diseases of pulp and periodontist Flashcards

1
Q

what is hyperaemia characterised by

A
  • increased blood supply in pulp chamber
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2
Q

what is a chronic sinus

A
  • get a big bubble forming which then bursts and get a bad taste and then goes away and is better for a while but then whole process will start again
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3
Q

what are symptoms of pulp hyperaemia

A
  • pain ;asting seconds
  • stimulated by hot/cold or sweet foods
  • resolves after stimulus removed
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4
Q

what causes pulp hyperaemia

A
  • caries approaching the pulp
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5
Q

what are the symptoms of pulp hyperaemia the same as

A
  • reversible pulpitis
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6
Q

what are the clinical symptoms of acute pulpitis

A
  • constant severe pain
  • reacts to thermal stimuli
  • poorly localised pain = often get referral of pain
  • no or minimal analgesic response
  • can have less symptoms if pulp is exposed as less pressure on it
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7
Q

how to diagnose acute pulpitis

A
  • negative TTP
  • pulp testing equivocal
  • radiographs
  • diagnostic LA = numb to see if pain goes
  • removal of restorations
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8
Q

what are the symptoms of acute apical periodontitis

A
  • pain easy to localise now because it has spread to apical tissues
  • very TTP
  • slight increase in mobility
  • tooth can feel high in the mouth for pt so they can’t bite down
  • tooth is non-vital
  • shouldn’t see a radiolucency on radiograph but can see loss of clarity of lamina dura
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9
Q

what are the causes of traumatic perio

A
  • parafunction
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10
Q

how to diagnose traumatic perio

A
  • clinical exam of occlusion
  • TTP
  • normal vitality
  • radiographs show general widening of pdl
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11
Q

what is the treatment for traumatic perio

A
  • occlusal adjustment
  • therapy for parafunction
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12
Q

what is the most common pus producing abscess

A
  • acute apical abscess
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13
Q

what are other causes of pus

A
  • periodontal abscess = formed in periodontium
  • pericoronitis
  • sialadenitis = infection of salivary gland
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14
Q

what organisms are associated with dental abscesses

A
  • polymicrobial
  • anaerobes play important role
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15
Q

what are clinical features of acute apical asbcess

A
  • initially almost identical to acute apical periodontitis - prior to eroding through bone and into soft tissues
  • severe unremitting pain
  • acute tenderness in function
  • acute tenderness on percussion
  • no swelling, redness or heat until spread into soft tissues
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16
Q

what are the 5 cardinal signs of inflammation

A
  • heat
  • redness
  • swelling
  • pain
  • loss of function
17
Q

what are the symptoms of acute apical abscess once perforated through bone

A
  • pain often remits as less pressure
  • swelling, redness, heat become increasingly apparent
  • there is an initial reduction in TTP as pus escapes into soft tissues
  • as swelling increases, pain returns
18
Q

what does the site of swelling depend on in acute apical abscess

A
  • anatomy
  • position of tooth in the arch, tooth length, muscle attachments
  • potential spaces in proximity = submittal. sublingual, submandibular, buccal, infraorbital, lateral pharyngeal, palate
19
Q

what is the treatment for acute apical abscess

A
  • provide drainage = soft tissue incision (I/o or e/o)
  • remove source/cause = extract tooth, pulp extirpation, periradicular surgery
  • AB’s determined by severity, absence of adequate drainage, medical condition
20
Q

what are local factors which determine need for AB’s

A
  • toxicity
  • airway compromisation
  • dysphagia
  • trismus
  • lymphadenitis
  • location = e.g. floor of mouth
21
Q

what are systemic factors which determine need for AB’s

A
  • immunocompromised pts = acquired causes, drug induced, blood disorders
  • diabetes = poorly controlled
  • elderly
22
Q

what is a periapical granuloma

A
  • mass of chronically inflamed granulation tissue at apex of tooth = plasma cells, lymphocytes few histiocytes with fibroblasts and capillaries
  • not a true granuloma because not granulomatous inflammation
23
Q

what is the aetiology of a PA radicular cyst

A
  • caries, trauma, PD
  • death of pulp
  • apical bone inflammation
  • dental granuloma
  • stimulation of epithelial rests of Malassez
  • epithelial proliferation
  • periapical cyst formation