Disorders Flashcards

1
Q

What would objective measures would you find with nodules

A

Increased NHR, VTI, jitter and shimmer
Decreased fundamental frequency due to mass
Decreased intensity from less subglottal pressure
Increased MAR from loss of air in phonation
Lower VSPL and VE

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2
Q

How would you differentiate nodules from polyps

A

Nodules are bilateral, on anterior 2/3, posterior 1/3 of folds
Arise over time

Polyps are mostly unilateral and have a blood supply
Can form with one incidence of phonotrauma

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3
Q

What are the 2 types of nodules

A

Acute which are gelatinous in appearance. From hyperfunctional voice use

Chronic which are more fibrous and arise over time with repeated phonotrauma

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4
Q

How do you treat nodules

A

Voice rest and good vocal hygiene

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5
Q

How do you treat polyps

A

Surgery

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6
Q

What are the two types of polyps

A

Sessile which are on the fold

Pedunculated that hang from a stem

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7
Q

What is laryngitis

A

Inflammation of larynx

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8
Q

How do you treat laryngitis

A

Good vocal hygiene
Usually heals itself within a week
Medical treatment if an infection is present

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9
Q

What are the symptoms of laryngitis

A

Hoarseness
Fever
Cough
Lower pitch from edema

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10
Q

What is Reinke’s edema

A

Edema that forms in the superficial lamina propria

Associated with smoking

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11
Q

What objective measures would you expect to see in Reinkes edema

A
Increased NHR
Increased jitter and shimmer
Reduced frequency
Reduced intensity, VSPL and VE
Increased MAR
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12
Q

What is a laryngeal cyst

A

Mucous mass found underneath mucosa in superficial lamina propria
Usually in transition area or vocal ligament
Can be anywhere
Need to make a differential diagnosis with contact ulcers

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13
Q

Describe intubation granuloma

A

Mass arising from arytenoid cartilage due to trauma from intubation
Iatrogenic

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14
Q

What causes contact ulcers

A

LPR

Laryngeal-pharyngeal reflux

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15
Q

What symptoms would you see with a contact ulcer

A

Hoarseness
Breathiness
Reduced pitch
Reduced loudness

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16
Q

What causes candida

A

Antibiotics
Illness
Chemotherapy

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17
Q

How would you identify candida

A

White spots on mucosa
Stiff irregular folds
Abnormal redness

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18
Q

What changes would you see in the voice with candids

A

Minimal to moderate changes due to swelling

Pressed, hoarse, or breathy

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19
Q

What is the viral infection that causes a mass in the supraglottal, glottal, or subglottal regions

A

Human papilloma virus 6 and 11

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20
Q

What is a papilloma

A

Mass due to HPV in the supraglottal, glottal, or subglottal regions

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21
Q

What objective measures would you find with a papilloma

A

Increased NHR
Reduced frequency
Reduced vital capacity

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22
Q

What is the primary symptom of a glottal web

A

Respiratory difficulties
Shortness of breath
Harsh and high pitched voice
Can be anterior or posterior

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23
Q

What causes a glottal web

A

75%congenital due to folds not separating during embryonic development in the 4th to 10th week
Acquired due to trauma usually iatrogenic

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24
Q

What is a laryngocele

A

Air filled dilation in ventricles

Congenital

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25
Q

What causes subglottal stenosis

A

Congenital: malformed cricoid cartilage formed in utero.
3rd most common congenital

Acquired: iatrogenic

Idiopathic. More common middle aged women

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26
Q

What symptoms would you see in subglottal stenosis

A

Stridor

Dyspnea, cough, significant chest wall movement

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27
Q

What are the symptoms of presbylarynges

A
Sarcopenia:thinning of muscles
Superficial layer of cord thins
Collagen in cord becomes more dense
Mucosal wave abnormalities
Possible bowing of folds
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28
Q

What is sarcopenia

A

Thinning of muscles

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29
Q

What is sulcus vocalis

A

Groove in fold
Bilateral and symmetrical
May protrude into vocal ligament

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30
Q

What is varix

A

Enlarged and dilated vein in folds

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31
Q

What is ecstasia

A

Fused lesioning of blood vessels in folds

On superficial lamina oropria

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32
Q

What causes muscle tension dysphonia

A

Phonotrauma

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33
Q

What contributes to poorvocal health

A
Smoking
Marijuana
Alcohol
Caffeine
Sleep deprivation
Vocal fatigue
Inappropriate vocal use
Obesity
Allergies
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34
Q

What does improper voice use result in

A

Increased tension
Inappropriate pitch
Ventricular phonation
Later-medial and posterior-anterior compression

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35
Q

What is the difference between primary and secondary dysphonia

A

Primary is direct result of disorder

Secondary is from compensation

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36
Q

What causes phonotrauma

A

Excessive loud talking
Straining during laryngeal inflammation
Coughing or throat clearing
Sports cheering

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37
Q

What are symptoms of phonotrauma

A
Hosrseness
Fatigue
Strain
Pain
Loss of voice
Poor projection
Loss of pitch/ loudness range
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38
Q

What are good differential diagnoses between muscle tension and neurologic disorders

A

Usually worse at the end of the day

Varies throughout day

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39
Q

What does a larynoscopy reveal in strain

A

Compression (lateral medial or anterior posterior)
Supraglottal strain
Hyper adduction
Elevated laryngeal position

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40
Q

What is a hard glottal attack

A

Rapid adduction of folds before a vowel

Increased subglottal pressure to overcome adductive forces which produces sudden explosive sounds

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41
Q

What happens when there is elevated laryngeal positions

A

Pitch increases

Perceive strain, hoarseness, and increased pitch

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42
Q

What are the symptoms of puberphonia

A

Hoarseness, breathiness
Pitch breaks, inadequate resonance
Shallow breathing, muscle tension
Lack of variability

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43
Q

What is ventricular dysphonia

A

Vibration of false folds
Secondary to vocal fold disorder
Gives rise to diplophonia

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44
Q

What are the characteristics of ventricular dysphonia

A

Low pitch because of added mass
Hoarseness
Reduced intensity due to poor pressure below folds

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45
Q

What causes psychogenic dysphonia

A

Emotional trauma, stress or attention seeking

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46
Q

How can you tell if someone is faking

A

They can laugh, whisper

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47
Q

What are the types of assessment

A
Laryngoscope
Perceptual
Acoustic
Aerodynamic
Quality of life
EMG
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48
Q

What are the 5 subsystems of motor speech disorders or dysarthria

A
Respiration
Phonation
Resonance
Articulation
Prosody
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49
Q

What do you call a lesion on a muscle

A

Myopathy

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50
Q

What do you call a lesion on a nerve

A

Neuropathy

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51
Q

What do you call a lesion at the junction of the nerve and muscle

A

Myoneural junction disease

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52
Q

What is loss of muscle mass called

A

Atrophy

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53
Q

What is muscle tension dysphonia

A

Any inappropriate use of voice

Overuse of laryngeal muscles

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54
Q

What are the two types of muscle tension dysphonia

A

Primary and secondary

55
Q

What is the primary concern of muscle tension dysphonia

A

Airflow especially laryngeal resistance

More resistance = less airflow

56
Q

What is hyperfunction

A

Compression of muscles that causes strain

On superior medial, lateral medial, or supraglottic

57
Q

What assessment do you use for vocal fold paralysis

A

EMG

58
Q

What can you expect to see in the assessments of muscle tension dysphonia

A

Auditory perceptual:strained, raspy
Acoustic: noise etc
Respiration: reduced airflow
Laryngoscopy: lateral medial compression, anterios posterior compression, supraglottal tension, aperiodicity, asymmetry

59
Q

What are the 3 parts of brain stem

A

Midbrain
Pons
Medulla

60
Q

What are the two UMN fibers

A

Corticospinal

Corticobulbar

61
Q

Where do the UMN originate

A

Precentral gyrus

62
Q

Where do LMN originate

A

From coricobulbar fibers

63
Q

What is dysarthria

A

Any speech disorder caused by neurological impairment

64
Q

What are the aspects of speech that a motor speech disorder would affect

A
Respiration
Phonation
Articulation
Resonance
Prosody
65
Q

What do LMN lesions cause

A

Flaccid dysarthria

66
Q

What do UMN lesions cause

A

If bilateral they cause spasticity

If unilateral do not affect voice

67
Q

What are the 7 types of dysarthrias

A
Spasticity
Flaccidity
Unilateral UMN
Ataxia
Hyperkinesia
Hypokinesia
Mixed
68
Q

What is ataxia

A

Loss of coordination

69
Q

What causes spasticity

A

Bilateral UMN

70
Q

What causes flaccidity

A

Uni and bilateral LMN

71
Q

What causes ataxia

A

Lesion in cerebellum

72
Q

What is hyperkinesia

A

Too much movement

73
Q

What is a classic disease with hypotonia

A

Parkinson’s

74
Q

What is a motor neuron disease

A

Disorder at the junction of LMN and UMN

75
Q

What is the origin of the LMN

A

Motor neuron

76
Q

What are the two braches of the Vegas nerve that innervate the voice production

A

Superior laryngeal

Recurrent laryngeal

77
Q

What des the recurrent laryngeal nerve innervate

A

All intrinsic laryngeal muscles except the cricothyroid

Sensory information below the folds

78
Q

What are the types of neurogenic voice disorders

A

Vocal fold paralysis
Spasmodic dysphonia
Organic voice tremor

79
Q

What muscles are affected by abductor paralysis

A

Posterior cricoarytenois

80
Q

What muscles are affected by adductor paralysis

A

Lateral cricoarytenoid

Interarytenoids

81
Q

What are the problems associated with adductor paralysis

A

Phonation
Aspiration
Subglottal pressure

82
Q

What is the primary concern of bilateral adductor paralysis

A

Aspiration

83
Q

What would be the voice quality of someone with bilateral adductor paralysis

A

Dysphonic with secondary muscle tension
Bresthy
Wesk

84
Q

What would the voice quality be like with unilateral abduction paralysis

A

Mildly dysphonic

Possible loudness issues

85
Q

What is the most common form of vocal fold paralysis

A

Unilateral adductor paralysis

86
Q

What type of dysarthria is spasmodic dysphonia

A

Hyperkinetic

87
Q

What is the origin of spasmodic dysphonia

A

Disruption in extrapyramidal fibers in CNS

88
Q

What are the symptoms of spasmodic dysphonia

A

Pauses in phonation
Effortful voice production
Strain
Uncontrolled spasms

89
Q

What voice qualities would you expect in adductor spasmodic dysphonia

A

Strain
Effortful
Vowels more affected
Intermittent normal phonation

90
Q

What voice qualities would you expect in abductor spasmodic dysphonia

A

Intermittent breathiness
Short breaks of aphonia
Intermittent normal phonation
Can affect consonant production

91
Q

What is essential tremor

A

CNS disorder

Results in involuntary regular tremors in limbs, head, larynx

92
Q

What is organic voice tremor

A

Essential tremor limited to larynx

Based on frequency deviations of 4 to 6 hz

93
Q

What is myasthenia gravis

A
Neuromuscular disorder
Autoimmune
Causes weakness and atrophy in muscles
Occurs at myoneural junction
Disruption of ACH receptors at myoneural junction
94
Q

How does myasthenia gravis affect voice

A

Weakness after 5 -6 minutes

Mucosal wave abnormalities

95
Q

Where are 4 places that you might see a LMN disorder

A

Motor neuron
Myopathy
Neuropathy
Myoneural junction

96
Q

What is the function of dopamine

A

Inhibitory

97
Q

What type of dysarthria is parkinsons

A

Hypokinesia

98
Q

What voice qualities would you expect in parkinsons

A

Monopitch
Monoloudness
Loudness biggest symptom
Dont have sensory info that they are too soft

99
Q

What is the voice treatment for parkinsons

A

Lee Silverman Loud

LSVD

100
Q

What would happen to reflexes if in the LMN

A

Reduced

101
Q

What would happen to reflexes if the lesion is in the UMN

A

Hyperactive reflex

102
Q

What are the different types of paralysis

A

Abductor or adductor

Both unilateral or bilateral

103
Q

What is a passymuir valve

A

A valve that redirects air through the folds for speech when there is a stoma

104
Q

What are possible causes of vocal fold paralysis

A
Surgery
Neurologic disease
Head or neck trauma
Viral infection
Tumor
105
Q

What are general symptoms of of paralysis

A

Breathiness
Low intensity
Low pitch
Intermittent diplophonia

106
Q

What is botox

A

A toxin that temporarily paralyzes the folds

107
Q

How can you provide biofeedback for a parkinsons patient

A

Sound level meter

108
Q

What are some examples of congenital neuroligic disorders in children

A
VF paralysis
Laryngeal stenosis
Laryngomalacia
Laryngocele
Webbing
109
Q

What are some prenatal reasons for congenital voice disorders

A

Maternal substance abuse

Poor embryonic development

110
Q

What are some perinatal reasons for congenital voicedisorders

A

Birth trauma
Delayed birth cry
Hypoxia

111
Q

What is laryngocele

A

Air filled sac in the vestibule

112
Q

What are postnatal causes of congenital voice disorder

A

Neonatal jaundice
Seizures
Encephalitis

113
Q

What is laryngomalacia

A

Epiglottis is flaccid and obstructs airway.

Concern is swallowing and aspiration

114
Q

What are signs of pediatric voice disorder

A
Dysphonia
Intermittent aphonia
Voice breaks in singing or speech
Excessively loud voice
Inability to sustain a note
Effortful or strained voice
115
Q

What is the primary acquired disorder in children

A

Muscle tension dysphonia

116
Q

What is the leading cause of acquired voice disorder in children

A

Phonotrauma

117
Q

What are actities in kids that may cause vocal nodules

A
Excessive use
Glottal attack
Not resting voice when ill
Crying, laughing, outbursts
Shouting, screaming
118
Q

What should you consider when working with children

A

Laryngeal development
Psychological/emotional development
Language speech
General development

119
Q

What are some anatomical changes in geriatric patients

A
Atrophy
Vocal fold thinning
Bowing
Discoloration
Edema of superficial lamina
Ossification
Decreased blood supply
120
Q

What are perceptual voice changes in geriatric patients

A
Altered pitch
Roughness
Breathiness
Hoarseness
Tremor
Weakness

Quality. Pitch. Loudness

121
Q

Why would loudness be affected in geriatric patients

A

Less air support

Less subglottal pressure

122
Q

What are the essential elements of a report

A
Background/case history
Laryngoscopy
Auditory perceptual analysis
Acoustic analysis
Aerodynamic analysis
Quality of life

Both subjective and objective

123
Q

What info is in a case history

A
The problem verbatim
Effect of problem on their life
Onset and duration
Variability or constancy
Other symptoms present
Medical history/ list of procedures etc
Medications
Vocal habits
Psychological issues
124
Q

What are components of a subjective aerodynamic assessment

A

Observe type of breathing
Number of words per breath
S/z ratio
Max phonation duration

125
Q

What are objective measurements for phonation

A
Habitual loudness and pitch
Loudness variability (contrastive stress)
Pitch variability, range
Tremor
Voice quality with severity
126
Q

What is LPR

A

Laryngeal-pharyngeal reflux

127
Q

What is LSVT

A

Lee Silverman Voice Treatment

128
Q

What is MTD

A

Muscle tension dysphonia

129
Q

What term is used describe functional disorders

A

Muscle tension dysphonia

130
Q

Why is throat clearing destructive

A

Increased subglottal pressure required to overcome adductive forces to produce a sudden explosive sound

131
Q

How does unilateral UMN present

A

Contralateral lower facial weakness

132
Q

What contributes to PVFM

A
Reflux
Exercise upper airway inflammation 
Exposure to irritants 
Environmental allergens
Stress
Change in temp
Vocal maneuvers
133
Q

How do you diagnose PVFM

A

Observe neck muscles
Visualization of folds
Symptoms
Thorough history

134
Q

How do you treat PVFM

A

Direct: relaxed throat breathing, panting, biofeedback
Indirect: education, relaxation, reassurance, hygiene