding points under scales

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16
Q

Ultraviolet Radiation Damage

A

UVA (not absorbed by ozone)
- Deep penetration, immediate effect (tan)

UVB (most absorbed by ozone)

  • Delayed response, more genotoxic (burn)
  • d/t free radical formation
  • damage to cellular proteins & DNA

-Effects of both are temporary and reversible but research links them to causing skin cancer

17
Q

Explain sunburn and SPF

A

Sunburn: mild to severe
- red, burning, peeling, itchiness, blistering

SPF (Sun Protection Factor)
Chemical
Physical

18
Q

Thermal Injury: 1st degree burns

A
  • Outer epidermis
  • Pink/red/dry/painful
  • Usually without blister (like sunburn)
  • Skin can still “function”
  • Heals in 3-10 days

More serious with infants/elderly

19
Q

Second Degree Burns

A

Epidermis & dermis

  1. Partial-thickness
    - Red, painful, moist, blister that weep
    - Sensitive to touch/heat/air
    - Intact blisters help maintain body fluids
    - Heals in 1-2 weeks
  2. Full-thickness
    - Epidermis and into deep dermis
    - Hair follicles and sweat glands remain intact
    - Mottled pink/red or waxy with flat, dry blisters, edema
    - Loss of sensation possible
    - Scarring
    - 1 month to heal
20
Q

Third Degree Burns

A
  • Involves subcutaneous tissue, possible muscle & bone
  • Waxy white/yellow or tan/brown/black
  • Extensive edema
  • Can be no pain but seldom exists without 1st and 2nd degree burn pain
21
Q

Complications of Burns

A
  1. Hemodynamic
    Injury to capillaries & surrounding tissues
    Fluid loss = hypovolemic shock (vascular, interstitial, cellular)
  2. Respiratory
    Smoke inhalation
    CO, toxins, ammonia, chlorine, sulfur dioxide
    Damage mucosa = bronchospasm, edema
  3. Thermal injury
    - Resulting pneumonia, pulmonary embolism, pneumothorax, etc…
22
Q

Burn Complications (cont)

A
  1. Hypermetabolic response
    Catecholamine and cortisol released in response to stress
    – muscle/fat wasting, glucose stores depleted
    - Heat production is increased d/t heat losses from burned area
  2. Sepsis
    - burn site ideal for microorganism growth
  3. Circumferential burns
    - Encircle body or body part
    -Healing to “eschar” constricts and must be removed/lyzed to prevent compartment syndrome
23
Q

Eschar

A
  • Remnant of burned cells/tissue
  • Leathery
  • Prevents normal healing
24
Q

Treatment of Burns

A
  • Active cooling
  • Fluids
  • Hemodynamic balance
  • Nutrition
  • Analgesia
  • Wound care (may include removal of eschar)

> Protection

  • Antimicrobial
  • Skin grafts
  • Autograft: from own body
  • Homograft: from another person (alive or not)
  • Heterograft: from another species
  • Synthetic

> Physiotherapy
Psychological support

25
Q

Decubitus Ulcers

A

Pressure

  • Over bony prominences
  • Immobility
  • Compromised integument (disease, age)

Shearing Forces

  • Skeleton moves; skin doesn’t
  • Cell injury and thrombosis

Friction
- elbow, heels

Moisture
-Weakens cell walls by changing pH of skin

26
Q

Nevi (moles)

A
  • Congenital or acquired benign skin tumors
  • Pigment or not
  • Flat or elevated
  • Hairy or non-hairy

-Melanocytic/junctional/compound nevi
Dysplastic nevi
- rough/pebbly surface, irregular shape
- capacity to transform into malignant melanoma, usually related to increased size

27
Q

Malignant Melanoma: risk & manifestations

A

-Melanocytic tumor typically sun-exposed areas
-Rapid progression
Risk
-Increased UV light exposure; h/o blistering sunburns
-Family history of MM; h/o dysplastic nevus syndrome
-Fair hair & skin, freckles
-Immunosuppression
Manifestations
-Slightly raised, irregular border and surface
-Independent or from previous nevi
-May have erythema, tenderness, ulceration, bleeding
-Often mottled (red/white/blue)

28
Q

Malignant Melanoma: Growth and diagnosis

A

Growth
1. Radial: horizontal spread in epidermis

  1. Vertical:
    - Invades deeper dermis
    - Raised dome lesion
    - Increased risk of metastasis
Diagnosis
A = asymmetry
B = border irregularity
C = color variegation
D = diameter > 6mm
E = evolution
29
Q

Basal Cell Carcinoma: risks & manifestations

A

-Neoplasm of basal layer of epidermis
-Most common neoplasm, rarely metastasizes
-Slow-growing
Risk
-Fair skin, h/o sun exposure
Manifestations
-Nodular: small flesh- colored/pink smooth translucent nodule enlarging over time

Superficial: scaly erythematous patch/plaque

30
Q

Squamous Cell Carcinoma & Risk and manifestations

A

-Malignant tumor on sun-exposed area
-Confined to epidermis for long periods, then converts to “invasive” stage
-Usually older population
Risk
-UV exposure
-Arsenic, industrial tar, coal, paraffin
-Men; rare if of African descent
Manifestations
-Red-scaling, slightly elevated, irregular border
-Shallow chronic ulcer, crusts
-Can metastasize if not excised early