Discuss / Evaluate one or more cognitive etiologies of one disorder (Alloy & N-H) Flashcards

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1
Q

Cognitive Etiology of MDD “The cognitive approach argues for patterns of thinking that are…”

A
  • Major Depressive Disorder (MDD) is a mood disorder that is characterized by prolonged (longer than a few weeks) feelings of sadness, loss of interest in daily activities and lack of appetite.
  • We categorize MDD as abnormal behavior – behavior that is deviant and maladaptive, causes feelings of distress to an individual

The cognitive approach argues for patterns of thinking that are responsible for our mental disorders.

→ Gender diff in depression cause of coping styles

→ Men = distraction, women = ruminate feeling, why? What is the meaning for this distress?

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2
Q

Cognitive Vulnerability Hypothesis

A
  • Cognitive Vulnerability Hypothesis — maladaptive thinking patterns that make a person more susceptible to a mental illness.
  • Pattern of negative schemas about ONESELF, OTHERS and THE FUTURE which leads to stress which triggers the disorder, but this is hard to empirically research
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3
Q

Theory of Rumination

A

Theory of Rumination — focusing on the patterns of neg thinking can lead to depressive symptoms (low self-esteem, hopelessness)

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4
Q

Cognitive Etiology Thesis

A

The cognitive etiology of Cognitive Vulnerability Hypothesis and Theory of Rumination in the likelihood of developing depression supports and provides an explanation for the origins of depression.

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5
Q

Claim 1: Cognitive Vulnerability Hypothesis

A

A negative inferential style will contribute to depression especially when individuals encounter negative life events

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6
Q

Alloy et al Aim

A

See if one’s thinking patterns could be used to predict the onset depression

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7
Q

Alloy et al Method

A
  1. Sample of non-depressed college freshmen with no other diagnosed disorders - half had a history of clinical depression, the other half did not
  2. Students given test to measure cognitive test (divided into High or Low Risk based on thinknig patterns)
  3. Used questionnaires + structured interviews to identify stressors, cognitive styles, and depressive symptoms
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8
Q

Alloy et al Findings

A

With the group with no prior history of depression, 17% of HIGH RISK students develop MDD and 1% of LOW RISK students - HR showed symptoms of minor depression and more likely to relapse

Suicide rates were higher.

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9
Q

Alloy et al Link

A

Correlation between negative cognitive style ←→ onset and relapse of depression

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10
Q

Alloy et al Strengths

A

Used method and data triangulation, increasing credibility. Usage of high standardized tests increases reliability. Ecological validity. The use of a pre-test, / post-test design helps to diminish the bidirectional ambiguity.

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11
Q

Alloy et al Weaknesses

A

Quasi-experiment (naturally occurring IV) so cannot establish C+E relationship, domino causality (Cognitive psychologists have the difficulty of explaining how this way of thinking leads to depression. Some argue that negative cognitions lead to stress - about interacting with others or the future. This stress may then be the actual trigger that leads to the disorder. In other words, it may be an example of domino causality - negative cognitions lead to stress, stress leads to a genetic expression, which may then lead to the disorder.)

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12
Q

Claim 2: Nolen-Hoeksema’s Theory of Rumination

A

The more we experience a symptom, the more cognition we use to evaluate it, the more we become depressed

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13
Q

N-H (Aim, Method, Findings, Link, Strengths, Weaknesses)

A

AIM: Role of rumination on depressive symptoms

METHOD:

  1. 1132 participants in San Fran
  2. Interviewed 2x 1 year with a 90-min clinical interview
  3. Tests – Beck Anxiety and Beck Depression (automatic negative thinking)
  4. Rumination and coping questionnaire (How much are you thinking about your negative feelings?)

FINDINGS: Potential MDD indivs had higher score of rumination.

Improvement of depression indivs had lowered rumination compared to chronically depressed

LINK: Rumination leads to an increase in exhibited depressive symptoms – persistently thinking negatively about your feelings

STRENGTHS: Supports N-H Theory, multimethod research - used both questionnaire + interview

WEAKNESSES: Self report – response biases. Self report – response biases, those with the strongest symptoms dropped out (introducing bias)

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14
Q

Limitations of Cognitive Etiology

A
  1. Weaknesses of cognitive is that the majority of research is correlational nature.
  2. It is hard to resolve bidirectional ambiguity - is it the cognition that leads to depression or is the depression causing these patterns of thinking?
  3. Helps to explain gender differences in the prevalence of depression.
  4. Explains the role of cognitive processes as mitigating factors in depression - explaining both depression and resilience to stressful life events.
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15
Q

Bio Counter-claim

A

Biological Approach - provides physiological reasoning - Neurotransmitters and hormones

Neurological phenomenon = neurotransmitters and hormones.

Neurotransmitters released from the Axon Terminal.

  • Excitatory (postsynaptic neuron dendrites fire another action potential, the process goes onwards) or inhibitory reaction
    • Neurotransmitters are cells which send electrochemical signals throughout the body -chemical messengers that occur psychological and physiological changes - released from the Axon Terminal as action potential builds up (terminal buttons)
  • It is an electrochemical process

Neuroreceptors OVERSENSITIVE/INSENSITIVE causing too much/too little production

As a neurotransmitter, serotonin carries messages between nerve cells in your brain (your central nervous system) and throughout your body (your peripheral nervous system). These chemical messages tell your body how to work. Serotonin plays several roles in your body, including influencing learning, memory, happiness as well as regulating body temperature, sleep, sexual behavior and hunger. Lack of enough serotonin is thought to play a role in MDD, anxiety, mania and other health conditions.

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16
Q

Caspi et al (Serotonin Hypothesis)

A

Serotonin Hypothesis: a deficit in serotonin could be the origin of depression

Caspi et al - role of gene mutation and epegenetics

AIM: Study gene-environment interaction in mutation and formation of MDD

Explores diathesis-stress theory of depression states that depression risk lies on a persons vulnerability, altered by their genetic predisposition (release of neurotransmitters in response to stressful situations)

METHOD:

  • 847 NZ adults who were assessed for mental health every single year
  • Divided into 3 groups based on their 5-HTT alleles (Group 1: 2 short, Group 2: 1 short, 1 long, Group 3: 2 long) 5-HTT gene has shorter alleles
  • Fill in “Stressful life events” questionnaire about stressors and assessed for depression

FINDINGS:

  • Version w short allele exhibited depressive and suicidal behavior
  • Strongest effect for those who experienced stressful life events

LINK: Just having the gene mutation is not enough → may increase your susceptibility to depression but it’s the stressful life events that increase likelihood of developing depression

STRENGTHS: Holistic approach that considers environmental factors

Data supports biological evidence - Serotonin (5-HT) neurotransmission has a key role in stress sensitivity and in vulnerability to negative affect.

WEAKNESSES: Correlational study so no cause + effect, no evidence that serotonin causes depression, self-reported data

17
Q

Cog Etiology Conclusion

A

Differences in thinking patterns - people’s thoughts and attitudes - is one explanation for the origins of depression

  • Rumination leads to an increase in exhibited depressive symptoms – persistently thinking negatively about your feelings – can be explained biologically as environmental factors may have alter how you talk about yourself
  • It is difficult to isolate cognitive factors as biological and social factors also influence
    Differences in thinking patterns - people’s thoughts and attitudes - is one explanation for the origins of depression
  • Question of bidirectional ambiguity: depression can make thinking more negative, and negative thinking can probably cause and certainly worsen depression
  • Nolen-Hoeksema’s theory has been challenged for temporal validity. Has society changed over time and made the theory less valid?